PLAINTIFF'S EXHIBIT USG-436 Operating Division PBDXHJCT PBOCEDOBE Approvedi SPECIAL BULLETIN COttFmgftTiAL DO MOT RZPHODUCf Oil COPY BEAUT MIXED JOIST COMPOUND JE-533A - DALLAS No. 61-2-24-2-D Date 8-29-66 Page 2 Caneela Dated NEW FORMULA - Mica JC Carey 7EF9 aaheatoe Methocel 6$BB - 4000 epe Excello clay Pic Pleat JP 100 aeah Possolanlo Filler (box Co.) i earnii ne pentahorate PH 674 plaatlolsed eaulalon Carbltol **.7. 0.6 Y Talne aa req'd - approx. 4.25* 0.9* 2.556 to sake 100* IT lbs. car 2000 Ibe R.7. batch 230 to 280 lbe. nozaal range about 85 lbs. 18 lbs. . 50 lbs.
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DEPARTMENT OF HEALTH Pathology Division Report: Demographic Data and Disease Rates for January - December 2000 Dr Jill Murray Dr Geoff Candy NATIONAL CENTRE FOR OCCUPATIONAL HEALTH NCOH Report 3/2001 P O Box 4788 JOHANNESBURG 2000 TABLE OF CONTENTS Executive Summary Section 1: Introduction Section 2: Data Description Distribution of autopsies by year & ethnic group (1975-2000) Number of autopsies by age & ethnic group Number of autopsies by type & ethnic group Number of autopsies by industry & ethnic group Number of autopsies by years of service & ethnic group Mean duration of service and mean age by type of mine & ethnic group (2000) Section 3: Active Tuberculosis Distribution of active tuberculosis by anatomical site Number and prevalence of pulmonary tuberculosis by age& ethnic group Number and prevalence of pulmonary tuberculosis by industry& ethnicgroup Number and prevalence of pulmonary tuberculosis by years of service & ethnic group Section 4: Silicosis Number and prevalence of silicosis by age & ethnic group Number and prevalence of silicosis by industry & ethnic group Number and prevalence of silicosis by years of service & ethnic group Section 5: Massive Fibrosis, Coal Workers' Pneumoconiosis, Mixed dust Fibrosis & Asbestosis Number and prevalence of asbestosis by age & ethnic group Section 6: Emphysema Number and prevalence of emphysema by age & ethnic group Number and prevalence of emphysema by industry & ethnic group Number and prevalence of emphysema by years of service & ethnic group Section 7: Mesothelioma Number of mesothelioma cases by age & ethnic group Number of mesothelioma cases by industry & ethnic group Section 8: Primary Lung Cancer Number and prevalence of primary lung cancer by age & ethnic group 4 5 6 7 8 8 9 10 11 12 12 13 14 15 15 17 18 18 19 20 20 21 2 Number and prevalence of primary lung cancer by industry & ethnic group22 Section 9: Causes of death Causes of death by ethnic group (2000) Appendix: Distribution of autopsies according to the last mine where the deceased worked 23 24 3 EXECUTIVE SUMMARY South African law requires the examination of the cardiorespiratory organs of deceased miners and ex-miners for compensation purposes, provided the next of kin agrees.
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UNITED STATES ENVIRONMENTAL PROTECTION AGENCY REGION 5 In the Matter of: Marathon Petroleum Company LP Canton, Ohio Proceedings Pursuant to Section 113(a)(1) and (3) of the Clean Air Act, 42 U.S.C. 7413(a)(1) and (3) ) ) ) NOTICE AND FINDING ) OF VIOLATION ) ) EPA-5-24-OH-3 ) ) ) NOTICE AND FINDING OF VIOLATION The U.S.
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The mechanisms and pathways leading to the presence of PFOS in human blood are not well characterized but likely involve environmental exposure to PFOS or its precursor molecules and residual levels of PFOS or PFOS precursors in industrial and commercial 3MComo^iv EPI-flO:i3 Pages o r336 products.
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Objective 2 - To determ ine if contam ination o f feed is leading to the low levels of PFOS seen in control rats in the two-year dietary study o f N-Ethyl FOSE conducted at Covance M adison.
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Review article Solitary plasmacytoma of bone and asymptomatic multiple myeloma Meletios A.
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EPA had previously assumed that human benzene metabolism proceeded according to nonsaturating (firstorder) kinetics at exposure concentrations well above 10 ppm, saturation of metabolism below 1 ppm "could lead to substantial underestimation of leukemia risks" in the general population (U.S.
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Toxicogenomics | Article Mechanisms of Benzene-Induced Hematotoxicity and Leukemogenicity: cDNA Microarray Analyses Using Mouse Bone Marrow Tissue Byung-IL Yoon,1 Guang-Xun Li,1 Kunio Kitada,2 Yasushi Kawasaki,1 Katsuhide Igarashi,1 Yukio Kodama,1 Tomoaki Inoue,2 Kazuko Kobayashi,2 Jun Kanno,1 Dae-Yong Kim,3 Tohru Inoue,4 and Yoko Hirabayashi1 1Division of Cellular and Molecular Toxicology, National Institute of Health Sciences, Tokyo, Japan; 2Kamakura Research Labs, Chugai Pharmaceutical, Co., Ltd., Kamakura, Japan; 3Department of Veterinary Pathology, College of Veterinary Medicine and Agricultural Biotechnology, Seoul National University, Seoul, Republic of Korea; 4Biological Safety and Research Center, National Institute of Health Sciences, Tokyo, Japan Although the mechanisms underlying benzene-induced toxicity and leukemogenicity are not yet fully understood, they are likely to be complicated by various pathways, including those of metabolism, growth factor regulation, oxidative stress, DNA damage, cell cycle regulation, and programmed cell death.
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Carcinogenesis vol.32 no.4 pp.605612, 2011 doi:10.1093/carcin/bgq286 Advance Access publication January 7, 2011 Chromosome-wide aneuploidy study (CWAS) in workers exposed to an established leukemogen, benzene Downloaded from carcin.oxfordjournals.org at Universiteitsbibliotheek Utrecht on April 3, 2011 Luoping Zhang, Qing Lan1, Weihong Guo, Alan E.Hubbard, Guilan Li2, Stephen M.Rappaport, Cliona M.McHale, Min Shen1, Zhiying Ji, Roel Vermeulen1, Songnian Yin2, Nathaniel Rothman1 and Martyn T.Smith Division of Environmental Health Sciences, School of Public Health, University of California, Berkeley, CA 94720, USA, 1Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892, USA and 2Institute of Occupational Health and Poison Control, Chinese Center for Disease Control and Prevention, Beijing, 100050 China To whom correspondence should be addressed.
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The mechanisms and pathways leading to the presence of PFOS in human blood are not well characterized but likely involve environmental exposure to PFOS or its precursor molecules and residual levels of PFOS or PFOS precursors in industrial and commercial H - 000006 3M Comcoanv EPI-00323 Page 5 or 236 products.
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FILE NAME: Visible Dust (VDT) DATE: 2005 DOC#: VDT022 DOCUMENT DESCRIPTION: Journal Article - The Navy and Asbestos Thermal Insulation A bstract Asbestos was once used in U.
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