Document vBLgeg2bd1aLQvpr49LyRDJM9

Castieman File: American Petroleum Institute w/c = with cover letter or memo If DATE = 0, undated CD-ROM Document #:API j DATE [9^/ Xl published article from medical journal __ published article from trade journal __ published advertisements __ newspaper article __ published government report __ government inspection results __ unpublished or internal report __ unpublished presentation from conference __ letter __ memorandum __ industry warning labels __ industry sales literature __ industry recommended practices __ meeting minutes (with attachments) __ membership list BC notes THE MEDICAL BULLETIN Vol. 5 OCTOBER, 1941 No. 2 EDITORIAL CONTENTS The Industrial Medical Service of the Standard Oil Company/of New Jersey and Affiliated Companies. . . Review of June 19^1 Issue of THE MEDICAL BULLETIN 92 The Necessity of Accurate Records on Hospital Cases. . (Spanish Translation) A. W. Schoenleber, M.D. 95 The Tuberculosis Program of Standard Oil Company (New Jersey)........................................................................................ James M. Adams, M.D. 99 The Use of Vaccine in the Prevention of Epidemic Influenza .................................................................................................... 106 Robert C. Page, M.D. The Medical Department's Role in the Education of Employees' Children ..................................................................... Felipe Ovalle, M.D. 115 Study of Physical Examinations of Employees of Compania Nativa de Petroleos, S. A. EnriqueA. Puzzi, M.D. (English) (Spanish) 122 135 Occupational Eye Hazards with Special Reference to Hazards Other than from Foreign Bodies and Traumatic Injury...................................................................................................... Roy S. Bonsib, M.A., E.M. 1^+8 Epidemic Conjunctivitis..................................................................... J. Thomas Diaz, M.D. 169 Problems of the Visiting Nurse.............................................. Anna M. Callahan, R.N. 173 Staff News................................................................................................. 176 disminuir'a 3, elevando In las ctras "T" al lado idica que el , al anotar de sifilis f+" indica un gente. MEDICAL BULLETIN 148 OCCUPATIONAL EYE HAZARDS WITH SPECIAL REFERENCE TO HAZARDS OTHER THAN FROM FOREIGN BODIES AND TRAUMATIC INJURY THEIR OCCURRENCE, EFFECTS ON THE EYES, AND NECESSARY PROTECTIVE MEASURES * Roy S. Bonsib, M.A., E.M. New York, New York. Introduction Industry, In Its efforts to obtain maximum efficiency for its national defense effort, must recognize that "eye crip ples may become unwitting saboteurs," Doctor Hedwig S. Kuhn told the Industrial Nursing Section meeting of the recent National Safety Congress in Chicago. Industrial efficiency now demands the utmost of human ef ficiency, consequently, since vision is essential to human efficiency, the early recognition of occupational eye hazards and the prompt provision of protective measures is of prims importance. Score of this Discussion Eye injuries occur over a wide range of industrial occu pations, and virtually none are exempt unless adequate mea sures of prevention are in effect. The most frequent cause of eye damage is the direct injury produced by foreign mater ials such as solids and corrosive substances such as acids or alkalies entering the eyes. Accidental injuries occur accord ing to the kind of work done, though they may be classified as contusions, punctures, incisions and lacerated wounds with or without infection, and the presence or absence of foreign bodies; and burns of the various structures of the eyes and pitB surrounding area. (3) Since these types of eye injury or jjpainage are generally recognized and have been discussed fre quently and voluminously, they will be excluded from this JttiBcussion. Although not as prevalent, there are other serijjpus occupational eye hazards, which, unfortunately have not jjgeceived adequate consideration. It is important to recog- tze the fact that poisonous dusts, gases and vapors may resented at the Session on "PROTECTION FOR THE EYES OF WORKIN INDUSTRY"-12th Annual Greater New York Safety Conven- |0n and Exposition-New York,N.Y. Hotel Pennsylvania 4/23/41 149 MEDICAL BULLETIN directly or systemically affect the eyes and vision; that# radiant energy in the form of ultra-violet and infra-reftf? rays, may damage the eyes of the industrial worker: that'll certain parasitic and Bacillary diseases may affect the eyu:|| and produce damage of this delicate organ; and finally that! lack of proper illumination may produce Miners' Nystagmua * and other diseases of the eye due prohahly to unusual effort, as a result of abnormal physiological condition.(4) Due to time .limitations it will not he possible to discusa all of these occupational eye hazards; neither will it he possible to discuss those war gases and war materials which have a marked effect on the eyes, such as chloracetophenone, and which industry will probably be called upon to produce within the next few months. However, we shall attempt to briefly consider some of the more commonly encountered haz ards, especially those from a few of the extensively used industrial poisons; because the growing use of poisonous chemicals in industry now presents one of the most serious hazards to the eyes of workers.(5) This hazard is aggravat ed by the fact that thousands of men and women who are work ing with these poisons, are wholly unaware of that fact, wholly unaware of the disease hazards to which they are ex posed, and unaware of the steps they should take to guard against the harmful effects of these poisons. Some attention will also be devoted to the eye hazards produced by weld ing operations due to the widespread and ever increasing use of this process. **** DISTINCTION BETWEEN OCCUPATIONAL DISEASE AND ACUTE IN DUSTRIAL INJURY - The definition of a subacute or chronic occupational disease as distinguished from an acute indus trial injury due to a single accident has been considerably broadened by state legislatures, (l) Effects of Gases, Vapors, Fumes and Dusts in the Eyes Toxic gases, vapors, fumes and dusts are met in a great many industries, both casually and as a constant menace to the eyes. They are encountered notably in chemical, acid, alkali, and solvent manufacturing and handling plants in textile industries involving the production of artificial silk, bleaching and dyeing, singeing in print works, in lphur plant ' locally 3; others, at is, the! g and is ex> pon the oute: ay act in bolvO the body, [locally, caus: that lines the cause total bl l nerve, and rel [mediate effect ; vision. w- The effects of blood in th severe inflamm by acid vapors notably chlori being very pen hydrofluoric a Diethyl sulpha posed to them. Some of the been produced fulminate of m percussion cap; have been resp< junctivitis aci The basic anil: produce inflaun Other aniline c and paranitroai vitis owing to dust may cause Spicules of pit corneal ulcer c chamber of the serious injurie to corneal opac exposure to the of the liquid i OCCUPATIONAL ENE HAZARDS 150 nd vision; that t and infra-red al worker: that y affect the eye and finally that Miners' Nystagmus to unusual effort .tion,(4) Dossihle to discuss oher will it he r materials which 3hloracetophenone, upon to produce shall attempt to encountered hazextensively used use of poisonous the most serious tazard is aggravatromen who are work'e of that fact, riiich they are exLd take to guard ons. Some attention 1 produced hy weld- i i ever increasing f 2ASE AND ACUTE INibacute or chronic; an an acute indus-| 3 heen considerably! fumes are met in a greavl constant menace t0| in chemical, aciJ handling plants :tion of artificial _n print works, ..g sulphur plants and in smelting operations. Some of them act only locally upon the eyes, like the fumes of acids and alka lies; others, like wood alcohol act upon the eye indirectly. That is, their effect follows absorption and general poison ing and is exerted upon the retina and optic nerve and not upon the outer structures of the eye. Others, like lead, may act in both ways, according to the mode of entrance in to the body. The fumes of molten lead may irritate the eye locally, causing an inflammation of the delicate membrane that lines the eyelids, or some time after absorption may cause total blindness through inflammation of the optic nerve, and retina. (6) Again they may cause temporary im mediate effect through the circulation, producing double vision. Irritant Effects The effects vary with the agent in operation from excess of blood in the delicate membrane that lines the eyelids to severe inflammation. Such a condition is frequently caused by acid vapors, especially hydrochloric, and by the halogens, notably chlorine arid bromine, and by ammonia; the latter being very penetrating. It may also arise from exposure to hydrofluoric acid, hydrogen sulphide and sulphur dioxide. Diethyl sulphate vapors cause eye affections to those ex posed to them. (7) Some of the most active types of eye irritation have been produced by certain industrial dusts. For example, the fulminate of mercury and silver used in the manufacture of ; percussion caps and Christmas fire-crackers respectively : have been responsible for outbreaks of a severe type of con - Junctiviiio accompanied by much pain and discharge of tears. The basic aniline dyes, methyl violet in particular, may produce inflammation of the cornea as well as conjunctivitis. : *)' Other aniline compounds producing conjunctivitis are tetryl Paranitroaniline. Chimney-sweeps suffer from conjuncti- |t Vltis owing to the irritation caused by the soot. Pitch 1 dust may cause severe conjunctivitis in those exposed to it. Spicules of pitch may penetrate the cornea, giving rise to fOrneal ulcer or an accumulation of pus in the anterior Chamber of the eye. The dust from grinding asphalt produces Serious injuries to the cornea. Nitronaphthalene gives rise f corneal opacities, either as the result of long continued l!?8Ure bhe vapor, or as a result of accidental splashing the liquid into the eye. Workers in pitch or asphalt MEDICAL BULLETIN dust, especially these breaking up pitch, should he suppuJ! with goggles of the proper type. (7) Systemic Effects of Industrial Poisons A large variety of industrial poisons gives rise to traiilf sient affections of the eyesight, and some of them to j manent effects. Various poisons may give rise to any one o*l more Of these conditions, depending upon the strength of the'J poison and duration of the exposure. Especially hazardous are the fumes of dinitrobenzol which is used in the manufac ture of high explosives. Other substances which formerly produced much eye trouble have been eliminated from modern processes of manufacture or the operators are better pro tected by the wearing of respirators and goggles, as well as by the installation of exhaust fans. Such substances are 1 iodoform, volatile coal tar products used in dyeing, and arsenic, formerly used in cosmetics and in the coloring of wall papers, artificial flowers, and paints. (6) Prominent among the group of protoplasmic poisons which produce blindness through inflammation of the nerve behind the eyeball, acute or chronic, are: alcohol, nicotine, car bon disulphide, tri-chlorethylene and the arsenical com pounds. There is another group of internal poisons which produce blindness through other forms of inflammation and disease of the retina. Poisons of this group, instead of acting directly on the nerve fibers affect the nerve secon darily through contraction of the small blood vessels of the nerve or retina, or through changes in the blood. In this group may be found lead, quinine, cocaine, sodium salicylate, snake, venom, silver salts, mercurial preparations, amyl ni trite and nitrous oxide gas. (8) Toxic Amblyopias Toxic amblyopia is a partial or complete blindness caus ed by a poisonous substance which interferes with the func tion of the retina, the optic nerve or the more central optic pathway. Every new drug used in medicine and every new chemical used in industry is a possible cause of toxic dimness of vision so that this subject is constantly growing in scope. Victims are becoming more and more compensation conscious and the ophthalmologist, in order to he fair to both employer and employee, must have a thorough understand ing of this subject. (9) **** Icarroll (9) w Those cause Ml blindness, Itire visual f |elds, and II] lively or chiei "In cortic most com] to light center, tion is 1 the blooc cause of whether 1 causes o; plete bl: alcoholi: probably ness occ the eye) is excel 25 hours type of "The seco caused b field bu Barbitur only whe found am ijf-* cementer j&fe and vulc zol also which oc below pa glob in a conditic both fox (remedy some cas "Methyl a 8:1 OCCUPATIONAL EYE HAZARDS 152 Classification of Toxic Amblyopia Carroll (9) divides toxic amblyopias into three groups, I - Those caused by poisons which produce cortical or cen tral blindness, II - Those caused by poisons depressing the entire visual field, but more especially the peripheral fields, and III - Those caused by poisons depressing exclu sively or chiefly the central visual field. (9) "In cortical blindness there is a complete or al most complete loss of vision but the pupils react to light because the lesion is above the light center. One of the several causes of this condi tion is uremia (presence of urine constituents in the blood). Acute carbon monoxide poisoning as a cause of toxic amblyopia is a definite entity, but whether chronic carbon monoxide poisoning ever causes optic nerve damage is questionable...Com plete blindness in a person suffering from acute alcoholism whose pupils react normally to light is probably due to acute alcoholic amaurosis (blind ness occurring without any apparent injury to the eye). The prognosis (prospect of recovery) is excellent and the patient is usually well in 25 hours. It is important to differentiate this type of poisoning from methyl alcohol amblyopia. "The second group of toxic amblyopias are those caused by poisons depressing the entire visual field but more especially the peripheral field. Barbiturates are usually a cause of amblyopia only when used in very large doses. A cause found among makers of artificial leather, rubber cementers, dry cleaners, engravers,lacquer makers and vulcanizers is BENZOL (Benzene). Dinitro-Ben- zol also may be a causative agent. Blood loss which occurs in patients whose general health is below par or whose red blood cell count and hemo globin are markedly reduced sometimes causes this condition. ...Carbon tetrachloride which is used both for cleaning purposes and as an anthelmintic (remedy for worms) may be a causative poison in some cases of amblyopia. Methyl alcohol is a recognized cause of optic 152. MEDICAL BULLETIN atrophy (a wasting or diminution in size) aa evi denced by the occurrence of this condition among painters who, when working in closed spaces, inhale the fumes of wood alcohol. The same danger exists for persons using wood alcohol In lamps... i "In the third group of toxic amblyopias are those poisons which depress, exclusively or chiefly, the central visual field. Carbon disulphide, used widely in the rayon industry and also in the preparation of rubber, explosives, hides, insecti cides and in the wool industries produces this type of amblyopia. It is usually accompanied by poly neuritis. ..and other evidences of toxemia. Lead poisoning is found to be a cause of amblyopia in patients who are compositors, plumbers, storagebattery makers and even in children who have eat en the paint from their cribs. "Thallium which is used by women having excessive hair growths as a depilatory and in industry as a rat poison usually produces peripheral neuritis preceding any visual disturbance. "Tobacco-alcohol poisoning is the most common and the most important toxic amblyopia seen in this country. ...Recent research has shown that it is possible for patients with this condition to con tinue smoking and drinking and recover completely providing an adequate amount of vitaminB is taken. " **** 1 Effect of Lead Lead poisoning, which is the greatest poison hazard in all industry may be acquired in a great many trades, is particularly liable to injure the eyes of painters, file cutters, plumbers, electro-typers, typesetters and cleaners, pottery glazers, storage battery makers, paint grinders, and makers of red, white, orange and yellow lead, makers of lead pipe, tinsmiths (solderers) and cutters of sheet lead or various lead article manufacturers. (6) The effects of poisoning by lead are very widespread in the body, but tend particularly to be localized in the nervous system. Inflam- fon of the optic jfin chronic leac __ of the retir |lysis of the oc |o been found in m Lead causes se `leases, such as _alysis. It may ptem through the Effe H Many volatile su )80rption may seri JCtly upon the str jrebral deficiency riaion centers. 0 fllcohol forms the m >st instantaneous, flrihaled in quantiti insolvents are absorb the body, the eyes swelling due to acc ,brain and other org ||eye tissues. This : ft blood vessels and i damage to the eyes, which we can actual physiological chang of the poisonous ma in the body, partic The alcohol whic methyl or "wood a blindness through i only chronic alcoho from severe amblyop persons who combine are the most frequei past few years our < ethyl alcohol and tc gators have shown tl vitamin B2 complex i OCCUPATIONAL EYE HAZARDS 154 mation of the optic nerve of one or Loth eyes is not uncom mon in chronic lead poisoning and is associated with inflam mation of the retina which may produce total blindness. Paralysis of the ocular muscles and dimness of vision has also been found in lead poisoning; recovery usually occurs. (7) Lead causes serious central, as well as peripheral, eye diseases, such as inflammation back of the eyeball and paralysis. It may be inhaled in fumes, or may enter the system through the mouth. Effect of Volatile Substances Many volatile substances when they reach the blood through absorption may seriously affect vision acting either di rectly upon the structure of the eyes or affecting it through cerebral deficiency in quality or quantity of blood in brain vision centers. Of this group of volatile poisons, wood alcohol forms the most striking example, as it produces al most instantaneous, and frequently, permanent blindness when inhaled in quantities. (6) Vapors of some of the volatile solvents are absorbed through the lungs and cause damage to the body, the eyes included. Any substance that will cause swelling due to accumulation of a fluid in the tissue of the brain and other organs will cause a similar response in the eye tissues. This may cause dilation or constriction of the blood vessels and if prolonged or repeated may result in damage' to the eyes. The eye is the only window through vhich we can actually look inside the body and see the physiological changes which are taking place as the result of the poisonous material, not only in the eye, but elsewhere in the body, particularly in the brain. Effect of Alcohol The alcohol which is usually responsible for blindness is methyl or "wood alcohol". Ethyl alcohol also produces blindness through its action on the optic nerve. However, only chronic alcoholic individuals are likely to suffer from, severe amblyopia, and even then it is well known that persons who combine the use of alcohol and tobacco to excess ftre the most frequent victims from these poisons. In the Pftst few years our conception of dimness of vision due to Otbyl alcohol and tobacco has been modified. Certain investi- 8&tore have shown that a deficiency of the vitamin Bi and Vitamin B2 complex in regular diet is actually responsible for 155 MEDICAL BULLETIN the "blindness, inasmuch as alcohol and tobacco may be coni tinued in the usual quantities after "blindness has set in yet complete recovery will follow the administration of ad^j quate amounts of these vitamins. (8) , Wood Alcohol Wood alcohol is soluble in such lipoid substances as are! found in nerve tissues, and after death the brain retains ep large percentage of this alcohol. A highly differentiated! nerve substance like that of the optic nerve is therefore! particularly liable to be affected by it. (6) It has been# thought for a long time that wood alcohol attacks the eyes! especially, but recent researches have demonstrated, exclu-1 sively that this is not a special attack on the eye but that the eye is merely indicative of the response that goes on in the brain and other tissues, and that the blindness produced or the affect on the vision is simply discerned more readily. There is nothing so outstanding or dramatic as any effect on the eyes. Effect of Carbon Disulphide Used in rubber and rayon industries, and in refrigerating plants, carbon disulphide produces a vapor which injures vision by affecting the retina or optic nerve. ., .A large num ber of eye injuries occur in the rayon industry to workmen in the spinning room where irritations are caused by a combi nation of mist containing about eleven per cent sulphuric acid in addition to the carbon disulphide. British investi gators found that, as the result of an isolated exposure to a concentration of only one part of carbon disulphide by volume in 3,000 parts air of a few hours duration, severe headache and mental dullness or confusion usually result. If such exposures are of daily occurrence, the symptoms be come increasingly severe with inflammation of the nerves, distorted vision and mental disturbances. (10) Effect of Dinitro-Benzol Used in manufacturing aniline dyes and the production of high explosives, injures the vision by inhalation and produces a distinct toxic effect on the vision similar in many respects to that caused by carbon disulphide. Its effects are found in varying degree in all those who suffer periodically from K |cute attacks c fsymptoms, but Jltively few coi Stably about one *|fall cases absei jlarying length c 111 alleviate the y Carbon monoxide _A80line may he s< gSopB as to produ< Vina. (13) ^ : contains a fair m jision. Drinker [Injury requires s< lyhicb there ia a tare rare cases wh: Itold that in most jqarbon monoxide a Ivital part of cel. I'effects may be pei ^damage due to carl ji; eient. When perr ^hysterical phenome f hy the presence c damage to the ner^ eyegrounds. ; The term "chroi : very loosely desc: ; exposure of an aci ; describe the ill 1 posures of an acul any symptoms rema: Occupatioi An occupationa. flammation marked of the cornea proc (15) states that 1 titis, without the OCCUPATIONAL EYE HAZARDS 156 subacute attacks of poisoning. Susceptibility may aggravate the symptoms, but no worker can claim perfect immunity. Com paratively few complain of any impairment to their sight, but probably about one in ten are unknowingly affected.Snell (11) In all cases absence from exposure removes these symptoms in a varying length of time, and usually a restricted exposure will alleviate them. (12) Effect of Carbon Monoxide Carbon monoxide produced by the imperfect combustion of gasoline may be so prevalent in garages and automobile repair shops as to produce disturbances of the optic nerve and the retina. (13) The literature on carbon monoxide asphyxia contains a fair number of reports of permanent damage to vision. Drinker (l4) has found that as a rule, serious eye injury requires severe poisoning, that is, poisoning in which there is a long period of unconsciousness. But there are rare cases which are exceptions to this rule. We are told that in most instances of visual disturbance following carbon monoxide asphyxia, the injuries (lesions) are in the vital part of cells of the different ocular nerves. The effects may be permanent, as in the case of other cerebral damage due to carbon monoxide, or they may be quite tran sient. When permanent, they are to be distinguished from hysterical phenomena, so often found after mild poisoning, ty the presence of nervous or mental symptoms indicating damage to the nervous system, or by definite lesions In the eyegrounds. The term "chronic monoxide poisoning" as usually used, very loosely describes the permanent after-effects of severe exposure of an acute nature or it is used just as freely to describe the ill health resulting from repeated minor ex - Posures of an acute nature but where certainly very seldom toy symptoms remain when the exposure ceases. **** . Occupational Superficial Punctate Keratitis , An occupational superficial punctate keratitis is an m- vfjflaumation marked with points or dots on or near the surface |of the cornea produced by occupational exposure. Davidson ;|l5) states that the symptoms of superficial punctate^ kera titis, without the use of the slit lamp, may give rise to 157 MEDICAL BULLETIN the diagnosis of acute, subacute or chronic conjunctivitis. This condition has been found among workers with lacquers in the furniture and metal industries, in the rayon industry, in mushroom canning, among workers in the straw hat industry and in agricultural workers on reclaimed marsh lands in California. Rankine (l6) who reported on 1,598 workers in the acid house in the rayon industry, listed the symptoms as blurred vision and halos, grittiness, abnormal intolerance of light, discharge of tears, eyelid muscle spasm, aching back of the eyes and headaches. Burning and stabbing pains increase toward the end of the day and working-week and the symptoms are most marked during the winter months when the ventilation of workrooms is poorest. All writers stress evidence of individual predisposition in the form of frequent recurrences in some persons and immunity of other neighbor ing workers. (15) The outstanding offending agents appear to be hydrogen sulphide, principally in the rayon and sugar industries, also mines and tunnels, benzol and similar solvents and diluents of varnishes, shellacs and lacquers. Many writers, such as Klein (17) and Buchlers (18) stress the effect of drops of chemicals on the cornea, and the drops scattered by spinning and spraying rather than the gases involved in artificial silk industrial processes as the factor respon sible for the eye conditions found. According to McDonald (19), hydrogen sulphide containing droplets of the acid bath are responsible for keratitis among the workers in the rayon industry; he found the condition more marked in damp weather and stated that a concentration of ten parts per million is permissible. Others of the important agents considered in textbooks and in the literature are hydrogen fluoride, tar, pitch, mercury and silver sublimate, certain kinds of wood and lead nitrate. The victims all get well with change of occupation, and there is complete return to health in from seven to ten days. (15) Occupational Dystrophy Davidson (15) asserts that true occupational dystrophy such as is reported in textbooks and in the literature, has not been observed. Cases of dystrophies as after effects of accidents have, however, been encountered. Eor example, examinations prove that a degenerative process goes on in the cornea with lime incrustation in the course of years in connection with lime burns. These manifestations may be ^' delayed and presen between Injury and Occi At the 1956 meel Dr. Lane (20) of ti port on an occupal tear apparatus, etc patients. The cat tumors or cancers g mentary deposits, report issued (21) in the United State where oils have a h have such high canc oils. American oi oils. Refining in process; the workme of the largest oil with records extend small number of can tensively to sunligl being exposed to kn and tar have more tl found in all the otl to the large number mas, as well as alai growths which can e: lieved that the occi carpenters is due tc composition roofing of building papers, to the sunlight. Pa tribution of ocular irritant and frequen A small distribution whose occupation inv Concentrated coal pr distillation of coal house workers show a parts of the body bu from ten to fifteen W\ OCCUPATIONAL EYE HAZARDS 158 unctivitia. lacquers in 1 industry, lat industry 1 lands in workers in symptoms as intolerance sm, aching ahting pains eek and the s when the iters stress of frequent r neighbor- he hydrogen industries, olvents and iany writers, effect of scattered involved in ctor respon- to McDonald the acid hath in the rayondamp weather million is sidered in oride, tar, ds of wood change of h in from dystrophy ature, has effects of 'or example, goes on in of years in itions may he delayed and present problems in the differential diagnosis between injury and disease. **** Occupational Cancer of the Eye At the 1936 meeting of the American College of Surgeons, Dr. Lane (20) of the University of Michigan presented a re port on an occupational study of cancer of the eye, lid, tear apparatus, etc., setting forth the results on 1,000 patients. The cases were divided equally between malignant tumors or cancers and tumors characterized by abnormal -pig mentary deposits. These studies were continued and a later report issued (21) from which we learn that oils produced in the United States except in the middle west oil region where oils have a higher paraffin and asphalt base, do not have such high cancer-producing properties as do foreign oils. American oils contain less sulphur than do foreign oils. Refining in the United States is largely a closed process; the workmen have little contact with the oil. One of the largest oil companies, employing thousands of workers, with records extending over many years show a surprisingly small number of cancers. Occupational groups exposed ex tensively to sunlight and the elements at the same time being exposed to known cancer-producing agents such as pitch and tar have more than 2.8 times the total number of cases found in all the other groups. Special attention was called to the large number of farmers and laborers having carcino mas, as well as a large number of carpenters having tumors or growths which can either be benign or malignant. It is be lieved that the occupational hazard in the case of the carpenters Ib due to the handling of creosoted shingles, of composition roofing which contains much tar and asphalt, and of building papers, also other lining products in addition to the sunlight. Painters have a potentially increased dis tribution of ocular cancer. Chrome is a, decided chemical irritant and frequently enters into the composition of paint. A small distribution of ocular cancer was found among those whose occupation involved a considerable use of chemicals. Concentrated coal products such as pitch and tar derived by distillation of coal are potent cancer-producing agents. Gashouse workers show a rather high cancer incidence in other Parts of the body but not in the eye. As a rule it takes from ten to fifteen years' exposure to an irritant before 159 MEDICAL BULLETIN precancerous or cancerous injuries (lesions) develop. Doctor lane's report is evidently a very interesting statistical study of the occupational "background of a series of cases showing cancerous changes in the eyes. However, it is questionable whether the figures given make any particu lar case as to the occupational origin of these cancers. The distribution of the cancer cases among the occupations mentioned is so varied and of such low order in those occu pations having a known exposure to cancer-producing materials as compared with the cases not so exposed, it is doubtful that this study shows any particular occupation to be a cau sative factor in the location of cancer in the eye. Effects of Heat on the Eyes One of the most frequently reported causes of the occupa tional type of cataract or the crystalline lens of the eye becoming opaque is exposure to intense heat as In the remov al of molten material from the furnace. However, heat is apparently being discounted more and more as a cause of cancer per se. Glass workers are probably the best known victims of this type of occupational eye hazard. Mehofer (22) be lieves that the excessive perspiration of glass makers plays an important part in producing this change in the lens. A third cause may be the brilliant white light that dazzles workers in glass and which, by fatiguing the retina of the eye, reflexly influences the lens. Molten iron and working near electric and other types of furnaces expose workmen to ex cessive heat which burn the lids and cornea and cause cata racts and degeneration of the retina. Protective glasses and helmets may prevent such occurrences. (23) Effects of Radiant Energy Radiation from an intensely heated solid or vapor may be divided into three portions: (a) invisible infra-red rays, (b) visible light rays, and (c) invisible ultra-violet rays. The presence of the infra-red rays, although they are invis ible, may be recognized by the sensation of heat which they produce on the skin. The visible light rays enable us to see; but we have no sense that detects the invisible ultra-violet rays, so we know them only by their effects. Exposure of the unprotected eyes to very intense light sources, rich in ultra violet radiant energy, produces an acute and painful inflam mation of the eyes. Long continued exposure or frequent short Photophthalmi duced by the bur of radiant energ posed to intense blindness. It i person Is expose< may lead to seve: known as "Klieg 1 to a certain ext< to the intense srays. (13) According to 1 light and heat m develop. ery interesting ound of a series es. However, it .ke any particulese cancers. The the occupations in those occu>ducing materials it is doubtful '.ion to he a cau;he eye. is of the occupa- Lens of the eye as in the remo'v- owever, heat is 3 a cause of cancer 3t known victims iehofer (22) be- Lass makers plays in the lens. A at that dazzles retina of the eye, and working near workmen to ex- and cause cata- ctive glasses and or vapor may he infra-red rays, Itra-violet rays, h they are invisheat which they enable us to see; ihle ultra-violet . Exposure of the es, rich in ultra - d painful inflamor frequent short OCCUPATIONAL EYE HAZARDS 160 exposures to ultra-violet rays of even moderate intensity may have a similar effect on the eyes. The disease produced is a form of inflammation of the lining of the eyelids commonly called Electric-Ophthalmia or Photophthalmia. (13) Black (24) says that the effect upon the eyes is practically the same as a sunburn when the unprotected body is exposed to the sun's rays, and, like a sunburn, is not noticed until several hours after exposure. The heat radiation from work where the ra diant energy hazard exists is so great that heat insulating material is required for all parts of the protectors which touch the face of the operator. A series of experiments have been made by the United States Bureau of Standards upon various kinds of colored glasses. It was found that most black, amber, green, greenish-yellow and red glasses are efficient for protecting the eyes from ultra violet light. For shielding the eyes from infra-red rays: yellowish-green, sage-green, gold-plated and bluish-green glasses are the most serviceable, although color is no cri terion of efficiency. Glasses having a gray or neutral tint are the most agreeable, as they do not alter the color of ob jects. It has also been found that workmen in welding pro cesses prefer a yellowish-green glass to any other color. The cobalt glass so commonly used by furnace men is not effective in providing protection from ultra-violet radiant energy.(25) In fact the cobalt blue is the least suitable color for this protection. (13) . Photophthalmia Photophthalmia is an inflammation of the conjunctiva pro duced by the burning effect of the harmful ultra-violet rays of radiant energy.(2) It is likely to develop in.workers ex posed to intense light or heat and is quite similar to snowblindness. It is not a serious disease in itself but if a person is exposed for long periods or frequent intervals, it may lead to severe trouble. A peculiar form of this disease, known as "Klieg Eyes", is found among'motion picture actors to a certain extent. In this case it is caused by exposure to the intense studio lights which are rich in ultra-violet rays. (13) Occupational Cataracts According to Hannum (13) continued exposure to intense light and heat may cause a slow development of cataracts. 1 l6l MEDICAL BULLETIN Because the disease is especially associated with workers in molten glass, it is commonly referred to as "Glass-Blowers' Cataract", although it has teen found to occur in other in dustries such as chain-making, smelting, tin plate making, gas welding and cutting. There seems to he considerable dif ference of opinion and controversy as to whether or not cataracts may he formed from such exposure. However, it is reported (13) that the development of a cataract, which is an opacity of the crystalline lens, is so gradual that often the vision is reduced to one-tenth before advice is sought. Such a condition can be prevented by the continuous use of suitable goggles or other visual protective devices during periods of exposure. (13) Eye Hazards Due to Electric Arc Welding There is a high localized energy discharge with emission of light and a temperature as high as 6300E. (3^82PC.) in the field of an electric arc. Thus one would expect radiant energy, volatilization of materials, gaseous fixation and decomposition products, and molten metal spray to be the most serious hazards present. Eye Exposure in Welding The changes in the eye due to radiation exposure have been discussed frequently and have been ascribed to various wave lengths. The welding flame, as encountered in the course of work, presents but little, if any, exposure to either infra red or ultra-violet, but it is now believed to injure the eyes chiefly through intense glare or radiation within the visible range. The length and intensity of exposure is of course important as is the distance from the sources of radiation. It must be remembered that the sources of radia tion include not only the welder's own work, but also direct or reflected rays from neighboring workers. Lipkovitch (26) has estimated that the welder has a potential exposure of 30 to 60 per cent of his work day from his own or his neighbor's operation. Pozzi (27) has reported inflammation of the cornea and the lining of the eyelids due to ultra-violet radiation occurring in an individual 30 feet lateral to the welder. The minimal safe distance has been estimated at 200 feet. (28) According to an abstract from the American with workers in "Glass-Blowers' ir in other in1 plate making, uisiderable difvhether or not However, it is ract, which is adual that often vice is sought, tinuous use of devices during elding e with emission F. (3l+82C.) in d expect radiant s fixation and ray to he the rposure have been to various wave in the course of to either infra- to injure the tion within the exposure is of the sources of sources of radia- but also direct Lipkovitch (26) al exposure of 30 or his neighbor's ammation of the to ultra-violet lateral to the estimated at 200 om the American OCCUPATIONAL EYE HAZARDS 162 Journal of Ophthalmology (September, 1935), Natason states that 35 per cent of 210 welders examined had inflammation of the conjunctiva, and 203 out of 295 eyes had diminished cor neal sensitivity. Acute Eye Injuries Due to Ultra-Violet Rays The usual injuries produced by ultra-violet rays from the electric arc during welding operations are typically a blepharitis (inflammation of the eyelids) and keratoconjunc tivitis. Symptoms occur from four to eight hours after ex posure and last from 24 to 48 hours. Prolonged exposure or secondary infection may cause prolongation of symptoms and even the development of corneal ulceration. This type of eye injury is rather common among welders and may result in the development of chronic inflammation of the lining of the eyelids. Most welders have noted a severe inflammation of the eye by rays beyond the violet of the spectrum at one time or another but have lost no time from work because of the difficulty. Tho exposure necessary to produce this con dition is variable, a mere "flash" has been reported to cause discomfort. Certainly individual susceptibility and the presence of chronic eye diseases predispose to the de velopment of such conditions. (29) Chronic Eye Injuries On repeated exposure a chronic indefinitely localized in flammation ofthe outer layer of the sclera has been reported. Other eye conditions reported to occur as a result of welding exposure are inflammation of the retina, pigment changes in the retina, inflammation of the cornea, and even detachment of the retina. Color blindness, blind or partially blind areas in the visual field, weakness or speedy tiring of the visual organs, attended by pain in the eyes and headache, dimness of vision and other visual derangements have also been reported. Sudden intense stimulation of the unprotected eye by the welding arc may produce blindness similar to snow or ice blindness (due to ultra-violet and visible rays) or electric, eclipse or flash blindness (due to the entire spectrum, ultra-violet, visible and heat rays). These con ditions may be only temporary; however, in the case of infra red rays a permanent heat, death or destruction, of the retina may occur. Roy (30) and Wurdemann (31) have reported permanent retinal damage and blindness in individuals !;H[1iI: 165 MEDICAL BULLETIN watching electric welders at a distance of a few feet withl the unprotected eye. ; Cataracts The occurrence of cataracts due to exposure in welding is not easily disposed of. Opinions are divided; it is gen erally agreed that the shorter ultra-violet radiation does not reach the lens (some of the longer ultra-violet rays may he absorbed and produce changes in the lens proteins) and. that shorter infra-red rays do reach the lens and are trans formed into heat and may cause some damage to the lens nu trition. However, one usually finds a combination of causes such as general disease or nutritional states of the body also may play a role.(29) Eyestrain Eyestrain due to fineness of the work, the diminished luminosity and sudden changes in light intensity are de scribed in the literature.(32) Chautin (33) noted some delay in visual adaptation in a small group of welders. Others deny that such a condition exists, and that protective screen and colored glass effectively protect the workers' eyes by absorbing ultra-violet, visible and infra-red rays as is shown in their examination of visual acuity and visual fields of many welders. They found no more abnormalities of vision in groups of welders than is found in any average group.(29) Other Eye Injuries Another source of eye damage is the transference of infec tions from one worker to another by exchange of goggles and other protective equipment. Each welder should be furnished with his own visual protective equipment which must be sterilized at intervals. Equipment which has been sterilized will be much more acceptable among the workers, since goggles are a very personal piece of protective equipment and work men do not care to use such an article that has been worn by others. It has been found that giving each person his own equipment, and having it sterilized frequently, adds immeasu rably to the worker's acceptance and use of the equipment. Foreign bodies in the eyes occur not only from molten metal and slag from his own or neighboring work but also from sharp fine slag particles which break off when the brittle MEDICAL BULLETIN______ S f OCCUPATIONAL EYE HAZARDS g- i--i ad g i 1 i--1 0 0 1 ra rP o 1 1 1 ft On CO P 0 rQ P o p <p ad 0 > o g 0 P p O ad 0 p P 0 /--l o 0 P tjD pP ro P p 0 P ft rH jd cr* 0 > H P O 0 P o p ft 0 P i P p ft p rH ad P P P o o ft rH p g H rH g Pi P g a1 0 Pi g P o o OP H 0 PI 0 rP rH P ,P > P P P o P 0 g 0 H `-P rP i--1 ft i--1 O 0 H o 0 0 PI P ft O P o H P O 0 rH 0 P 0 P 0 0 P u o p p P P O P o H P P t"i 'd P P ad 0 o o H xi g > g om i--( og og Ch O i--1 g g rH -p 0 ft *H -p 0 I--I r^ P 0 P P 0 P 0 i--1 P P OJ O tsl o < CO 'o p' 00 p od 0o 0o p ad P rQ P go o p hg p 0 op gp g ft g *ri 1--1 ft p 0p 00 w H P ad P 0 rH o H p> 1 P P P rH P 0 rP > H P Po P p ad 0 i--1 ft 0 H H rP 0 0l p0oP pi MO ad 0 Pi rHp P P P 0 P ft P0 ad P PP o H r\ P H ft rM PI p ad O 0 V_0^ P P ad .P P > pg H 0 gg o p rH P Ch oO 0 ft rH 0 ad 0 i--1 r^ P rH rH 0 P5 p > o 0 p 0 0 rQ ad P P P CO p p o H P 0 P Pi P *N P* m OJ 0 ft o s o 0 ad 'aBd ad 0 P 0 P 0 P 0 P P ft o P H P P P }>a *s ad P P p 0 rH O rH P o ft 0 0 i--1 ad O H P rH 1--) P rH O P P 0 O Jh 0 O 0pP >1 P P > O S0 P > P 0P W ft 0 0 0 dP g o 0 rP P ft o p T 0 o 0 P 0 P P H 0 rP P ft o 0 0 p P o 0 PQ P0 0 oP H pO Pp o 0P rH p 0P P 00 a H P0 0 gP O P0 ft rH Pi 0 t>i ad PP P P P ft 0 Pi P ft o p rH M 0 0 Pi P 0 P O 0 P Po P rH p P H ft P P ft P rH 00P 0 rH 0 0 0 ft o ps o P00p PP p o H P r-1 P P00 0 H 0 0 *r4 P ad P P M 0 Pi P P 0 P *H ad 0 P rH 0 P >0 rrd o p 0 0ft u0 ft rH ,P P 0 rH > P ad 0 P rH p M 0 P 0 ad 0 P P O P 0 P P o H ad g H rd r-1 P P 0 0 0 O Q P P a p rH o P 0 x\ ft rH 0 rP P > oPM oP n P 000Oo O P 0 0 Pp Po >p tJO P 00 P i--1 0 p i--1 p 0 0 ft s P H H ft 0 P P0 H P d 0 P p P 0 P -P 0 P P a1 P P m> rd 0 ad n P rH P P P 0 rH ft o H ad 0 g O ts P t--1 ft a ft CO ft O 0 P P ad P t>=, PW Pi g P 0 fot k Jo--l > P P P P W CQ P O H P P P rH 9 P i--1 i--1 H P P 0 d O H ft ft P 0 P H ft O 0 P O 0 ft ft ft (D I ) 0 fftt H 0 03 Ps Ti I rH P 0 P g cq 0oa gg ad O t* to P0 ad g P2 -p 0 f0t g H CQ Ia--d1 0 H p fOt ^ ra- gg CD cd g g 0 P H ' 0 pi ,P g rH O -P >pppg g 0 <P g *H h g 'd o o g od 0 go p< a rtf *H ' g *h g P> -P 0 TCi P g > I CD CQ o q cd Pg g g g -P H 0 ft O CQ ;h -p g 0 0 fot !> Hi--1 -P rH 0^P CJO o -P 0 * xi o g 0 P O CQ o -cPpd CQ r-1 ad cd ft P 0> fot g g i g pp ad m 0 -P o p g 0 CQ +3 p ud fogt .hg 0 ro 0 ad o SOra *aHM ipd> p p p CQ g -P H gg 0_ -p 0o to rd 0 > p CQ rd p -p cd o O cd P Pi -p o go o g P rH P PP a 2> P P P P 0 0 CQ P 0 ia--d1 0 -Pp g ra P o ft CQ >> ^ 0 o ad 0 P ad O o^ Oa 00^--CM- P 00 > O Ip ra ' o0 rH P ad P0 oP 0o 1--1 0 0P O 2pP rH P 8p< ft p o ttO| p 0 0 0P CQ p 0 P fPt g g P s H p ad o p o O ft 0 ch P 0 p Ph -P rd 00 p p H ad p 0 f0t ad P P g H P f0t P > i--1 0P >P O0 p f>s p p o o o ^ g h 0P P tog ad O ad f0t P 0 P P O o p *rpH w . p 0 1--ft1 W) 0 ft ft 0 p 0 g P ^0 rd P i--i g3 P O 0 ft P ft 0 P P ad 0 P o H P rH 0 P ft o o CQ 0 0 *H 0P HP*"3 0 W P r0P P o Pi O ii--d1 P 0P -p 03 Ch [Q Jh 0 X f0t W p O O -P P cq -p in ad H P H p ft ^ CQ CQ cd cd O rd *H 0 ft ft H H cd 0 -x -p ^0 -p fd P CQ P {>3 rH 0 P P 0 Pr~i, rf0t, 'PH rd 0 o -P g P ft d CQ -p CQ i: H o cd CD 5> 0 P CQ O -o 0 t-1 OPo0 0 rma OQ 2 m & 1-D P 0 rP ad g -p 0 ag P *H 0 cd p> CQ !> .rfdt aftd ft m -p m g *0 o o g <D 0 CQ g-tPo 0o S ?H <H o g g g g p > g f0t O m g m g CD -P o 0. g oM CD -=J- ftCQ g > N~\ H S>i *--s g g CQ O rd CD g cd or) g 6 0 g id g ^ t> g a g h g p g g . -p o CD cd & *t>5 -rPa ing H1 O0 OH oo OO P ft Q O ft P rH P> 165 MEDICAL BULLETIN Necessary Eye Protective Measures Since eye damage may Be due to the direct action of auh stances such as solids or corrosive liquids, toxic gases,va pors or dust which produce occupational poisonings (intoxica tions); or to various forms `of radiations or to infectious 0: parasitic agents, or, finally, to fatigue and occupational eye strain, it is obvious from a consideration of these causa tive means that the control of toxic gases, dusts, and vapo: in industry will control the eye damage due to such toxic': substances. Toxic materials in industry should therefore bei controlled by proper enclosure or isolation, ventilation anil substitution. Protective goggles and shields of various: types should be provided to prevent the entrance of solid1 material into the eyes. Special types of goggles are novi available to protect the eyes from acids and alkalies andb their vapors. Protection against radiant energy, as has al ready been indicated, may be achieved only by the use of suitable radiation-filtering screens and goggles. In thel| case of infectious and parasitic agents, protection may be - achieved by the methods usually employed by health authori ties against parasites of man and animals and, finally, in the case of occupational eye fatigue and eyestrain, protec tion may be achieved chiefly by lumination in adequate amounts. the provision of suitable . il -MW According to Resnick (35) preventive hygiene when" toxic substances are used in industry consists mainly of complete cleanliness in all parts of plants where the poisons are used or stored. Floors, walls, machinery, clothing and skin of exposed workmen should be thoroughly cleaned as often as practicable. When the toxic substance is in the form of fumes or vapors, adequate ventilating systems should be installed, and all pipes, tanks and receptacles of any kind should be absolutely leak-proof. Close-fitting, airtight goggles are advisable when other means of protection cannot be employed. When the toxic substance is a liquid, floors, should be made of cement or of some other relatively inert material, and should be protected by paint resistive to the liquids present. They should also be so constructed that the liquid, escaping from a receptacle, will not present danger to nearby workmen. Certain employees are abnormally susceptible to certain poisons; such workers should be transferred to other employ ment. Men and women with respiratory diseases or skin di seases, or who use alcohol to excess or who are generally allergic, are usually predisposed to the harmful effects of MEDICAL BULLETIN OCCUPATIONAL EYE HAZARDS__________________ 166 ft ftp ft . ft0 tPo cd 0Pa P 0 fctd f0t oP 0a to ft ft ftCjD CO rP pT ft ftp P XI P .H cd oo 0P0Pp ft R P ftrQ CQ CM rH o0 R J3o CO ft g ft p to P0 ft ft PrH p H p ft ft ft ft NCQ H ft s ft Pp o p rfPp CQ 0 ft ft ft R ft P ftPM cd p rH . ft P ft ft0 ft<D P 0 P o cd ftp ft i P ^ ft P R ft ft ft-(PD o P p 0p PM p ft CQ pH ft p P ft ^ -P ft m -- P0 -P > R g m 1--l tt) >p cd o ft J5 ft R P -p R --i &CD ,i--1 xpi o P CoQ Xi CQ RXI P ftcd CQ cd cd xoJ -P Pi &p PM P 0 ft ft fT --i 0 P P ft ft P ft0 CQ CQ ft S o ft O PH ' * P ft0 H ft ft o ^ P P ftP P P o p CQ p i--1 O po 1 opp fftt 1 opo fot 0 o P0 1 pcd M xl Pcdi to Pi PI --ooPH X> Pi 1 <p ft o p tPoi op P0 a 0 t> 1 1 P 0P m xRl Xl fot nRH -P 0 0 p 0 r--1 O -P O -H oo -p cd p -p p p H ft O p o p o ^ Vh Pi p ft m -h CROh R Q i--0l P *P u0 00 p 00 *p P P 0 i--1 R P 0 XftI 0 XftI 0 P P nPH O P 0 K*a P0 >0 0 oR p0 0p H rH P 0 Ai o ft 0 0 ap ft ft XI ft o P O p rH P o ft o Pi -P ft i--1 0 p p ft ft 0 ft l--1 R cd ft -p o P p p -p 0 p 0 23 Is 0 rH 1--1 pp xl xt P ft 0 ft tt P *P 0: cd 0 o ft o P>a ft 0 O p ft CQ P P *P d P Po -op fctd -p p cd Po cd CQ 0t>a 0 x0i -P O p O P fot 0 fot p p P0 s ft cd 0 pp X-pI 0 cd opffptt i-- rP CQ 0 po p 0 ft cd p ft o opo p p ft p *p o 0 o p p pfoat *p 0 i p a ft 0 xl p -p Pi tt 0 0 to-> cd P 0 l>a i--1 0O ^-p H rH 0 ft i--1 0> -P H "R cd Xi mUj Pi o o o x! p 0 0 h P {>a H O Cs) 0 M P R R PI xl O O cd 0 Is -H SO R P R R rH O ""3 Cd 0 p '*f0pt oo p o op H -P 0 > 0 P ft p p 0 0O o opp cd CO R O ft p p 0p W *H H cd -P |H cd 0 rH m O PH 1H o pft cd oH Ro fot H Cp CisO>-P Xi -p p R QS rH CHO oR PH R x) i--1 Xi tO 0 P p o o ft p p p ft o0 p p o o o p o ft 1--01 p p o p 0 01 1 0p p 0 p ft op P > o pto aoo 0 0 0p o rH pP ft e, 0 0 i> o rH 0 P p p X! Ri 0 0 >o o R p 0 a cd S R o p p pR o> P p rM CO R rH P O P v>a CQ fc> P P P ft 0O00 > 0 0 XI ft ir----11 xi p p p i *p a p ft 0 ao ft ft P 0 P P rHPh 0 fPt O 0 P^ 0 0 P 0 P P xoi 0 0 0 O rH a0 f0t 167 MEDICAL BULLETIN BIBLIOGRAPHY ( l) DAVIDSON, M. : Occupational Keratitiaes and. Corneal Dystrophsis, Arch, of Ophth. 21: 673-683 (April I939I ( 2) DORLAND, W.A.N. : The American Illustrated Medical Dictionary 13th Ed. W.B.Saunders Co., Philadelphia and London. (1925) ( 3) LANZA, A. J. and J. A. GOLDBERG: Industrial Hygiene, Oxford University Press, New York, pp 210-229. (1939)1 ( 4) GREENBERG, L. : Priceless Possessions, National Safety | News, Jan. 1940 p. 26. i| ( 5) RESNICK, Louis: Fifteen Years' Progress in Eyesight | Conservation in Industry, Sight-Saving Review, 7 Vol. IK: 3-11, (June 1931). 1 ( 6) THOMPSON, . G. : The Occupational Disease, D.Appleton &-j Co., N.Y. (1914). ' ( 7) HOPE, E, W. : Industrial Hygiene and Medicine, Wm.Wood & Co., New York pp. 415-430 (1933). ( 8) BENDICT, W. L. Ocular Disorders Due to Exogenous Tox emia, Digest of Treatment, 3: 546-548 (jan. 1940). ( 9) CARROLL, E. D. : The Toxic Amblyopias, Digest of Ophth. & Otolary. II: 350-354. (Mar. 1940). (10) LEAFLETS. Dept. Scientific & Indust. Research. His Majesty's Stationery Office, London. (1937-1939) (11) SNELL, Simeon: Brit. Med. Journal (1894) Quoted by Thos. Oliver in Dangerous Trades. (12) OLIVER, Thos.: Dangerous Trades, John Murray, London (1902) p. 482. (13) HANNUM, J. E. ; Eyesight Conservation Survey, The Eye Conservation Council of America. New York (1925) PP* 114-144. (14) DRINKER, C. K. : Carbon Monoxide Asphyxia, Oxford Uni. Press, London, pp 60-6l. (1938) (15) DAVIDSON, M. : Occupational Keratitises and Corneal Dystrophsis, Archives of Ophthalmology, 21: 673^683 (April 1939). (16) RANKINE, D. : Artificial Silk Keratitis, Brit. Med. Jour. 2: 6 (July 4, 1936) (17) KLEIN, E. : Les lesions oculaires dans les fabriques de soie artificelle. Arch, d'opht. 45: 686 (Nov. 1928) (18) BtfCKLERS, M. : TropfschenfUrmige Niederschlage auf der Hornhautoberflhche bei Mobilarbeitern.-Klin.Monatsbl f. Augenh. 99: 676 (Nov. 1937)' 'Mm McDonald, r.; Ophthai bon Disulphide and Viscose Rayon Indr ease Prevention Di : try, Harrisburg, I |) LANE, L. A. : An Occup J; and Adnexia. Surg. If) LANE, L. A. : Occupati ' Eye and Adnexia. / |P (March 1939) fib} .INDUSTRY REPORT, Ret? P (June 1939) |23) :INDUSTRIAL EYE HAZAR] jp National Safety C< 24) BLACK, N. W. : The Ey< the Extens. Div.,1 || Gen'l. Series No. 1(25) NATIONAL BUR. OF STA1 p ard Safety Code I i; and Respiratory 0 I (1938) 1(26) LIPKOVITCH, I. G. : D If- gieneischer Stand I 286-302 (1934) 1(27) POZZI, N. : Les dange If' Mai. profess. 1: (28) QUERIES & MINOR NOTE - Med. Assoc. 109: '(29) BRITTON, J. A. and E Jour. Ind. Hyg. & : (30) ROY, N.J.: Monocular Origin. Am. J. Op (31) VURDEMANN, H. V. : Tt f Light Burn From I Am. J. Ophth. 19: (32) GRANJOU, R.: Safety ing and Oxygen Gi ' gene 27: 8-10 (IS . (1936) (33) CHAUTIN, S.: Visual Arc Lamp. Sirret stracted in Jour. (34) LYLE, D.J. and C P.I tional Diseases.! (35) RESNICK, Louis: Eye (From manuscript be published by ( i.nd Corneal -683 (April 1939) 3d Medical , Philadelphia rial Hygiene, o 210-229.(1939) ational Safety in Eyesight g Review, se, D.Appleton & icine, Wm.Wood & Exogenous Tox8 (Jan. 1940). Jigest of Ophth. isearch. His . (1937-1939) t) Quoted ty Murray, London urvey, The Eye 3w York (1925) PP- ia, Oxford Uni. and Corneal ogy, 21: 673r683 1, Brit. Med.Jour. les fabriques de : 686 (Nov. 1928) .-'schlage auf der srn. -Klin. Monatail OCCUPATIONAL EYE HAZARDS 168 (19) MCDONALD, R.: Ophthalmological Aspects in Survey of Car ton Disulphide and Hydrogen Sulphide Hazards in the Viscose Rayon Industry. Bull. 46, Occupational Dis ease Prevention Div., Penna. Dept, of Lator & Indus try, Harrishurg, Pa. (1938) (20) LANE, L. A.: An Occupational Study of Cancer of the Eye and Adnexia. Surg., Gynec. & Ohst., 64: 458 (1937) (21) LANE, L. A. : Occupation in Relation to Cancer of the Eye and Adnexia. Amer. Jour.Ophtha. 22: 267-278. (March 1939) (22) INDUSTRY REPORT, Retail Credit Co., Atlanta, Ge. IV: 8 (June 1939) (23) INDUSTRIAL EYE HAZARDS Health Practices Pamphlet No. 6, National Safety Council, Chicago, 111. (1934) (24) BLACK, N. W. : The Eye In Industrial Accidents. Bull, of the Extens. Div.,Univ. of Wisconsin. Serial No. 919 Gen'l. Series No. 707- Madison, Wise. (I9I8). (25) NATIONAL BUR. OF STANDARDS HANDBOOK H24: American Stand ard Safety Code for the Protection of Heads, Eyes, and Respiratory Organs. Washington,Govt.Print.Office. (1938) (26) LIPKOVITCH, I. G.: Die Elektroschweissung vom gewerbehy- gieneischer Standpunkt. Arch. f. Gewertepath. 5: 286-302 (1934) (27) POZZI, N.: Les dangers de la soudre a l'arc. Arch, des Mai. profess. 1: 389-4l4 (1938) (28) QUERIES & MINOR NOTES: Electric Arc Welding. Jour.Amer. Med. Assoc. 109: 975 (1937) (29) BRITTON, J. A. and E.L.WALSH: Health Hazards of Welding. Jour. Ind. Hyg. & Toxicol. 22: 125-151 (April 1940) (30) ROY, N.J.: Monocular Blindness from Light of Electrical Origin. Am. J. Ophth. 12: 822-824 (1929) (31) WURDEMANN, H. V. : The Formation of a Hole in the Macula. Light Burn From Exposure to Electrical Welding. Am. J. Ophth. 19: 457-460 (1936) (32) GRANJOU, R.: Safety and Sanitary Measures in the Weld ; ing and Oxygen Cutting of Materials. Rev.Soudre Auto gene 27: 8-10 (1935) Abstracted Chem. Abstr.30: 1347 ' (1936) (33) CHAUTIN, S. : Visual Adaptation Among Workers With the Arc Lamp. Sirret Viestnik Ophth. 5: 239 (1934) Ab stracted in Jour. Ind. Hyg. & Toxic. 17:83 (1935) (34) LYLE, D.J. and C P.McCORD: Eye Symptomatology in Occupa tional Diseases.Nations Health.p.613 (Nov. 1923) (35) RESNICK, Louis: Eye Hazards in Industrial Occupations (From manuscript of forthcoming revised edition to be published by Columbia Univ. Press, New York,N.Y.),