Document rpMMkYyRYG2O9rp7J5MEZXErG

'Industrial Hygiene Digest - 55 January, 1950 and frequently progressive Inflammatory reaction In the exposed animal. ----- By employing native, In contrast to autoclaved Bagasse and its extracted - resins minerals, It could be demonstrated that the fiber Itself calls forth a foreign body reaction which la amenable to healing; The progressive Inflammatory reaction and death of the animal, however, are due to micro organisms attached to the bagasse. The authors conclude that the Inorganic part of the Inhaled bagasse dust produces a long-standing tissue reaction vhlch Is primarily a foreign body response. It is unlike silicosis and Is amenable to healing by resolution. Superimposed an these lesions, , there occurs in those animals which are more susceptible to the causative micro-organisms acute branchlolltlc and pneumonic changes which, If t sufficiently extensive, may cause death of the animal. A similar combina tion of etiologic factors seems to be the most plausible explanation of the complex picture of human bagasse disease. -- Condensed from Arch. Ind. Hyg. & Occ. Med. 112 Removal of Mists and Dusts from Air by Beds of Fluidized Solids. H.P. Meissner and H.S..Mickley. Ind. & Eng. Chem. 3-1, 123^ (June, 1949). These studies were made on the principles of the technic by which sulfuric acid mist may be removed from air by beds of fluidized solids. Exploratory work an removing ammonium nitrate dust by this method Is also described briefly. -- Condensed from Arch. Ind. Hyg. & Occ. Med. / 115 Lung Lesions In a Corundum Smelter. H. Gartner. ZtBchr. f. d. ges. Inn. Med. u. d. Grenzgeb. 2, p. 761 (1947)* This article Is a discussion an the pathological findings In the lungs of two oven workers In the process of corundum smelting. 3he con clusion arrived at 1b that the substance which causes the damage to the corundum smelter's lungs Is really mullite (slUlmanlte), possibly In combination with amorphous silica. -- Condensed from Bull. Hyg. 114 Empyema of Kasai, Sinuses As An Occupational Disease Due to Dust Inhala tion. W. Schmidt. Pract. oto-rhlno-laryng, 11, p. 282 (1949). Schmidt says that In the course of animal experiments carried out over a period of years attention has been given to the effect of Indus trial dusts an the mucous membranes of the nose and of the nasal sinuses. Babbits exposed to Industrial dusts frequently presented empyema of the Twmal sinuses. This empyema does not differ in Its effects on the mucous lining, from the empyema of the nasal sinuses of human subjects. Consequently when dealing with workmen who are exposed to dust the physi cian should give special attention to the nasal sinuses'in addition to the main cavities of the nose. -- Arch. Ind. Hyg. & Occ. Med. 115- Contribution to Study of "Car Pulmonale" In Silicosis. P. Lavenne. Acta cardlol. J+, p. 245 (19^9) Lavenne's report concerns three coal miners with silicosis who died from cardiac decompensation. Two had pseudotumoral silicosis complicated with vesicular emphysema. In the third the disease was still In the nodular