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i I i ro Table V. X-Ray Appearance ASBESTOSIS SILICOSIS Diffuse lesion* limited to lower halve* t Nodular lesions distributed more in of lungs--Hyperventilation in upper upper and mid-Jung fields or general- By the same token, extreme care should be taken to eliminate contact with tuberculosis from these work ers. This can only be accomplished by repeated chest x-rays of all those exposed to hazardous dust and removal of the ones showing evidence of infection. portions. iuid-empbyaem* in lower halves. Obliteration c! the diaphragm. No nodulation- Shortening of long diameter of cheat with adhesion* and tenting of dia phragm. Marked nodulation. Bibliography It Kionshbebc and Home: Health Httirds of Occupational En vironment*, Department of Public Health Circular--164. 2. Saffinctok, C, O.: Essential* of Indoitrial Health. Lfppincott, "Ground glass1* appearance. Discrete nodulation to manive con 1946. pp. 166-187, glomerate shadow*. 6. Ganb, Lu U-: Pathology of Silicosis. Second Symposium on May be unilateral. BDeteraL Silicosis, Saranac Lake. Hew York. 1966. 4. Dunkb and Hatch : Industrial Dust. McGraw-Hill Book Com- pan?. Inc.. Hew York. 1966, p. 2. Certain other conditions such as fungus infections, 6. JOBNKOttE, R, T.; Occupational Diseases. TV.B. Saunders. Phila miliary tuberculosis, miliary calcification and miliary delphia. 1942. p. 104. carcinoma produce shadows in the roentgenogram 6. Panooast, H. K.: Siiieosls and Asbestosis--Introduction. Lanza. Oxford Medical Publications, 1988, which may be confused with those of silicosis. In these 7. Lanza, A. J.: Siliooels and Asbestos!*. Oxford Medical Publica instances careful study of the film and previous occu tions, 1083, 8. Gakonu, L. U-: Symposium on Silicosis, Saranac Lake, 1634. pational history will usually be sufficient to make an 9. Sayers, XL B-: Reaction to Mixed Dusts: Atmospheric and Clin i accurate diagnosis. As in all diseases due to dust, treatment is an en ical Finding* In Hard Coat Mining, Fourth Saranac Laboratory Sym posium on Silicosis, 1989. 10. Dkessen, W, G,, Dallavalle, J, M., hbWAftPS. T. I., HltLEft, J. gineering problem rather than a medical one. Once fibrosis is established in the lungs, it is permanent. Progression of the disease is very slow and chronic, IV,. Savers. R. R.: A study of Asbestosis in the Asbestos Textile In dustry. United States Public Health Bulletin--241, 1038. 11. Gardner. L. U.: Recent Developments in Relation to Silicosis. Industrial Medicine, 9:45, February, 1940. except in some instances where infection occurs. Con 12. MSttWETHEiu E. R. A., and Puce* C. W,: Report on Effect* of Asbestos Dust on the Lungs and Dust Suppression in tbe Asbestos In i! ( i tinued exposure to hazardous dust appears to be the biggest factor in the advancement of fibrosis. When dust is kept at a safe concentration in the air of work ing places, there is no good reason why a person with dustry. H, M. Stat. Office, London, I960. 13. COOK. W. E.: Asbestos Dust and the "Curious Bodies" Found in Pulmonary Asbestosis. British Medical /camel. 2 :$78, 1929. 14. Gardner, L. U.: Saranac Lake, New York, Personal Communica tion, 1943, uncomplicated silicosis cannot continue his occupation. 15. Sampson, H. L.: Third Symposium on Silicosis. Saranac Lake, New York, 1937. i t SPNY 000233