Document n9816z22GDj7oEqpR5DZ1oEzG

II 31135401 THIS IS AN OFF-LINE BIBLIOGRAPHIC CITATION LIST GENERATED BY HEOLARS II N.L.H.'S NATIONAL INTERACTIVE RETRIEVAL SERVICE 3CLER0DERHA NUMBER OF CITATIONS PRINTED IS THIS SEARCH NAS PERFORMED ON THE MEDLINE SORT 3 SORT FORMAT t'AUTI'). (ASCENDING) NAS SPECIFIED REQUESTED BY R. N. WHEELER MAT 31. 1984 FILE. PLEASE SEND THIS LISTING TO 00019215 SCLERODERMA PAGE 1 5C2S S liC 1 AU - Blseecla EP > Scarborough DA ; Lowney ED TX - Atrophoderma of Pa*> nl and Plarlnt and syitamlc sclarodanna [latter] - Eng - Adult ; Atrophy ; Casa Report > Female ; Humwt - Scleroderma, 3ytamie/*etIology 1 Skin/apathology - Arch Dermatol 1982 JanillAtl):l-2 Bon 1 no 11V ] Btancht C ; Blanchl 0 Garcia Garcia A - [Post-traumatic nodular scleroderma] - Spa - Scleroderma of rare appearance In children appears In minor scale as to the five per cent on the whole Incidence of this collagen disease. The children usually present localized scleroderma and at times associated with other pathologies, traumatisms and Injections were referred. Two patients aged 5 and 13 years old are presented, both with nodular lesions on anterolateral thigh area, and In the right buttock respectively. The patches of a side bigger than the palm of hand were only touchable and the skin that covered them only showed a slight hyperpigmentation In the edge In one of the cases. The limits were not precise and the nodulose surface was Irregular. The evolution was a* of two and three years, right after Inoculation of antit*tanteal vaccination and puncture thorn of Yuca leaves. The histological control showed Intensive phenomenons of fIbrohyallnosls covering almost all the dermis. The studies of the laboratories didn't produce Interesting data. The histological and clinical set of symptom* shows differences with the esclerodermlc like states as a consequence of excipient of vitamin K, B 12, norhydroxprogesterone and anti-tetanic serum. In which they settle In the cellular subcutaneous tissue and they Involution spontaneously. There are also differences with the panlculttts artefacts and with the linear morphea associated to bifid spine. At last the authors make special mention of the ease described by Desmons of progressive linear scleroderma right after the triple vaccination. The nodular or subcutaneous scleroderma Is a clinical form In which the histological alteration decays In deep dermis and superficial hypodernl*. The eases shown suggest a conection between a previous traumatism and the nodular scleroderma. - Adolescence i Case Report } Child, Preschool i English Abstract - Female I Human i Injections, Xntradermal/*adverse effects ; Male - Scleroderma, Clrcumscr1bed/et1ology/pathology - Mounds, St ab/*complteattons - Med Cutan Iber Lat Am 1983ill(5>>329-32 81112 00019216 3CIER00ERMA -------------------------------------------------------------------- ucc PAGE 2 065039 3 AU - Brentnall TJ t Kemeelly D I Barnett AJ I de A1 zpurua HJ AU - Lolatt 3J i Ashcroft R I Toh BH TX - Autoantibodies to fibroblasts in scleroderma. LA - Eng AB - Sera from 33 patients with scleroderma were examined for Immunofluerescent reactivity with vldble or acetone-fixed fibroblasts. All 33 sera reacted with the cell surface membranes of viable fibroblasts. 23 of 33 sera (702) also reacted with 00019216 SCLERODERMA PA6E 2 ^ 3322111 3 All - Brentnall TJ i Kertneally D t Barnett AJ J da Alzpurua HJ All - Lolatt SJ i Ashcroft R i Toh BH TI - Autoant Ibodies to fibroblasts In scleroderma. LA - Eng AB - Sara from 33 patients with scleroderma were examined for Immunofluoraaeant reactivity with viable or acetone-fixed flbroblaiti. All 33 sara reacted with the call surface membranes of viable fibroblasts. 23 of 33 sera (70X) also reacted uith nuclei of acetone-fixed fibroblasts. The commonest nuclear staining pattern was homogeneous (46Z) followed by nucleolar (36X) and speckled (23'/,). 707. showed more than 1 staining pattern In the same serum. Antibody tltres of homogeneous and nucleolar staining patterns (1*8 to 1:1024) were generally higher than those of the speckled pattern (1:8 to 1:256), No change In pattern or tltre was noted In sara from 4 patients over a 10-12 month period. Antibody In sera with homogeneous or nucleolar staining patterns belonged to one or more of the 3 major antibody classes, Xg6, Xgfl or IgA while antibody In sera with a speckled nuclear pattern belonged to the IgH class only. No correlation was found between the pattern of anti-nuclear reactivity and visceral Involvement. - Adult ; Aged ; Antinuclear Factors/analysls - Autoant1bod1es/anelysis/*biosynthesis ; Female Fibroblasts/analysis/immunology > Fluorescent Antibody Technle - Human ; Xmmunoglobulinsi Surface/analysis I Male } Middle Age - Scleroderma, Systemle/etlology/almmunology - Support, Non-U.3. Gov't - J Clin Lab Immunol 1982 May;8(l>:9-12 - Garza-El1sondo MA J Dias-Jouanen E ! Franco-Cas1 qua JJ - Alarc'on-Segovia D - Joint contractures and scleroderma-like skin changes In the hands of Insulin-dependent juvenile diabetics. - Eng - He studied 34 unselected Insulin-dependent juvenile diabetics by seeking contractures at the proximal interphalangeal (PIP) joints and scleroderma-like changes of the hands or elsewhere. He found 14 contractures of only the 5th PIP In 7 and of the PIP of other fingers as well In the others. Nine of these patients also had scleroderma-like skin changes. Only one of 34 age and sax matched healthy controls had a minimal eontracture of the 5th PIP joint but had no skin changes. All patients found to have these abnormalities had had diabetes for 6 or more yew's and the difference In the disease duration between those with hand changes and those without was significant Ip less than 0.01). There was no correlation of these changes with renal or ocular vascular changes (n this small group of patients. - Adolescence l Adult ; Contracture/aetiology - Diabetes Hellltus, Xnsulln-Oependent/ecomplIcatlons t Female - F1nger Joint t Hand Deformities, Acqulred/oetlology - Hand Dermatoses/etiology t Human ; Male - Scleroderma, C1rcumsertbed/*et1oiogy i Support, Non-U.S. Gov't 23311 ucc 065040 00019217 SCLERODERMA PAGE 3 SO - J Rheumatol 19S3 Oct;10(5):797-000 3 AO - Graham-Brown RA > Sarkany 1 TI Scleroderma-like changes due to chronic graft-versus-hoit disease. LA - Eng Ml - Adult ; Bone Harrow/transplantation ; Casa Report Ml - Child. Preschool > Chronic Disease i Female MH - Graft vs Host D1sease/complicat1oni { Human i Male MH - Scleroderma. 3ystem1c/*et1ology SO - Clin Exp Dermatol 1903 Sep 18(5) 1331-0 6 AU TI ,U MH MH MH MH SO - Grebennikov VA l Challmova RA - fTrantformatlon of scleroatrophic lichen Into systemic scleroderma! - Rus - Case Report ; English Abstract > Female 1 Human - Lichen Planus/*complteat ions 1 Middle Age - Scleroderma. Clrcumscrlbed/complleatlons - Scleroderma. Systemtc/e11ology - Vestn Dermatol Venerol 1903 Apr>(9):60-3 7 AU - Guseva N8 TI - tEtlology and pathogenesis of systemic scleroderma] )A - Rus >91 * Adenosine Cyclic Monophosphate/biosynthes1s . Animal >91 - C-Type Viruses > Collagen/blosynthesIs S Flbroblasts/eetabollsm HH - Human ; Microcirculation S Review MH - Scleroderma. Systemte/et1ology/fam111al 0 genet1 c/1mmunology physlopathology t Virus Dfseases/complfeat Ions SO - Revmatologlla (Moskva) 1903 Apr-Ju!(2)53-10 0 AU - Ha RH TI - (Report of t cases of primary hypothyroidism with accompanying scleroderma arid Raynaud's phenomenon (author's transit] LA - Chi MH - Adult > Casa Report ; Female I Human MH - Hypothyrotdlsm/compl1 eat1ons/1mmunology . Middle Age MH - Raynaud's Dtsease/*at(ology i Scleroderma. Systernic/*ct)ology SO - Chung Hua Net Ko Tsa Chih 1902 Febi21(2):7S-7 9 AU - Jlm'ene* SA TZ - Cellular Immune dysfunction and the pathogenesis of scleroderma. LA - Eng Ml - Animal > Collagen/biosynthesIs I Flbroblasta/lamunology/pathology Ml - Graft vs Host React1 on i Human I Immunity. Cellular Ml - LymphoktnesXimmunology I Monocytes/Immunology ! Rats i Review Ml - Scleroderma. Systeml c/et 1 ology/<*1mmunology/pathology HH - Skln/spathoiogy I Sujsport. U.S. Gov't. P.H.S. Ml - T Lymphocytes/Ptwwunology SO - Semin Arthritis Rheum 1903 Aug>13(1 Suppl 1)1109-13 00019210 SCLERCOERMA PA6E 9 ucc 10 AU - Lee EB I Anhalt GJ | Voorhees JJ ) Dias LA 065041 TX - Pathogenesis of scleroderma. Current concepts. LA - Eng Ml - Autoimmune Dlleases/immunology I Collagen/analysll/metabol1 tm Ml - Hunan } Scleroderma. Cl rcumicr1 bed/et1ology/1 mmunology/pathology MH - Scleroderma, Systemlc/*et1ology/lmmunology/metabol1sm 00019218 SCLERODERMA PASE 9 8222223 10 AU - L* EB t Anhalt SJ ; Voorhees JJ ) Diaz LA TI - Pathogenesis of scleroderma. Currant concapta. LA - Eng - Autoimmune D1seases/lmmunology i Collagen/analys1s/metabolts Human i Sclarodarmai Ctrcumicribed/*etiology/immunology/pathology - Sclarodarmai Systamic/*etiology/1mmunology/matabol1sm - Skin/analysis/blood SupplyAllTraitrucTura - Supporti Non-U.S. Gov't t Support. U.S. Gov'ti Mon-P.H.S. - Supporti U.S. Gov'ti P.H.S. ! Vaaeular Diseases/complieat Ions - Int J Oarmatol 1989 Mar;23(2):8S-9 - LaRoy EC - Pathoganaata of iclarodarma (systemic aclarosls). - Eng - Increasing Interast In tha vascular faaturas of iclarodarma has lad to tha hypothesis that tha blood vassal Is tha major targat tissue and that tha endothelial call Is tha principal call targat. Usaful observations stemming from tha vascular hypothasls Include tha use of mlerovascular abnormalities in tha early detection of tha patient destined to develop classical sclarodarmai tha discovery of a serum protaasa selectively cytotoxic to endothelial cellsi and tha study of a serum mitogenic activity for fibroblasts In scleroderma patients. Immune events related to the vascular lesions are under active study but have not as yet provided a unique immunological lesion in scleroderma patients. The possibility that Immunity to basement membrane (type IV) collagen may be selective for scleroderma patients deserves further study. Persistent Iwrncnlty to endothelial basement membrane structures would provide a basis for continued endothelial injury. Techniques to quantify endothelial Injury are useful to assess activity of the vascular lesions and to monitor therapies designed to block further vascular Injury. The definition of pre-fibrotle vascular lesions may have future therapeutic and preventive Implications for scleroderma. - Blood Vessels/pathology i Collagen/blesynthesis - EndothellMi/pathology ; Fibroblasts/metabolism i Human - Sclarodarmai Systemic/aetlology/metabolism/pathology - J Invest Dermatol 1982 Jul;79 Scppl l:87s-89s 8232 12 AU TI 1A AB - Lukaschek E - [Radiologic changes In the bones of the hand In progressive scleroderma) - Gar - 27 patients suffering from progressive systemic sclerosis on both hands were examined through x-ray in two levels. The findings have been evaluated with regard to eight diagnostic eriterions and then related to clinical and laboratory data. The results allow tha differentiation between two groups of patients: groiqs A revealing obvious radiological and laboratory evidence of Inflammation and severe symptoms as well as a relatively short dvs-at 1 on of the disease) group B showing fewer Inflammatory sljjss ucc 065042 00019219 SCLERODERMA PAGE 5 with lii developed symptoms and ligntflcantly longer duration of tha disease. The clinical and prognostic tlgnlflcanca of thasa tuo dlffarant courses of PSS is dfscussad with ipacial rafaranca to ovarlapplng syndromes of eonnactiva tlssua dltaasas or subclasses of PSS. MH Adolescence S Adult ; Age factors i Aged m Bone and Bones/*rediography > English Abstract ; Female m Hand/*radiography ; Human > Mala ; Middle Age MH Raynaud's Dlsease/complleat ions Scleroderma Sys taraic/at 1ology/*radiography so Z Hautkr 19S2 Nov 15!57(22):1649-63 13 AU TI LA AB MH W MH SO Ml ike T i Ohtani Y > Hattori S > Ono T ; Kageshlta T ! Matsuda 1 Childhood-typa myositis and linear scleroderma. Eng A 5-year-old girl had linear scleroderma on tha flexor surface of tha right arm; muscle wasting Included tha shoulder girdle. IgM fluorescence on blood vessels and along dermal-epidermal junction was observed by direct Immunofluorescence in blopslad skin. Biceps muscle underlying the plague of the scleroderma showed atrophy of entire fascicles* perl fascicular atrophy* and cellular infiltration around blood vessels that are quite similar to those found In childhood-type dermatomyos1t1s. In addition* various abnormalities* Including edema and thickening of basal laaiina* were found on blood vessels in muscle tissue. The results suggested that the autoimmune collagen vascular disorder is responsible for this condition. Autoimmune Diseases/eompllcations } Case Report ; Child* Preschool Collagen Olseases/etiology i Female ; Henan Myos111s/compl1 cat 1ons/et1ology/*pathology Scleroderma > Sys tern1c/compl1 cat t ons/et1ology/"pathology Vascular Diseases/etiology Neurology (NYJ 19S3 Jul*33(7)>928-30 14 AU - Rankin JA t Matthay RA TI - Pulmonary renal syndromes. II. Etiology and pathogenesis. LA - Eng AB - Numerous systemic diseases share immunopathogenic mechanisms. This article reviews the proposed etiologies and immunopathogenic mechanisms of a yovp of diseases which share pulmonary and renal abnormalities. Spedfieally* we dlseuss the following diseases: Good-pasture's syndrome* systemic lupus erythematosus> progressive systemic sclerosis* Megener's granulomatosis* lymphomatotd granulomatosis* and Churg-Strauss syndrome. MH - Animal t Anthropoidea i Antigen-Antibody Complex/immunology MH - Antinuclear Factors/Immunology j Autoimmune Diseases MH - Basement Membrane/Immunology t Collagen 01seases/*et1ology tffl - Diseases in Twins * Eosinophi11 a/1mmunology IVt - Blomerulonephritis/tmmunology > Goodpasture's 3yndrome/et1ology ttl - Human ; Hypersensitivity* Delayed/immunology > IgE/immunology ' m - Kidney Glomerulus/Immunology ; Langarhana Cells/cytology MH - Lupus Erythematosus. Systemic/etiology ; Lymphoktnes/tmmunology MH - Lymphomatold Granulomatos i s/*et t ology ) Macrophaget/immus-tology 5 D ) > 3 3 ^ 'h J| II. ucc 065043 82222 SCSfcg S 2 2 2 ;iS 8222S 00019220 SCLERODERMA PACE 6 - Neutrophili/1mmunology t Pulmonary Alveoli/cytology i Review - Scleroderma. SystemIe/et 1oloqy > Sheep ; T Lymphocytes/1mmunology - Vasculitis. Allergic Cutaneous/*etfology - Wegener's Sranulomatosi*/etiology - Yale J Biel Mad 1962 Jan-FebiS5(1>>11-26 - Rlmbeu E i Aub'ia J i LIorach 1 ; Llovera* J ; Hesram'on J - O-flla MA ; Lloraeh M - (Effectiveness of tha angiotensin converting enzyme Inhibitor in clarodarmic crista (latter)} - Spa - Case Raport > Human i Ktninasa II/antagonits 2 Inhibitors i Mala - Middla Age ; Raynaud's Diaaaaa/complications/enzymology - Sclerodarma> System1c/*enzymology/ettology - Med Clin (Bare) 1983 Jan 29i80(2):89 - Sat tar MA i Cawley MI - Scleroderma and carcinoma of uterus. - Eng - Adenocarc1noma/*compl1cations i Aged i Casa Report - Cervix Naoplasms/ecomplicat tons i Female ! Human - Scleroderma! Systemic/eatiology - Br J Clin Praet 1963 Feb;37(21:69-70. 72 - Toyota T ; Unezu M i OIKawa N ; Sanoyama R t Suzuki S J Suzuki H - Nakajima Y i Goto Y - Diabetic scleredema. - Eng - Many skin lesions are specific for diabetes mellitus. Necrobiosis lipoidica. ltpoatrophy and Idiopathle bullae (bullosis diabeticorum) are usually associated with diabetes. However> diabetic scleredema has not been noticed by internists, although dermatologists have paid attention to such a cutaneous manifestation. He reported a clinical case of a female diabetic patient aged 15 who had been afflicted with diabetic scleredema. She had been treated with insulin since 5 years of age. She noticed stiffness of the skin In April 1986. Skin biopsy showed thickness of the dermis and accumulation of acid mucopolysaccharide. After control of blood glueose with continuous subcutaneous Insulin infusion (CS111 and attaint strati on of tocopherol acetate and hyaluronldase. the skin lesion Improved. Etiology of dlabetie scleredema is unknown. Such skin lesion which Is observed frequently In insulin dependent obese patients is different from a category of selaredema of Buschke. - Adolescence t Case Report > Diabetes MaliItus/scomplteatIons - Diabetes Mellitus. Insulln-Depandant/complIcatlons/pathology Female t Human S Male S Scleredema AdultonWet1 ology/pathology - Skin/pathology - Tohoku J Exp Ned 1983 Dec 1141(6>>657-61 83122 ucc 065044 00019221 SCLERODERMA PAGE 7 813 18 AU * Urbeno-Merquez A TI - EosInophlllc fasciitis evolving into scleroderma [latter] - Eng - Adult ; Case Report i Fatcltt1*/*compl1cations ; Human ; Male - Scleroderma. Systemle/eetlology > Time Factor* - Ann Intern Med 1983 Sepi99(3>:412 END OF OFFLINE PRINT ucc 065045