Document jRKQaqm9w99pqnpQQkq2VMBp

; !Jf C '* is %!rm* eituaj . The American Journal of Medicine e:t l.i A. fi4*' A1 Krut.i VOLUME 42 APRIL 1967 NUMBER 4 arj*' m r nd tcrux, uuf* I. rr. ) t^irar t. ^rr.tu *. Cm. ol?*n- tirinr. *i. btm>> . IU-. i.Thc a &Jebe ttude r, 19: .-rrtity {eo> dy of (Otifttt luoro- <UBUO Porter Dam, hock hymai il cit- |y. b Editorial a Asbestosis and Neoplasiav il least one fourth of the adulu who die still an assumption, an extrapolation from the in urban areas in the United States and observed data since we do not know at this time 1other industrialized countries asbestos bodies whether or not the small amounts inhaled at the ut found in the lungs (/-]. This observation result of asbettot air pollution have an clfrct has led to the recognition of ubesto-rcUted comparable to the much larger amounts inhaled duease at a potential serious modem urban haz ard I7-P). in industrial or intimate environmental eircunutances. The question will nut be resulted I'or Indeed, Thomson (d) has predicted that tome time because of the long latent period asbestos-induced neoplasms will rival cigarette- between initial contact and the jppr.ir.uirr ul' induced lung cancer in the next several decades. clinical tlisr.ise. Although Ihrrr are t .iri.itiuns This prediction is based upon five observations based upuu individual idiostucrast. imtm.icv. >nd one assumption. The known data may be summarized as follows: (1) Asbestos inhaled under industrial conditions will produce a serious pneumoconiosis (pulmonary asbestosis) |10| *y*n with relatively light exposure 111]. (2) k'ndcr similar conditions of heavy [/?) or light duration and coiitimutv of exposure. and per haps also upon such factors as tlie t.irirtt uf asbestos used tchrssotile. .inM>ur. nuruMitc. aulhuphtlitr. treiiiuliirl .md die pre-encr uf ullwr coiicuuiitani dusts. Itv .uni l.uce it u..it It said that divase caused In astiem* i- r.uelv (UJ industrial exposure, workmen die of neo- iiianifrslrd in lest than ten lu fucutv tears hum plasms more frequently than expected. (3) Widespread contamination of the environment onset of exposure. t,J-15| may occur bod) from industrial sources (uch as asbestos-containing dusts from con- ruction tites) and from disintegration or use of "ktioi products (such as asbestos-containing r tiles and brake linings (W). (4) Such tneirouiuental contamination may result in w'dejprrad inhalation and retention uf small numbers of the mineral fibers |/-0'|. (5) Under Cfnain conditions (for example, residence within * *aali-<uilc of uu asbestos factory or in the house- "W uf an asbestos worker) eitvircmtitrut.il 'xPU.iirc has l>ccn shown to produce serious dijcase |/7 -jmJ. The prediclinu that widespread 411"<* eoiit.uitiiiiition of city air will be assort*lr<l cviilt widespread neoplasia, liuwrver. is 1`i'i.tuix.tut t-msiu-i The case re|x*ri uliicli y.itr die Jw.ise n> name [J/| was uf a tiumnii asliesto- irtul.- operator who lud Uipm this stork tuentt te.us In-litre. Alerted lv this .mil siiuilar c.w. a survrv of Dritish f.icturirs n.is uululake:-.. evidence ol nslvsiinis u.is louml lu It . on. hut uulv in thtisr islio-e ivenp.itiou.il ixjiueiir to aglvstos hail Ivcim m.mt t,-.n- lietoi,- Similar ev|*eiieuves sure -non ieruiue,i m dir United Siaies |.V|. l'lie l omuri p.m oi u-m o.i- Ctnis was almost nr\ri uhteis-tl, e\r*'p* ;t,ilu|i. uiidri tin* must tnmsii.tl riiciiinu.tMi e- 1 Tins silo.moil utl olil.utis In a M..-;e -i..,t <87 HFM-011699 8004 0038 SCF-FA-5920