Document Y6Vdmmn9dwm93e7RwQwE349K
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1. EPA has already taken steps that will largely remove lead from the nation's atmosphere.
Leaded Gasoline. In 1981, gasoline consumption accounted for 85.9 percent of the 40,739 tons of lead emitted into the atmosphere in the United States. EPA has since promulgated new lead phasedown regulations that will all but eliminate leaded gasoline, and is considering a total ban.
Industrial Emissions. In 1978, EPA adopted National Ambient Air Quality Standards limiting atmospheric lead con-
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centrations to 1.5 ug/m . At that time, lead concentrations around major sources averaged as high as 17 ug/m"* or more -- almost 12 times the level permitted by the new lead standards. Plans for implementing the lead standards haye been adopted in all but two states. As these plans are carried out, lead industrial emissions, which accounted for less than 15 percent of the U.S. total in 1981, will drop to a fraction of their former levels.
2. Despite the steps it has already taken, EPA is now considering reducing the national lead standards still further.
The Present Standard. EPA adopted the existing 1.5
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ug/m lead air standard on the basis of three principal findings:
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First, anemia occurs in children at blood lead levels
of 40 ug/dl, and a blood level of 30 ug/dl would assure protec
tion against anemia with an adequate safety margin.
Second, one microgram of lead in air produces two
micrograms of lead in blood (the so-called air lead/blood
lead ratio).
Third, in order to bring the blood lead levels of
virtually all O.S. children below 30 ug/dl, the mean blood
would have to be 15 ug/dl. Since there was evidence that the
average child's background blood lead from non-air sources
was 12 ug/dl, the permissible air concentration was 3 ug/dl
(15-12*3) which, employing the 1:2 air lead/blood lead ratio
dictated a standard of 1.5 ug/dl.
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EPA1s Current Review of the Lead Standard. EPA is
completing work on a revised lead criteria document. Early
drafts of that document and of a staff report recommending a
possible reduction in the lead standards based on the document,
indicate that EPA may lower the 30 ug/dl blood lead threshold
for adverse effects to 20-25 ug/dl based on seven blood lead
effects observable, or alleged to occur, at lower levels.
As the accompanying papers show, four of these effects
-- inhibition of Py-5-N, ALA and FEP elevation and reduction of
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vitamin D activity -- are detectible at lower blood lead levels,
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but the Criteria Document does not state in what way these
effects are harmful. Indeed, because of improved laboratory
analysis techniques, a number of enzymatic and other bodily
reactions to lead can be detected at blood lead levels as low
as 5 ug/dl, far below the 30 ug/dl threshold for adverse health
effects established by EPA in 1978, but this does not mean
such effects are harmful.
As to a fifth effect, on cognition, an expert panel
appointed by EPA found that there were "no studies that, to
date, have validly established ... a relationship between
low level lead exposure and neurophysiologic deficits in
children."
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The remaining two effects, hypertension and electro-
physiological responses, are highly debatable and have been
questioned by experts.
4. The costs and benefits of reducing the lead
standards are being investigated. Separate cost studies are
being done by the Bureau of Mines (for primary smelters) and
by the Lead Industry Association (LIA) for primary and secondary
smelters and battery plants. An LIA study of the number of
children with blood lead levels above 20-25 ug/dl who might
be affected by a reduced standard is being done by an LIA con
tractor, TRC. These studies will be completed in 1986.