Document VKKoKRRn9wvGOgwO62mx6EZrj

FILE NAME Talc TALC DATE 1995 DOC TALC039 DOCUMENT DESCRIPTION Autopsy Record . AUTOPSY NO 95-04 ' CANTON POTSDAM HOSPITAL AUTOPSY REPORT NAME HOUSE ARTHUR AGE 69 SEX Male OCCUPATION Retired RACE Caucasion , ; HOSPITAL E. J Noble Gouverneur N.Y. DOCTOR Dr. Reason ] ADMITTED 02-14-95 DIED 03-17-95 TIME : DATE OF AUTOPSY 03-20-95 | AUTOPSY PERFORMED BY Spurgeon S. Smith M.D. | ASSISTED BY AUTOPSY PERFORMED AT French Funeral Home DATE COMPLETED 05-01-95 FINAL DIAGNOSIS 1 Pneumoconiosis with pulmonary fibrosis and numerous doubly refractile mineral fibers 2 Acute aspiration pneumonia 3. Pulmonary emboli SPURGEON S. SMITH M.D. _ PATHOLOGIST y 05 ar HOUSE ARTHUR 95-04 GROSS DESCRIPTION The body is that of a thin developed appearing white male The body was opened with a shaped incision and the lungs were removed Both lungs were very firm and the external surface had a slight nodular appearance There were some pleural plaques noteodn the diaphragmatic surface of the right and left lungs along with some plaques noted on the visceral surface of the right and left lung Upon sectioning the lungs had a slight combed appearance There exuded some greenish purulent fluid from some areas of the lung upon sectioning There were also multiple pulmonary emboli noted within the blood vessels HOUSE ARTHUR 95-04 MICROSCOPIC DESCRIPTION Sections of the lung showed a marked neutrophilic infiltration withn some of the alveoli acute bronchopneumonia There is some fibrin deposition within some of the alveoli There are also some histiocytes and multinucleated giant cells There are rare ferruginous ~bo~ dies noted.- There are numerous doubly refractile crystals noted - polarization These are noted free within the alveoli within some histiocytes within some multinucleated giant cells within some of the rare ferruginous bodies and within the fibrous tissue between the alveoli Most of these refractile particles are very small and splinter in appearance There are also a few larger somewhat spherical doubly refractile particles The ferruginous bodies are most noted in block 5 are Thersoe me areas are some macrophages containing of fibrosis Within some brownish these areas there granular pigment Focally there are some shaped eosinophilic amorphous bodies Some of these appear to be surrounded by histiocytes There are large areas of fibrosis There are relatively acellular hyalinized fibrous plaques on the surface of the lungs There is rupture and dilation of some of the alveoli Some of these foci are adjacent to the fibrous areas Within some of the blood vessels there are layered fibrin and red blood cells pulmonary emboli There are rare food particles noted within some of the alveoli with acute inflammation indicating aspiration pneumonia Special stains for Acid Fast and fungal organisms are negative ru HOUSE ARTHUR 95-04 FINAL SUMMARY Results of the autopsy Grossly revealed an increased firmness of the lung along with visceral and parietal pleural plaques Microscopically there was foci of fibrosis within the lungs along with adjacent expansion of alveoli There was marked acute inflammation within numerous alveoli The presence of rare food particles would -_ indicate an aspiration pneumonia There were also multiple pulmonary emboli present There were numerous doubly refractile splinter crystals noted on polarization There were also some giant present These could have been present due to the aspiration pneumonia They also could be present because of the foreign crystals present Tuberculosis can also not be completely excluded However Acid Fast stains were negative and" the foci do not have the classic caseating tuberculoid appearance Ferruginous bodies are seen with mineral deposition within the lungs They are most common with cases of asbestosis However they can be seen in exposure to silicates The presence of refracticle crystals within the ferruginous bodies would suggest this as the possibility The exact etiology of the mineral fibers can not be determined without rays refraction studies and electron microscopy Based on the results of the autopsy it is concurred that the acute agonal cause of death was due to aspiration pneumonia and multiple pulmonary emboli The patient was predisposed to this condition due to pneumoconiosis with numerous doubly refractile mineral fibers