Document VJdLoBdg3opEVNN03mYr26m5j

Industrial Hygiene Digest - 34 January, 1950 ` "blood raised the viscosity of the "blood, --Condensed from Brit. J. Ind. Med '! 108 Blood and Bone Marrow Studies cm 21 Workers With SIIIcobIb. F. Trcncbetti, and C. Conti. Med. d. lavcro 39) 237-244 (Aug. 194ttJ. Examination of hlood and hone marrow of 21 silicotic workers revealed a slight normochromic or hypochromic anemia with bobinophllia In the peri pheral blood; and a moderate hypoplasia of hematopoetlc tissue with histio cytosis and eosInophllia in the sternal marrow. These changes are said to he due to the sclerotic effect of free silica on hematopoetlc tissue. -- Authors' English Summary 6j,109 Silicosis In Workers Packing Cleansing Powder. (Accelerating Effect of Oxalic'Acid?) J. Strutek. Caaop. Lek. Cesk. 343-346 (Mar. 26, 19^8) Czechos lovukla. The author Investigated eighteen persons who worked at packaging by hand, a cleansing powder containing 96 per cent silica and four per cent- oxalic acid. Masks could not be worn, as dust containing oxalic acid accumulated beneath the face piece and caused dermatitis. Ten case histories are given, together with x-ray findings, which shoved the presence of silicosis up to the third degree. The time of exposure to dust ranged from nine months to eight years. The condition developed rapidly and was present In three subjects after 1 3/4 years exposure. Concentrations up to 380 x 10particles of less than 10 micron dieuneter were found In the air. It is considered that oxalic acid was responsible for the unusually rapid development of silicosis, perhaps by depressing the concentration of calcium In the tissues. It.also produced pallor and dryness of the res piratory mucosa, may also have been responsible for the marked loss of weight, which is not characteristic of silicosis In the absence of tubercu losis. -- Condensed from Bull. Hyg. 110 Industrial Dust Diseases, Becent Developments and Compensation Aspects. E. Mayer and I. Eappaport. Coinpens. Med. 1, 3^10 (Dec. 1943). This is a general paper on dust diseases, including notes on the following new developments: tentative classification of beryllium dust poisoning as a pneumoconiosis; severe dust disease In bauxite workers; In compensation problems, more attention to disability symptoms and less complete acceptance of fibrosis as the deciding factor. Ill The Pathogenicity of BagaaBe: II. Effect on Babbits of Prolonged Expo sure to BagasseT B. OerBtl, M. Tager. and L.W.fizczepaniakl Proc. Soc. Exper. Biol. & Med. JO, pi 697 (April, 1949). Oerstl associates say that various factors have been suggested as etlolog'lc. agents of bagasBe disease, such as the compoa ition of the -fiber Itself, fungi and micro-organisms attached to the fiber, and the high Blllca content. The authors describe experiments vhich were carried out an rabbits in order to throw more light on the etlologlc factors. Bagasse, Irrespective of the route of administration, produces a complex