Document RjZ9gdERVL6pJ4jw0zE3zJ3R8

fewaxialio:? ;i[ SDsiDg HUBB-Q069784 -.3 - S'. 2* < References to Recent Literature - 10 42 LEAD POISONING. SECTION II. SI FATAL CASES OF SUSPECTED LEAD POISONING* AND 46 OF NON-FATAL LEAD POISONING* DIAGNOSIS* TREATMENT* SUMMARY. V.A. Gant. Ind. Med. 7, 679-99 (November 1958). For Section I 3ee Abstract No. 845, November 1938 bulletin. In the second section of his paper the author devotee considerable space to case histories.of patients which are classified as lead poisoning among children, as chronic industrial cases, as doubtful and unjustified cases, and as acute cases, with discussions in each instance. In the concluding sections of the paper, the author repeats that since there are no symptoms pathognomonic of lead alone, diagnosis without chemical findings (determination of lead in blood or urine) is incomplete. Disodium phosphate is proposed as the best therapeutic agent in the treatment and should be used in conjunction with .a normal calcium.diet. 44 SOME OBSERVATIONS UPON THE EXAMINATION OF MEN EXPOSED TO A LEAD HAZARD AT MOUNT ISA, INCLUDING AN ORIGINAL METHOD FOR THE EXAMINATION OF BLOOD FOR POLYC1IR0MA5IA. L.A. Wir.dsor-McLcan. Med. J. Australia , 367 (aeptembor 3, 1936). (Australian). During 1937, only 6 of tho 1231 men employed in all departments of the Mount Isa Minos in Australia were adjudged to bo incapacitated by plumbism. In connection with his own examinations of 115 of these workurs for lead poisoning, the author found only 1 worker suffering from the positive symptoms, tho remainder of the workers proscr.ting various symptoms not attributable to any action of lead. A conclusion is drawn that hemoglobin percentage provides no indication of load absorption and that tho ratio between tho large and small lymphocytes is of little or no value. The technic for estimating tho number of polychromatophllic cells per million erythrocytes is explained in detail but no conclusion is drawn as to the significance of the reeulte. 45 DETECTION OF LEAD POISONING IN A MOTOR CAR FACTORY. P.Molitor, M. Arnolds* and G. He- -ser. Arch.Mai.Profos. 124-8 (1938). (French). A soi-.r containing 30 per cent load was used on assembled automobile parts in a French factory. When two workers who came under notice were found to have load poisoning, the remaining workers were examined. It was found that 10 out of 39 men examinee had clear cases of lead poisoning with an additional 4 classed as doubtful. The men wore employed in smoothing and rubbing down soldered parts. In another factory this work wae boing done in separato compartments where the men wore respirators, rubber gloves and impermeable clothing and upon examination no cases of poisoning wore found in this group. 46 STABILITY OF ANEMIC CONDITIONS IN EXPERIMENTAL LEAD POISONING. Loureau, O.S. doSacy and A. Arthus. Compt. rend. Soc. de biol. 128. 512-4 (1938). (Fronch). In some tests on rabbits subjected to experimental lead poisoning, variations in individual susceptibility to tho poisoning and reoponso to troatment were found to be a predominant factor. Mnrkod difforenoes wore noted in red cell blood count and in powor of rocovery of the animals, DISTRIBUTION OF LEAD IN THE CAT AFTER INTRAVENOUS INJECTION OF A COLLOIDAL LEAD PREPARATION, AND THE EFFECT OF IRRADIATION ON THIS DISTRIBUTION. B.L. Crawford, H.L. Stowart, C.F. Willoughby and F.L. Smith. Am.J.Cancor , 401-22 (1938). Cats receiving i.\-.ravenous injections of a colloidal lead preparation generally remained in good clinical condition without loss of weight or anemia, and the organs of these animals showed slight, if any, structural changes. When, however roentgen irradiation was given in addition, toxic manifestations ensued and pro nounced structural changes in tho ergans occurred. These were of the some nature as were observed in animals treated by irradiation alone, but were more marked and are ottributed to the combined toxic effects of the two agents. The lead in jection brought about significant increase in the lead concentration of most