Document QXv0wKvwyM8rg5NJZRa73DkbL
FILE NAME: Ford (FD) DATE: 1977-1983 DOC#: FD046 DOCUMENT DESCRIPTION: Workmen's Comp Claim with Medical Records
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PART I.
W ORKM EN S COM PENSATION T H E INDUSTRIAL C O M M ISSIO N O F O H IO
Claimant' Report of Occupational Diaeaae
C ltia No.
O.D._
Xacjtlae (Om.) *
Tetefiepenfiled'
WOODIE THOMPSON
CLAIMANT'S N A M E _____ 1 2 5 1 8 E ir.^ i^ R o a d
(M iC ilt LaJm l)
(Last)
2. Horn* Address (Number)
S__o_c_ia_l
(Street)
(C.ty)
FO RD (M<5!? '6 r CO
3. Age.... ...........Security N o........ X 6 - - - 3 0 O 4 1 3 ------------- Employer:------------------------------------------------
otto ef DUeb.
W eld er
Occupation: .... - ...................................................No. and relationship of d e p e n d e n t s __________________
ui
A sb a sto a is 4. Prom what disease art you suffering?.......... ......................................................................................------------- ---3. What parts o f the body are affected?-- ------- llir u ^ S .......... - -- ........-- ------------------ ..-- - --
TtoSTT
Give date your symptoms first ap p eared ...3.r..-L&2-J--Name of employer when symptoms appeared: ............... ................................How lone in employ of this employer before symptoms first appeared?______
FORJ>-K< 2 I OBc-eapeiie
6. Location of plant where you worked, when disability began:.. CkLyAhg>^A
--
M arch, 1975
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7. When were symptoms first reported to employer?--------
Describe exactly how you were exposed to the substance, which, is your opinion caused your disease:_________ _______ ______I w a s w e l d l n c e n g i n e b l o c h t . v .___We u s e d a b a e t o a t.a k e e p tfr
.... Heat olf. The abestos would turn Into dust and I inhaled
I t 7"
"CfenST XrvwFTRewiy
Give name and address of physician who first diagnosed your disease as being occupational, and date dagnoas
nude: ___________ ~ G r o v e __ Cleveland ,,Clin.lc_.S.5.ftP...JEi-WCl-,.l*.|4-.AVfi__ClftYfi.,..
10. Have you previously itiffirraed from the disease for which this application is made, or any other occupational
disease?______ l x ^ O t o , _give details (dates of attacks, name and address of p b yricias)________ __ - ..
1 1 . D ite quit w ork:__
* r e t u r n e d ______ p or w tut rtu o n ? ____ U n a b l e t o ._ w o f k ___________
12. Date returned to work:.-- _______
13. Occupation when disability began;... when disability began:........................--
...... -...Type of work:.
W eider
___
W eld er
..Type of work you were doing .Xength of tima is this type of
work (a) immediately prior to onset of symptoms:----- . 2 3 y r s
(b) During llf ttlm :____ ?iS ?-------------------------------
14. Were you exposed to dust in youx work?....
Typs of dust:_____ ^ . .I t O _ B ______
Describe briefly how you were exposed:..........h5..-h9.fc....i?.iSjSiS_______________________________________
____
T h e s h o o t s t u r n e d t o d u s t a n d I i n h a l e d t h e d u s t , _____________
SIWT h lV , Tye et Dltokto Hemrt e( Diub.
hn el Bedy
WJWh . "
Over what periods were you exposed in your employment?---------- ----------------------------------------------------------------
A m in We
Date of last exposure to dust:.
~, 1 9 7 7
13. If you were exposed to the substance which allegedly caused this diseaae in other employments, give the names
and addresses of the other employers, and dates of employments:---------------- ---
.................... ..............
16. Have you filad a previous application concerning this or any other occupational disease?
If to, giva claim number:.
--
-- ------------------------- ----- ------
By plat tkie sratketfe* I prtetly wtlvt. ee btbslf ef mytU s*4 si any per** S* alult beve tny totereet la thli clslw. sTI frewUlese of I orkidiaa aar fbyticiaa ec atber aerees vb* bee boretefere etiea4*4 er ettAtoeC . r h ktttthvt stia4 * ! at, fta eidMin< eey baawitCt er iafemenM vbtcb they tbereby eeeeire. 1 h in reed ell the uie^eoi caau(a4 bere u 4 bae tbe lame te k tree iM terree- I ktreky eatboria a Udiertoi C-- > t yy am* mrafut which/G} A ewvUeS Swaet to Ike | i m a riaSartss ecmee p a n sa i to ikle sptlitMto* fe f j m m t ai at toe ftuto O y y < itoiJ D>feee 1 ^ `
"KF^VTCSr jrv Law
(XI ersltoeat elaae by toerk rwe witaeeeei at
her.)
--------------------------------------------------------- ---------------
D it . report >nt to tm ploytr__________________________
(Sicweiare el tkfLeeat. II aaekl to eifa^berb wM be wSe.)
Strt Addrtw----------------- 1-- 1 2 5 X 8 _ E o a r y --A v e City or Town_______________ C l e v e l a n d . O h i o
1(135'
PART 1L
Employer' Report of Occupational Duease
1. Nomo ot .m pl o y - r : .- c ^ e ) . 2. O Ses addraaa:
^
j_________________________
Of? u sbew^ e Uaaraaee Certiieete)
--
- -
_______-- - -
X Nature of business:.
(Perniai, cael alaLsf, eu.)
Type of orgaaltatioa:
(Cr^. perumtoip.)
4.1 The ctruings of the iaJurd<tstployM for sarrieta rendered at time of occupational Utas are being reported on
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Tii'. m rU ST R IA L COI M is s io n OF OHIO . ...
OFJGTTAL RECORD OF PROCEEDIIIGS
. . V -- -- ** Sticet and No----- :-- S-:----------
City
Employer ------ T ^ '
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Street and No.----
City --
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.Claim No-
.(SUte) ____ L _ _ -- _ ------ r '? 7
.Date oi Injury..!--: ,'------
.(SUte)
r-*-> r v
Findings oi Facts and Minutes
Present (or Claimant-------- --
Present lor Employer-------- E li--
On this day tils
was heard and findings and orders were made as follows:
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Dried at______ lutrctrinl Coinmlgaton of Ohio
.tills.
-doy of___ : --Bureau oi Wooers' Cou"*. !!' .
Ad'*
$007 04-50
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* '-- I-* r ~ ~ t t r z " , tr-o, ecrrecil7 recorded and thet this record msy be retained In the files of th To:d
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; ' . 2 . v '.;ru j'c'fh a fa 'Jccir.r'tirsacae or conditions: ..
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- hpjrttnrn I __ AtthtUl----- LlLi
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Arridenti____h i P .: ___ Tuberculosis___ h i U
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D:.'. - ^ ~ ^ 1 * S ^ _____________ --Interviewer.
-Reinstate.
. PRE-EMPLOYMENT PHYSICAL EXAMINATION
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L>C<\ _____ lIndirac. Weight- \ ]S[ Lbe.
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-With Glasses. -W ith Giessen.
Murmurs.
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Abdcm eri-- (Nots scars, tenderness, tension, tumors, etc.).
A geJ2 _!^ -------Se--
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-Development: Good_L
-Pali.
-Poor-
__________ Nfear Vision__________ :____
A m w iitaH nn
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1______n________ * . -
-Rhythm____________L iJ____ ; ii--*-:'--__!___!____________ __
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In a''r s l R c jlo r (Note Glands, Condition of rings, hernia, sice and type, etc.).
f \ v\ | L f t / * ' .
_______________
Evtrriv.Ulre and T ru nik -- (Norte anv dslonnlties, limitations of motion or aammputstlons.). j a J O .flC^S !pp*wf V
t V r f c o i f t ' ^ _____
Temeiei DX.M
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JPciiodn.
-Regular--!____ Scantgr.
U unrnlnweit ,..
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Hliwrt Hamlngv
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C crrrrnrated Injuriee--(Describe and giva pat cent disability ii "y ) *1 ^
JHeaty-
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.Dlmbling.
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CL:vC`-.Ai!L) CLIM'C HC'3riTAL
THOMPSON, Hr. Woody
( )
' ______ Age________40 ' , ' Coie
Bronchoscopy; left thoracotomy; biopsy
Ptfl<4.rD5.-.7 7 ----
V . . --- -----G-e--n-e-r-a-l-------- - ' ""'_______ :________ R oom
Dr. L.. K. G r o v e s Jt: on____ --- ------------------- ---------------------------
A s s is ta n ts ____ P T.-- Mj-- P filLfir^Mr. F .s ky
FP.LOPSRATIVE DIAGNOSIS:
Left sid e d p l e u r a l e f f u s i o n * e t i o l o g y urt'deterrined.
rOSTOPERATIVE DIAGNOSIS:
Mesothel ioma.
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PATHOLOGICAL DIAGNOSIS:
OPERATI
F 1 i s T 0 R*Y :
The p a t i e n t is a 4 6 - y e a r old black male ytho has
had progressive shortness of breath on the left side with
pleural e f f u s i o n on c h e s t x-ray. T h i s was tapped and 2000 cc.
of fluid was r e mo v e d from there. This was c h o c o l a t e colored. .
and cytology was negative. Bronchoscopy was unremarkable.
When the chest was opened, a b o u t '2500 cc. of chocolate colored .
fluid escaped and there was diffuse infiltration of fibrosis
all over the pleural surface. The left lung was contracted
and small in size. A b i o p s y showed on frozen section m e s o
thelioma.
.
PR0CEUDRE:
Under general anesthesia, bronchoscopy was
performed using a rigid bronchoscope. This was unremarkable.
The patient was intubated, positioned for a thoracotomy on the
lateral side with the left side up. Routine thoracotomy incision
was made and the chest was e n t e r e d in the fifth interspace.
On o p e n i n g the p l e u r a , a b o u t 2 5 0 0 cc.- of c h o c o l a t e fluid e s c a p e d '.'
This was aspirated. The left lung appeared small in size and
had diffuse f i b r o s i s a r o u n d it. T h e e n t i r e pleural surface was
found to be studded with m u l t i p l e i n filtrative lesions. Three of
these lesions were biopsied and frozen section showed mesothelioma
Two chest tubes were placed for drainage purposes and the chest
wound then closed in r o u t i n e fashion. The p a t i e n t tolerated the
procedure well and was transferred to the Intensive Care Unit
in good condit i o n .
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Dictated: Typed : I .D .
Dr. M. P e t e r for Dr. L. K. G r o v e s / 0 4 - 0 5 - 7 7 04-08-77/mal 04-05-77/001
S037
M A R S .1970
f / i t : : c v .o c : : a l p x r c r j
thcmpsc;:, Hoodie
___R.cc'rn No_____
.Clinic No__ 1-42G-475-2 Service Dr, " Hev/lett
J 'otil. I Ic*. Date__ ll:
PATHOLOGICAL DIAGNOSIS: .
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. FINAL PATHOLOGIC DIAGNOSES
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550-8772A
370-8772A
t20-nn?.A
2/5-P77ZA 060-8772A 659-0772A 657-8772A
350-4971
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MESOTHELIOMA EXTENSIVELY INVOLVING MEDIASTINUM,'PLEURA, PERICARDIUM,
DIAPHRAGM, AND PERITONEUM, WITH SEROSAL COATING OF ADCOMJUM
VISCERA. .METASTASIS TO PARA-ESOPHAGEAL AND LEFT SUPRACLAVICULAR
LYMPH NODES AND EPISTERNAL SUBCUTANEOUS TISSUE. LUNG BIOPSY
April 1977, Surgical Path. #77-5123.
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INTRA-PULMONARY ASBESTOS BODIES. . . . .
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410-13X 410-900.6
CARDIOMEGALY (580 gm.) WITH LEFT VENTRICULAR HYPERTROPHY ( 2 . 0 cm. ) . `
Dilatation of right ventricle, right atrium, and left atrium." ;
Congestion of liver (1550 am.), kidneys, spleen, and lungs; : 1
Edema, right lung (660 gm.).
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Atelectasis, right lower lobe. , ... ;`
Cerebral edema (1780 gm.).
`
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Arterial and arteriolar nephrosclerosis. ; ; ' 1
Cortical nodular hyperplasia, adrenal glands, mild.
Divertlculosis, sigmoid colon. '
Hydroceles, bilateral.
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Hyalinized granuloma (1 cm.),' liver. '.
UfL
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KP/ Howard S. Levin, M.D..
wt: n r
='.'L \::d CLINIC lOriTA!. f PATHOLOGICAL REPORT
________ _ _C lln lc No.. 1-426-475-2 , Pnth M ^ 1 0 9 8 5 5ervlcer>r Hewlett ________;________ , Dn>a 12-20-77
PATHOLOGICAL DIAGNOSIS:
Date and time of death: Date and time of autopsy: Restrictions : Performed by:
12-20-77 at 1 :35 a.m 12-20-77 at 11:00 a.m None K. Pontius, M.D.
CLINICAL DIAGNOSES Mesothelioma, respiratory failure, and metabolic acidosis
GROSS EXAMINATION
S b OCR
EXTERNAL EXAMINATION: The body 1s that of a 49-year-old, black male,
w
weighing approximately 160 pounds and measuring 66 inches In length. The
v;
body appears wasted, except for a protuberant abdomen, the hair 1s black and
shows a rale balding pattern. The conjunctivae are clear but show marked edema.-
The irides are brown; the pupils are unremarkable. The ears, nose, and mouth
show no abnormalities. The teeth are natural and in fair repair. The neck ..
is of normal configuration and there 1s a palpable node withtithe left supra-
clavicular fossa. The thorax is symmetrical and normal 1n configuration. . .
There is a well-healed thoracotomy scar running around the left thorax and
measuring 30.0 cm. in length. Inferior to this are two well-healed scars, one
measuring 1.0 cm. and one measuring 2.0 cm., apparently representing tubal.'
orifices. Tho breasts are of normal configuration and there are no palpable ..
masses. The abdomen is markedly protuberant and a fluid wave is present.
There is a 3.0 cm. ecchymosis located Inferior to the umbilicus 1n the midline.
The external genitalia are of normal conformation, and there are no external
lesions. The extremities appear normal, and the joints are not deform,ed. There
is a trace of pedal edema.. The skin is of normal pliability and texture and
shows some depigmentation across the upper extremities fn that there are
patches of lighter colored skin fronting on the darker pigmented skin. There
1s no icterus. Other than the described supraclavicular node, there 1s no
.
palpable lymphadenopathy. There are numerous ecchymoses present In both antecubltal
fossae and on the radial aspects of both wrists.
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INTERNAL EXAMINATION:
Pleural Cavities: Upon removing the sternal plate,
there is noted to be numerous nodules of gritty white neoplasm present both
\
beneath the sternum and external to the sternum. The tumdr likewise encases
the ribs. The right pleural cavity contains approximately 1500 cc. of a :
slightly cloudy serosanguineous fluid. The right lung 1s matted to the apex ....
and vertebral side of the pleural cavity. There are numerous small tumor "
nodules studding the lower lobe of the right lung as well as studding the . .
parietal pleura in the lower aspect of the pleural cavity. The left lung 1s .
rot observed due to encasement by tumor. This 1s filling the pleural space from
apex to base and adhering to the pericardium. ,,
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; ` Mediastinum: .Numerous tumor nodules arc present .
within the mediastinum and adherent to the sternum, as previously described.
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Peritoneal Cavity: There 1s an accumulation of.
approximately 5000 cc. of slightly red-tinged serous ascites. The peritoneum ' - '
r ' " . ' ! P CH'V. . , P A T H o i O F i r A 1. r:rr,o::r
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t.'oo'lio -Room No..
-Sorvlco .Clinic No.- V -^ .R - `17T>~?.
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PATHO LO GICAL DIAGNOSIS; Gross Examination - Pngo two
as well as the omentum, mesentery, and sorosnl curfncrr. of nil pbdomlniil Vl?l?vrfl .
arc studded bv nodules of tumor. Tho abdominal vlscrrn occupy their noNial
anntr;;'ii:o.l positions. The diaphragmatic leafs nro clovntod, The r-l'jlr Of
\
th- li'cr is in proper relationship to tho costal margin find llin r-Hfnin of Lit *
spleen is nondetoctable due to encasement by tumor, .
.
r r.r ; : 015ruLAi sYSTrfi; .
Heart: Weight CCI) prams.. The coronary m tert^.
Fa "normal anatomic distribution and multiple cro.18 sections shov/ no Slt/Alfi*
c e n t narrowing of lumlna and no evidence of thrombosis.
"f:c epicardium shows numerous adhesions to the rorlcnrdlliiii find, 1n faet*
th: rricardial space 1s obliterated by these adhesions. Thst'fi 1 no t' nr.*r ' ''
p i : 't cn the rplcardlal surface. There 1s o notrol amount of
r*fCj r d i ^ l
fal i its distribution 1s normal. The myocardium 1s of norm'll con51 r - ' -.i r rr.ercnce. The right atrium, right ventricle, and thi left fltilim U C 1
r:'-' *,.j dilatation. The left ventricle shows a normal configuration, fill' h ' M h . . . ;
l ! . . ! 1 ft ventricular v;all Is markedly hypertrophied. Thp pre-Jt VfiS'nla n t f
e r d 1 rive the hr:rt In a normal narr.er. Tho septa aro 1 n U c t, find ibfh* dr 719 y
c r - *: -.Hal atrerr?! itles. The left and right ventricles are 2,0 c/'.nnd 0/i ,c."i y
, rccccctivrly. The heart va'vc: are thin, pHfiblr find diHGSt*, t i i
cc
ty. Valvular r^surc'ents m ts
fisrtls 7 6c*/# >
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3007 :459 PQ
r v : . f'W . D c n ; ;<j n u r 'iiAL PATHOLOGICAL REFOKT
Ml \%. r.i:TN rSON, Wood0
.ClinicNo.. 1-426-475-2
-Path. Nil.,,
Age_Jl2_--RoomNo____J_____ServiceDrJ!f!!l^-------------'-------!-------;-----Date-----V'~P ~ ? L
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PATHOLOGICAL DIAGNOSIS: Gross Examination - Page three.
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present and on cut surface shows part of the parenchyma to be replaced by firm ,
white tissue.
.
'
. '
. Bone Marrow: The vertebral bone marrow presents a ''
normal appearance.
.
PASTRQiNTESTINAL TRACT: The esophagus 1s free of lesions. The stomach has a Jv norma'Pconfiguration and the serosa of the stomach is coated by tumor nodules. .> Tko mucosa is thrown into rugal folds. There are no areas of ulceration. The V \ \ retroperitoneal portion of the duodenum shows no involvement by tumor; however, . the peritoneal extension of the duodenum shows serosal coating by tumor nodules.' The jejunum, ileum, and colon show marked coating of their serosal surfaces by tumor. Numerous diverticula are present within the sigmoid colon. The appendix' " likewise shows marked serosal wrapping by tumor. The anus 1s normal. . , .
LIVER: Weight 1550 grams. The capsule 1s studded with tumor nodules, the
largest measuring 0.5 cm. The liver configuration is normal. Multiple cross
sections reveal a hyperemic cut surface but no accentuation of the lobular
architecture. The inferior surface of the liver shows a thick tumor coat. ' '
The gallbladder 1s of normal size and configuration. The wall is thin.
and the mucosa 1s bile-stained. The viscus contains a normal amount of green-
. black bile. The cystic duct, the right and left hepatic ducts, and the common
duct are patent and free of calculi and other Intrinsic lesions.
''
... '
; "
PANCREAS: The pancreas is soft and mushy 'and on cut surface the lobulated
patiern is present but the parenchyma appears yellow and soft with areas of
hemorrhagic discoloration. There is no evidence of fat necrosis. The ducts
are patent.
.
... .
'
GENITOURINARY SYSTEM:
Kidneys: Right 140 grams. Left 150 grams. The
n 5 * 0 .OOP
right and left kidneys are s i m i l a r . T h e capsules strip with moderate difficulty
to reveal finely granular subcapsular surfaces. There 1s a 1.0 cm. depressed
wedge-shaped scar 1n the cortex of the left kidney. The renal arteries and
veins are patent and free of stenoslng lesions. On section, the renal cortices
are of normal thickness and the cortico-medullary demarcations are distinct.
The cut surface of the kidneys shows marked hyperemia of the parenchyma. The
pelvo-calyceal systems are normal in appearance. The ureters are unremarkable, c k
Bladder: The bladder is of normal configuration. .;- l y
The pelvic fat surrounding the bladder 1s studded with nodules of tumor. The *
mucusa is intact and free of ulceration. There is a small .submucosal hemorrhage
present in the posterior wall of the bladder.
.
. ='
.v "
Prostate and Seminal Vesicles: A portion of
~
the prostate is examined and sections of this reveal rubbery, firm, gray-white
parenchyma, free of unusual characteristics.
.... ; .
,
..
Testes: There are bilateral hydroceles present '!
but otherwise no abnormalities.
. .
. '
r ;l . - ;.
ENDOCRINE SYSTEM: '
Adrenals: Right 10.0 grams. Left 15.0 grams. '.'V*"'-
T h e adrenal cortices show marked convolution and nodularity. They.are bright yellow
. In color. The medullae are unremarkable.
. '
.>
Thyroid: Weight 23.0 grams. Multiple c r o s s ' * :
sections of the lateral lobes and Isthmus reveal no parenchymatous nodules or
.
.
. '
.i _
*
nsrop.r
Vi'A'1''SUN, Vluo.Ma
J V w n No..
.ClinicNqj -426-475-2 .Scrvico br Hewlett
P o t h . N ^ - l 1 ' 1 '
^ 77_
f* \i .'!' *L'!CAL IMAGNOSIS: Cross Examination - Page four
'.V kstuns.
. ;
.
.
Parathyroids: Two parathyroid glands pro
: ' iikd, l.hich era grossly unrc-arl.ab)e.
P itu ita ry : The p itu ita ry 1s unrcrrorhoMc,
i ' .;*1 I f J l v r M S V S TT M :
C r a i n : .H e ig h t 170 g r ? r ; . T h e r e fr, r - ' !" * ?
.
ning'oi t;/h tyr1* There is no uncal notching tut the right ccr-^;)ter
tchoil is somewhat soften?.!?,
'
'
Tl.e 'toajor cerebral arterlES show no significant atherosclerosis cr
Ci .OiiiO l i e s *
Spinal c/rd: The spinal cord end i t s c o y e rtrg s
1'i'C unremarkable.
> '
'
"
1_NUSITT05,kTLETAL SYSTEM: The axial and .appendicular skeleton show VO t.jnonnaliFies. The exposed musculature .is unreniankab]^
.. . ' ' , , `
' *
.. \ *'
:
ADD^NbUr*: :Lcft_ILi,JTiL: The left lung 1s removed from the ployrel (Cfrv.fty v/ltth - * ;
greet difficulty due to an encasement by neoplasm.. The Tuny tis formal An
; '" ,
Thr>-'Mated for three days prior to sectioning.. Crocs sections through
.r\
erilfi'O lung Shov; a '0.5 ito 'S.O om. coating by gritty -.white rvoplcsm,. The f.urror -_.
hue el so 'encased the pericardium amd the diaphragm. The -.ent1rp iplteiWH Purfoce v" .
-ov U'o Tung is covered '.by tumor, and tumor nofliiliis -extend fnto the snibpTenrol
'plfporidhyma at several -'areas. In one -of these areas there fc a -r.'tm -of rcompr?r>r.ftS
lulig parenchyma which is -unite firm as tcormrared to ..the surrounding tlung -fctesiie.. ''
The lung .'parenchyma away from the tumor shows oiniform-ei.r spaaes vwfthput <$v1.tlenp
<bf -siibpieural buTlae. The bronchi are pulmonary .arteries show rPP S ^ n i f l s a n t
'abnormal ity. Several neribronchial lymph nudes are present pt ihe b:1furoofcipn
`of-the'main stem bronchus and on cut *urfame show 'sigrificont antjhrecQklc
pigmentation. Sections -of the Hung are .submitted for microscopic rstudy.
W.-eul
5007 04 1
* *
'
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i.:;uLAr ciJ'C Lr/ :t\
TATKOLOGICAL REPORT (
.
THOMPSON, Hoodie
' . Kl 1-420-475-2
A g c _ _ i f ________ Room No_____________ _____Service D r .- ftgwl eA t ----------------------
PATH O LO GICAL DIAGNOSIS:
MICROSCOPIC DESCRIPTION . .
,
1
Path. N o . ^ 2 ^
O 'i*
- 1 2 -2 0 -,
v .
CARDIOVASCULAR: Sections of the left ventricular wall and Interventricular septun show hypertrophy of the muscle bundles manifested by an Increased amojnt of cyto plasm and enlarged nuclei. The epicardial surface shows fibrous scar.
, RESPIRATORY: A section of trachea shows it to be wrapped by mesothelioma
' which 1s characterized by dense fibrous tissue containing malignant colls with
vesicular nuclei and basophilic cytoplasm, which are arranged in papillary
structures and tubular structures. Special stains for hyaluronic a d d show
that the major secretory component of the tissue is Indeed hyaluronic acid.
A section of diaphragm shows neoplasm to be coating both its pleural and
peritoneal surfaces.
Sections of left lung reveal the pleural surface to be replaced ly neoplasm
which superficially invades the lung parenchyma. Sections of the left lung taken
away from the tumor show severe congestion of the alveolar septal capillaries
.
and edema fluid in the alveolar spaces. There are frequent asbestos todies
present. These stain positively with an Iron stain.
. .
Sections of the right lung show similar severe congestion and edema as
well as organizing intra-alveolar exudate and areas of hyaline membrare
formation. The parenchyma of the right lung likewise shows frequent asbestos
bodies. There is no evidence of interstitial fibrosis, however.
LYMPH NODES: A paraesophageal lymph node and left supraclavicular lymph node show metastatic mesothelioma.
LIVER: Sections of hepatic parenchyma show centrilobular congestion v/ithout necrosis. The capsular surface of the liver shows studding with tumor nodules. These do not penetrate the capsule into the parenchyma. In addition, there is a hyalinized granuloma present 1n a subcapsular location.
SPLEEN: The capsular surface of the splenic sections is coated with mesothelioma. The parenchyma of the spleen shows severe congestion.
PANCREAS: Sections of pancreatic parenchyma show autolytic changes.
ADRENAL: Sections of adrenal gland show a nodular cortical hyperplasia characterized by small nodules of adrenocortical cells s.tudding the adrenal cortex.
GASTROINTESTINAL SYSTEM: A section of peritoneum shows' its surface to be
entirely replaced by mesothelioma. A section of jejunum shows serosal meso-
^
thelioma. A section of appendix likewise shows it to be entirely enveloped by . '
mesothelioma.
GENITOURINARY SYSTEM: Sections of the right and left kidneys show numerous cortical scars and hyaline sclerosis of the renal arteries and arterioles. There is moderate congestion of the glomerular capillary loops. '. -
< v
;
S007 C442
c s i k o t :-:;
- .......... T n.--
,
Ci-HVLI-At'D ClUC HOSPTA*/ '
: *
. ^ PATHOLOGICAL REPORT
.
'`* **'--:
>
. *
** .
'
K'nm: THOMPSON, Woodie Ago__12_____ Room No..
_ _ C lln )c Kft M 2 6 - 4 7 5 - S :_____ Poth. Nof a o ? C ,, -Servlco Dr. Hpwlet.t. -- !------------------:-----Dota..---T & ib .7 7
ci
PATHO LO GICAL DIAGNOSIS: Microscopic Description - Page two .
V
DONE MARROW: A section of vertebral body bone marrow 1s nortnocellular with `
a slight erythrold hyperplasia.
'
.....
..
***
SKIN: A section of subcutaneous tissue taken from the eplstemal area shows '
nodules of mesothelioma.-
DRAIN: Routine sections of rectus gyrus, basal ganglia, hippocampus, mammillary bodies, midbrain, cerebellum, pons, medulla, and spinal cord show no significant changes.
K P :eat
(ama ere
%/
iOO7 0^63
'^JLI-VELAND C M N I C HOSPITAL/-^
.
PATHOLOGICAL REPORT
'."r;
. ^Nome--TH--O--M-P--S--O-N--,----W-o--o--d-i--eA g e _4_Q _____ Room No..
_______ __a,nic
1-426^475-2
_ pQth
Service r>- Hewlett________________ ______Dote___ 12-2Qr7i
PATHOLOGICAL DIAGNOSIS:
BRATN AFTER FIXATION: The dura shows no lesions, and the sagittal sinus Is .
: patent. THe" leptbmeriTnges show some thickening and clouding over the frontal
and temporal aspects of the brain. They are not thickened, however, at the
--
. base of the brain. The convolutional pattern shows some flattening of the gyri.
There 1s no evidence of subfalcial, uncal, or cerebellar tonsillar herniation
.present. The major cerebral arteries show mild segmental atherosclerosis
and appear to be patent throughout. Serial coronal sections through the cerebral
hemispheres show poor fixation of the brain but a grossly normal cortical
- ribbon and underlying white matter. The basal ganglia and diencephalon show
. no gross abnormalities. Serial cross sections through the brain stem and .
sagittal sections through the cerebellum fail to show any gross lesions or
, abnormalities. The ventricular system is symmetric and of normal size and
configuration.
SPINAL CORD: Serial 1.0 cm. cross sections through the spinal cord and the
cauda equina show no gross abnormalities.
.
.
KPreat
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007 0466
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___ ____ ____________________________ ^ fE iiC v v__________________ 1]_______________________________ a: ' lO'-sG - - . ;
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r -P WAS HCWAUZtD -OR Al rOl'RS 01 lO'-OlR CO/.'.sLET REVERSE C-r. M T a'Z C;17'-M0 C- S^V-vtR^
^.VSE'C N V.a StER RECORD
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3007 0465
A D `.-.!O-ON-DL'CKAP.GrRFCr:? ar***.'. ^ z> ' .
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MARS W O
L.-
'JAMES bi WISMAR.. M.D.
0 1 OI.D OAK rOmtVANR W i l l *H
. MlDDKIUnO HtlOHTt OriHI..O..***110
'TWwtWW M tlll
:..i
Fobruaj^ 10, 1978
*
f.
Robert K. Hianay
i-
Supervisor, Ins, Relations Dept.
Cleveland, Chio Wtlhl
Re: Kr. Vioodie Thompson
.
Pear I'r. Ilisnay:
Enclosed please find copiec of hr. Thompnon'n
aar.ittlnf, history and physical exam, hocpltal
discharge suir.r.axy and letters iron the Cleveland
Clinic relative to his care at that Institution,
iy last contact vith hia was on i-'arch 19, 1977,
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3037 0t7C
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Cl . F A L I A N D CLINIC.
TH l CLINIC C f N R J0 0 EU Cim A V fN i;i. C llv E .A N D . OH 44I0A. U S A
it.- -v; M or heMMoiOGr
A V V t.'.C A i O N G G I^ -
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. 21G-444 2200 ~CAIE. C lt V C l'S .. r
January 9, 1978
Janes D. Wismar, M.D. 7225 Old Oak Boulevard Middleburg Hts., OH 44130
Dear Doctor Wismar:
RE: Mr, Woodie Thompson CLINIC //1-426-476-2
I regret to inform you that your patient, Mr. Woodie Thompson, with the malignant mesothelioma was admitted to the Cleveland Clinic Hospital through the Emergency Room on December 17,' 1977 in a preterminal condition. He was given supportive care but expired on December 20, 1977. Permission for an autopsy was granted. We regret that there was nothing we could do for Mr. Thompson at this late stage of his disease.
Sincerely yours
JSH/ha/M-6395
b(aq7 0471 PO
W OOf-IE. ,114-2 ISO
77-31C32-3 .
:
: ..Admitted 3/4/77 ` .
Dr. '.-.'iGmar
` '
1 * >'
*> ,
' )**%
\ -y
. r *
M w* ./ . -
4 3 year old Black male admitted to the hospital because of left pleural
effusion.,
.
'
'
' ' '.
HISTORY 0? PRESENT ILLNESS: He states that he was well until approximately x nesl: prior to admission to the h o s d t i l when he began to experience cough and exertional dyspnea, These s;/i.im&o;r.o ineecasel, were not associated wit.i a n y chills or fever and promote 1 aim to be seen in the E.R. on /o/77. At tr.it time, it was felt that his problem was tracheobronchitis. He was stirtel on antibiotics and cough expectorants. He was seae by the u.ileP3i;-nei on the day prior to admission to the hosnital with no significant i-rrover.ent. AT that time, clinical sir^s were consistent witn left pelurai offuiion and x-ray examination showed .massive density of the- left hemitnorax with siilft of the mediastinum to the right. It was felt he snouli be .i)sriauli/.e 1 for further investigation and treatment.
?Ar7 HISTORY: is significant in that the patient has had known hpperteu3ion i--., ^_ p_e_r__i_o_d of o -v years. he was additionally treated with antinyperter.sive
cnerinf/ anj nus not- taxon any medication in the oast six months. In 1973, '
'? wis hospitalised at SWRK for a ''enechup'1. At that time, he was told
- iat 'ho nal a
ii.ibetes and was treated with diet, but did not followup
any `.further medical care for tills problem and has not had any symptoms . '
sug-eative of pplyuria, polydipsia or unexplained weight loss. Hls_ health .' '
nas otnerlrlse been .pood. NTo allergies. No injuries.
HABITS: DOes nbt use tobacco. Drinks alcohol only socially and currently
ills 'only medications include Erythromycin and courh expectorant from the
emergency room physician.
-
FAMILY HISTORY: His father died in hi i 00s from c o m m u t a t i o n s of hypertension.
Motner is living at 6p yrs. in Rood health. He has 3 male and 1 female s i d i n g
all of whom are well. Patient is married ,has 3 children, ail of whom are
in good health. The patient works as i wilder for ^ord Motor foundry.
.
SYSTEM REVIEW: HEENT- doe3 relate some diminished vision especially in
the.rlgnt eye recently, but no other symptomatology. C-?- his only distress
has been slight right sided pleuritis like chest pain, cough has been
productive ofl only scant amount of mucoid 3putum. GI - negative. 0U- not
remarkable. MS- not remarkable.
,
*
''1 'V \\ ''\ .* i\
007 C4?2
11^-2 LlO
2/V77 :ar
w e n developed, well nourished, alert, cooperative Her,ro male In no acute
diotresc. Vital signs - B/P 200/130 in both arms; pulse rate 88 regular'
^
rcrrirations lb but not labored at rest. '
' " ' *'* '
S'.T`: No remarkable findings;
.'
'' ' \
t `-vf ~
V-,E: .Bilaterally responsive pnrils. Arcus sinilis was present. \ Funduscbpic '
examination showed copper wiring with r.arked tortuosity and AV crossing
defects an 1 scattered waxy dxddates, bit no microaneurysms or nemormages. ' .
ho papnlliedema. . *
.
.
i *
nothin,", remarkable.
;C" : Supple, no thyromegaly or ude.nor athy present. ;rc.- ai3e.it on the left side,' praent or. the right.
Supaaclavicular pulsations
i: Expansion appeared to be somevrhta limited with aefinite diminished r-jiBfi .sounds and dullness over tnc left hemithorax witn markedly reduced ' .r.isntrea emonuonatc ami E to A change. No rales or rhoacni were audible.
EAST: difficult to aso e 3s , mediastinum was definitely shifted to the right :
.nd tne nearfl sounds were more pronounc id on the right than on the left". HHBere'. ,
s no manifest enlargment. First and second neart sounds appeared to *,
normal. 32 was more pronounced over the pulmonic region. S3 Sd were
' .
.bsent and no murmurs were present.
'
.
.
,BD0:i3W: Showed slight tenderness to deep palpation in the right upper
uadrant. Mo definite organs,. masses were felt. Bowel sounds appeared to
e nornoactlve.
.xternal genitalia not remarkable. '
:
IXTRSillTICS- peripheral vascular examination was within noraal limits.
'.ZCTAL: No.remarkable findings.
*
'
MPuESSIOM:
Left pleural effusion, etiology unknown.
, R/0 underlying tuberculosis, malignancy"with
' metastatic disease, empyema etc.
associated with bronchopneumonia.
'
2. Hypertensive cardiovascular disease.
3. Diabetes mellitus, historical.
'
i. Hypertensive retinojathy.
'
3/b/3/b/77
,0C7 C<73 PQ
J
Wianar,
M i
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.
1.77
.Left pleural effusion, ezlolory unknown Hypertensive vascular disease Diabetes mellitus, noniusulin dependent Leukopenia, mild, possibly related to Dx. #1
i.
; >
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_ Ne ideal and surgical including repeated thoracentesis,
'^arjnostic workup
. `
t ; V
3007 047
_ The patient is a U6 year old black male admitted to the hospital on 3-4-77
arid discharged on 3--19--77. He eos admitted because of chest oaln and exertional
dyspnea. History and physical examination are as dicataed.
' Laboratory data are as follows: Chest x-rav showed a massive left pleural
effusion with shift in the mediastinal structures to the. night. Intravenous
pjrwlograms including aamlnograohy, oral cholec^stogran, upper 01 series.
5ariun enema and liver acan were all within the normal range and chest x-ray
following adequate thoracentesis revealed no significant new findings tnd no
expansion of the left chest was noted. EKO was within normal limits. Examln- . -
it ion of the pleural fluid showed no evidence of any malignancy. The L?H was
j)2 units and amylase was 126. Hematocrit of the pleural fluid wa3 2.:. ` . '
Cultures were nonreactive. A tuberculin test was negative." Hemograms showed.
persistent mild leukopenia with a white count of 3700. Hemoglobin was normal
m d differential showed no abnormal cells with the exception of the third
"
nomogram which showed a loJC eoeinophll count on differential. Electrolyte .
:
pattern showed nothing remarkable. SMA-12 showed creatinine to be 1.5. all ,
rther parameters were essentially within range. S e rum amylase was 8**. Serole . '
r.v was nonreactive. Rheumatoid factor was negative. Urinalysis showed nothing --
.ignificant.
'
. ''
Heepital Course: The patient was tapped on two occasions with 600 ar.r'1230- i-
's of fluid removed and sent for studies. The fluid was a dark, reddish, -.
.logany appearing color. The patient tolerated the poocedure well. Kith a
.egatlve workup otherwise, it was felt that thoracotomy with exploration was . / *'
ndicatel and arrangement were made and ne was seen in consultation by
(CONTINUED)
..
-
r.lThmflVE SUMMARY THOMPSON V'OODIC 77-01832-3-,
.'rtcrp to Qo this, procedure, however, the p a t i e n t 'refused to`i ! .
- V.- 'cratlon done on the dayi-of its schedule. Accordingly he will
-; ".mi : red at hlo request to the Cleveland-Clinic for probable exnlorn-
n and further dirfrnostlo workup as indicated; T h e .llkllhood ,Of this
\ Tf-- .PhoTna appears to-be quite high.
' .: .' '
r
on Dlsch'arne-- Unchanged
' ", i
Pi?charfe n?dicatlon6 include HydroDIURIL. 50 rrir,. b i d `for control .of
hvner tens ion.
'" ' - . `
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i: -rv 3-21/ -ld-77
);
`-TD.' Wlsnar; M. D.`
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'**?r,1*' -007 C W 5
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, *
Casting Division Ford Motor Company
February 1978
Cleveland Casting Plant
5600 Engla Road
Brook Park, Ohio
Mailing Address
.
P.O. Bo* 9900
Cleveland, Ohio 44142
J. E. Slingluff, M.D.
'
2223 Fulton Road, N. V.
Canton, Ohio +709
Ret
Voodie Thompson
s.s. /A16-30-0I113
12518 Emory Avenue
Cleveland, Ohio U+135
Dear Doctor Slingluff:
.
*
Prior to his demise, our employe, Voodie Thompson, vas seeking lost
time and other benefits from us for a physical problem vhich he vas ex
periencing. Ve understand he had been under your care for this problem.
In order to properly evaluate his claim, it would be very helpful if you would provide us with a report of your findings, including Hr, Thompson' history.
Enclosed is a waiver signed by Mr. Thompson authorizing release of this informdtion. Please enclose your fee bill for the compiling of this infor mation along with your report and it will have our prompt.attention.
Very truly yours,
FORD MOTOR COMPANY
'C ( 0 \ ./ / / / / y >/
Robert M. Hisnay
/ ,
Industrial Relations Department
Cleveland Casting & Epgine Plants
R M H ipr . E no.
30I7 ?476
r
;i)fp' nif'rt'sun
ll/OB/77 11/00/77
Name
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J 111 -.in .f a | NnnMAU RAMCJKr n COMMENT
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96
, L MI O/Li : ; to routin ''
*'...,'9y 0*- 10-0'*ME.*i.*v;>
: :" UM .'' _ 136 MEQ/L
135- 145 ME * AUTOMATA MEDICAL SERVICE^
POTASSIUM
3.1f MEQ/L
3.5- 5.0 ME '
OF OHIO, INC.
'l.lf.tlUiLYtC, RAT* 68
1*1 tIS
MaatlUU. OkW44901
INffrpff'OIMULE SEE-A'TTAUCHMENT iu : 00- 25 IU
E SLiNGLUFF.DO
TRIGLYCERIDE' 119 MG/DL
10- 190 MG
23 FULYON AVE.N.W
nil FERENTTAL SEE ATTACHMENT
UON
OH .
i.o 2
28 HMOL/L . 2l- 31 MM '
*4720
SPOT .
167 IU/L "
SOOT
^ -A'NQRM.M 17-55 F 13-A5
tNo bilirubin (.80/ H MG/OL
0.00-0.60 MG
ACCT. NO. 520*127
OlRECT BILIRUBIN 770 HG/DL 0.00-0.40 MG PAOB NO.
|UR CHARACTER CLOY
*.J TH ABBT. M.O.. F . O . P . . F.A.S C .P
UR COLOR.
ornge
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1 RA898* HOODIE THOMPSON
| Ac/; No 1 ,
NAM E
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j RESULTS
UR SP. GRAV*
1.010
Ur PH
5.0
UR ALBUMIN
NEG
UR SUGAR
NFG
Ur ACETONE
NEG
UR RILE
' .NFG
R OCCOlT BLU NEG
UR HOC
NEG
Ur roc
NFG
Ur EPtTH
NEG
Ur bacteria
H
Ur amorph
*
Ur crystals
NEG
Ur casts
NEG
lUR MUCOUS NEG
UR Mt SC*
NEG
[j|11f/f0*0T.fV890/77|j11/P04W8/771
NORMAL BANOr OB COMMENT
! AitS
.015-.030 - 6.0 -NEG -NEG -NEG -NEG -NEG 0- l 0- l ' 0- l -NEG -NEG -NEG -NEG -NEG
jtiuii
AUTOMATED MEDICAL SERVICES OF OHIO, INC.
* AtS
AUntfiU, Ohi. 44KI
!1 * E SLINGLUFF*DO
t 23 FULYON AVE.N.W
NTON
OH
**720
ACCT. NO PA SS NO
520*127
A J. THABfcT. M D . F C.A * . F A tS>C*rr*r>
5*898*' HOODCE THOMPSON
A tC NO ]
NAMt
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bkSulTS j
UR ME SC*
NEG
nUN/CREAT RAT*
TO FOLLOW
CALCIUM SFRUM 9** . MG/OL
PHOSPHORUS
3.9 MG/OL
GfUCOSE
H MG/OL
run
13 M6/L
URIC ACID
7*3 MG/DL
C i. C L `STEROL
171 MG/DL
V I PROT
G*6 GM/DL
a'.tl'mTn
0 * i 3 X Cn/DL
`MLlRURtN TOTAL 1^0 MG/DL
I.K PHOS
/ 2 13\ h mu/ ml
LHH
's~?8*
mu/ ml
SGCT '
2L. . MU/ML
GLOBULIN
<!*>>> GM/L
AG
0.9-1
11/o0*8*r/77.11/r>0-8/77!
*rrfVTb | 'proffMro
normal danoe of comment
". *
AjiS;
/
8.5-10.5 MG 2.5- 4*5 MG 65- 110 MG
10- 20 MG 2.5- 8.0 MG 150- 300 MG 6.0- 8.0 GM 3.50-5.00 GM 0.2- l.D MG 30- 115 MU 90- 200 MU 10- 50 MU 1.3- 3.2 GM
AU70MAJID MEDICAL SERVICES
.OF OHIO, INC.
1*1 41$ .
IReniti*U,Dkn 41101
E SL.INPUTFF,00
OH **720
ACCT. NO. PACK NO.
520*127
4 J . T N A B ET. .D . P.'C.A.P.. FJk.S C N c** otvwcrm*
I >y
( )
DOCTC f> HOr'FlTAL MV'.ILIO/J, OHIO x-r.AY r e p o r t
EKin-.maTion cl Thompson,
X - A Y NO.
77-13403
. Hoodie
DATE
11/26/77
Report:
N&UC
H O SPITA L NO.
119865
Chest, Abdomon
.
ROOM NO-
O .P .O .
%#
on.
.
160-2
Slingiuff
Chest: 1n tv;o projections revealed the presence of an increased density noted within the left lower lung field. Infiltrate was noted throughout the left lung ` , field. On the lateral projection, the suggestion of a left pilural effusion /as noted. A left lateral decubitus projection 1s recommended for further evaluation. Discoid streaking was noted within the right mid lung field.
Adomen: 1n supine projection revealed the presence of homogenous density noted
throughout the entire abdomen. This may represent evidence of peritoneal
!
fluid. An absence of gas was noted within the left mid abdomen. This may represent
the presence of a mass 1n this area and an IVP 1s recommended for further evaluation,'
Hypoplastic ttth ribs were noted bilaterally. Other variation not Identified.
RPL/gp
Name T h o mpson H o o d i e
...Rt owojm, mN0o...... l1600r-0022 ,, ,, .S P Lab. No,7_7-f-rA3Cl
Resident, Dr......... ........ ......................
Intern, Dr.___
Pre-Operative Diagnosis (Clinical).
Operation.............................................
Post-Operative Diagnosis (Clinical) Tissue Removed. P a r a c e n t e s i s fluid
MICROSCOPIC:
l.:e I d e n t i f y n u m e r o u s hi st iocytes tog et her wi th e r y t h r o c y t e s and small cell clusters with somewhat Indistinct nuclei. Others of these c e l l s appear having a vacuolated-type cytoplasm and the nuclei appear displaced to the peripheral margin. Some of the cells also appear to have prominennucleoli. Inflammatory cells are also seen, being part of the background-
Thc large clusters of cells described appear most consistent with adeno-,
carcinoma, mucus-producing type cell.
...
IMPRESSION:
'
PARACENTESIS FLUID. POSITIVE CYTOLOGY.
( 2Y60-0143)
M. R. Du r l s h l n , D. 0
Pathologist 11-29-77
Ik
N
< 19/8
0G7 04?9.
'Jr >
-
DOCTC.TS HCrTITAL
, r.w i'!u .o r/. oh io
<. . X-flAY REPORT -
of Thrrpcon.
X.HAV NO.
77-13403
1/rcdfo
DA T Z
12/12/77
Report:
NAME
. .
H O SP IT A L NO.
'
120260
t \
Chest, ABdomon > ;
. '
ROOM NO.
O .P .O .
" 1
142-1
Sllrgluff
Chest: 1n AP projection utilizing the portable apparatus:
..
Cc-par1son made to previous examination on 11/30/77. The loft lung was unchanged
as compared to previous examination. Pleural effusion with left lowor lobs
1
Infiltrate was again noted. Right lung field revealed marked Increaso In
'
Infiltrative changes throughout the portion of the lung as compared to previous
examination. Consolidated Infiltrate was noted within the right baso extending
up tov/ard the apex, this was most likely secondary to pneumonitis or pulmonary
Infarction. The carxllomedlastlnal silhouette was unchanged.
Abdcr.cn: 1n supine projection revealed a generalized haziness notod throughout.
the abdomen. Fsoas margins wore not noted. Tho U v e r border or splenic border .
could not be visualized. We could not vlsuallzo the renal contours. Those findings
suggest peritoneal fluid. No evidence of gaseous distention was noted. Other 1
variation not Identified.
"
.
'
t i ^ i r a r i 4 ftor* r'
ft
/
rnm uz
1:1'
5007
/
1.. V- lidCLKAR r'LDICirilZ i(
'!:: if:'1, '.'m-llc _ ______
It--.
'
/ / - ] I i2/u/77._- _______
1.!'.:-.; scan: `
. Lu.'iy scan; Liver/spipeii s jim
I >**< '+ iw.
J m ; . i ;i .. s t
m' v.
\ 142-1 .. Lsnii'jiuff- :
. **
.
,
'
I. t>iii1u1 s'.^eci perfusion soon in noli': mper 1 field and inright base.
There is infiltration and offuuion i these areas on the chest x-ray and
differential diagnosis would exist tween tumor versus Infarction versus-
iniau. :onitis.
......
Left lini'j;
1. Lircronsud ii_r*fus1on 1s scon
lower portion of loft lung field.
.
flier:. is effusion and Infiltration present on chast x-r/iy and these findings
chain present differential div ..sis exists between pneumonia Versus Inf rctlo.i.
Clinical correlation Is sunoeslad.
..
.
.Llver/Spluon Scan:
1. Sn.u areas of Increased uptake are seen 1n loft lobe of liver.
This is scvetliros Sean with ut-srruc ;icr. of the superior veoa cava.
' A s t u d y of t iio superior vena c:\:a u ;il Icing Technetium is raceamended fo.`
furt'nor evlaiiaticui.
'
Liver is otherwise unrorxtrbaLlo.
2* Spleen oppuars to be within normal limits.
.
flJr/gp
"'" ' W 1978
PHYSICIAN'S COPY
<* ... I
o037
c
- : |;c-
a! Mission no'tes/ discharge sum. iy
T j- ;;?A!Tn 0=11-70___:__
W. Thompson - 120260 ' . '. '
MASS'UOii, u:;:
: II!.G P7ACN05T.S? 1, Carcinoma of liver and lungs.
'
-, ` '
-. .L DIACI OSIS. 1. Carcinomatosis with hepatic lesions primary site lung.'
' l'
'
.
'.
?he patient was admitted because of abdominal distention with shortness of breath A s c l r TM
believed to be tbe p r i o r y difficulty. He as seen on consult by Dr. T h o ^ L l i e r " jo
reccr.-cnded a paracentesis.
.
,r
Ills 2 hour postprandial blood sugar was 110. BUN was 19. Electrolytes showed
ml
potassium at 4.4. chlorides at 104, C02 at 26. VDRL was nonreacl" he urine aT e U ^ i y
hazy, pH of 5, specific gravity 1.030. Protein 1+. White blood cells occasional e? ly
20 hyaline casts with eosinophils occasional and 1+ uric acid Th calcium ,, / a q ! h?WCC
was 4.7. Total protein was 8.0. The albumin was 2.9. G l o b u l i was 5? T t o C
^
0.6. Choiesterol was 166. Uric acid was 8.3. Total bilirubin 0.2, SGOl'was 18 LDH w a s ^ i ` Alkaline phosphatase was 99. CBC showed 5.5 thousand white cells 4 2 million r J 14;`
10.0 grams of hemoglobin, 31Z hematocrit. 74 MCV, 23.3 MCH wich is very 1TM , 31.4 S c H C ^ t h
3 Begs, 2 bands, 21 lymphocytes, 4 monocytes. 1 nucleated red blood cell per 100 white cell Adequate platelets. 1+ hypochromia. 2+ anisocytosis, 2+ poikilocytosis
slight stippling. Repeat studies were essentially unchanged. Paracentesis una a * ?fS7 Z**
The patient had had a previous paracentesis which was positive for malignant cellsmi1hihed'
previous admission to this hospital. Chest x-rays were taken and werel e n t i a III TM cL ' ed
as far as the left lung was concerned and question of pleural effusion of the lower lffi ? *
was noted with an infiltrate and ascites was suspected. A lung scan was taen showedI L
ed piofusion of the right lung upper field and right base. Left lung showed decreased profu *
sion throughout the lower portion of the left luna The i
K
ea aecreased profu-
in tbe left boba of L livar. < .
S! aal" t t a
COw rSe W3S nV f continued difficulty with this ascites and complications of y ' WaS Cransferred t0 cl - e l a n d Clinic for further orb up and t o u t e d
d: 1-7-78 t: 1-11-78 JES/gl
: - W A K V 1970
J.E. Slingluff, D.O.
NOTES MUST BE SIGNED BY PHYSICIAN 8007 04*2
PQ
< [. ' I
;
^
- *' r:
( . r
'. : Ji
~
r)T 7-20-77
r V.ISSSON* NOTES/DISCHARGB SUi('' *ARY
UocUic Thompson - 119G65
MA::-! LO
:
* .
Lr:.r. of A'lr.isfion: H - 2 5 - 7 7 ... r aCc of D ischarg e: 12-6-77
. .' -
'' " '
.
.
. .
. \ ...
'
This patient was admitted with obvious ascites. History of carcinoma of the lung. Gave hint
c f having had a thoracotomy at Cleveland Clinic. Was seen on consult by Dr. Halter who felt
lhat a paracentesis was indicated, as the ascites was probably secondary to.metastasis to .
liver. -
-
'
. . -
.
The Urine was yellow and clear with pH of 5. Specific gravity 1.018. Protein negative, ougai negative, acetone negative, blood negative, bilirubin negative, urobilinogen negative. The urine was loaded with uric acid.The serum sodium was 142, potassium 3.7, chloride 99. C02 was 28. The glucose was 83. The BUN was 18, calcium was 8.7, phosphorus 3.3, total protein 6.1. Albumin low at 2.3, globulin high at 3.8, A to G ratio was 0.6, also abnormal. Cholestt 171, uric acid wa9 8.3, which is elevated. Total bilirubin was 0.4. SGOT was 12, LDH was 13i alkaline phosphatase was 80. Triglycerides were 153. CBC showed 8,100 white cells per high powered field, 4.16 million red cells per high powered field. 9.9 grams of hemoglobin. 30.2 hematocrit, 72 MCV, 23.9 MCH, 32.8 MCHC with 73 neutrophils, 21 lymphocytes, 2 monocytes and
4 eosinophils showing hypochromia poikilocytosis, microcytosis and anlsocytosis. A protein electrophoretic study was abnormal showing total protein low at 5.7, albumins low at 2.49, globulins showed slight elevation of the alpha 1 at 0.45, normal alpha 2 at 0.82, normal bet at 1.05, normal gamma at 0.99. A to G ratio abnormal at 0.7. 13 was 30.4, T4 was 7.7, T7 v;as 7.8. Repeat study showed continuous low albumin. He was supplemented with IV albumin: which improved until the time of discharge. Chest x-ray showed increase density noted within the left lower lung field. Infiltrate was noted throughout the left lung field and discoid streaking was noted in the right lung. The abdomen was suggestive of ascites. Repeat chest x-rays showed the cardiomegaly to be evident. Some tortuosity of the thoracic aorta. Suggest
of effusion was noted. Lung scans were taken showing almost no uptake in the left lung field Decrease uptake in the right lung field suggestive of pleural effusion. EKG showed ao abnorm EKG with generalized nonspecific ST-T wave changes consistent with Ischemia and/or electro lyte Imbalance. Paracentesis was accomplished twice by Dr. Halter and the paracentesis fluid showed a positive cytology consistent with adenocarcinoma of the mucous producing type.
The patient's course was some improvement with relief of 6ome of the symptoms following rema of fluid. He was discharged to be followed aB an out patient.
d: 12-23-77 t: 12-28-77 JES/gl
19TB
a
o o~n| o
J.E. Slingluff, D.O.
CO
NOTES MUST BE SIGNED BY PHYSICIAN
Inter Office
Personnel and Organization Staff
Mr. Paul E. Toth
April 28, 19 78
Subject: Asbestos Exposur-Cleveland Casting PlMt
An investigation of Mr. 'Woodyihompson* s death was conducted at the Cleveland Casting Center on April 25, 1978. Short meetings were held with D. Simmons, D.O., Messrs. G. Short, Security Supervisor, K. Hisnay, Supervisor Worker's Compensation, R. Finnessy, Worker's Compensation Administrator/also former floor supervisor in Mr. Thompson's department, and h. McDaniel, Floor Supervisor, Department V 7B9 .
In general, the operation of concern was a two-shift operation consisting
of repair welding on cast parts. This operation was initiated in 1951 and was terminated in November of 1976. (The job was free effort and no
production standards existed for it.) Approximately 35 to h5 people were employed on this job at a time. A total number of employes holding this
classification was not obtained. An attempt was made to determine if all employes formerly employed in this operation could be identified. This
question could not be fully answered but it appears that identification will be somewhat difficult requiring review of individual records.
.nn attempt was made to define conditions of the operation (furnace welding) which would influence employe asbestos exposure. A plant layout of the cleaning room is attached with the position of employes doing repair welding
shown in red.
Mr. Thompson's job was repair welding on heads or blocks. These parts were preheated in a furnace to facilitate welding (maintain welding temperature
and stress relief of parts). To provide radiant heat shielding while performing the operation sections of asbestos millboard was used. The sections of boards would vary in size and shape due to the type of repairs being made. The asbestos board would have been shaped by the employe to expose minimal surface area of the part.
Information concerning medical responsibilities in this caser was received from Dr. Simmons, a review of the record indicated that oh-May 6, 1977> Cleveland Casting Plant medical department was informed oF the diagnosis
of Mr. Thompson's mesothelioma.
Mr. Thompson's medical jacket contains five chest x-rays, the most recent being 19 7 ^* It was normal procedure to give yearly x-rays to employes in this area. It would seem that procedural problems (difficulty with scheduling, noncompliance by production personnel with medical requests)
precluded getting x-rays for every year of Mr. Thompson's exposure.
hr. Faul E. Toth
(2)
April 28,-1978
I was informed that medical records of other employes in the area should contain the same type of data.
Dr. Simmons also checked the OSHA log during our interview and found that Mr. Thompson's death was not reported or recorded. This case was not considered recordable.
Discussions with Mr. Hisnay revealed that no other cases are pending on asbestos
related disease from this or any other area of the plant. However, it was reported that two other deaths of former department 1*789 employes have occurred. These do not appear to be asbestos related. Death certificates are enclosed.
Discussions with Mr. Vi. Kosicky, Material Control Clerk, revealed that the
material used was asbestos millboard, 1*2" x 1*8" x
supplied by Allan hacking
Company. The current manufacturer is Philip Carey.
Allan Packing Company has been the supplier for the years of production use except for an approximate three-week interuption of supply.
Mr. Kosicky's estimate of usage was 50 sheets per month during the production welding operation.
lb attachments
Keith Lee
Attachment
The shaping of the board was done by simply scoring the asbestos millboard with a sharp object and breaking it. It was stated by both Messrs. McDaniel and Fimnessy that no grinding, sawing, or power cutting of any type was performed on the boards.
Mr. Finnessy and Mr. McDaniel both stated:
a. The area was clean relative to other areas in the plant
b. Employes did not wear respirators. However, welding faceshields or goggles were worn by the welders
c. Housekeeping in the area was above average
d. No training in the hazards of asbestos was conducted for the employes
e. Employes were supplied with Company issued coverallo and those may have been taken home
f. Little abuse of the asbestos boards occurred, except to write names of messages on them with the acetylene gas (this left a black residue on the boards)
g. The boards were not used as bumpers or parts stops h. Some employes, as personal preference, ate or smoked at their
job location i. Each employe did his own shaping of the asbestos boards. In some
instances, the set-up man might prepare a few boards for the welders but generally each man made his own
j. No air blowoffs were remembered
k. The area was well supplied with air-supply ventilation to provide air for cooling. There was no exhaust ventilation
l. Neither man could remember any habit unique to Mr. Woody Thompson which would have increased his exposure greater than other employes in the area
m. The asbestos was disposed of with any regular rubbish- generated in the department, no special provisions were made 'for it
n. Furnaces used in the area were not lined or insulated with asbestos
o. Material was delivered to the area in U-2" x lv8n x sheets
'007 0^P6
T Y P F '< P R I N T IN P E R M A N E N T INK
if'
f
1341 C umilia
USUAI RESIDENCE
W H U ! DECEAS10 |(V0 IF DEATH
o ccu rred in
INSTITUTION. C'VE RESIDENCE IEFORE AOMISSlON.
o m sz
csrnr
c3'iilX vaTiuu. "SHOT
Beg Dut No ________
P n m if) Hr* D>f N o .
O H IO D E P A R T M E N T OF H E A L T H ; ''7
DIVISION O F VITAL STATISTICS S'`" F'" N" "
C E R T I F I C A T E OF D E A T H Krfmart No
s f v a (a )~
D E C E A S E D -----N A M E 1m l
Middle
Grover
R A C E W htt. negro, omencun
A G E ---- L n i
U N D IC I TE Ab
indiun. //. tSProff)
htrthju) (ytun) A lo i. J<j
JlidL So
CiTT. yiU A G E . 0 LOCATION OF OEATM
Sb
INS-Of ClTT UMiTS
fSpecih fft or no)
7b Middlehurg Heights
7t Yes
JlA T f 01 IH IM III <M/ /H U.S.A.,
citizen of what c o u n t s
8* ftorth Carolina
S O C ' A l S E C U t i T r NUMftER
USA_______
w a s DECCASEO CVt in U. S T) et, hi), or unknou n)
Lati t
SEX
DATE OF DEATH
U jj. Yt*
GRAVES , Male 2__ April 25, 1977
UNDER 1 0 *T o u t 01 K IT H I W0.1//.. D.n C O U N T Y O F D E A T H
lionet Sc
'1 1 1 .
June 28. 1916
n_
Cuyahoga
M O S H A I OR O I h J I i n s t i t u t i o n -- * a ( / I f *i'W t t h t r , g n f % t t f f t u n d
)
u _____ Southwest General Hospital_______ ___
io Married A*ARf I( 0 N ( Vf A lt (0.
w iOOw id Oi>CCED (Specif))
Su r v iv in g S' Ou SE (If utte, gut muiJen nume)
i. ' Sophronla K, (Wesley)
A I mEO FORCES' (f yt p i# dotes of s0 1 )
Welder USUAL OCCUPATION f(,ite bind ol uoeb done during mott Of
u n rh n f
/if if re'i'fd)
.. _ .
n.
Ci n O OF I u Si n ESS 01 INDUSTRY
iJo Ford Motor Co.
RESIDENCE-- STATE
He OhlO
fa TmCR--n am e fm i
COUNTY
lib Cuyahoga
MtJJle
CiTr. VILLAGE OR LOCATION
Berea wi es 7 2 h Shelley U (
tN$>0E C ity l im is (Speeify i t ; 0 ' no)
STREET A*iD N j J [ R u.
U u mO ImLR--NAION NAMf
/ ,r,<
y\imjit
Parkway
Lau
Morris
I N F O R M A N T -----N A M E
Graves
MAILING AOCRESS
Rosellne - Simoson
tSttttl V' K.f ,U. i til O' .4.*A* ifu lf. up )
l?0 Mrs, Sophronla Kt Graves
1 7r 1 2 h Shelley Parkway Berea, Ohio 44017
A R* I Q *
` J N * L t A
u n >; 1 ANQ DEAi**
ie m u r m u r c rv AA iVtc)ct Condition 1 . if ony. /, u hi(h gai e tutto \
1
" C tfA jo U . C
l -r\
OUE TO. OR AS A CONSEQUENCE OF; 1
lb)
C-c U a ' H iL c v c L i (L i W i \ : la _
l ju .v !
(c) ttuting tbt under- )
i) m g (jute un
y
O ut TO. OR AS A
FAtT U. QTmCR S 'G N i,/ i CANT CCOOnNODiITIONS (
CONSEQUENCE
^/yVln'A* k-
co>ttnbufiflj to deutl> but not rtloted to luutt gn en in puil i*7j) ! a J*.OFS 'i'* "1IF t ES u t't ) ei 0 ' no;j HI J r t f '" lin in g
ip i.u r i - t / 0> J e . . '-
vjj^oyV^CW c.v^c. ACCIDENT. Su'C 'DE. h o V ' C i OE. D a t e o : i n ; u r \_
O R Un O ETER m i n CO O p f u h J A Io h i //. O u ) . V / u r JCb
HOU 33-
o
n*e
mQ w i n j j I t OCCUR RED 11`Ht* nuth if *! in-u'f in pu't I o' pu*t If .r1 / *
M 704
_
|NJUT AT W0L RlACE OF INIURT 4.' h-nne. f.iern. itffft. lutto' ( S p e c i f ) >*> o t n 0 iiHue bUg.. ttt. (Specif))
IC C a T'On
i "i n t e: m H l . 1 ) . 'io .. 1 >t i t i tl.'. i i t i : - t t . H P 1
Duy U.i) CERTIFICATION^ Month
Rh t S ` CAn
IATTE OED n the
TO
if - n -7~? h' i \ - 2s - 7 1 J'o d ecea sed from
L CE*Ti Fi Ca TiO n -- CORONE R O n (he bum ol (he exunttnuiton
of the bod} ,ind. o* the tut enigution, in nit opinion, deuth Onu'ffd on (he dute unJ due to the tuutt(t) tluted.
?0q____
) eur
AND l* S T Saw Mi / MI s
Alivfc C*i
Month
'j4
) eur
|. I M Z t-0* . : ` A t - OCCURRED i Vi w - r O ir, iOuR)
'A^TLRDEa 'h I
i
1
-n , \
'1
71 ( Ij - ( [, - f I , ? !3
n * C v /lM
Hour of dt.tth l h t d t i t J e n t u w> h* , n. t - o r .i
M onth
U .I>
) I
11 it t'-e ptu.e. On
/'/
und. to
ib e ? e
o* m *
inou tJge. dut tj the t+iue* t) ttuttJ
HOU'
??o
CERTIFIER-- n am e (Type ot print)
John A. Pupillo
m a il in g ADDRESS-- CERT/Fi ER
SIGNAtUIE- j y) ,.
lift rt O' title
73b
J . !yj- yn VX-
STREET OR * F 0 NO M
Cl t r 0 v il l a g e
Cate S'GnED - 27/77
^lU RiA l CEm aTiO NomApr. 28, N A M EO FCEm ETER YO RC R EM A TO R Y
LO CA TIO N
M i.'i ii.-.ige,orcount)) tStjtee
irSirlal 7b 1Q77 ji, Woodvale
/ J A KlddleburgHeights. Ohio___'____
N A M EO FfM IA lM EI
U 'CN O1
s siqnau
^
u*c N0 *
?5 Richard A. Baker
58^0-A
77 Baker Funeral Home 206 Front Street Berea. Ohio W)17 OATli FrEiI*mii Ti ti nssguico SiCN A tutt P FltSO N .jSSuiNG RtlMH
O'ST. NO
30 ' / / j ? / ? ?
3- 7 7 / / ) m J ./ j ? /... . / / y ......... y ......................
/J/s>. --
i \ -i
iiL2 y.i(u7 7
/
. d . / * .<f
3007 0C7
UwhSUiAlLeoteSclOcCAsNiCo!
LIVED IP DEATH O C C U ltfO IN in s t it u t io n . G ive ICSiDENCI iip o ie ADMISSION.
K** Dim. No---- ------ j T Ptimtty fire Dit No. V / T
OHIO DEPARTM ENT OF HEALTH S u it fil N o ._ (V IS IO N O F V IT A L S T A T IS T IC S C E R T I F I C A T E O F D E A T H RcX'tietr No..
ns
^ D E C E A S E D ----- N A M E I m i
Middle
Lett
SEX
D A T E O F D E A T H f.Mouf/, D*j, Y tor
,
JOSEPH
FESGHSON
L HAL]
, JA N U A R Y 1 3 . 1 9 7 7
Cit y .
omerieon If.-
NEGRO
Vi ll* G E . 01 VOCATION Or
A G E ----- L*,t UNDEt > T (A f
bitihdoy yt'iti) M0i. Oort
s kr>
>6
DEATH
INS'OI c u r lim its
no)
7b
v a r h e n s v il l e
STATI O f ftilTH III *'/l in U . S . A . , n o m e
tountry )
,
GERGIA_________
CITIZEN Or WHAT COUNTIT
,
USA
UNDEt 1 OAT
Hoort Se.
ATia .
Date OP I i IT h fMonth, Ooy, C O U N T Y O F D E A T H ) to*)
3 -2 R -1 9 2 3
7. CU YAHO GA
H O I'IT A I o n O T n ff INSTITUTION--NAME (if not tm m/htt. g,,t ttreet ond mrnmbtr)
S U B U R B A N C O M M U N IT Y H O S P IT A L
m a MICO. N C v Il M AIIUO W lOOwfD. DivO tCED Ibpetify)
,o KAftRIED____
SU.VIVING S'O U St III u lit. fnt mtiJrit n*m,)
11 kary fanning
SOCIAL S tC U I'U NUMIt
w a s DECEASED EVI in U. S. A Im EO P O K E S '
iV o , wo, or nnknou n)
(j yet. give dotet of tenner)
2 5 5 -3 6 -1 0 6 7
. J b ______________
NO
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LOCATION
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r'StiRIAL ,.b . 1-18-77
NAmE OP EmI a im EI
}4< HIGHLAND PARK CEI-1ETERY 74. CELEVELAND
I1IC NO.I
PUN E / ^ D lU C T O I S SlO N A m iE n _
OHIO
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,,___EDWARD E. STRONDER
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8l8 EAST 105th STREET CLEVELAND OHIO
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Inter Olfice
Mr. D. Schultz Safety Engineer Cleveland Casting Plant
Personnel and Organization Staff
25 July 1983
Subject: Material Analysis
A sample of material that you submitted was analyzed by the Industrial Hygiene Laboratory. You expressed concern that it contained asbestos.
The material is comprised of a red fibrous matrix which included a granular gritty material. The fibers appear to be organic which was confirmed upon ignition above a Bunsen Burner. Approximately one-third of the material ignited leaving no ash or residue. A course granular material remained which was examined microscopically. The particles had the appearance of sand which was subsequently confirmed by your office to be core sand. Asbestos fibers were not demonstrated microscopically. Since asbestos is non-ignitable, the fibrous portion of the sample is assumed to be a material similar to wool, cotton, or wood. No fiberglass was indicated.
Rogev Vabeke, Supervisor Industrial Hygiene Services Employee Health Services
Jb
007 c;$9
FORD MOTOR COMPANY INDUSTRIAL HYGIENE LABORATORY SAMPLE IDSNTIFICATIO$l,ANALYSIS REQUEST & RESULTS REPORT.
By Vhom taken:
Date taken: / l 5 " Date submitted to lab: ? / / ?
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Name of Plant:
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Date reported froa lab: 7 / 0 . 2 / 0 Remarks:
Analyst(s& ( { L W
TV S
Concur:
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