Document MGDX227p6J6BL5zRKpqB1bgVx

. -:r MU.' . >; c O^iis considerable gap in the theory impedes the solution of such major practical problems as the standardization of asbestosbearing dust,- the individual preventive medicine and therapy of asbestosis. For many years the special fibrous structure of asbestos was examined as the basic reason for its pathogenic eTfect. Gardner, Cummings (1931), Mottura (1939), King et al (19^6), Behrens * (19^9), Vorwald et al (1951) and many others consider that only . rigid, relatively long asbestos fibers, introduced in the tissue of the lung, cause its traumatization and, as a consequence, sclerosis. Seemingly testifing in favor of such hypotheses are such facts as the more considerable fibrous changes during the intro eduction of long filaments (15-20 v) and the insignificant changes * during the introduction of particles of respirable fractions ... -(<...7; p),..(Vorwald, 1951, Szimczikiewicz, ..Wicek, 1962;* Klosterkbtter, 1 around the place of the implantation of filaments, peribronchialarly, which is explained by the mechanical trauma of tissue during respiratory movements.. Also given are such facts as the absence; of fibrosis in lymph nodes and the development of fibrosis under - according to the head physician of Canadian enterprises Cartier ; . (19*l9`, 1955), a large number of patients sick with asbestosis FTD-MT-24-HI77-72 2 eooe t>86X PRODUCED BY FORT)