Document EvxOgVrzgyaR6bn5mMQr9orJV

| PERSONS- EXPOSED g PERSONS AFFECTED 20 cases reported 6 affected SYMPTOMS ANIMALS EXPOSED ANIMALS AFFECTED Alleged birth defects and malformations (anencephaly spina bifida). Study indicated that the number of cases reported were not significantly different from expected; no correlation found with spraying of 2,4,5-T. Chloracne SYMPTOHS REFERENCES 68,86 38 A cases reported Increase in perinatal death & birth defects. Study indicated the number of peri natal deaths are within normal frequency for the region; no correlation found with spraying of 2,4,5-T. Study conducted on animals in the area. 21 68 No increase in illness. ! i iO rH to ro c* o TABLE 3: Summary of Incidents Resulting in Possible Environmental Contamination with 2,3.7,8-TCDD or other Polychlorinated Dibenzo Dioxins (PCDDs) DATE OF INCIDENT 1944-present; reported in 1975-1976 LOCATION New Zealand INCIDENT RESULTING IN ENVIRONMENTAL CONTAMINATION Shrub control by spraying defoliants. SOURCE OF CONTAMINANT 2,4,5-T CONTAMINANT LEVELS 4x10 * g/arre EXPOSURE ROUTE 1945 Berlin/Germany 1949-1971 1950-present reported in 19751977 Australia-Union Carbide of Australia - Limited Yarram,Victoria/ Australia Frying of vegetables in a fat-like sub stance found in the bonb ruins. Landfilling of wastes from production of 2,4,5-TCP and 2,4,5-T Shrub control by spraying defoliant. Substance thought to have been hexachloronaphthalene N. A.a Dioxin contaminated 2,4,5-TCP and 2,4,5-T wastes. 2,4,5-T 16-30kg dioxin. N. A. Ingestion NO INCIDENT OF F.XPOSl'RI REPORTED a. N. A. - not available 0 PERSONS EXPOSED 0 PERSONS AFFECTED SYMPTOMS None reported from None reported. consumption of chic ken . 0 ANIMALS EXPOSED 0 ANIMALS AFFECTED Millions of chickens ?eneral N. A. Increase in mortality to certain cancers. Study showed no increase in overall mortality or cancers due to 2,6,5-T use. Many South Vietnamese Various maladies alledged and American due to "Orange" exposure veterans exposed. including: birth defects, increased abortion rates, skin disorders, CNS dis orders, cancer, liver disorder, etc. Many exposed. SYMPTOMS REFERENCES Dyspnea, wpbbly gait, stunting, subcutaneous edema, sudden death. 2,86 At necropsy revealed ascites; hydropericard ium; hemorrhages; swollen, pale, irregular livers. 3 ---------- . ' ------ Various 2,11,21,30,68,76,R6 -- - -- <f! TABLE 3: Summary of Incidents Resulting in Possible Environmental Contamination with 2,3,7,8-TCDD or other Polychlorinated Dibenzo Dioxins (PCDDs) DATE OF INCIDENT -, 1957-1960 hw . LOCATION United States INCIDENT RESULTING IN ENVIRONMENTAL CONTAMINATION SOURCE OF CONTAMINANT CONTAMINANT LEVELS Chick Edema Disease. Adulteration of chicken feed with low cost fats to increase caloric value of feed. Fats pooled, from many N.A. sources. Upon processing they had to be heated.... residues of commercial- chlorophenols were probably converted to dioxin, the chick edema factor. EXPOSURE ROUTE Ingestion 1957-1972; reported in 1972 Sweden Railway workers 2,4,5-T and 2f6-D exposed to phenoxyacetic acid herbicides and others during use on the rail roads. . A. Dermal, possible inhalation 1962-1970 South Vietnam Spraying of Agent Orange defoliant to control vegetation during war. 2,4,5-T and 2,4-D concontained 2,3,7,8-TCDD as contaminant !xl08kg of 2,4,5-g prayed on 1.2x10 ha; rontained 167kg TCDD. Possible dermal, inges tion, inhalation NOTE: Many studies have been conducted on the populace of South Vietnam and American veterans. Many are conflicting in their findings and in genera! the validity of the conclusions reached from these epidemiologic studies maybe questionable.