Document B5m70MoY6L2Y87ORZG1zg1ke8

Having developed our experimental research, wo even could not. confirm the hypothesis about the chemical-toxic action of asbestos. During the analysis of the materials given by its supporters it is almost never possible to reveal the correlation between the manifestation of asbestos fibrosis and the 510^ content in individual persons. Extrapulmonary changes In other organs being ascribed to the action of resorbed SiO^ are completely explained by the migration of the asbestos fibers detected, in particular, in parenchymatous organs. On intraperitoneal Introduction of different types of asbestos (1968) we found its filaments not only in the liver and kidneys, but also in the lungs. Roe and Harington (1967) upon introduction subcutaneously of three forms of asbestos, into mice found fila ments and inflammatory changes, and also swelling in the pericardium, pleural cavity, myocardium and mesenter. The filamentB can penetrate yen, through the ,wall of the ^bowels and reach^the ^(Westlake'etal.. 1965>.^-rjt V`. ` We Compared further the. solubility of the different forms of ' .asbestos and asbestos-bearing dust, and also serpentine and *brucite. Thus, after 15 days of contact of the dust of chrysotile- asbestos with O.lJf of Ringer's solution* the concentration of S102 ' - in solution comprised at most 3*8 mgjf, and of anthophyllite - . .? k.8 mg/m^ (see appendix).J,.This "agree s^withj&he- results^o'f^Ciark^.^ ' and Holt (1961)',' who established the low solubility of chrysotile- , ... asbestos; The corresponding index for asbestos-cement dust turned out to be equaXjb.o...?. 8 mg?, .for-,tremoliter.jr.2 jngl. 4+4. flbrogenous turned out to be the duussit which 1 east vY./Aa-.- . * 1 ' ,r!V " *vt* *.%: 8000 0869 ji PRODUCED BY FORD