Document 7Rz2907nYDk28E0dN1VR8Yvj6

SI135401 *******4H.*<t****tt**)t*****<HHHHMH.***#*******#******#****##* * THIS IS AN OFF-LINE BIBLIOGRAPHIC CITATION LIST GENERATED BY ** * imAElJt * A N.L.M.'S NATIONAL INTERACTIVE RETRIEVAL SERVICE ****W***************************4t#***tt*tt*IIHHH*#** NUMBER OF CITATIONS PRINTED = 18 THIS SEARCH MAS PERFORMED ON nZ'jgjjgjffa SORT = SORT FORMAT ('AUTI'). (ASCENDING) HAS SPECIFIED REQUESTED BY R. N. WHEELER MAY 31. 1984 FILE. PLEASE SEND THIS LISTING TO SHIRLEY D. COMER UNION CARBIOE CORP. PO BOM 8361 SOUTH CHARLESTON. MV 25303 ucc 020678 II 00019215 SCLER00ERMA PAGE 1 1 AU - Blsaeeia EP > Scarborough 0A i Lowney ED T1 - Atrophoderma of Pas ini and Ptarinl and systemic- scleroderma flatter] LA - Eng MH - Adult ; Atrophy ; Casa Raport > Female Human MH - Sclarodarma, Systamic/*etiology > Skln/epathology SO - Arch Darmatol 1982 Jan;ll8(l):l-2 2 AU - Bonino MV ; Blanch) C > Blanch) 0 ! Garcia 6arc)a A Y^^-^^^L^traumatlc nodular sclarodarma] 'i f"'> AB - Sclarodarma of rara appaaranea )n chtldran appears In minor seals as to tha flva par cant on tha whole tncldanea of this eollagan dlsaasa. tha children usually present localized sclarodarma and at times associated with other pathologies, traumatisms and r- injections ware referred. Two patients aged 5 and 13 years old are presented, both with nodular lesions on anterolateral thigh area, and In tha right buttock respectively. The patches of a side bigger than tha palm of hand ware only touchable and the skin that covered them only showed a slight hyparpigmentation In tha edge In one of tha cases. Tha limits were not precise and the nodulose surface was Irregular. Tha evolution was as of two and three years, right after Inoculation of antitetanieal vaccination and puncture thorn of Yuca leaves. The histological control showed Intensive phenomenons of fIbrohyalinosis covering almost all the dermis. Tha studies of the laboratories didn't produce Interesting data. The histological and clinical set of symptoms shows differences with tha esclerodermic like states as a consequence of excipient of vitamin K, B 12, norhydroxprogeeterone and antl-tetanle serum, in which they settle In the cellular subcutaneous tissue and they involution spontaneously. There are also differences with the panlculltts artefacts and with the linear morphea associated to bifid spine. At last the authors make special mention of the case described by Desmons of progressive linear scleroderma right after the triple vaccination. The nodular or subcutaneous scleroderma is a clinical form In which the histological alteration decays In deep dermis and superficial hypodermls. The cases shown suggest a coneetlon between a previous traumatism and the nodular scleroderma. MH - Adolescence i Case Report i Child, R-eschool t English Abstract hH - Female i Human ; Injections, Intradermal/adverse effects i Male MH - Scleroderma, Clrcumscrlbed/set 1ology/pathology Ml - Hounds, Stab/*ccmplteatIons SO - Med Cutan Iber Lat Am 1983,`11 (5).'329-32 II ucc 020679 00019216 SCLERODERMA PAGE 2 S113313 } ill Brentnall TJ 5 Kenneelly D 1 Barnett AJ > da Aixpurua HJ AU - Lolait SJ '. Ashcroft R i Toh BH T1 - Autoantibodies to fibroblasts In scleroderma. LA - Eng AB - Sara from 33 patiants with sclarodarma wars examined for immunofluorescent reactivity with viable or acatona-fixed fibroblasts. All 33 sara reacted with the call surface membranes of viable fibroblasts. 23 of 33 sera (70X) also reacted with nuclei of acetone-fixed fibroblasts. The commonest nuclear staining pattern was homogeneous (46/0 followed by nucleolar (36/!) and speckled (23)0. 70% showed more than 1 staining pattern in the same serum. Antibody titres of homogeneous and nucleolar staining patterns (i:A to 1)1024) were generally higher than those of the speckled pattern (lift to 1`.256). No change in pattern or tttre was noted in sera from 4 patients over a 10-12 month period. Antibody in sera with homogeneous or nucleolar staining patterns belonged to one or more of the 3 major antibody classes* IgG> IgM or IgA while antibody In sera with a speckled nuclear pattern belonged to the IgM class only. No correlation was found between the pattern of anti-nuclear reactivity and visceral involvement. - Adult ; Aged ; Antinuclear Factors/analysi* - Autoantibodies/analysis/biosynthesis i Female - Fibroblasts/analysis/immunology > Fluorescent Antibody Technic - Human > Immunoglobulins* Surface/analysts S Male i Middle Age - Scleroderma* Systemie/et1ology/*1mmunology - Support* Non-U.S. Gov't - J Clin Lab Immunol 1902 MayJ8(11:9-12 - Garsa-Elizondo MA { Dlax-Jouanen E > Franco-Casiqua JJ - Alarc"on-Segovia D - Joint contractures and scleroderma-like skin changes in the hands of insulin-dependent juvenile diabetics. - Eng - He studied 34 unselected insulin-dependent juvenile diabetics by seeking contractures at the proximal interphalangeal (PIP) joints and scleroderma-like changes of the hands or elsewhere. We found 14 contractures of only the 5th PIP in 7 and of the PIP of other fingers as well in the others. Nine of these patients also had scleroderma-like skin changes. Only one of 34 age and sex matched healthy controls had a minimal contracture of the 5th PIP Joint but had no skin changes. All patients found to have these abnormalities had had diabetes for 6 or more years and the difference in the disease duration between those with hand changes and those without was significant (p lass than 0.01). There was no correlation of these changes with renal or ocular vascular changes In this small group of patients. - Adolescence > Adult ; Contraeture/vetlology - Diabetes Mellitus* Insulin-Dependent/"cornpl1 cattons i Female - Finger Joint ; Hand Deformities* Acqu1red/*at1ology - Hand Dermatoses/etiology > Human * Mala - Scleroderma* C1rcumscr1bad/*etiology ; Support* Non-U.S. Gov't 13131 H 00019817 SCLEROOERMA PAGE 3 SO - J Rheumatol 1983 Oct;10<5):797-800 5 ill Graham-Brown RA > Sarkany I TI - Sclerodarma-llke changa* due to chronic graft-verus-ho*t dl lease LA - Eng MH - Adult ; Bone Marrow/trensplantation > Casa RaporT MH - Child* Preschool > Chronic Disease Female MH - Graft vs Host D1sease/*complteat Ions > Human * Hale MH - Scleroderma* Systemie/*etiology SO - Clin Exp Dermatol 19B3 Sep*8(5):531-8 6 AU - Grebennikov VA > Challmova RA TI - [Transformation of scleroatrophie lichen Into systemic scleroderma] r - Case Report * English Abstract > Female > Human MH - Lichen Planu*/*complieat Ions Middle Age MH - Scleroderma* Cireumscribed/complicatlons MH - Scleroderma* SystemIc/et1ology SO - Vestn Dermatol Venerol 1983 Apr>(9):60-3 7 AU - Guseva HB TI - (Etiology and pathogenesis of systemic scleroderma] isina Cyclic Monophosphate/biosynthesis > Animal MH - C-Type Viruses > Collagen/biosynthesis ; Fibroblasts/metabolism MH - Human Microcirculation ; Review MH - Scleroderma* Systemic/eetiology/fami11al A genetic/immunology physiopathclogy > Virus Diseases/complicat tons SO - Revmatologila (Moskva) 1983 Apr-Jun;(8):3-10 8 AU - He RH TI - [Report of 2 cases of primary hypothyroidism wtth accompanying scleroderma and Raynaud's phenomenon (author's transit] SffiHt S Case Report > Female Human MH - Hypothyroidism/Pcomplicatlons/lmmunology > Middle Age MH - Raynaud's Disease/Settology * Scleroderma* Systemic/*etiology SO - Chung Hue Mel Ko Tsa Chth 1988 Feb;81<2>=75-7 9 AU - Jlm'enex SA TI - Cellular immune dysfunction and the pathogenesis of scleroderma. LA - Eng ftf - Animal ; Collagen/biosynthesis > Fibroblasts/immunology/pathology MH - Graft vs Host Reaction * Human t Immunity * Cellular MH - Lymphokines/immunology Monocytes/Immunology * Rats t Review MH - Scleroderma* Systemte/ettology/*1mmunology/pathology MH - Skin/epathology > Support* U.S. Gov't* P.H.S. MH - T Lymphocytas/elmminology SO - Semin ArthrJtie Rheum 1983 AugSl3(l Suppl 1JS104-13 II ucc 020681 00019215 SCLERODERMA PAGE 9 seas s w a m 10 1U - Lee EB ! Anhalt 6J i Voorhees JJ ; Diaz LA TI - Pathogenesis of scleroderma. Currant concapt*. LA - Eng - Autoimmune Diseases/immunology i Collagen/analys 1 sAietabol 1 sm - Human ; Sclarodarma* Cireumscribed/#etiology/immunology/pathology - Sclarodarma> Sy*temie/etiology/immunology/metaboliim - Skin/analysis/blood supply/ultrestructure - Supporti Non-U.S. Gov't * Support* U.S. Gov't* Non-P.H.3. - Support* U.S. 6ov't* P.H.S. > Vascular Dlseases/complicationi - Int J Dermatol 1954 Hari23(2):85-9 - LeRoy EC - Pathogenesis of scleroderma (systemic sclerosis). - Eng - Increasing interest in the vascular features of scleroderma has led to the hypothesis that the blood vessel is the major target tissue and that the endothelial cell is the principal cell target. Useful observations stemming from the vascular hypothesis include the use of mtcrovascular abnormalities in the early detection of the patient destined to develop classical scleroderma* the discovery of a serum protease selectively cytotoxic to endothelial cells* and the study of a serum mitogenic activity for fibroblasts in scleroderma patients. Immisie events related to the vascular lesions are under active study but have not as yet provided a unique immunological lesion in scleroderma patients. The possibility that Immunity to basement membrane (type IV) collagen may be selective for scleroderma patients deserves further study. Persistent immuiity to endothelial basement membrane structures would provide a basis for continued endothelial Injury. Techniques to quantify endothelial Injury are useful to assess activity of the vascular lesions and to monitor therapies designed to blocK further vascular Injury. The definition of pre-fibrotic vascular lesions may have future therapeutic and preventive implications for scleroderma. - Blood Vessela/pathology i Collagen/biosynthesis - Endothelium/pathology Fibroblasts/metabolism ; Human - Scleroderma* Syatemle/eetiology/metabclism/pathology - J Invest Dermatol 1982 Jul)79 Suppl 1:87s-89s 12 AU - Lukasehak E TI - [Radiologic changes in the bones of the hand in progressive ients suffering from progressive systemic sclerosis on both hands were examined through x-ray in two levels. The findings have been evaluated with regard to eight diagnostic criterlons and then related to clinical and laboratory data. The results allow tha differentiation between two groups of patients: group A revealing obvious radiological and laboratory evidence of inflammation and severe symptoms as well as a relatively short diration of the disease* group B showing fewer Inflammatory signs s iii 00019219 SCLERODERMA PA6E 5 822233 with less developed symptoms and a signifieantly longer duration of tha disease. Tha clinical and prognostic significance of thasa two different courses of PSS is discussed with special reference to overlapping syndromes of connective tissue diseases or subclasses of PSS. - Adolescence } Adult > Age Factors l Aged - Bone and Bones/aradiography > English Abstract > Female - Hand/aradiography ; Human ; Mala i Middle Age - Raynaud's Olsease/complicat Ions - Sclerodermai Systemic/etiology/aradlography - Z Hautkr 19B2 Nov 15J57122).'1649-63 Mi ike T ; Ohtani Y ; Hattori S S Ono T ; Kageshita T ; Matsuda I - Childhood-type myositis and linear scleroderma. - Eng - A 5-year-old girl had linear scleroderma on the flexor surface Of the right arm; muscle Masting included the shoulder girdle. IgM fluorescence on blood vessels and along dermal-epidermal Jwiction uas observed by direct immunofluorescence in blopsied skin. Biceps muscle underlying the plaque of the scleroderma showed atrophy of entire fascicles, peri fascicular atrophy> and cellular infiltration around blood vessels that are quite similar to those found in childhood-type dermatomyosttis. In addition> various abnormal)tlas. including edema and thickening of basal lamina* were found on blood vessels in muscle tissue. The results suggested that the autoimmune collagen vascular disorder is responsible for this condition. - Autoimmune Diseases/complications > Case Report ; Child, n-eschool - Collagen Diseases/etiology ; Femala i Human - Myositis/eomplteations/etiology/apathology - Scleroderma, Systemic/eomplteattons/et1ology/pathology - Vascular Diseases/etiology - Neurology (NY) 19S3 Juli33(7)1928-30 - Rankin JA ; Matthay RA - Pulmonary renal syndromes. II. Etiology and pathogenesis. - Eng - Numerous systemic diseases share tmmunopathogenfc mechanisms. This article reviews the proposed etiologies and immunopathogenic mechanisms of a group of diseases which share pulmonary and renal abnormaltt1es. Specifically, we discuss the following diseases: Good-pasture's syndrome, systemic lupus erythematosus, progressive systemic sclerosis, Wegener's granulomatosis, lymphomatold granulomatosis, and Chirg-Strauss syndrome. - Animal ; Anthropoidea ; Antigen-Antibody Complex/1 mmiriology - Antinuclear Factors/immunology ; Autoimmune Diseases - Basement Membrane/Immunology ; Collagen Diseases/aetiology - Diseases in Twins ; Eosfnophllia/immunology - Glomerulonephritis/Immunology t Goodpasture's Syndrome/*et1ology - Human i Hypersensitivity, Deleyed/immunology ; IgE/immunology - Kidney Glomerulus/Immunology i Langerhans Cells/cytology - Lupus Erythematosus, Systernie/etiology > Lymphokines/Immunology - Lymphomatold Granulomatos I s/*ett ology ; Maerophages/1 mmu-iology 5C2S 822333 liliilili 5 w C C S2223S 8222IS H C C 82233 00019220 SCLERODERMA RASE 6 - Neutrophils/Immunology i Pulmonary Alveoli/cytology i Review - Sclerodermai Systemic/etiology i Sheep ; T Lymphocyts/immunology - Vasculitis! Allergic Cutaneous/*et1ology - Wegener's Granulomatosii/et1ology - Yale J Biol Med 1982 Jan-Feb;55(l):ll-26 - Rlmbau E i Aub'le J ; Llorach I S Lloveree J i Mairam'on J - Orfile HA ; Llorach M - [Effectiveness of the angiotensin converting enzyme Inhibitor In sclerodermic crisis (letter)} Report ; Human ; Klnlnase XX/*antagon1sts 8 Inhibitors i Male - Middle Age Raynaud's Dlsaase/complicatlons/anzymology - Scleroderma> Systamic/*anzymology/etiology - Med Clin (Bare) 1983 Jan 29)80(2)-'89 - Sattar MA ; Cawley MI - Scleroderma and carcinoma of uterus. - Eng - Adenocarctnoma/acomplleatlons ; Aged Case Report Cervix Neoplasms/*complicat ions > Female i Human - Sclerodermai Systemic/et1ology - Br J Clin Pract 1983 Feb;37(2X69-70! 72 - Toyota T i Ubiezu M i OIKawa N i Sanoyama R ; Suzuki 3 S Suzuki H - Nakajima Y ; Soto Y - Diabetic scleredema. - Eng - Many skin lesions are specific for diabetes mellltus. Necrobiosis llpoldlcai lfpoatrophy and Idiopathic bullae (bullosis diabeticorum) are usually associated with diabetes. Howeveri diabetic scleredema has not been noticed by internists! although dermatologists have paid attention to sueh a cutaneous manifestation. We reported a clinical case of a female dlabetle patient aged 15 who had been afflicted with diabetic scleredema. She had been treated with Insulin since 5 years of age. She noticed stiffness of the skin In Aprtl 1980. Skin biopsy showed thickness of the dermis and accumulation of acid mucopolysaccharide. After control of blood glucose with continuous subcutaneous insulin Infusion (CSII) and administration of tocopherol acetate and hyaluronldasei the skin lesion Improved. Etiology of diabetic scleredema Is unknown. Such skin lesion which is observed frequently In Insulin dependent obese patients Is different from a category of scleredema of Buschke. - Adolescence i Case Report ; Diabetes Hell1tus/*eompl1cat Ions - Diabetes Mellltusi Insulln-Dependent/complIcat lons/pathology - Female i Human t Male ! Scleredema Adultorum/*et1ology/pathology - Skin/pathology - Tohoku J Exp Mad 1983 Oee;141(41:457-61 81213 ucc 020684 00019221 SCLERODERMA PAGE 7 TX -la All - Urbano-Marque* A EojinopHtltc fasciitis evolving Into scleroderma [lttr] LA - Eng MH - Adult i Case Report ! Fascl1tls/eompl1cat Ions ; Human ! Male MH - Scleroderma. System!e/et1ology ! Tima Factors SO - Ann Intern Med 1983 Sepi99(3>:912 *tt END OF OFFLINE PRINT UCC 020685