Document 6wyLowDJwNJE1dbExymzGy7RE

Industrial Hygiene Digest - 24 January, 1930 of alkali far several days, together vith a high vater Intake. A diet low In salt and high In protein Is desirable. Inclusion of vitamin C In large amounts Is considered advantageous. -- Condensed from Chem. AbstB. 75 Preliminary Observations of Uranium Metal On Wound Healing With Beference To Gas FormatlonT W.F. Neuman, Roberta Allen and B.J. Muiryam"! " Atomic Energy Commission Unclassified' Documents-2476 (UB-50h 5pp. (Jan.. 13, 1949). t Small scale tests vere conducted to determine whether uranium caused gas-format Ion when exposed to tissue fluid. In-vitro experiments and in- vivo experiments using rabbits vere carried out. It is clear that uranium metal did not result-'in gas-formation when exposed to tissue fluids. The apparent Absorption of gas In the In-vitro experiment probably resulted from the solution (a3 a bicarbonate complex) of some partially oxidized uranium. Clinically, If gas-formation Is observed In wounds containing uranium metal, it appears Justifiable to assume that bacterial contamination Is responsible far the gas-formation. -- Nuclear Scl. Absts. 76 Mercury Poisoning from Fingerprint Photography. J.N. Agate and M. Buckell. lancet 237* 431-434 (Sept. 10, 1949). The standard method of developing latent fingerprints with mercury-with- chalk powder is described. Out of 32 men engaged regularly on such work, seven were found to have evidence of chronic mercurlalism. This appears to be.a new form of a long established occupational hazard. Exposures In excess of 230 hours per year are considered to constitute a definite risk. Urinary mercury estimations vere of no assistance In diagnosing Individual cases or estimating exposures, but the average excretion of the group was abnormally high. -- Authors' Summary 77 Acute Phosphorus Poisoning. H.J. Bubltzky and B.M. Myersan. Arch. Intern. Med. 03, 164-178 (Feb. 1949). "Acute phosphorus poisoning constitutes a serious medleal and public health problem, as indicated by Its high mortality and the easy accessibility of substances, especially rat poisons, which contain yellow phosphorus. A brief review of the historic aspects of phosphorus poisoning and a description of the properties and preparations of phosphorus are presented. The three clinical stages of acute phosphorus poisoning are. described, with stress an the Importance of the garlicky odor to the breath and the presence of lumi nescent vomitus. Data an`l6 patients with acute phosphorus poisoning admitted to the Boston City Hospital are presented and the clinical course of three described In detail. The pathologic changes taking place In acute phosphorua poisoning and the changes occurring In the metabolism of carbo hydrates, flats and proteins are described. The treatment of acute phosphorus poisoning la presented." -- Bull. Hyg.