Document 37JQg5LBDx7X2VdbR9YjBLq3n
j4J IE [' i C E A?i O liw'joi ii Y
MECHANICAL ENGINEERING
Published by The American Society of Mechanical Engineers
55Volume
3Number
Contents for Marche 1933
THE PROBLEMS OF PUBLIC ENTERPRISE
. . Walter Rautenstrauch
FREIGHT CARS....................................................................
. . C. B. Peck
PUBLIC WORKS AS A STABILIZER.................................. FUTURE DESIGN OF PASSENGER EQUIPMENT .
, . . E. Ogur . . C. E. Barba
LARGE PRESSURE TANKS FOR OIL-GAS STORAGE DUST IN INDUSTRY............................................................
. . L. S. Stiles . A. E. Russell
THE PLACE OF THE UNIVERSITY IN INDUSTRIAL RESEARCH FN^ME-Um-
vatftst^Y......................................................................................B. M. Woods
a new high-speed engine indicator
E. d\ Taylor and C. S. Draper
recent steam research in Europe.......................................... J- H. Keenan
the American inch....................................................................Samuel Russell
147 151 156 157 162 163
167 169 172 177
PLAINTIFFS EXHIBIT ASM-4
editorial..........................................
180
correspondence................................................... 197
SURVEY OF ENGINEERING PROGRESS .
182 RESEARCH--CODIFICATION .... 201
SYNOPSES OF A.S.M.E. PAPERS
. ..
193 BOOK REVIEWS AND LIBRARY NOTES . 203
what's going on.......................................................................................................................2071
DISPLAY ADVERTISEMENTS PROFESSIONAL SERVICE .
1 CLASSIFIED ADVERTISEMENTS ...
20
18 INDEX TO ADVERTISERS...................................22
OFFICERS OF THE SOCIETY:
A. A. Potter, President
Erik Obbro, Treasurer
Calvin W. Ricb, Secretary
PUBLICATION STAFF!
Gborob A. Stetson, Editor
Frederick Lask, Advertising Aigr.
COMMITTEE ON PUBLICATIONS:
L. C. Morrow, Chairman
S. W. Dudley
S. F. Voorhebs, Vice-Chairman M. H. Rodhrts
W. F. Ryan
C. E. Davies, Secretary to Committee on Publications
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for in sectioo 1103, Act of October 3, 1917, suthorixed on January 17, 1921. . . . Copyrighted, 1933, by The American Society of Mechanical Engineers.
DUST in INDUSTRY
The Economic and Hygienic Aspects of Silicosis
By ALBERT E. RUSSELL, M.D.1
' WOULD be difficulr to estimate with any degree of claims which are filed against them are allowed. In
riccuracy the number of people in the United States most of the industrial states the number of claims filed who arc engaged in dusty trades; in fact, with the has greatly increased in recent years. present varied record systems of the different states, It is clear that dust-prevention work, like any healthsuch an estimate would be practically impossiblep. romoting activity, would pay many times its cost in
This the author learned when he was preparing hismoney, good health, and efficient operation.
paper for the International Silicosis Conference. How- Strangely enough, only during the past 15 years has
era-, an idea of the frequency of exposure to dust may dust received much attention as an etiological factor c/
be found from the statement that more than half a disease. The rapid development of industrializati
million workers are engaged in the coal-mining industry within that period partly explains the impulse tow
in the United States, and over 10,000 workers are em the study of this source of industrial hazard. The
ployed in dusty occupations in New York City alone. advances which have been made in all branches of
It is evident that more people are exposed to dust, which industrial medicine have helped to center attention on
constitutes the greatest single industrial hazard, than is dust and its effect upon the health, inasmuch as the
generally supposed. Certain dusts contain poisonous number of workers in the dusty trades is larger than
dements, such as lead, arsenic, mercury, ere., which that of any other group exposed to a single industrial
give rise to general conditions resulting from their hazard; and an additional reason for increased interest
absorption. This paper, however, will deal only with in this subject is gathered from the study of mortality
those dusts which are known to be direct factors in the statistics, which reveal the fact that workers in the
production of pulmonary diseases.
dusty trades have an excessive death rate from tubercu
Dust is possibly the oldest source of industrial hazard. losis, as well as from other respiratory diseases.
Knowing as we do how dust is generated, we may The most notable studies of silicosis have been made
assume that as far back as the stone age and farther, in the gold mines of South Africa, where the disease,
people have been affected by it. Certainly the Greeks, with its complications, has been most commonplace
Romans, and Egyptians must have encountered this among the miners. It became necessary either to study
hazard in connection with their extensive construction the disease and devise methods for its control or else to
work with stone. .The harmful effects of dust arc men abandon mining. Consequently funds, personnel, and
tioned in Greek literature, buc the Italian writer, clinical material were made available for the first time
Ramazzini, gave us in the 18th century the first detailed on a large scale. Investigators in South Africa have
account of the action of this industrial factor upon the become foremost in this field of research; their scientific
worker.
work is widely known and has served as a basis for
ECONOMIC ASPECTS OF SILICOSIS
The economic problem presented by silicosis is tre mendous. The suffering and loss of life due to it cannot be adequately measured. In the gold-mining area around Johannesburg, South Africa, during the1 period 1911 to 19x9, 11,108,015, or more than $53,000,000, was paid in compensation alone. The costs of medical care, hospitalization, and legal proceedings further augmented this enormous figure.
Mines in Australia arc reported to have been bank rupted by payment of silicosis compensation. The author knows of a company in our own country which has claims amounting to more than a million dollars for disability due to dust inhalation. There are other plants in the Ease which will be bankrupted if all the
legislation, compensation rates, and further siudy of the disease silicosis.
There are almost 300,000 workers in the South African mines. It was because of the excessive incidence of silicosis among the miners around Johannesburg that the International Labour Office of the League of Nations selected that city for the first International Silicosis Conference.
In the United States the first study of the effect of dust was made by Dr. A. J. Lanza, an officer of the Public Health Service serving with the Bureau of Mines. His observations, made on miners in the Joplin, Mo., district and published by the Bureau of Mines and Public Health Service, have been confirmed by subsequent studies. Agencies which have done and are now doing investigative work are the U. S. Public Health Service,
'Surgeon, U. S. Public Health Service; Chief Surgeon, U. S. Bureau of Mines, Washington, D. C.
Presented at the National Process Meeting arranged by the Process Industries Committee of the A.S.M.E. and held under the auspices of
the Buffalo Section of the Society, Buffalo, N. Y., June 6 to 8, 1932.
the Bureau of Mines, and the Universities of Harvard, Yale, and Pennsylvania. Private persons have also contributed valuable information on silicosis.
The word "pneumonoconiosis" is of Greek origin
163
164
Mechanical Engineering
and means "dusty lungs." It is an inclusive term and is being used now more chan previously with a designa tion as to the type of dust responsible for the pathology. There are few instances where a pure dust, i.e., one unmixed with other elements, occurs in industry.
The intelligent consideration of the relation of dust to the etiology of pulmonary disease must include the following:
i Chemistry and petrography of dust i Condition of worker at beginning of exposure 3 Length of exposure, concentration, and size of par
ticles 4 Effects produced.
The dusts most frequently met with--and, incidentally, the most harmful--in the industries are inorganic. It has been shown conclusively that silica, one of the most common dusts encountered, alone produces more per manent pulmonary damage than all other causes com bined.
CHEMISTRY AND PETROGRAPHY OF DUST
In the earlier days all dusts were thought to be capable of producing serious damage. Subsequent investigation showed that this was not the case, and that no two types of dust produced the saine results. Clinical and experi mental work has proved that silica is the arch-offender. Therefore any paper on the subject of pneumonoconiosis must necessarily be concerned principally with silicosis. The first theory relative to the effect of dust was that its harmfulness is due to sharp, angular particles. This theory has been disproved by several investigators. Gardner's work on animals showed that carborundum dust, a harder substance than silica, would not cause the massive pulmonary fibrosis that silica produces. The size of dust particles will be discussed later, but it might be said here that only the very small particles reach the alveoli of the lung, many of them, as small as bacteria and of unimportant physical hardness. Collis2 stated that, "Generally speaking, dusts arc more injurious as their chemical composition differs from that of the human body or from the elements of which the body is normally composed, whence it follows that animal dusts are less injurious than others."
With this in mind, it may readily be seen that dust from calcium, carbon, etc., common constituents of the human body, would not be expected to produce the same effect as silica or quartz, an uncommon constituent in our make-up. Research has shown that silica in its free or uncombincd state is capable of producing serious pulmonary damage, whereas in its combined state, as in silicates, it is relatively innocuous. Obviously, it is important when studying a dust to know the percentage of quartz or free silica in its composition. This is accom plished by petrographic analysis. The chemical analysis of cement dust indicates the presence of about 2.1 per cent of silica, while petrographic analysis shows a content of only about i per cent of quartz. Likewise, the chemical
* E. L. Collis and Major Greenwood, "Health and the Industrial Worker."
analysis of Darre granite indicates the presence of ap proximately 70 per cent of silica, while by petrographic analysis only 35 per cent of quartz is found. It is not surprising that chronic conditions of the upper respira
tory tract may be produced by the physical or mechanical action of dust and that possibly a chronic bronchitis may result, but only silica produces massive pulmonary fibrosis and a strong predisposition to infection with tubercle bacilli.
The theory that silica becomes soluble after remaining in the tissues lias been generally accepted, and it was also agreed at the International Silicosis Conference that "there is experimenral evidence char the solubility of silica in the tissues is an essential factor in the causation of silicosis." It is generally accepted that the silica particles are removed from the alveoli by the macro phages, which are sometimes called "dust cells," and are stored in the various parts of the lung. Apparently, the gradual dissolution of the silica stimulates an exces sive production, with time, of fibrous tissue. Kettle5 contrasts this slow action of silica in the lung tissue with the more rapid reaction which occurs when silica is injected into the subcutaneous tissues, in which case .an inflammatory reaction is produced and in a short time necrosis takes place.
It is also well known that tubercle bacilli grow well in a medium rich in colloidal silica. Gardner'* showed that guinea pigs inoculated with a low-virulent strain of tubercle bacilli and exposed to siliceous dust of known quantity and concentration developed clinical tubercu losis with a dissemination of the infection, whereas the controls inoculated with the same strain and dosage of bacilli failed to develop the disease to such, an extent. Obviously, the effect of silica in the tissues aids in the growth and proliferation of tubercle bacilli.
Apparently, there is not the same effect in the action of other bacteria in the pulmonary tissues that is found in the case of tubercle bacilli. Likewise it has not been shown that the growth of tubercle bacilli is augmented by the presence of other types of dust. An excessive amount of other respiracory diseases can be accounted for by the lowered local resistance caused by the presence of dust and the excess of bacteria carried into the lungs by the dust itself.
CONDITION OF WORKER AT BEGINNING OF EXPOSURE
Reference has been made to the manner in which a tuberculous infection is augmented by the presence of silica. It follows that a worker having a tuberculous infection, either latent or active, will suffer more from the effects of dust than a person who is not so infected. It is highly necessary for workers to be examined thor oughly before being permitted to engage in a dusty trade. Experience with miners in South Africa has shown that a worker having a latent tuberculosis or one in whom such infection was not demonstrable, or if he was of the so-called "phthinoid" type, he developed
5 E. H. Kettle, "Experimental Silicosis," Jl. InJ.
toI. $
(1926), pp. 491--495
* L. U. Gardner, American Ktvinc tf Tnimnltsir, Dxembcr, 1929.
March, 1933
163
Silicosis more rapidly and a tuberculous complication respiratory diseases; it may be seen that these rates are
earlier than did the normal worker.
not in the same ratio as the total amount of sickness.
LENGTH OF EXPOSURE, CONCENTRATION, AND SIZE OF PARTICLES
The length of exposure to dust is evidently a great factor in the production of disability. It has been found that the occurrence of silicosis is in direct relation
COURSE OF SILICOSIS AMONG WORKERS NO LONGER
EXPOSED TO SILICA DUST
In the study of silicosis, one of the most important questions which has arisen is the health history of those individuals who have worked in the granite industry for a number of years and then left, to find occupations in other trades where there was no further dust exposure. It was possible during the course of the study at Barrc to observe 14 such cases.
The latency of silicosis lias been referred to by other investigators. The South African workers found that "a steady fall over a period of years in dust concentra tion is not associated; with a corresponding fall in the silicosis incidence."6 Dr. Watkins-Pitchford also re cords cases of miners who had been exposed to dust for a long period of time, apparently sufficient to develop silicosis, but who went into the war apparently not silicotic. He asserted chat such cases were liable to
FIG. I PREVALENCE OF TUBERCULOSIS (ACTIVE CASES) BY
LENGTH OF SERVICE AND DUST-COUNT GROUPS
(Percentage of workers diagnosed on physical examination as having tuberculosis.)
to length of service and concentration of dust. This is illustrated in Fig. i.
EFFECTS PRODUCED
In order to determine the effects produced by dust inhalation, a number of scudies were made. These observations included a study of the sickness of workers with especial reference to respiratory diseases, physical examinations supplemented by X-ray and laboratory tests, and a study of the mortality records. _ Morbidity. Prior to the discussion of morbidity rates, a brief reference will be made to the extent of dustiness to which groups of workers have been exposed. The principal information presented in the morbidity studies was taken from observations made on a group of granite workers exposed to a known concentration of dust. These workers were classed in four general groups according to intensity of exposure per cubic foot of atmosphere, as follows: Groups A and D, 30 to 60 million particles per cubic foot; Group C, 2.0 to 30 million particles per cubic foot; Group D, less than id million particles per cubic foot (Fig. 1).
The frequency of disability lasting longer than one week, exclusive of that from accidents and from respira tory diseases in specified industries, is shown in Fig. z. The lower portion of the graph shows the incidence of
~
A3
C D Hurd Rock
Iron General
u
Granite Workers
Cold Cement and Sick
Mining
Steul Benefit
" 20 15 -
19.5
12.8
(b) ANNUAL DEATH RATE
PER 1000 PERSONS FROM TUBERCULOSIS
10 -
5
0 A
36*
. ?;, feS&SI
o
3 C3 4* I*
,4 i--------1
Rural Vermont
Deaths
Deaths
Deaths
Age 20"59
* From Beginning of Study to Working Up of Report - about Three Years - among 972 Workers A,$/4;3,/04;C, M6;l),/08.
FIG. Z (_a) ANNUAL FREQUENCY OF ABSENCE FROM TUBER CULOSIS (EIGHT DAYS AND MORE). (if) ANNUAL DEATH
RATE PER IOOO PERSONS FROM TUBERCULOSIS
develop tuberculosis. Dr. Pancoast presents a case of advanced pneuinonoconiosis in a quartz miner who had been exposed to dust for eight years. He had been out of the mining industry for 10 years, yet the X-ray showed extensive pneumonoconiosis with irregularities of diaphragm, and by fluoroscope he found the dia phragm restricted on each side. There was, perhaps, a
tuberculous infection intervening at the time.6
1 South African Institute Medical Research, Annual Report for year
ending Dec. 31, 1925.
i
* Henry K. Pancoast, `'Roentgenological Studies and Other Fibrosing
Conditions of the Lungs," Analysis of Clinical Miilicim, July, 1923.
166
Mechanical Engineering
Britton7 reports two cases of workers who had been exposed to dust between seven and eight years. They changed occupations and had been away from siliceous dust for eight and nine years. They developed pulmo nary symptoms and were found to be suffering from sili cosis and tuberculosis. Tattersall8 also observed cases of latent silicosis in his studies: "Some of the men (rock drillers), moreover, had changed their occupation for various reasons quite apart from health, but in due course the inevitable dyspnoea came on. One man, for instance, worked eight years regularly with rock drills from 1906 to 1914, then joined the Army, was passed as A-i; but in spite of his open-air life, dyspnoea came on in 1918, and from then until his death six years later his illness was a typical case of silicosis."
MORTALITY
The influence of dust on mortality from tuberculosis is clearly indicated in the following table. New methods
Vermont, shows that there has been an excessive death rate from pneumonia and other respiratory diseases (tuberculosis excluded) during this period.
Fig. 3 shows the age curve of mortality of workers using hand pneumatic tools as well as that of males in rural Vermont. The rate of the latter may have been in fluenced by the fact that many disabled cutters move to
I AH Pneumatic Hgnd'Too! Operators
Surface Cutters with Eihoust
Y///////A
Tool Grinders
1i
Genera/ Plant Atmcso^iere
Sandblast Operators
BP
Plants without Yert/t/often m P/ants with Ventilation E,/-) , \
10 cO 30 40 50 60 Average Oust Count in Millions of Particles per Cubfc Foot
70
no. 4 SHOWING THE DIFFERENCE IN ATMOSPHERIC DUST
CONDITIONS DETWEEN A GRANITE PLANT EQUIPPED WITH EFFICIENT LOCAL EXHAUST VENTILATION AND PLANTS NOT
SO EQUIPPED
rural districts. However, the contrast between the two races is great.
3FIG.
TREND OF MORTALITY FROM TUBERCULOSIS AMONG
GRANITE CUTTERS COMPARED WITH THAT IN RURAL
VERMONT
of manufacturing stone which created excessive dust by the use of pneumatic tools, were introduced in the granite industry about the beginning of the present century, and the tuberculosis rate has increased rapidly with their use. The rate has risen in direct proportion to the length of time during which they have been employed, as follows:
1.5 per 1000.............................................. 10.8 per 1000.............................................. ______ 19.5 per 1000..............................................
* During period of our observations.
1890-1894 1910-1914 1924-1926*
A consideration of the mortality statistics of Barrc,
7 James A. Britton, M.D., "Silicosis, a Modern Factory Hazard,"
Jl. Ind. Hy&itne, September, 1924. ' Norman Tattersall, M.D., "The Occurrence and Clinical Manifes
tations of Silicosis Among Hard Ground Workers in Coal Mines," ]l. Ind. Hjgiint, November, 1926.
SUMMARY
Morbidity and mortality records indicate that workers exposed to dust have excessive rates of respiratory diseases.
Inorganic dusts produce more pulmonary disabilicy than organic dusts.
Silica (SiOj) of the crystalline variety (quartz) is the dement in dust which produces the greatest pulmonary damage, and it is present in varying extents in most industrial dusts.
Silicosis is insidious in its onset and progress, and presents but few symptoms or physical signs in its early stages.
Silicosis is almost invariably complicated by tubercu losis after a number of years of exposure, which time depends upon the concentration of the dust and the percentage of free silica which it contains. Latent tuberculosis is usually activated after shorter exposure to siliceous dust, and has more characteristics of this disease in persons not in dusty trades.
Masks frequently give a false sense of protection. Removal of dust at its source is preferable (Fig. 4).
There has been a great reduction in the death rate from tuberculosis among the general population during recent years, while among workers in dusty trades there has been a great increase during the same time in the death rate from this cause.