Document v1dg34OnoqwG12VMr0JGJJQrY
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IN THE CIRCUIT COURT OF COOK COUNTY, ILLINOIS COUNTY DEPARTMENT, LAW DIVISION
BOARD OF EDUCATION OF THE CITY OF CHICAGO,
Plaintiffs,
NO. 92 L 9934
vs. A, C AND S, INC., et al.,
DEPOSITION OF: DR. ARTHUR M. LANGER
Defendants.
:
--------- - X
EVANSTON COMMUNITY CONSOLIDATED:
SCHOOL DISTRICT NO. 65, et al.,:
Plaintiffs,
NO. 92 L 9933
vs. :
A, C AND S, INC., et al.,
:
Defendants.
:
--------- - X
BOARD OF EDUCATION OF HIGH
:
SCHOOL DISTRICT NO. 221, etal.:
Plaintiffs,
NO. 92 L 9932
VS.
A, C AND S, INC., et al.,
:
Defendants.
:
--------- - X
BOARD OF EDUCATION OF TOWNSHIP :
HIGH SCHOOLS, et al.,
:
Plaintiffs,
: NO. 92 L 9927
vs.
:
A, C AND S, INC., et al.,
Defendants. -------- -
:
: -X
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TRANSCRIPT of the Examination Under Oath of the witness called, in the above-captioned matter, said deposition being taken pursuant to Federal Rules of Civil Procedure by and before BERNADETTE H. MASTRANGELO, a Certified Shorthand Reporter and Notary Public of the State of New York, at the law offices of WILSON, ELSER, MOSKOWITZ, EDELMAN & DICKER, ESQS., 150 East 42nd Street, New York, New York, on Wednesday, May 31, 1995, commencing at 10:00 a.m.
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APPEARANCES:
6REITZER & LOCKS, ESQS., 1500 Walnut Street 22nd Floor Philadelphia, Pennsylvania 19102 BY: JONATHAN W. MILLER, ESQ., Attorneys for the Plaintiffs.
STICH, ANGELL, KREIDLER & MUTH, ESQS., The Crossings, Suite 120 250 Second Avenue South Minneapolis, Minnesota 55401 BY: ROBERT D. BROWNSON, ESQ., Attorneys for the Defendant, Asbestospray
Corp.
PEPPER, HAMILTON & SCHEETZ, ESQS., 3000 Two Logan Square Eighteenth & Arch Streets Philadelphia, Pennsylvania 19103-2799 BY: SUSAN A. KING, ESQ., Attorneys for the Defendant, Pfizer Inc.
MORGAN, LEWIS & BOCKIUS, ESQS., 2000 One Logan Square Philadelphia, Pennsylvania 19103-6993 BY: DENNIS J. VALENZA, ESQ., Attorneys for the Defendant, United States Gypsum 6 Company.
W.R. GRACE 6 CO., One Town Center Road Boca Raton, Florida 33486-1010 BY: RICHARD A. SENFTLEBEN, ESQ., Attorneys for the Defendant, W.R. Grace & Co.
BURKE, WEAVER & PRELL Xerox Centre 55 West Monroe Street Chicago, Illinois 60603 BY: DANIEL C. COLE, ESQ., Attorneys for the Defendant,
Union
Carbide.
23 HAYTHE 6 CURLEY, ESQS., 237 Park Avenue
24 New York, New York 10017
BY: CLINTON B. FISHER, ESQ.,
25 Attorneys for the Defendant, T 4 N pic. ***
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1 2 WITNESS
INDEX DIRECT CROSS REDIRECT RECROSS
3 DR. ARTHUR M. LANGER
By: Mr. Miller
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4.
5
6
7
8 9 10 IDENT. 11 P-1 12 P-2 13 P-3 14 P-4 15 P-5 16 P-6
EXHIBITS DESCRIPTION Curriculum vitae Expert witness disclosure One-page document Diagram sheet Cover letter Report by Dr. Langer
PAGE 14 55 98 116 116 163
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18
19 REQUESTS FOR INFORMATION
PAGE
20 Electron micrograph data sheets
123
21 Air sampling for work sites
139
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23
24
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DK. ARTHUR M. LANGE R, doing business at Bedford Avenue and Avenue H, Brooklyn, New York, having been duly sworn, testified as follows:
DIRECT EXAMINATION BY MR. MILLER: Q. Dr. Langer, good morning. My name
is Jonathan Miller. I'm an attorney with the firm of Greitzer & Locks in Philadelphia. It's important that you hear and understand everything that I say today. If you do not hear me or do not understand me, please stop me and I will be happy to repeat my question.
Do you understand that, sir? A. Yes.
Q. I represent two of the three plaintiffs, and they've consolidated the asbestos case pending in Chicago. I represent the Board of Education of the City of Chicago and a number of suburban district captions, generally under the title of Evanston.
Do you understand that, sir? A. Yes.
Q. I understand that you have been retained, you are an expert consultant for Asbestospray in
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this case, is that correct?
A. Yes.
Q. You are also an expert consultant
for another defendant named Conwed in this case,
is that correct?
A. Yes.
Q. It's my understanding that your
testimony today will focus on your being
consulted by Asbestospray, and so it will
include Asbestospray-specific questions, as well
as general questions about your view, but I will
not go into any Conwed specific questions.
MR. MILLER: Is that your
understanding, also?
MR. BROWNSON: Jon, so we're clear,
Dr. Langer was obtained by Conwed. As you
know, Conwed is a defendant in District
2007, 211 cases which are being handled by
Elaine Siegel.
MR. MILLER: That is another reason
why I am not going to ask anything about
Conwed. Conwed is not a defendant in whom I
have interest. I understand there's a
future date for your deposition with
Ms. Siegel that will focus on Conwed.
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MR. BROWNSON: Specifically, we made
those arrangements with her.
BY MR. MILLER: Q. Doctor, how many times have you
testified in an asbestos property damage
deposition? A. I don't know, maybe a half dozen or so.
Q. Have you ever testified in a trial
in an asbestos property damage case?
A. Yes.
Q. How many times?
A. I testified in the West Virginia property
damage case, I may have testified in one other
case, but I'm not sure, maybe several other
cases.
Q. At least one of the asbestos
property damage depositions you gave was in a
case for Robert Sweeney on behalf of the
plaintiff, is that correct, sir?
A. Yes, that'scorrect.
Q. Aside from that, have all your other
asbestos property damage depositions and trial
testimonies been as an expert consultant by a
defendant?
A. That couldbe true, yes.
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Q. Approximately, how many times have
you testified in depositions involving asbestos
personal injury cases?
A. Several dozen, probably. Q. How many times have you testified at
trials involving asbestos personal injury cases?
A. Maybe one dozen, maybe more.
Q. How would you divide those
testimonies between an expert consultant by the
plaintiff and expert consultant by the
defendant, sir?
A. These are only testimony, correct?
Q. Both deposition and trials.
A. Just testimony, you're talking testimony,
not consultant?
Q. Exactly, just testimony, precisely
right.
A. That's an evolution. At the very
beginning, I represented plaintiffs only. Then
an equal number of plaintiffs and defendants;
and as of late, it's mostlydefendants.
Q. Have you ever testified before the
EPA?
A. Yes. Well, I'm just trying to think if
EPA's ever heldhearings,which onewould
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testify. Have I? Indirectly.
Q. I'm not talking here about the times
that you were a reviewer.
A. No, no, I understand the question.
Q. Have you ever done such direct
testimony with regard to asbestos?
A. Yes, that would be before a committee
hearing on the advisability of passing the Toxic
Substances Control Act. It would be Senator
Tunney's (phonetic) committee.
Q. What year was that, sir, that you
testified?
A. Could be 1973 or thereabouts.
Q. What was the general subject of your
testimony?
A. Well, the sources of fiber which may enter
into the environment.
Q. You were still --
A. I'm sorry, that's all right. Ask your
question.
Q. You were still at Mount Sinai at
that time?
A. Yes.
Q. Were you testifying on behalf of any
particular group?
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A. No.
Q. That's the only time you've
testified directly or indirectly regarding the
EPA, is that correct, sir?
A. Well, this is my recall, but there may
have been other instances in which testimony was
then used by the Environmental Protection
Agency, such as testimony before OSHA.
Q. How many times have you testified
before OSHA with regard to asbestos?
A. I think three times, two or three times,
something like that.
Q. Please tell me the years and the
subject matter of each one.
A. Well, the foremost that is easy to recall
is the last piece of testimony which focused
on -- well, what was the year? The years 19 --
1991 or thereabouts, '90,'91.
Q. it concerned what, sir?
A. It concerned the subject of whether or not
OSHA should remove cleavage fragments from the
asbestos standard.
Q. Your position was, in summary, what?
A. Cleavage fragments were different from
asbestos fibers. Experimental data indicated
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that they had a much, much reduced
carcinogenicity, and they should be removed from
the asbestos standard.
Q. Did OSHA so remove them?
A. Yes.
Q. Mas thatin the1994 regulations?
A. Which?
Q. The asbestos regulations that came
out in August of 1994?
A. Well, the --yeah. Imean, all of these
are published in the Federal Register. These
are all public documents. You can read them
yourself and find out what the outcome was, but
they did effectively remove cleavage fragments
from the standard.
Q. 1990 testimony, were you testifying
on behalf of any organization?
A. I was testifying on behalf of my own
laboratory. We had provided data which were
collected during the support from the Vanderbilt
Tagging Company, but we represented ourselves.
When I say "we," that's Langer, Nolan
who testified. He's the associate director of
my crew, and John Addison from the Institute of
Occupational Medicine in Edinburgh.
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Q. Is this the second time you testified before OSHA?
A. Yes, that would be the hearings held in
19, I think, 84. Q. Have those hearings led up to the 1986
OSHA asbestos regulations?
A. Yes. Q. What was the general subject of your
testimony in those hearings?
A. Well, I wrote a document which was
submitted as supporting a documentation
concerning the nature of asbestos fibers and
whether or not certain materials should be
included in the standards.
Q. Was that testimony on behalf of any
organization?
A. No. Actually, it was on behalf of the
Department of Labor. We were asked to -- we, my
colleagues and I at Mount Sinai, were asked to
generate a number of documents which were to be
used as the underpinning support for OSHA
asbestos regulations.
Q. Is there a possibility of a third
time you testified before OSHA?
A. I seem to recollect that in the late '70s
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I appeared before one of the hearing committees
with Irving Selikoff.
Q. Do you remember the general subject
of that testimony? A. No, I think it was general mineralogy.
That was my expertise, and thatwas whyI was
present. I appeared with IrvingSelikoff.
That
was my expertise, mineralogy.
Q. Okay. Are there any other Federal
government organizations that you have appeared
before in giving testimony before, sir?
A. Here in the United States?
Q. Yes, sir.
A. Yes, I appeared before the Consumer
Products Safety Commission.
Q. What year was that?
A. 1989, perhaps, 1990, something like that.
Q. What was the general subject of your
testimony then?
A. I appeared as an expert for the Consumer
Product Safety Commission. The issue at that
time was a carbonate place, and in whether or
not this material was contaminated with asbestos
fiber.
q. So, that's the only time you
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appeared before them?
A. Yes. Q. Any other Federal government
testimony appearances in this country that you
can recall at this time?
A. I can't recall. Q. Have you ever testified before any
State or local legislature or municipal or
governing bodies in Illinois?
A. In Illinois?
Q. Yes, sir.
A. No.
MR. MILLER: Okay. Exhibit 1,
please.
(P-1, Curriculum Vitae, received and
marked for Identification.)
Q. Dr. Langer, marked asExhibit 1 is
a copy of your CV. This copy of your CV has
materials going up through 1994, has numbered
pages through page 43, and then additional pages
called funding source.
Is this the most up-to-date copy of
your CV, sir?
A. Well, it's reasonably up-to-date, yes.
Q. Please tell me if there are any
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itaas that should be added to it in any of the
categories.
A. As an example, I attended a conference at
Pennsylvania State University last week and
presented a paper on modification of chrysotile
fiber and biological activity. It was called a
Goldschmidt Conference. It was held under the
auspices of the Mineralogical Society of the
American -- the Mineralogical Society of
America, and the Geochemical Society, among
others.
Q. Anything else which should be added
to Exhibit 1?
A. I don't know. I would have to examine it
in detail.
Q. How about in terms of peer-reviewed
articles, are there any that have appeared more
recently than your last one, which is on Page
16, and it's your New York City article with Dr.
Wilson, et al.?
A. No. I don't think so.
Q. Okay. Are there any more recent
publications under symposium proceedings?
A. I think that is current. It looks
current.
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Q. Are there other publications and
published abstracts? A. No, we have two abstracts that were published in proceedings of the Goldschmidt
Conference, and we have a paper which has been submitted to -- as soon-to-be-published text on
Occupational Environmental Medicine, a chapter
in the book.
Q. What is your chapter on, sir?
A. It's on mineralogy, the nature, the
materials to which workers are exposed in the
workplace.
Q. Who's the editor on that book? A. Phillip Harber, from Los Angeles.
Q. Do you know which publishing company will be publishing the book?
A. Not offhand.
Q. Do you know how soon it will be
published?
A. I think publication is about as imminent,
as they say.
Q. Good.
Do you have any other peer-
reviewed articles or other typesofpublications
in which publication isimminent,namely,
before
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October?
A. No. Q. On Page 30, you have a list of
manuscripts and preparation. Are they still in preparation or --
A. Preparation changes with time because
there is the famous item, crocidolite paper,
that's on the back burner because I don't have
time to do it. The tremolite dust is exactly
the same. It's just the data there. The fibers
in human lungs of an exposed worker, that is all
done, and that has to be written up. The
surface mechanisms and the anion transport
paper, that is up to Nolan to write up. He has
pretty much a draft worked out.
Basically these papers are in
preparation, meaning the data are in the
laboratory and they are in the process of being
written up.
Yes, they're all pretty much
manuscripts and preparation -- work in preparation.
Q. The last two manuscripts in
preparation are on the top of Page 31, and they
both concern asbestos bodies.
A. Correct.
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Q. If I were to ask you your general
opinions about asbestos bodies, particularly as
indices in the disease risk, would you be able
to tell me even though the manuscripts are not
published?
MR. BROWNSON: That's a yes or no
question.
A. Would I be willing?
Q. Would you tell me your opinions?
A. Only if you're decent to me today; yes, of
course.
Q. What are your opinions, Doctor,
about asbestos bodies as indices of disease
risk?
MR. BROWNSON: Well, this is a vague
and general question, and I object to it
on that basis, but to the extent you can
answer, go ahead.
A. The asbestos body has been used as a
marker of exposure, as an early marker of
exposure. It has -- the asbestos body has
evolved in time in terms of nomenclature and
specificity. The asbestos body, depending on
occupation, is an index of exposure to fibers,
generally, and on the basis of my work.
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following various groups of individuals, there
is a quantitative and proportional trend which
indicates that among workers that are intimately
involved with asbestos-containing products,
their asbestos body concentration tends to be
high, and the prevalence among that population
tends to be universal. Meaning, an insulation
worker who works in cramped spaces exposed to
aerosols of asbestos-containing products, all
asbestos workers have what we call asbestos
bodies in pulmonary tissues, and concentrations
tend to be very high.
My data showed that the shipyard
worker population tends to be lower in
prevalence, meaning, only 75 or 80 percent of
shipyard workers have what appear to be asbestos
bodies in their pulmonary tissues, and that the
concentrations of these are very much less than
present -- are present if the insulation worker
that is followed by a group that we call
construction workers, that I called construction
workers, in which the prevalence, again,
decreases, and numerically, the quantities
decrease.
This is followed by a number of
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other occupations which deal with automotive trades, mechanics, brake maintenance and repair
workers, and this is followed by white-collar
workers, in general, and then followed by
white-collar housewives who have never been
engaged in work outside of their own home,
followed by neonatal deaths.
So, there is discontinuum, and the
asbestos body is, according to our technique,
and the technique, of course, was light
microscopy and the certain volume of tissue
waves analyzed according to a certain
technique. So, the asbestos body is a rough
index of "exposure," and, of course, there's all
these other embellishments, meaning that there
are variables and confounders and so on and so
forth.
Q. Did you find asbestos bodies in the
cases of neonatal deaths?
A. No.
Q. Did you find asbestos bodies in the
case of the white-collar housewife who never
worked outside the home?
A. Approximately 30 percent of these women
had what appeared to be asbestosbodies in their
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tissues. They tended to occur numerically,
according to our protocol, maybe one or two
asbestos bodies per unit volume of tissue.
Q. Did you find asbestos bodies in
white-collar workers?
A. Approximately 40, 45 percent. Again,
there were very few, maybe one or two or three.
Q. Have you reached any conclusions
from this body data that you just summarized?
MR. BROWNSON: Again, I'll object to
the form of the question as vague, but go
ahead.
A. What have I concluded? I have concluded
that the asbestos body is a gross index of
exposure to mineral fiber, that shipyard workers
and construction workers appear to be exposed to
higher levels of fiber than others in the
working populations in the United States.
Whether or not risk can be determined on the
basis of asbestos bodies is more problematic and
I'm not sure it's possible.
Q. If you do not find asbestos bodies
in the lungs of the person who has died, can you
then conclude that that person did not have an
asbestos-related disease?
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A. You' re talking to me as a mineralogist,
not as a pathologist?
Q. Based on your experience, either in
mineralogy or if you wish to opine on pathology,
let me know.
MR. BROWNSON: I'll state for the
record that we're not going to opine about
pathology because he's not a pathologist.
Q. Well, limited to mineralogy, if
you're comfortable to, then?
A. Sure.
Q. Do you know the question, sir?
A. Not only do I know the question, I know
the answer.
Q. May I have the answer, please?
A. As you know, there is a spectrum of
"asbestos diseases." The mesotheliomas,
meaning the sites, the multiple sites in which
mesothelioma occurs, lung cancer, and cancer of
other sites, and, of course, asbestosis.
Now, there are some who believe that
the asbestos body is a necessary marker for each
and every one of those diseases. I, personally,
believe that one should look for uncoated fibers
rather than asbestos bodies. In that sense, the
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aibMtos body is a more -- I guess the right
word is a more coarse index of exposure.
Q. Are you aware of any person who had mesothelioma, who, upon appropriate examination.
was not found to have had uncoated fibers in
their body?
MR. BROWNSON: I'll object to the
form of the question.
Are you talking about his
examination or --
Q. I'll start with your examination.
A. My experience?
Q. Yes, sir, your experience.
A. Have I ever found a mesothelioma without
asbestos fiber in the pulmonary tissues?
Q. Yes.
A. Yes.
Q. How many times did that happen?
A. We're talking about asbestos. This means
even below what I would anticipate as
background?
Q. Yes, sir.
A. Perhaps twice.
Q. Okay. Did you reach any conclusion
regarding the cause of the mesothelioma in those
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two cases? A. Other than unrelated to asbestos, no.
Q. Going back to your CV, on Page 3 you
state that you were a fellow with the Collegium
Ramazzini, and elected a fellow in 1983?
A. Correct.
Q. What is the Collegium Ramazzini,
sir?
A. Collegium Ramazzini was an organization
which came into being through the efforts of
Irving Selikoff and a number of other
individuals, and came about because of the
perceived time lag between research in areas
affecting occupational health, "the publication
of seminal papers," put that in quotes, in the
peer-reviewed literature and the implementation
of those data to ameliorate conditions which
might have been dangerous in the workplace.
There was a perceived need to bring
together scientists from around the world
working on these peculiar problems and to use
these new findings to better understand problems
of the workplace and to bring about change in
perceived hazards.
Q. When was the Collegium formed?
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A. You mean, when was it incorporated?
Q. Yes, sir. A. I don't really know. I'm thinking out
loud. That's a good question to ask Shelly
Samuels. Perhaps 1981, '82, something like
that. It's about that time.
Q. Have you been a member since it was
incorporated?
A. Yes.
Q. After you left Mount Sinai, which was in 1988 , is that correct. Doctor?
A. Correct.
Q. Have you kept up with Collegium Ramazzini in terms of doing anything with regard
to it?
A. No.
Q. Did you attend the conference known as the Third Wave, here in New York City?
A. No.
Q. Why not? A. I don''t know.
Q. Were you invited? A. No.
Q. Did you ask somebody if you could
go?
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A. No. Q.
Did you know that you could attend
simply by paying an admission fee?
A. That, I'm certain of. Oh, that tickles me.
Q. The general subjects discussed at
that conference appear to me, as a mere layman,
to have had some relevance to work that you have
done for many years, and I wondered, given that,
why you didn't attend.
A. I can't for the life of me think why. I
could have been out of townthat week.
When was it, anyway?
It was sometime in 1991. I really
do believe I had commitments elsewhere, as a
matter of fact.
MR. FISHER: June 1991.
Q. Doctor, I've handed you a book which
is titled: "The Third Wave Of Asbestos Disease:
Exposure To Asbestos InPlace PublicHealth
Control,H published by the New York Academy of
Sciences in 1991.
Have you ever had an occasion to
review this book?
A. To review it?
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Q. To read any of the papers in it?
MR. BROWNSON: You mean, as a peer
reviewer? Q. No, to read it.
A. Oh, have I read some papers in it?
Q. Yes, sir.
A. Yes, of course.
Q. Okay. Do you have any opinions
about the usefulness in your work of any of the
papers that were represented in here?
MR. BROWNSON: Well, again, that's a
general and vague question. I'll object
to the form of the question on that basis,
but it's a thick book.
Are those papers useful in your
work, is that the question?
MR. MILLER: I think so.
A. Well, I have to translate what you mean by
"useful."
Do I occasionally cite papers that
are in that volume? Yes.
Q. Okay. Which papers, if you recall,
have you cited in this volume?
A. Do you want to go through the volume with
me?
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Q. Yes, sir, just the table of contents
would be sufficient, I think.
A. You want to know what I've read in here or
what I cited, what I've quoted?
MR. BROWNSON: Well, let's get the
question in mind.
THE WITNESS: Well, let's start
with --
MR. BROWNSON: Whoa, let's hear the
question. Are you asking him papers he has
cited in his publications?
MR. MILLER: Yes.
MR. BROWNSON: That seems to be the
question.
Q. That's correct.
A. Which of the papers that I, myself, have
read that I've cited and found of interest?
Well, the very first paper, the one
by Selikoff and Seidman on "Asbestos-associated
deaths among insulation workers in the United
States and Canada," that has the most recent
data on the national survey. Very interesting.
I've read the paper on the
"Carcinogenicity of chrysotile asbestos:
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evidence from cohort studies." A paper by
Decent. That has some interesting chrysotile
commentary in it and other interesting features.
The third paper I have quoted, this
is the paper by Kohyama and Suzuki, "The
analysis of asbestos fibers in lung parenchyma."
That's also of interest.
I've also read the fourth paper,
which is Ron Dodson's paper, and colleagues, "A
comparison of asbestos burden in lung parenchyma."
I've read this paper, the fifth
paper, as well, which is Baker's paper, on the
"Limitations in drawing etiologic inferences
based on measurement of asbestos fibers from
lung tissue." I've read that, too.
MR. BROWNSON: Wait a minute. Now,
you're asking as to papers you read. I
think the question was papers you cited.
MR. MILLER: I appreciate the way
the doctor is doing it. It's making time
shorter. I wish he'd continue that way.
MR. BROWNSON: The only thing I was
going to say is let's continue with the
answer to the question of papers you've
cited in your publications, and if you want
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to note whether you've read it from
something or not, I think you can do that,
that's fine. I don't want to get the
answer confused with the question.
MR. MILLER: I appreciate that the doctor
is being very clear, as he always is in
his answers, about which he has read, and
in addition to which of those he has read,
he has also cited, and I wish he would
continue that same way. I think that's
good.
MR. BROWNSON: Okay, fine. I want
to make clear as to what we're doing
here.
A. I will state which papers I've read and,
for example, the Kohyama paper, the Dodson
paper, the Baker paper are referenced in the
manuscript that I'm generating now on asbestos
fibers in 83 cases in the United States.
The Dose-Response papers,
Nicholson's papers on "Comparative dose-response
relationships with asbestos fiber types," I've
read that. I haven't quoted anywhere as yet.
I've read Murray Finkelstein's paper
on exposure response relationship with
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DMOtbe1 ioma among the cement workers in
Canada.
I've read the paper on the
radiographic abnormalities by Christine
Oliver and colleagues. I've read that but I
haven't quoted it anywhere*.
I've read the paper by Sluis-Cremer,
"Asbestos disease at low exposure after long
residence times." I haven't quoted that anywhere,
either.
I've read Friedrich Potts' paper,
"The neoplastic findings in experimental
asbestos studies," which is very interesting,
but I haven't used that as yet.
I've read Suzuki's paper of
"Comparability of mesothelioma in humans and in
experimental animal studies." I have not quoted
that, as well.
The experimental studies, I've read
the study of Raymond Begin, "The asbestosis in
sheep."
I've read Marie-Claude Jaurand's
papers, "The observations of carcinogenicity of
asbestos fibers."
I have read Landrigan's paper on the
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children at risk. I haven't quoted that anywhere
as yet.
I've read Mancuso's paper on
"Mesotheliomas among railroad workers."
I've read Jacques Ruttner's paper,
"Mesothelioma in Swiss railroad workers."
I've read this paper by Seidman and
Selikoff. Lou Joubert as a single part, I don't
know why.
"Mortality experience and family
contacts of asbestos factory workers," I've read
that, as well.
I read "Asbestos in lungs of
children." This, again, comes out of Ron
Dodson's group. It's an interesting paper. I
don't know what it means, though.
I've read the paper, "Pleural
plaques in the general population," Hillerdal's
paper. I haven't used that in any paper.
I've read the paper by Boutin, and
"Pleural effects of environmental asbestos
pollution in Corsica."
I've read David Lilienfeld's paper,
"Asbestos-associated pleural mesothelioma in
school teachers." I haven't used that in
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anything.
I've read Jack Harington's paper on
"The carcinogenicity of chrysotile asbestos." I've read John Davis' paper on
"Variations in the carcinogenicity of
tremolite." I've read Bruce Case's paper on
"Health effects of tremolite: Now and in the
future." I haven't used that, as well. I have read "Asbestos exposure from
activities in buildings with asbestos-containing
materials," by Keyes and Jim Millette.
I read the "Mineral fiber contents
of lung tissue in patients with environmental
exposures." That's the paper by Roggli and Longo. I haven't used those.
I read the paper by Christine Oliver
on maintenance workers.
I've read the paper by Levin and
Selikoff on radiological abnormalities.
I think I reviewed John Balmes'
paper. The original paper was submitted to the
Peer Review Journal, so I did not review it here.
I read Henry Anderson's paper on
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"Mesothelioma among employees with likely
contact with in-place asbestos-containing
building materials." I have just about looked at every
paper in here. I mean, whether I've read them
all in detail or not, is more problematic, but
those are the ones that I've read or looked
over.
Q. Thank you. Doctor.
Do you have a general opinion about
the breadth or scope of the people who submitted
papers that are in the Third Wave?
MR. BROWNSON: I'll object to the
form of the question.
What do you mean, "the breadth or
scope of the people"?
Q. Do you believe that the people who
submitted papers are all plaintiff-oriented
experts in asbestos-oriented litigation?
A. I have no idea.
Q. Do you respect the scientific work
of some, at least, of the people who submitted
papers in the Third Wave?
A. Sure.
Q. Is it fair to say that you respect
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the scientific work of those people vhoss papers
you have just said you cited in your own work?
A. That doesn't necessarily follow. q. who are the people whom you just
mentioned as having read, and in some cases
cited whose work you respect?
If you would like the book back to
look at the index again.
MR. BROWNSON: I'm going to object
to the form of the question. If you're
asking him does he -- first of all, I
object to the form of the use of the term
"respect."
Secondly, when you say "respect the
work," you're not limiting it to the work
which comprises those papers. So I think
the form of the question is objectionable.
That being said, you can attempt to
answer the question.
A. Well, one of my concerns is that it's --
when you say "respect," there are words written
on Page 1, accepts or rejects. It's one on Page 1
that reads the data, how old the data derived
are, these data, robust data, are these data
weak data, are there conclusions in keeping with
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the data presented, is this a contribution to
the field, is it just a rehash of old materials,
and so on and so forth. I mean, you could read
papers and read them different ways.
Do I respect the people? I mean,
some people I respect, some I don't respect, but
you have to take each individual contribution
and read it with a fresh eye.
Selikoff -- I cannot think of any
group in the world that has reproduced the work
that Selikoff, Hammond, Seidman, et al.,
produced in terms of the insulation workers.
They stand on -- without peer, or peers, sure,
wonderful work, great work.
Q. Are there any other papers in this
Third Wave that you would say are contributions
to the field?
A. Well, I happen to like the paper by
Kohyama and Suzuki, because it is the first work
to come out of Mount Sinai that shows that
insulation workers were exposed to crocidolite.
I thought that was first-rate.
There are others like that. I've
considered John Harington -- Jack Harington as
one of the more brilliant and insightful
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investigators in the field. I mean, there are
lots of people out there.
Q. Doctor, are you familiar with a book
called the "The Identification and Control of
Environmental & Occupation Diseases, Asbestos
and Cancer," edited by Ira Mehlman and Arthur
Upton . I'm showing you the book now.
A. No. Q. Do you know anything at all about
this book?
A. No. Can I look at it?
Q. Certainly.
A. Oh, my goodness, this has finally come
out. Isn't that something. This was in the
works for about 10 years.
Q. Are you familiar with some of the
papers in that on a pre-publication basis?
You're looking at a picture of Dr. Selikoff.
don't mean to interrupt you.
A. Quite a guy.
I'm sorry, what was that question
pending?
Q. Are you familiar with any of the
papers that appeared in this book on a
pre-publication basis?
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A. No.
MR. MILLER: Off the record. (Whereupon a discussion is held off
the record.) Q. Dr. Langer, moving on with your CV,
reference Page 5, that you worked with the EPA in
the Orange, Purple and Blue Books. Did you have any connection with the
Green Book? A. No. Neither Green Book -- either Green
Book. Q.
I am speaking about what I believe
you had called the second Green Book.
A. 1990? Q. Yes, sir.
A. No.
Q. Okay. Why not?
A. Why not what?
Q. Why didn't you havea connection
with the Green Book, given that you had a
connection with three prior rainbow books that
in some ways led up to it?
A. No oneasked me.
Q. How would you describe your
contribution or your status with the three prior
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books as the peer-reviewing consultants or what
would you use for either one of them?
A. Both, basically.
Q. Each of the three books?
A. Each of the three books.
The Orange Book, I wrote a section
in one of the appendices with Arthur Rohl and
Ann Weill (phonetic). Arthur Rohl and I were at
Mount Sinai at the time, Ann Weill at the
University of Maryland. And we wrote on the
identification and characterization of asbestos
in bulk asbestos-containing products found in
buildings. We wrote that section.
I also reviewed part of the document
which meant read over certain sections, and I
was asked in the Purple Book, in the Blue Book,
I was one of the committee members when we put
these documents together.
Q. On Page 9 of your CV, under the
heading "Consultants," it refers to you being a
consultant for Safe Building Alliance, asbestos in
buildings, 1987.
Was that the symposium held at
Harvard University, sir?
A. No.
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Q. please?
What was that consulting about.
A. I met with John Welch, in Washington.
John Welch, he. was the president of the Safe
Building Alliance, and we discussed certain
problems that they perceived with
asbestos-containing materials in buildings.
Q. Did you write any document for the
SBA?
A. No.
Q. Do you know if your views were
reflected in any SBA publication?
A. There probably are, but when you say my
views, they are not so unique as to be easily
recognizable as having my imprimatur;
many people think the way I do, concerning
asbestos products in buildings.
Q. Okay. Who were some of those people
whom you would say think the way you do
concerning asbestos products in buildings?
MR. BROWNSON: Now, there's a vague
and general question, but I'm assuming it
will lead to --
MR. MILLER: Assuming I'm responding
exactly to what he said.
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MR. BROWNSON: I'm sure you are.
I'm objecting to the form of the question,
and we'll proceed from there.
THE WITNESS: I'm sorry?
MR. BROWNSON: Go ahead, proceed,
and tell us who the people are who think
as you do.
A. As an example, the generic paper which
appeared in Science; Mossman, Gee, Corn, Bignon,
others like that. Q. Can you think of the names of any
others at this time?
A. Who, what, who think the way I do?
Q. Yes, sir.
MR. BROWNSON: I assume you're
limiting the question to researchers in
the field and not men on the street?
MR. MILLER: I'm limiting it to any
way that Dr. Langer meant it, though I
assume he meant the researchers in the
field as opposed to the average dumb
lawyer on the street.
A. Not so dumb.
Those in the field, if we were to
approach this problem as it should be
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approached, the Mossman article, or articles,
because there was another article which appeared
in, I guess, the New England Journal of
Medicine, Mossman & Gee, they are responsible
for bringing to the United States that which
already existed elsewhere in other countries.
Q. That is what, sir?
A. That is that low-level exposure to mineral
fiber, such as fibers in buildings, represents a
very small risk of developing the asbestos
diseases.
Now, when we talk about the
individuals who would support this, I would
think that the following individuals in, let's
say Europe, Richard Doll, Julian Peto, Chris
Whamel, Allan Gibbs, Jean Bignon, Molly
Newhouse, Lorenzo Simonato.
i served on a committee with John
Gillson, and of course, Gillson passed
away, for the regenerated document in Hanover,
Germany, for the internationalprogram for
chemical safety on asbestosfibers inthe
ambient environment, and presented really a
European view of the importance, or lack of
importance of fibers in the ambient
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environment.
There are a number of others in
Europe. The general consensus among the
scientists working in the field was that the
focus on asbestos in buildings in North America
was misplaced. The others who also feel this
way presented their views for the Ontario Royal
Commission. That is part of the Dupres report
on fibers and buildings in Ontario.
Q. I'm familiar with a report that was
done at the Ontario Royal Commission. I am not
used to the Dupres report.
Is there only one Ontario report?
A. Yes. Stefan Dupres is the senior
committee member.
Q. Okay.
A. Stefan.
MR. BROWNSON: Be careful, try not
to talk when Jonathan's talking because
our reporter here does not like that.
MR. MILLER: I was equally at fault.
Q. Doctor, who in the United States
feels this way, the way you just described the
certain Europeans, and now certain people who
took part in the Ontario Commission field in the
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asbestos in buildings? MR. BROWNSON: Again, I'm posing an
objection. I assume you're referring to
researchers in the field?
MR. MILLER: Of course.
A. Brook Mossman, Morton Corn, Bernard Gee,
certainly the people who work with me, Robert
Nolan, Marvin Kuschner, Richard Wilson, Jack
Spengler. A number of others have been at the
Harvard School of Public Health.
Robert Sawyer, I'm thinking Jerry
Kliaman as a pathologist. I believe he also
supports this view. There are others, and I
don't know why I'm blocking them, but there are
many others who support this.
Q. Doctor, we started this particular
part of the discussion by starting with your
reference to people who think the way you do
about asbestos in buildings.
Will you please tell us, summarize
for me how you feel or believe, how you think
about asbestos in buildings?
A. I don't think about it. One of my
opinions concerning asbestos in buildings is --
you want to know all of my opinions or just
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45
general, just a little brush stroke?
Q. If you could summarize your opinions
in asbestos in buildings, that would be good,
sir.
A. Asbestos in presence in buildings are in a
number of forms. Each of these forms serve some
function and purpose. Some of these functions
and purposes are very important. The presence
of asbestos-containing products in buildings
need not indicate the presence of asbestos fiber
in the air.
The levels of fibers found in
buildings, now many hundreds ofbuildings and
thousands of air samples, have demonstrated that
in the very large majority of these buildings,
the fiber levels in air, if found in air, the
fiber levels, even using the most pessimistic
risk model, represents de minimis risks to
building occupants. I think that's areasonable,
short summary.
Q. What is that de minimus risk using a
pessimistic risk model?
MR. BROWNSON: Before you answer the
question, I'm going to object, but I
just -- since we're about to take our
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break. I'll incur the wrath of Judge Ito and make a speaking objection, but what I
wanted to say, Jonathan -- MR. MILLER: -- Thank you. I'll accept
that.
MR. BROWNSON: -- Jonathan, is that you know we've identified Richard Wilson as
our expert on risk assessments in this
case and generally with respect to Chicago
buildings, so he's going to testify about
that area and I have no objection to you
getting into that a little bit with
Dr. Langer, but in terms of moving his
deposition with what he's going to testify
about, that's not the area that he's talking about in this case.
Q. I appreciate it. Doctor.
Very briefly, what is the de minimis
risk to building occupants using the most
pessimistic risk model?
A. Using the fiber levels found in the air,
it would range from one in a million plus or
minus in order of magnitude, one in 100,000, one in 10 million.
Q. By "building occupants," you mean
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passive building occupants, is that correct?
A. Yes, correctomundo.
MR. MILLER: Thank you.
Break?
MR. BROWNSON: Sure.
(Whereupon a brief recess was taken
at 11:00 a.m.)
(Whereupon the proceedings resumed
at 11:10 a.m.)
BY MR. MILLER:
Q. Doctor, on Page 36 of your CV, you
state at the very bottom that you gave an
invited seminar and lecture to the Armstrong
Defense Group in Memphis, Tennessee. That it
was called the "Asbestos Saga and the Star Wars
Trilogy: The Empire Strikes Back." It was
given in 1991.
Which, if any, asbestos tests were
part of the Armstrong Defense Group?
A. I have no idea.
Q. Okay. Did it concern vinyl asbestos
tile as opposed to other types of asbestos in
buildings, if you recall?
A. I believe so, yes.
Q. Why did you choose such a catchy
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48
name, aside from the fact that it's obviously a
catchy name, for your talk? A. I thought people might come.
It's hard
speaking in an empty room, the reverberations
are deafening. Q. What did you mean when you said,
"The Empire Strikes Back"?
A. 1991, what was the date of that
presentation?
Q. Thursday, April 25th, 1991.
A. The asbestos in buildings passion play was
being played out in North American courtrooms,
and the only group, "recognizable group," put
that in quotes, which could not embrace any of
the concepts and principles of low-level
exposures, was the group at Mount Sinai which
was then under the leadership of Phil
Landrigan. The more North Americans brought the
issue of low-level exposure and its low-level
risk to the attention of administrators in
certain government agencies, the more the Mount
Sinai group resisted.
In fact, there was some speculation
that the Mount Sinai group had not acted in the
best interests of the community by actions and
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by words. Q.
What do you mean by that, sir?
A. Just that I've spoken sentences, and it
means just that.
Q. How have they not acted in the best
interest of the community?
A. I believe that if one reads a data set,
that is how delicately I phrase this, a data
set which runs contrary to your beliefs and one
attacks ad hominem, rather than the data, that,
to me, is not serving the best interests of the
scientific community.
So if a young woman publishes a paper
which someone at Mount Sinai finds offensive
because they don't like the conclusions, one
should present data and the data will speak for
themselves, oneshould notproceed toattack
this person as anindividual, itserves no
purpose.
Q. Are you referring specifically to
the paper by Mossman, et al., and Science?
A. You can use that as an illustration.
Q. Do you believe she was attacked
ad hominem?
A. Yes.
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Q. There were certain letters that were
published in, I think Science, and maybe elsewhere, as follow-ups to the Science article, and I believe one of them was by Dr. Nicholson, at least. Are these the documents to which you were referring that she was attacked ad hominem, or
something outside that?
A. Outside that.
Q. What is there outside those
published letters that you're referring to, sir?
A. This is hearsay of people present at meetings at Mount Sinai.
Q. Who was it? Who did this attack,
according to your source of interest?
MR. BROWNSON: To the extent if you know.
A. To the extent that I know, this is all
hearsay, and it's unfair because some of the
principals are no longer living.
q. Selikoff?
By that, you're referring to Dr.
A. He was one, yes.
Q. Do you believe he was one who
unfairly attacked directly Ms. Mossman?
A. Yes.
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Q. Excuse me, Dr. Mossman.
Who else?
A. Who else what? Q. Who else attacked her, do you
believe?
A. You would have to depose Jack Harington to
find out details, since he was present at some
of these meetings.
Q. Now, when you were referring to the
empire striking back, who, in your mind, in
1991, was the empire?
A. Who constituted membership in the empire?
Q. Yes, sir.
A. Well, the center of the empire was at
Mount Sinai, of course.
Q. Do you remember anything you said at
that particular discussion in 1991 before the
Armstrong Defense Group, other than what you
have already referred to here today?
A. I don't think I mentioned this ~ well,
what I've spoken about today.
Q. What did you tell the Armstrong
Defense Group in 1991?
A. Actually, I outlined what was known in the
field, I outlined what was known in Europe, I
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outlined what was brought over to the United States, and I outlined what the perceived
response was from Mount Sinai. Q. Anything further than that that you
recall?
A. About what?
Q. What you told theArmstrong Defense
Group?
A. Well, I don't remember everything, of
course not.
Q. Okay.
A. Since it was more spontaneity than
pre-thought.
Q. I'd like to ask you a couple
questions about the projects for which you have
provided a very nice graph at the back of your
cv under "funding sources."
Starting with the most recent one first,
sir, on the last page, there's a project at the
Ecaterinburg workshop, project chrysotile.
You're co-investigator for The Asbestos Institute
and the W.R. Grace Company.
What was that all about, sir?
A. About five years ago, Favel Kogan wrote a
paper which was published -- actually a
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commentary which was published in the American
Journal of Industrial Medicine on the occurrence
of mesotheliomas in chrysotile-exposed workers
in Katerinsburg. It used to be Verdslosk, and
I'll spell Katerinsburg later.
The commentary was of interest to me
because its wording suggested there were more
mesotheliomas there than reported among the
miners and millers from Canada. I wrote to
Kogan and we communicated, and through a mutual
colleague, we were able to obtain tissue blocks
from Kogan, obtained from individuals who died
with asbestosis, and we began a joint -- and we
arranged with certain individuals in the Russian
federation, we arranged to participate jointly
in a study of tissues obtained from certain
kinds of workmen.
We are presently obtaining tissues
from individuals who succumb with mesothelioma,
which interestingly, more mesotheliomas occur
outside this region than inside the region, occur
from other areas in the Russian federation.
Q. Will this someday be a published
article?
A. It certainly will.
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Q. Do you have any tentative conclusion
you have reached based on your research thus
far?
MR. BROWNSON: Well, I'll object to
the form of the question. Again, it's broad and it's vague and requires speculation, but go ahead.
A. You just want to hear two basic
conclusions, let's say?
Q. If you reached them, sir, yes.
A. The first conclusion is that they have a
lot of the dust in their lungs, high concentrations of chrysotile, no tremolite.
Q. You're referring to dust in the lungs of these people that died from asbestosis or --
A. Yes.
Q. Are there any who died from mesothelioma?
A. Not yet.
Q. Okay. Is the Katerinsburg a
chrysotile mine?
A. Katerinsburg is themajor city which is
east of the Ural Mountains. It is the jumping-
off point to a small town of approximately 60
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kiloaeters to the northeast called Asbestos,
like asbestos, Quebec. Basically, this is
asbestos and this is where the largest pits are
located. Q.
Have you concluded that the chrysotile
mine in Asbestos does not have tremolite as a
contaminant or that, whatever word you would
like to use in it?
A. No.
Q. Do you know if it does have
tremolite in it?
A. No.
Q. Turning to the prior page that I
guess is Page 48, there's another project funded
by the W.R. Grace Company, titled "Asbestos in
the lungs of exposed workers," starting March of
'93, going to the present.
What is that project all about?
A. That's part of the Katerinsburg project.
MR. MILLER: Exhibit 2, please.
(P-2, Expert witness disclosure,
received and marked for Identification.)
BY MR. MILLER:
Q. Doctor, I've had marked as Exhibit
2, an expert witness disclosure in this case
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given by the defendant Asbestospray. I believe
this is similar to some expert witness
disclosures I have seen in the past, and I hope
I will not have to spend too much time on it.
Have you seen this before, sir?
A. Yes.
Q. Can you tell me if, in the last five
years, you can think of any particular area of
your testimony where your views have been
significantly changed?
MR. BROWNSON: I'm going to object
to the form of the question, but are you
speaking here to see if we can at least --
so I understand, your testimony expressed
in this disclosure?
MR. MILLER: Exactly.
MR. BROWNSON: If you look at the
areas in that disclosure, the question
is: Have your views changed in any of
those areas in the last five years, as I
understand it?
MR. MILLER: Yes.
MR. BROWNSON: I still object to the
question, but go ahead.
A. You want to do this line by line or just
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topic by topic? Let's do it topic by topic.
I see
you are shaking your head to the topic by
topic.
The testimony in the first
paragraph, he will testify there's been a great
deal of confusion in the medical and scientific
literature concerning the term "asbestos" and
that it is often improperly defined.
Well, there seems to be a little
less confusion now since OSHA has taken cleavage
fragments out of the asbestos standard. Okay.
Q. What is the nature of fiber release
from asbestos fireproofing?
A. That's rather broad and generic.
Q. Well, that's something that you're
going to testify to in exactly those words. I'm
interested in what you're going to say about it.
A. It depends on whether Mr. Brownson asks me.
Q. Tell the jury, in your opinion,
whether or not fibers are released in
asbestos-containing fireproofing, and if so,
under what circumstances?
A. I have not examined allasbestos-containing
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fireproofin?. Asbestos-containing fireproofings
have a range of compositions, asbestos-containing
fireproofings have a range of agents that are
called binders.
I, personally, have examined many
structures which were sprayed with a cementitious
material. For example, Asbestospray formulation
T, which is an amosite-containing material.
These applications have existed in construction
for the past 30 years or more. The buildings
that have vibrated with mechanical stresses have
been subject to large noises, have had bombs go
off in their basements -- no, that's not
asbestos, I'm thinking of the World Trade
Center.
Materials are in place. The fiber
levels found in the area of these structures are
either not detectable below a certain volume, or
when fibers are detected, they suggest, given
various risk models available to us, the risks
vary between one in 100,000 and one in a
million, that these fireproofings function
according to their specifications, and that they
present a little or no risk to occupants of
buildings.
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Q. When you say "occupants of
buildings," you were referring, again, to what
you call passive occupants? A. Or Cl occupants, as we referred to them in
the AGI report.
Q. Do you have any opinion about risk
to other categories of building occupants other
than the Cl categories as referred to in the
HEIAR report?
MR. BROWNSON: Well, again. I'll
object to the question in that Dr. Langer
is not going to testify for Asbestospray
in this case about risk, as he previously
stated, but go ahead and answer.
A. Again, it's a broad question because there
are any number of activities that could be
performed inside of a building or by maintenance
workers. If a material is sprayed on a surface,
on a decking, on cementitious material that's
behind a suspended ceiling, would people who
sweep the floors be at any greater risk than
people sitting in a room? The fiber levels
would be identical and the risks would be
identical, as well. The risks would approach
zero. Others might change lightbulbs. If the
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lightbulb is below the suspending tile
ceilings, they constitute zero risk. If there's
someone who pulls cables above a suspended tile ceiling, it depends on the integrity of the
material whether or not there are any activities
that have dislodged the spraying application.
So there are any number of scenarios.
I mean, I'm not saying it approaches an infinite
number, but N is a very large number, and one
has to specify a kind of worker and an activity
and the circumstances under which this person
performs these activities and the nature of the
installation of which these are carried out. I
mean, there's no way to really do it other than
with some specificity associated with them.
Q. One of the suggestions or
recommendations of the HEIAR was that research
should be accomplished on theseother than Cl
building occupants.
Do you believe that that research or
any has been done, sir?
A. Yes, a great deal of it's been done. Not
all of it, but a great deal.
Q. Would you please cite for me certain
articles or other reports on which you rely,
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particularly for the conclusions you just gave
about people who sweep floors are not at
different levels, people who change lightbulbs may not be at different levels, et cetera?
A. The HEI had a meeting in Boston, the proceedings of which was published in Applied
Occupational Environmental Hygiene in November
of 1994. There's a number of interesting papers
in here which I am now reading.
Q. That means you have not read them
yet, sir?
A. No, I haven't finished. Q. Okay.
A. I know this is hard to believe, but it's true.
Q. Okay.
A. The title of the conference was called
"Proceedings of Operations and Maintenance
Programs in buildings containing asbestos,"
subtitled"Workshop organized by the Health
Effects Institute in asbestos research."
Editors are Morton Lippmann, John Samet, and
Rashi Shaikh.
There's a number of papers that were
presented, interesting papers, with data
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touching on the issues of maintenance workers in
buildings, and whether or not they are exposed
to fiber and various settings.
Q. Are those your own handwritten notes
in that volume of the journal, sir?
A. Yes.
Q. Can you tell me which articles, I'm
sorry?
A. Yes.
Q. I mean, can you tell me which
articles you have read, thus far, that you found
to be a particular interest or value to you and
your opinions?
A. You mean,the articlespertaining to
asbestos? There are some very interesting ones
in here on mortality amongconstruction laborers,
if you're interested.
Q. I mean only asbestos?
A. Onlyasbestos, thus far, Millette's paper
on TEM, transmission electron microscopy,
"Analysis of asbestos structures," and his
microvac technique and so on.
An interesting paper by Rashi Shaikh
and colleagues on fiber levels found in
buildings with maintenance activities.
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There are a number of others that I started to read but put down because I haven't
had time to really focus on these things. Q. What is your opinion about the value
of surface dust sampling for asbestos?
A. Not much. Q. Why is that, sir?
A. Well, it doesn't tell you anything,
basically. It tells you that you have asbestos
in some settled dust. It doesn't tell you the
nature of the original size distribution. It
doesn't tell you anything about the respirability of the fibers. It doesn't tell you about the
nature of the fibers in the air. It tells you
that it used to be in place A, it's now in place
B, and we find it there, and people generate
these very large numbers which are generally
misleading, and they have very little biological relevance.
Q. When you speak about people generating
these large numbers, they are generally in terms of structures per cubic something? A. Area.
Q. Some area?
A. Correct.
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Q. Do you know, if the information were
analyzed in terms of weight, if that would have
more relevance for the issues you're concerned
with?
A. Worse.
Q. Why?
A. Weight doesn't tell you anything except
weight.
Let's say that -- let's use an
example. A fiber has just fallen off the
ceiling. The fiber dimension is 10 microns in
diameter, and it isa millimeter in length, and
the weight is 100 micrograms. Let's say it
falls to this table, someone comes in and does a
dust assay, and say there's 100 micrograms of
dust on this table. My question is 100
micrograms of what? What arethe dimensions of
the fibers?
Now, I just told you it was a single
fiber, but let's say we were to calculate on the
basis of a preparation technique which alters
the size distribution. We grind it up. We
sonicate it. We do a lot -- we manipulate this,
so instead of that one particle, we now have
100,000, and that 100 micrograms now represents
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100,000 fibers.
Now, that carries a different
meaning to you in terms of hazard and risk than
that single fiber. So you have to know
something about size distribution in order to
come to some understanding of risk.
So mass is awful, don't bother using
that because then mass may be, in part, the
matrix materials, it could be mixed with vitreous
fiber, could be mixed with a cementing agent,
Portland Cement, it could be mixed with bentonite
clay, whatever.
So mass means nothing. Well, it
means something, but it doesn't tell you very
much, and when one looks at the microvac
techniques and sonication and the alteration of
size distribution, it is misleading. So it's
worse than telling you very little, it's
actually misleading.
Q. Would your opinion regarding the utility or usefulness of surface dust sampling
vary depending upon the method by which the dust
is taken off the surface?
A. Taken off of the surface?
Q. Yes, sir, microvac as opposed to
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scraping as opposed to any of the different
techniques?
A. No, no, no, the artifact is not in
removing from the surface so much as in the
preparation of the material for examination by
electron microscopy, that's the problem.
Q. The way it is removed from the
surface is not something which would enter into
your judgment or surface dust sampling in
general?
A. I don't think -- in general, that's too
much of a problem.
MR. BROWNSON: Try to wait until the
question is done.
THE WITNESS: I know, I'm getting
antsy. I have to apologize.
Q. Would your opinion about utility of
dust sampling change if the preparation were
done by direct preparation as opposed to
indirect preparation?
A. Better.
q. Okay. How is it better?
A. A direct preparation would be a
preparation in which the material in its state
on the surface is not altered when examined by
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transmission electron microscopy; that would be
better, yes.
Q. Do you think it still has some
limits?
A. It depends on the nature of the matrix.
Q. Explain.
A. Well, do the fibers stick out? Can you
see a whole fiber? Is this particle respirable?
If a fiber is sticking out, is it coated with
some of the matrix materials?
It's more meaningful, but there are
always interesting questions that come up. I'm
not dismissing it, of course. Merely, I'm
saying if you remove it from the surface and you
prepare it so that there's no artifact introduced
in terms of size distribution, that's better.
Q. Is one of the disadvantages of
direct preparation that it could hide the
asbestos fibers in other material?
A. That's possible.
Q. Are there any other disadvantages to
direct preparation that you can think of?
A. The major disadvantage is that these
clumps are so large that you can't see
anything. So this material is broken down so
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you can see what's there. That's one of the
reasons people went into the indirect method;
that the direct method, that was just so much
junk there, you couldn't see through it.
Q. Any other disadvantages you can
think of at this time, sir?
A. Well, no. There are probably others X
just can't think of.
Q. Have you, by any chance, read the
book by Millette called "Settled Asbestos Dust
Sampling & Analysis"?
A. No, I have not.
Jim Millette is such a handsome
devil.
Q. Well, I've seen pictures of you,
Doctor, that flatter you.
A. Aren't you nice to saythat.
Q. Doctor, you said that at one point
recently in your testimony that you had examined
various types of cementitious fireproofing.
What other types of fireproofing,
cementitious or not, have you, personally,
examined?
MR. BROWNSON: I'm going to not
interpose an objection to the question,
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per se, but just state that Dr. Langer is
here to talk about his examination of
various asbestos fireproofings. While
other examinations he may have done will
be of interest, and you're certainly
. welcome to question him, we're not going
to ask him at trial.
MR. MILLER: Thank you.
A. I don't know whether I'm going to give
this back to you. It's very interesting. I'm
only kidding.
Q. Doctor, it only costs about $70 and
some of the money goes --
A. Only $70, well...
Q. What is your current -- while we're
speaking, what is your fee for expert
consultation?
A. Well, it depends. $225 an hour, general
background work; $275 an hour for a deposition;
and usually $325 an hour for trial testimony.
Q. It has not gone up in a number of
years?
A. Indeed, it is the same.
Q. Very good. I'm going to be --
A. How good that is, I don't know.
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Q. What other asbestos-containing
fireproofings have you, personally, examined?
HR. BROWNSON: Other than asbestos?
A. You mean, over my career?
Q. Yes, sir.
A. Holy mackerel, I think about all of them.
I've looked at specimens of Kafco blaze shield, I've looked at a number of products from W.R.
Grace, I've looked at material -- this is just
fireproofing, correct?
Q. Yes, sir.
A. I would say on the average at Mount Sinai
we would examine a half a dozen to a dozen
specimens submitted to the laboratory weekly
over a period of several years.
Q. Using the EPA definition of friability, do you believe that allthe
fireproofings which you've examined, including
those specifically which you named here, are
friable?
A. You mean, the definition, crumbled or
easily crumbled?
Q. By hand pressure.
A. By hand pressure?
Q. Yes, sir.
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A. When you say all of the products, you mean
blaze shield?
Q. Let's start there.
Is that friable?
A. I have to have the specimen in front of
me.
Q. How about the Asbestospray type T?
A. That's fairly cementitious, especially
when tamped.
Q. Would you say that is not friable?
A. I said generally not friable.
Q. How about Grace Monokote-3?
A. I would have to see it in front of me, and
I just don't recall. I've looked at them, but
when you talk about friability, that requires a
specimen in front of you.
Q. Are there any others you can recall
now without having a specimen in front of you?
A. Actually --
MR. BROWNSON: The question is: Can
you recall if any others are friable or can
you recall seeing any others?
Q. If any others are friable?
A. I'm sorry, now I'm confused.
Q. Can you recall if any other
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fireproofings that you have had personal experience in, are, in your opinion, friable?
You understand the EPA definition?
A. Well, there were some that I examined at
Mount Sinai that under the definitions would be
considered friable. Q. Do you recall the names?
A. I can't recall.
Q. Okay.
A. These are just fireproofings, we're not
talking about decorative?
Q. Just fireproofings, sir. A. Okay.
Q. You gave a definition or your opinion of risk a little while ago, and that
definition or your opinion of risk was based
upon air levels, is that correct, sir?
A. In part.
Q. Was it based on anything other than
air levels?
A. Well, of course.
Q. What?
A. Well, the air level is only one aspect of
risk assessment, the other involves the
response. There's a dose and there's a
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response, and the response models, which are
used by various agencies. Q. Was your opinion regarding risk
based at all upon the actual condition of any of
the asbestos-containing materials in any of the
plaintiff's buildings?
A. I don't understand the question, I'm
sorry.
Q. Do you know the actual condition
today of any of the asbestos-containing
materials in any of the plaintiff's buildings?
MR. BROWNSON: We're speaking of the
Chicago buildings?
Q. Yes, this case.
A. I've seen photographs of the application
of Asbestospray in some of thesebuildings'
photographs.
Q. Aside from the photographs, do you
have any additional information regarding the
actual condition of any of the
asbestos-containing materials in these
buildings?
A. Yes.
Q. What is that?
A. Some of the documents that I've read of
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the individuals who inspected these facilities.
Q. Aside from the inspection reports
and the photographs, do you have any further information regarding the actual condition of
the materials in the buildings today?
A. You mean, like a videotape or something?
Q. For example, fireproofing?
A. Fireproofing, no, I don't believe so. Q. Do you have some knowledge about the
types of asbestos-containing materials in the
Chicago and Evanston plaintiffs' buildings other
than fireproofing?
A. Yes.
Q. What is that, what type?
A. As you know, I'm also representing Conwed,
a manufacturer of a ceiling tile. Those
materials are present in some of the buildings.
Q. I will not be asking you questions
specifically about Conwed.
Other than the Conwed specific information, do you have any additional
information?
A. Other than that which I read, meaning that
other experts examine materials and carry out
what is commonly referred to as product ID;
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other than that, I have no firsthand
information, correct.
Q. Did the information you received
through photographs you mentioned and the
inspection reports that you mentioned form some
part of the basis for your opinion about risk in
the buildings?
A. In part.
Q. Was the information that you saw and
you've described, thus far, namely, the
photographs and the inspection reports, aside
from the Conwed, which I'm not going to ask you
about, but other stuff you saw regarding our
building, was that Asbestospray-specific, to the
best of your knowledge; in other words, it was
in buildings where Asbestospray products were
alleged to have been?
A. Yes, that's right. There are three
buildings there we're talking about, correct.
Q. in what document could I find the
most up-to-date statement that the EPA's published
on asbestos in buildings, if there is such one
document?
A. Yes, there is. What is that document
called? You say asbestos inbuildings or
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asbestos in schools?
Q. I said in buildings.
A. Asbestos, I have two recent EPA documents,
one published in June of '91, which is called
"Asbestos in schools, evaluation of asbestos
hazard emergency response act, a summary report;
and in January of '92, a U.S. EPA document
called "Communicating aboutrisk, EPA,
and
asbestos in schools."
Q. Is there anything more recent than
those two documents, more generally referring to
buildings?
A. You know, there is one and I'vebeen
trying to get it and Ican't get it.
Q. What is that, sir?
A. There is a most recent document, it
postdates the second Green book, andit
postdates many of the statementsthat you hear
about it's safer to leave it in place than to
take it out, so on and so forth.
There is another document which I
have not been able to get as yet, butit's my
understanding that it's floating around at the
Environmental Protection Agency. It might be a
draft of a document rather than a final report.
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Q. Can you give any further or a more clear description of what the document might be?
A. I wish I could. Q. Does it concern generally asbestos
in buildings, as you understand it?
A. That's my understanding, yes.
Q. It has not been published yet in any
form, to the best of your knowledge?
A. Apparently so, correct.
Q. Doctor, do you have any AHERA
certifications?
A. No.
Q. Do you have any certification from
any Illinois Department of Public Health or
other governing bodies concerning asbestos?
A. No.
Q. You are not a certified industrial
hygienist, is that correct?
A. Correct.
Q. Would you call yourself an
industrial hygienist?
A. No.
Q. Okay. Given that, would you please, in some summary form, tell me why you believe
you have the expertise to testify as stated in
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Exhibit 2 concerning proper responses to
asbestos in buildings including, but not limited
to, the necessity of removal, the advisability
of removal, and operations and maintenance of
asbestos in buildings?
MR. BROWNSON: Well, I object to the
form of the question, but go ahead, you
can answer.
A. I object to the form of that question,
myself.
Do you want to ask part of it, what
makes me think I can advise people about
anything, is what you're asking me?
Q. Advise people specifically, and
what, if anything, should they do about asbestos
in buildings?
A. I'm one of those people who believes that
advice of an expert is best when from an expert
who knows a lot about a subject area. I'm not
saying that an industrial hygienist -- a
hygienist tells you one part of the problem, but
expertise requires some understanding of what is
asbestos; what are the fiber types; what are the
risks associated with the different fiber types;
what is known about those responses; what is
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known about fiber levels in the industry; what
are the implications of cigarette smoking in
asbestos risk; how much fiber is in the air; how
do we ever get these models that the EPA uses.
And in their hazard analysis, what do these
models mean; what is it based on; what is
epidemiological studies; what are the fiber
levels; how do you measure the fiber levels;
what do you use in electron microscopy; why do
you use an electron microscope rather than a
light microscope; what are the fiber levels;
what are your interpretations and meaning of
these data.
Now, if you find me an industrial
hygienist in the United States who can answer
all of those questions and give you a reasonable
dissertation on each of these, then I will back
out of the litigation, meaning, that there's
someone out there who really knows something.
On the other hand, one doesn't have
to be an industrial hygienist to gauge
situations where there may or may not be a
risk. In fact, one doesn't need all the tickets
in certain instances, but you have to have some
broad-based knowledge.
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Q. Do you believe that a medical doctor
would be more qualified than you with your qualifications to give advice to a building
owner about what, if anything, they should do
about asbestos in buildings? MR. BROWNSON: I'll object.
A. It depends on the doctor.
Q. Are there some doctors whose names come to mind who you think would be at least as
well qualified as you to give advice to building
owners regarding asbestos in buildings?
MR. VALENZA: Objection.
MR. BROWNSON: I'm going to object
to the form of the question, and I'll tell
you specifically why, which may help you
understand the objection.
Dr. Langer will be asked in this
case to give testimony as to what
response, perhaps, will be asked, what
response would be appropriate in the three
Chicago buildings which are at issue with
respect to Asbestospray, and I don't know
what doctors may or may not have reviewed
the data he's reviewed with respect to
those buildings, and therefore, I think
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the question is objectionable on that
ground. MR. MILLER: I appreciate that. Let
me change my question slightly.
Q. If a medical doctor were to review
the same data that you reviewed regarding the
three Chicago buildings, is there any particular
medical doctor whose name comes to mind who you
think would be at least as well qualified as you
to give advice to building owners regarding
what, if anything, to do about the asbestos in
those buildings?
A. Very, very complex and compound question.
First, you're asking: Is there a
medical doctor who's as qualified as I am in my
field? Nay, not so.
Are there medical doctors who know a
great deal more in pulmonology than I do? Sure.
Are there doctors who are certified
to be readers who can read chest x-rays? Sure.
Sure, there are many physicians who are better
qualified in the medical field.
Now, talking overall asbestos field,
that's more problematic now.
Are there physicians who could read
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the data and come to the sane conclusion as I would
cone to, qualifications aside? Yes, there are
physicians who would come to the sanegeneral
conclusion as I.
Q. When you refer to your field, were
you referring to what you subsequently call the
overall asbestos field?
A. I think so, yes.
Q. Who, in your field, namely, the
overall asbestos field, do you believe is as
qualified or perhaps even morequalified than
you to give advice to a building owner if
they were to review the same information you
were to review? And this is advice concerning
what, if anything, to do about asbestos in
buildings.
A. Well, I don't normally give advice to
people on what you do in buildings. All I could
do is present the problem to them and they make
their own decisions.
That's not the question. The
question is: Are there individuals who -- well,
there are people who do it to a greater or
lesser extent.
q. Who, among the people who do it in
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your field, do you believe, if any, are as
qualified as you or more so to give either the
advice or the information that you give to
people? A. Well, there are a number of people who
give the information to building owners. I
think Roger Morris, as an architect, is very
well qualified to render advice and opinions to
building owners and operators. I think that
Morton Corn, who is an industrial hygienist or
engineer, is qualified to render advice to
various people. I think Richard Lee, who's a
physicist, is qualified to render opinions and
advice to people.
The people have slightly different
areas of focus in slightly different percentages
in areas of expertise. I mean, there are lots
of people who are not physicians, like myself,
who you can categorize as basic scientists, who
give advice, and the advice comes from a
slightly different perspective.
Are they as qualified in my field?
I don't think so. In their field, yes, they're
as qualified as one could be qualified, yeah.
Q. It's fair to call your field, then,
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that of a basic scientist, is that right?
A. That's fair enough.
Q. Who else, specifically in your
field, would you believe is a basic scientist
who is as qualified as you, or perhaps more so,
to give advice to a building owner regarding
what should be done, if anything, about asbestos
in buildings?
MR. BROWNSON: I'll interpose the
same objection, but again, the original
question was based upon the data he's
reviewed here?
MR. MILLER: Right, and I will amend
it.
Q. To me, if they were able to look at
the same data you looked at, who do you think.
as a basic scientist, would be as qualified as
you?
A. Well, Richard Wilson.
Q. Anyone else whose name comes to
mind. sir?
A. I'm thinking.
Q. I'm sorry.
A. Maybe Richard Lee, Robert Nolan.
You're talking just about basic
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scientists now?
Q. Yes.
A. Who are in the field, who have a feeling
for all these data and what they mean?
Q. Yes.
A. Malcolm Ross.
Q. Where is Malcolm Ross, sir?
A. He is just retired from the United States
Geological Survey. He's in Virginia.
Q. To your knowledge, is he now in the
business or expertise of giving advice to building
owners about asbestos?
A. No.
Q. I interrupted you, I'm sorry.
Were there any other names that you
had in mind?
A. I'm thinking. Well, there are others who
are epidemiologists who are interested in risk
analysis. There's Janet Hugh and Philip
Enterline of Pittsburgh. I'm sure there are many
others, but I just can't recall.
MR. MILLER: Off the record.
(Whereupon a discussion is held off
the record.)
(Whereupon a luncheon recess was taken
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at 12:00 p.m.) (Whereupon the proceedings resumed
at 12:55 p.m.) *
*
*
AFTERNOON SESSION
DIRECT EXAMINATION BY MR. MILLER CONTINUED: Q. Doctor, before lunch, you referred
to the physicist Richard Lee. Is that the same Dr. Lee who does
product identification in this case, he's in Pittsburgh, at ETC, or are you referring to somebody else? A. I'm referring to the Richard Lee who is the president of the Lee, I guess, and Associates in Monroeville, Pennsylvania.
Q. That's the same person? A. Oh, yes.
Q. That's exactly? A. Oh, yes, yes.
Q. Thank you. You have been consulted by
Asbestospray in this case.
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Is it your understanding that they
are alleged to have an asbestos-containing
product in three schools in Chicago in this
case?
A. There are three buildings.
Q. Three buildings, that you think?
A. There are three buildings, one is the
South Shore High School, one is the Leif Ericson
School, and the other is the District 2 Offices.
Q. What is your advice to the building
owners as to what, if anything, she should do
about the asbestos-containing Asbestospray,
allegedly Asbestospray material in those
buildings?
A. No one's asked me for my advice.
Q. I'm asking you.
A. What would I advise them to do?
Q. Yes, sir, this is a hypothetical
question.
A. This is a hypothetical. Well, based on
the surveys which have been completed, based
upon the description of the utilization of the
spaces, based on a description of where this
material occurs in relation and regard to the
spaces in which people work and are educated,
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given the purpose which it serves and given the
experiences, and given the air datawhich have
been obtained in other circumstances,in other
structures, my advice would be to leave the
material in place. Q. Do you know what type of asbestos is
alleged to be in the fireproofing in those three
buildings?
A. Yes, the formulation approximates
Asbestospray T, which is an amosite-containing
formulation.
.
Q. Have you seen the formula for
Asbestospray T?
A. Pardon?
Q. Have you seen the formula for
Asbestospray T?
A.
I have.
It's been reported to me. I have
notes and it is my understanding that it is a
formulation consisting of approximately 25
percent amosite fiber,approximately 60 percent
vitreous fiber, a wool of some kind,
approximately seven and a half percent Portland
cement, and approximately seven and a half
percent bentonite clay.
Q. Do you have any reason to believe
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that any of the information that you have that
the material in any of those three buildings is
not Asbestospray T? A. Do I have information that it is not?
Q. Yes, sir. A. You mean, do I have an understanding that it is not, or do I have some impression that the assays performed are limited or provide me with
some reason to be skeptical that this is
Asbestospray, is that what you're asking me?
Q. Yes.
A. Yes. Q. What is that?
A. I have looked at some of the assays from, I guess, Dr. Longo's group, MVA -- no, excuse
me, that's not MVA -- Longo's group, and it is
the assays, as presented, are similar to the
Asbestospray formulation, but the assays have limited -- the assays described certain materials as being present in amounts which are
similar to Asbestospray products, and I'm not at
all certain that the interchanging of bentonite
and montmorillonite.
So, there are certain characteristics
of the assay which are not -- haven't fulfilled
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my own requirements for a proper product ID. I
would have preferred to have seen a bulk x-ray diffraction tracing. I would have preferred to have seen other kinds of data presented in order
to come to the conclusion this is Asbestospray
T.
Q. Thank you, Doctor. MR. BROWNSON: For the record, as
you know, Dr. Langer is not being
presented as a witness on this question,
although you might find his comments
interesting.
MR. MILLER: I always find the
doctor's comments interesting.
MR. BROWNSON: Off the record for
one moment.
(Whereupon a discussion was held off
the record.)
BY MR. MILLER:
Q. Doctor, one of the bases for your
advice to the building, I understand the three
buildings, was where the material occurs?
A. Correct.
Q. Does the material occur above some
type of suspended ceiling in these buildings?
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A. Yes, it is my understanding from the description in one of the schools in which there
is a suspended ceiling in a pool area, maybe the pool area wasn't opened and the pool area wasn't
used, but there are areas described in which the
spray product is present above a suspended tile
ceiling. I would have to check the details.
Q. Do you believe that the owner
should, even though the owner is keeping
material in place, have some sort of operations
and maintenance program with regard to that
material?
A. Well, if they're not using the room, no.
If it's above a suspended tile ceiling, other
than you might do some air sampling once in a
while under the ceiling, no, I wouldn't
recommend any extensive kind of O and M program,
no.
Q. Is any of the air sampling that you
have seen thus far, air sampling, taken above
the suspended ceiling inthesebuildings?
A. No.
Q. Do you have any information as to
how often maintenance or custodial persons in
the course of theirnormal duties must gain
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access above the suspended ceilings in these
buildings?
A. The inference drawn from the description
of what exists above the ceiling with the
existing services indicates that they would have
very little, if any, need to go above these
tiles.
Q. If they should have any need to go
above tiles, would you recommend any form of
protective clothing or other practices that they
should follow in there above the ceiling?
MR. BROWNSON: Just so we're clear
on the question, you're speaking of
buildings that he's examined and talking
about, you're not making this a more
general question?
MR. MILLER: I'm speaking about the
buildings in this case that you are
referring to that have the alleged
Asbestospray type T above some form of
suspended ceiling.
A. If it makes the workers feel better, then
they should wear some kind of respirator, an
approved respirator, dust respirator for fibrous
dust.
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Q. Do you believe there's any health-related reason as to why they should have
any type of precaution, whether it's a respirator or anything else when they go above
ceilings in these particular buildings or
buildings where there is alleged Asbestospray
type T above ceilings?
A. A health reason, specifically?
Q. Yes. A. Are they at risk, is what you're asking
me?
Q. Yes.
A. Or are you asking me are they at risk?
Q. Yes.
A. If there's a risk, it is a very small
risk. If the risk exists, it is a very small
risk.
Q. Could you quantify that risk?
A. Well, I've stated for the record that I
know of no air data taken above those tile
ceilings, so I don't know that.
You can give me another
hypothetical.
Q. Given the presentstate of your
knowledge, which includes no air data, would you
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recommend that any type of precautionary
measures be taken by custodians or maintenance
personnel if they must access above the ceiling
in the same building or buildings that we're
talking about? A. The absence of data invites extrapolation.
The extrapolation would include a worst-case
scenario. I would recommend the use of a
respirator.
Q. Okay. Would you recommend that the
maintenance or custodial personnel who are going
above the ceiling in the same buildings we've
been talking about take care not to in any way
contact the fireproofing itself?
A. Of course.
Q. Okay. Do you have any knowledge as
to whether there is any debris or dust or other
portions of the fireproofing that has fallen
from the fireproofing and is currently on the
top side of the suspended ceiling in the same
buildings we've been talking about?
A. According to my recollection and an
inspection carried out by Don Hunter, there was
some debris on top of the tile ceilings, and I'm
thinking that there was debris, also, in a power
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plant, which came about for other reasons. Q. Assuming that that debris, in fact,
did come from the asbestos-containing material,
do you have any recommendation as to what, if
anything, should be done with that debris? A. You mean, other than place it in some kind
of a bag and take it away? Q. Well, that's the first thing.
Should you leave it there, put it in
a bag, take it out, or what?
A. That would depend on the situation. I
mean, you might leave it there.
Why would you want to move it?
Q. If a maintenance personnel or
custodian has to go above the ceiling to do some
work above the ceiling, sticking his or her head
above of the ceiling, and all around there's
some debris, do you think in that situation the
debris from the fireproofing should be cleaned
up?
MR. VALENZA: Objection.
A. I still don't know. One needs an air
measurement of the fibers in the air. The fact
of the matter is we can have a box of asbestos
on this table, the question is: Does it produce
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any hazard or associated or attendantrisk by
its presence? Are there other factors that come
into play here? So, its mere existence is no prima
facie evidence that it hasn't existed and
recommendations thus follow.
Q. Do you believe that
asbestos-containing dust or debris on a surface
can be re-entrained into the air?
A. Could be, sure.
Q. Do you have opinions as to what
types of activity would re-entrain
asbestos-containing dust surfaces into the air?
MR. BROWNSON: Object to the form of
the question.
Are we speaking again of these three
buildings or ~
Q. More generally, but if you care to
limit it, I don't care, Doctor. My question was
more general.
A. The only data I have seen are data
involved or generated on clean-up activities,
such as sweeping, specifically dry sweeping, and
fibers can be re-entrained for short periods of
time.
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Q. Therefore, would you recommend, going
back to the three Asbestospray buildings, that if
a custodian were going above the suspended
ceiling, that that custodian not dry-sweep, or
in any other way, similar way, push around the
debris from the fireproofing?
MR. BROWNSON: I'll object to the
form of the question.
A. Pushing around is different, of course.
Should he sweep the top of that tile? No, I
wouldn't recommend it. I wouldn't recommend
anyone going up there and sweeping the debris on
top of the tiles.
Q. Would you recommend that it would be
a prudent health measure for a custodian going
above the ceiling in the samebuildingswe've
been talking about to use some sort of water
spray to spray down the debris to help stop any
possible re-entrainment?
A. I'm not so sure.
Q. Why not?
A. Once you get water above the tiles and the
limited amount of air circulation above those
tiles, you might begin to harbor certain
organisms in dangerous environment molds, for
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example; so I'm not certain that would be an
efficacious method. Now, you've begged the question, is
there a health risk? You have to measure fibers
in the air. MR. MILLER: Mark this 3, please.
(P-3, one-page document, received
and marked for Identification.)
Q. Doctor, I've had marked as Exhibit
3, a single page document which is headed the
following words: "Dr. Langer will rely on the
following materials in this case." This came
from a pleading filed by Asbestospray.
Does this document, in fact,
summarize the specific materials in which you
expect to rely for your testimony in this case?
A. May I read them?
Q. Of course. I assumed you had seen
them before, I apologize.
A. No, that's all right.
I think that I have seen this report
before, yes. I have read reports of Longo,
Millette and Hatfield. I have read the
deposition transcript in this matter. I have
looked at the product ID reports, and we've
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DR. LANGER - Direct
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already discussed some of my reservations.
Air sampling data, yes, I've seen
those. Most of these instances, dust samples,
I've seen those.
Photographs in buildings, yes, air
sampling data. We are in the process of
completing those.
As a matter of fact, documents
pertaining to Sprayed Mineral Fibers, yes, I've
read those.
Air consultants and other parties,
yes, I have those.
Proceedings of Harvard, yes, I've
read those.
Spengler and colleagues on the
"Summary of Symposium," yes. I've read that.
The Health Effects Institute, yes,
I've read that.
Wilson, Langer, yes, I've even seen
that paper, too, yes. I've seen all of these.
Q. Thank you.
Are there any building-specific
documents that are not on this list on which --
or which you have seen -- let's start with that,
relating to our building, the three buildings at
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DR. DANGER - Direct
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issue. A. I have.
I am now completing review of
documents from Conwed, so there may be other
buildings. Q. Putting aside Conwed, again --
A. Oh, you mean this particular or three
little litigations grouped into big ones?
Q. Fairly big.
A. No. Q.
I'd like to go quickly through as
many of these references as we can.
Number 1: Reports of William Longo,
Jim Millette, and William Hatfield, are they
product identification reports?
A. Well, generally so. I mean, you're asking
me the questions. Let me see if I can give you
something more specific.
Q. Do you have with you the actual
reports that you looked at?
A. No, no, no.
Okay. Go ahead and ask me the
question. I can give you the specifics.
I've seen a report in a letter of
November 9th, 1992, HAS to Mr. Tucker, Counsel,
concerning samples analyzed in the District 2
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Office. Material is identified as Asbestospray T.
I've looked at the TEM reports.
I have looked at the report from law
engineering. This is Hatfield's report of
November 16th, 1994, concerning settled dust.
I have looked at Hunter's
Environmental Survey, 10 to 11 November, 1994.
Air sampling data. Those are the
data I have looked at for the District 2 Offices
in Kenwood.
I have also looked at a report in a
letter of July 6th, 1993, Dr. Longo to
Mr. Tucker, concerning identification of the
specimens, some of his TEM reports. Interesting.
I have the Hunter, Incorporated
Environmental Assay, 9 through 10, November of
'94.
MR. BROWNSON: Which building is
this that we're speaking of?
A. This is the Leif Ericson building -- I'm
sorry, this is Leif Ericson, also known as Leif
the Lucky.
Then it's Glenbrook South High
School, Glenbrook. We're not interested in
them.
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How about South Shore High School?
Q. Let's stop right there.
We said earlier, and Mr. Brownson said that there were three buildings in Chicago.
You also mentioned Glenbrook South and
Hawthorne.
Are they under the Evanston, Heading?
MR. BROWNSON: Yes, you can question
all you want about that. We have not had
to look for those specifically because
those are the ones that -- where there
have been removal already, so air sampling
has not been analyzed there.
Q. Very simple: Have you looked at
Glenbrook South or Hawthorne, Doctor?
A. I have looked at some documents.
Q. You have?
A. Yes. Many of these reports contain data
from different buildings, so if you read Report
A, it may have some on Hawthorne and South Shore
for reasons which escape me; but nonetheless,
this is the reality.
Q. The air sampling you see relates to
South Shore, Leif Ericson and District 2 Offices?
A. Yes.
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Q. Air sampling from Glenbrook or
Hawthorne, is that correct? A. May not be correct at all, no.
I have some data that I've looked
at.
MR. BROWNSON: Just to help you out,
we did not have him analyze air sampling
from those two schools because, as you
know, its materials have been removed.
So, whether he has seen some in one of
these Longo reports, I don't know.
you.
Q. I apologize, Doctor, I interrupted You were telling us* the documents you've
seen, and I wish you would finish the recitation.
A. I've seen documents from the South Shore
High School. This is a Longo report to Mr.
Tucker. It's dated January 1st, 1993. It
consists of a bulk assay of what these materials
are, its formulation. It consists of a TEM
assay of different components.
There is also a report from Hunter Environmental, a survey done on the 14th and
15th of November, 1994.
There is also, for some reason --
oh, as I said, sometimes there are reports with
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DR. LANGER - Direct
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different schools. Here is a report that had
Webster Elementary and South Shore High School
as part of the same report. The South Shore High School, there
is an assay by polarized light microscopy what
the bulk material is. There are a number of
settled dust assays.
There are South Shore air samples
analyzed by the NIOSH, OSHA technique using
phase contrast microscopy. These were
undertaken by McCrone and air data from South
Shore.
Q. You earlier talked about your
general opinions about dust sampling results.
Is there anything in addition about
the specific dust sampling that you looked at
here that you feel is worthy of comments or do
all your prior comments fairly cover what you
say about the dust sampling that you see here?
A. Well, that's an awfully broad question.
What is it that you would like me to
do? You want me to take a single specimen or
single report and just "sally forth," as we
say?
Q. Please, I would appreciate it.
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A. Okay. Let's take a look at the Leif
Ericson report here, a report from Longo's group
to Mr. Tucker, five samples analyzed as a bulk
material.
What is this stuff, basically, is
the question? MR. BROWNSON: Wait a minute.
MR. MILLER: Dust sampling is what
I'm talking about.
MR. BROWNSON: The question is
settled dust?
MR. MILLER: Surface dust or settled
dust.
Thank you.
A. Okay. Let's stay with the District 2 --
let's do District 2, and let's look at a report
dated November 16th, 1994, which consists of a
dust sample log from Hatfield which is sent to
materials analysis, whatever, Longo's group, and
Longo produces an assay and sends this assay to
someone on -- well, that doesn't matter.
The material is collected by
Hatfield and sent to Longo. Longo receives it
November 21st, which is five days later.
There are three dust samples. There's
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a certain area which has been microvaced, and
these materials are processed and a number of
objects are counted and given in asbestos
structures per square centimeter.
Of the 102 objects which are
described as amosite, only one of these 102
objects have any diffraction data available,
which is not included in that assay.
Now, of the 1.6 million, only
770,000, approximately one half are greater than
five microns in length, which I#m interested in
because this relates to risk analysis and the
OSHA data sheets that we use.
Now, the values given, concerning
the distance between the ceiling and the top of
the tile, from the data given, I have
recalculated, based on 30 years, 365 days a
year, 24 hours a day, 60 minutes as a continuum
that the fibers falling, if these were
individual fibers, those greater than five
microns in length, that a concentration per
cubic centimeter of air should have been
approximately 0.3 fibers per CC, 3/10 of a fiber
per CC. It was a continuum over that 30-year
period. Failure began day one, and it has never
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changed.
Now, if you look at the air data,
this is too high by three orders of magnitude,
so obviously, one cannot reconcile the settled
dust data with the fiber levels found in the air
of buildings.
So, the fact of the matter is you
can find "stuff" on the top of a surface, but it
begs the question: How did it get there? Was
it ever in the air to begin with? Other than
traversing point A to point B, were there ever
any fibers in the air? The fact you find fibers
on the settled surface, what does it mean in
terms of risk to building occupants, that begs
the question.
So just based on raw data, based on
the fact that there are a certain number of
fibers found, and given the circumstances that
it's landed on top of this surface, you cannot
reconcile fiber numbers with fiber levels found
in buildings, that is the bottom line.
Q. Is it your conclusion that the
figures found by Dr. Longo mean nothing in terms
of risk to building occupants?
MR. BROWNSON: With respect to
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settled dust? HR. MILLER: I'm asking the
question. A. No, of course not. I didn't say nothing.
Every piece of data has some meaning. The fact
of the matter is: I'm interested in risk
calculation and I'm interested in fiber levels
in the air. Now, tell me whether this pertains
to fiber levels in the air. Based on what this
technique involves, and based on what is
presented to us, I don't think that you can rely
on these data to form any conclusion regarding
risk to building occupants.
Do they have meaning? Yeah, I
suppose they have meaning. It means that it
went from there to here. I'm pointing up and
I'm pointing down. It went from there to here,
but it carries no other significance without
other data. One has to measure fiber levels in
the air.
Q. You indicated that your calculations
include the distance between, I guess, the
fireproofing above and the sample below.
How far was that distance, sir?
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A. The distance that I used in this
calculation was 10 centimeters.
Q. Okay. Are you willing to conclude
that the amosite found in the dust sample, 10
centimeters below the amosite containing
fireproofing, came from the fireproofing?
A. Might have.
Q. Is there any place else that you can
realistically think that it came from?
A. Well, let's say you have a maintenance guy
who's going to go up into the ceiling and he's
just come from the boiler room and he did a
repair job in the boiler room.
Is it possible that the jacket he's
wearing will be contaminated with stuff from the
boiler room? That's a possibility, but let's be
reasonable here. You look at the material on
top of the ceiling tile and you look at the
material in the ceiling, and you should
characterize it on the basis of fiber type,
fiber chemistry, size distribution. You come to
a better understanding as to whether this stuff
on top of a ceiling tile really came from the
ceiling.
Let's say it did. Is there any
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other way of getting it -- people talk so much about maintenance workers knocking this stuff
around, it could have come from any number of
sources.
Do I believe that that's it?
No, I think if you can find
something on top of the ceiling tile, it
probably came from whatever is sprayed on top.
That's just sensible.
Q. You indicated that the diffraction
information was not written down on a report
that you saw.
A. It was not provided.
Q. Not provided.
Do you believe that a light
microscope report should always have optical
diffraction information written down?
MR. VALENZA: Objection.
A. You mean, TEM report, a transmission? You
mean light microscopy?
Q. I did say light.
A. You did say light.
Well, what is diffraction?
Q. That's only for -- I'll rephrase my
question.
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Do you believe that a light
microscopic report should always have written
down on it optical qualities of the asbestos-containing material, such as indices
and refraction?
MR. VALENZA: Objection.
A. It depends.
Q. On what?
A. See, if someone gave me a sample and I
said there was amosite there, I think there's a
pretty good chance there was amosite there. If
there's someone that's just been trained in the
laboratory, let's say, a big laboratory, as in a
laboratory, and someone got three weeks training
how to identify asbestos, then I'd like to see
most of the characteristics written out, yes.
Is it necessary to do that?
Generally so.
in many of the reports there's an
index of refraction given parallel to the fiber
bundle and right angles to the fiber bundle and
some optical characteristics which indicate and
support the otherwise visual observation that
this stuff is an amosite, crocidolite or
chrysotile. So, generally so, you'd like a
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little bit of quantitative information. Q. You did some calculations to come up
with a .3 fibers per CC level on a piece of paper in front of you, is that correct?
A. Yes. MR. SENFTLEBEN: Objection. I
thought he said .03, but I stand
corrected.
Q. I thought it was 0.33.
A. Let's say it's .03, what does it matter?
Q. What does it matter?
A. It doesn't matter. Calculations could be
off.
MR. BROWNSON: Wait for a second. Q. Now, would you have any objection if
I made that piece of paper an exhibit, Doctor? A. Would I object to it?
Q. Yes.
You can certainly have a copy of it
or we can take a copy.
MR. MILLER: I'd like that to be
Exhibit 4.
MR. BROWNSON: Let's mark it as an
exhibit.
MR. MILLER: We'll do it after a
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break. Good. Q. Then this will be the next exhibit.
A. I assumed a ceiling height of 10 feet -- I
misspoke. Ten feet would yield a fiber level of 0.01. Ten centimeters, it would be 0.3, but this
is written down and so you may copy it.
MR. BROWNSON: We'll make that as
Exhibit 4.
Q. If you assume the ceiling height of
10 feet, does that mean that the dust sample was
not taken above a suspended ceiling; rather, it
had to be taken below a suspended ceiling?
A. Correct. Let's say you found it on a
floor, in a carpet, whatever.
Q. Given that circumstance, Doctor,
namely, you have the asbestos amosite-containing
fireproofing separated by a suspended ceiling
from the normal occupancy area, and somewhere
down on the floor of the occupancy area the
sample is taken. So you have about a 10-foot
distance and you find the 107 or 102 amosite
fibers in that dust sample.
Do you still believe that the
amosite in that sample probably came from the
fireproofing 10 feet above it?
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MR. BROWNSON: I'a just interposing
an objection to the form of the question
because it really is hypothetical, it's
not fact, but you can answer it.
A. May I answer it as a hypothetical?
Q. You can answer as a hypothetical.
It's how you are looking at it, but hypothetical
is fine.
A. The more, as the physicians would say, the
more proximal this sample is to the source, the
more you are secure in yourconclusion.
The more distal this is, like if you
find it on a floor under some surface embedded
in the carpet, there the issue of tracking dust
in a building is raised, and it could come from
any number of sources. So, the closer you are
to that source, the more likely it came from
that source.
Q. Given the hypothetical I gave you,
to what degree of certainty would you say that
the amosite on the floor came from the
fireproofing 10 feetabove?
A. I have no idea, I've never been in the
building.
Q. Okay. If you had a dust sample
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taken on a ledge that's about two feet below the
surface of a ceiling, and the ceiling, itself,
was asbestos-containing, and the ceiling has
chrysotile and you have chrysotile dust samples
taken a couple feet below that level, this is
all in a normal occupying area of a building,
like a hallway, say --
A. Right.
Q. -- would you be willing to make any
conclusion regarding the source of the chrysotile
in the dust sample being the material two feet
above it?
MR. VALENZA: Objection.
MR. BROWNSON: I will object to the
form of the question as an improper
hypothetical, but go ahead. Improper in
the sense that I don't think it fits any
of the facts that we're dealing with here.
Q. Okay. You may answer.
A. Chrysotile is more problematic. Obviously
it comes from a greater number of sources, and
there are those who believe that it is -- there
are many other sources of chrysotile fiber.
Amosite is more restricted in consumer products,
meaning products found outside of a boiler room
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or power plants. So there's only a limited number of sources, whereas chrysotile, you enter
into a "where did it come from" kind of thing.
MR. MILLER: The next exhibit
number, please. (P-4, diagram sheet, received and
marked for Identification.)
(P-5, cover letter, dated May 26,
1995, received and marked for
Identification.)
Q. Doctor, I've handed you Exhibit 5,
which is a cover letter from Lynnae Waskosky,
dated May 26, 1995, to Dan Albers. There are
attached reports that are, I believe, reports
prepared by you.
Are all the attached reports
prepared by you?
A. No.
Q. Did you prepare any of them?
A. These reports were prepared by Dr. Nolan
of my laboratory.
Q. Do you have any idea why Ms.
Waskosky would say in the letter that the
reports were prepared by Dr. Arthur Langer?
A. Well, she's a paralegal at Mr. Brownson's
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lav firm, and I suppose she assumed that. Q. Okay. Now, did Dr. Nolan do his
work under your direction and supervision and
control?
A. I received the samples. The samples were
prepared by my laboratory, by our laboratory
technician, and I asked Bob Nolan, who has 15
years' experience in this field, to scan the
grids.
Q. Okay. Now, it appears to me that
the work is not completed, is that correct?
A. As we sit here today, no, we're still
working on this.
Q. Well, this provides a bit of a
problem, since I'm supposed to ask you opinions;
since your work isn't completed, it may be hard
to ask you about opinions.
MR. MILLER: Mr. Brownson, what do
you think we should do about this problem?
MR. BROWNSON: Work not completed
largely because of the fact, as you know,
our second round of sampling was just done
less than a month ago.
My solution is, and has been, that
you can ask him all you want about the
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work that's been completed. As soon as
the additional analysis from the recent
sampling is completed, you'll get it. If
you want to ask him about that, you're
welcome to do it.
MR. MILLER: Okay.
MR. BROWNSON: I suspect that you
can ask him enough questions today
concerning the technique and such that
when you see the additional data, it
probably will not involve a lot of
additional questions, but maybe it will,
that's up to you.
Q. Let's look at the first page, HES
identification number refers to Hunter
Environmental Services, sir.
A. Which school are you in?
Q. The first page, as I have, table
one, it should be the same that you have.
A. Description of sites and types of second
air monitoring in South Shore High School.
Q. Right.
A. HES, Hunter Environmental -- yes.
Q. Do you have back-up data from them
detailing the dates, the times, the exact
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location, that type of information?
A. Yes, of course. MR. MILLER: Has that information
been provided to us, Mr. Brownson?
MR. BROWNSON: Yes, if you look at
this previous exhibit, which I think was
3. MR. MILLER: It was provided. I'll
take your word for it.
MR. BROWNSON: I'm trying to look
here very quickly, which I don't see it on
that exhibit. It was provided. It was
the Hunter report.
MR. MILLER: Okay. Fair enough.
Q. Doctor, I'll ask you a few more
general questions to see if you can give me that
information.
Were these samples all taken during
normal school hours, according to your
understanding, Dr. Langer?
A. I don't recall. I would have to reread
Hunter's report. I think so, but I'm not
certain.
Q. For the samples taken into the
specific building, were they all taken on the
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sane day except, of course, for the reference to
second-day air monitoring, which I assume would
be taken on a different day? A. I think they were taken over two days, if
I'm correct. Q. Okay. Getting down to the bottom
line of these, let's see, the sixth page of the
report, as I count them, is: "Table 2: Results
of the analysis of the membrane filters by
transmission electron microscopy for asbestos in
Leif Ericson Elementary School."
Do you have that page, sir?
A. Yes.
Q. The columns that are towards the
right center are headed "Chrysotile asbestos
fibers detected."
Do you see that?
A. Yes.
Q. Some columns simply have asbestos
fibers detected.
Was there a distinction or reason
why some columns on some pages use the word
"chrysotile," and others do not?
A. No, I don't know.
Q. If the amphibole fibers were
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detected in this specific school we are looking
at, which has a heading of only chrysotile asbestos fibers, would there be some indication
of amphiboles or should there be?
A. Normally, we indicate asbestos fibers
detected, and if an asbestos fiber is detected,
then we note which asbestos fiber type.
So we made reports before in which
we indicated it was either chrysotile or amosite.
We never found any crocidolite in the air, nor
have we found any other fiber by either amosite
or chrysotile. Why that says chrysotile, I'm
not certain, other than the fact that this was
awfully rushed when we made this report.
Everything that takes time is the actual
scanning, of course.
Q. As I look through these reports and
I look at the columns where it's either
chrysotile or just asbestos fibers detected,
where there is a numerical result, it is always
zero.
is that how you read it, also?
A. No.
q. sir?
Where do you see fibers detected,
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A. Well -- Q. Asbestos fibers detected?
A. Well, asbestos fibers detected, there were
no asbestos fibers detected. I think you were
referring to the airborne concentration. It's
never referred to as zero, of course, less than
a certain limit of detection based on the amount
of air actually scanned on the filter, the
amount of particulates deposited from air
scanned.
Q. Thus far, Dr. Nolan has found no
asbestos fibers whatsoever in those that he has
reported results on, is that correct?
A. These are as reported, correct.
Q. Do you have any back-up data of any
typewritten data regarding these air samples
that have not been presented?
A. What do you mean, the scientific data?
Q. Any type of spectra charts, any --
you broadly included Dr. Nolan when you did
these air samples, were there documents prepared
that we don't have?
A. Well, I don't know what you can prepare if
you don't see the asbestos fibers. That's what
you're counting. You don't just count the
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particulates or photograph particulates, what
you do is look for fibers.
Q. You don't have, for example,
electron micrograph data sets of some sort?
A. You mean, an actual printout of the grid
shown in the various areas scanned?
Q. Among other things, yes.
A. I don't have those with me.
Q. Okay.
A. Since the project is not finished as yet,
normally those are a part of a file and they're
kept in one place.
Q. Would they normally beturned over
to your clients when the project is done?
A. Sure.
MR. MILLER: Okay. I would
appreciate, of course, receiving
everything.
MR. BROWNSON: Right, you'll get all
that.
MR. MILLER: Okay.
Q. Let's move on a little bit,sir.
Do you have with you the photographs
that you looked at of the three buildings?
A. No.
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Q. Category 8 says that you will rely
on documents pertaining to Asbestospray and/or Spray Mineral Fibers Manufacturing Association.
Is. it all right with you if I call
it SMFMA for short? A. No, I would say it's most certainly not
correct. Q. Not correct?
A. No. I want you to call them anything else
but that. As a matter of fact, you mean the sprayed mineral fiber, whatever?
Q. Manufacturing Association, SMFMA?
A. Can't we agree on some other term? Q. What would you prefer?
You're serious? A. Sprayed Fiber Association.
Q. Doctor.
Okay. Sprayed Fiber Association,
I'm looking at what's been given to me. I believe it's supposed to be Exhibit A.
It's a fairly thick pile of papers, generically,
about Asbestospray or Sprayed Mineral
Manufacturers. I don't see anything in here dealing specifically with this case.
Tell me how this is going to support
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your opinions, this Exhibit A information.
A. What opinions are there? Those that are
going to be objected to are going to be supported?
Are we talking the Sprayed Mineral
Fiber Association? Then they will help me. If
you're going to ask me about Mr. Herbert Levine,
who is president of the company, these will help
me. If we are interested in whether this
organization participated in asbestos research
at Mount Sinai, these documents will help me.
If you're going to suggest to me that this
organization was a nefarious organization with
suppressed data and injured workers, then these
data will support me in my opinions.
Q. Doctor, it's obvious that the large
number of documents in there could be relevant
for many different issues.
A. Exactly correct.
Q. The point is, though, that it has
been presented to me by Mr. Brownson as material
on which you will specifically rely, so I think
it's fair to ask you: What is this type of
information which in some way is generically
different than everything else we've talked
about today?
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A. Correct. Q. How do you expect this to be
relevant to what you expect your direct
testimony to be? I don't mean relevant in a
legal sense, where does it fit in so I get a
fair shot to ask you a question without looking
through hours -- to go through page by page?
MR. BROWNSON: I can probably move
that along.
MR. MILLER: Appreciate it.
MR. BROWNSON: Dr. Nolan had certain
contact with Herbert Levine, who is
president of Sprayed Mineral Fibers
Manufacturing Association. Dr. Nolan
worked at Mount Sinai and --
THE WITNESS: You misspoke.
MR. BROWNSON: I meant Dr. Langer.
MR. MILLER: That's right, you did
say that.
MR. BROWNSON: To some extent you
can appreciate that we don't know what the
evidence may be on various theories with
respect to liabilities against Asbestospray,
but to the extent that any of the claims
involved analyzing conducting Asbestospray
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during the years the material was applied
in these buildings, we may ask Dr. Langer
to respond to that in terms of what he knows about the activities in Asbestospray during
those years, and that's why he's reviewed
this.
MR. MILLER: Thank you.
BY MR. MILLER:
Q. Can you, in summary, tell me what
you would tell the jury if you were asked: What do you know about Dr. Levine --
A. Mr. Levine.
Q. -- Mr. Levine, and his activity
about asbestos during the years covered by these
documents?
A. That's a fair question.
Mr. Levine frequently came to Mount
Sinai to discuss issues of asbestos and its
health effects. He frequently met with Irving
Selikoff. Mr. Levine, through his organization,
Sprayed Mineral Fiber Manufacturer's
Association, contributed to some of the ongoing
projects at Mount Sinai. Mr. Levine generously
gave of his time, he generously gave of his
knowledge in the field, he provided investigators
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in our laboratory with information concerning
ongoing spray activities, and allowed members of
our research group to visit sites and to perform
air sampling exercises.
He was forthcoming. He participated
in several of our early courses on asbestosis at
Mount Sinai. I found him to be reliable and
honest and open, generous with his resources,
and a refreshing individual in terms of a
representative of asbestos concerning the late
1960's, early1970's.
Q. Doctor, before we broke, you made a
comment that Dr. -- excuse me -- Mr. Levine was
refreshing, I think, in terms of people in this
issue at that time.
Whom did you contrast Mr. Levine to?
You said he was refreshing. Refreshing in terms
of comparison to X?
A. Well, let's invoke one of the great demons
of modem industry. A lawyer, by training,
called Vandiver Brown, who is quoted or is
reported to have said, "The less said about
this, the better, in terms of diseases,M so on
and so forth, that would be one side of the
fulcrum; and on the other side is Herb Levine,
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who is willing, able, open and generous with
resources, as I have said. Q. Okay. When did you first get this
pile of documents which is supposed to be No. 8
as listed on this Exhibit 3?
A. I don't remember. Several months ago, I'm
sure.
Q. You agree with me that these
documents are not in chronological order? Do
you agree with that?
A. I don't remember, but I put mine in
chronological order.
Q. Did you notice whether or not there
were documents which appeared to be missing
because Bates stamp numbers would jump from one
document to another?
A. Well, there are some documents that didn't
belong in there, actually, which I pulled out,
so that might account for it. This enterprise
is carried out by someone who is going to take
the 5 o'clock train home orsomething.
Hopefully, the documentsare put into some order
that make them useful, but it doesn't always
happen that way. So you have to look at a box
of documents, see what's there, and do your own
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kind of recategorizing, if that's a word, and
this has to be done. Are there documents missing? I
don't know. I haven't thought about looking. I
mean, are there jumps from 1967 through 1971? I
didn't notice any, but there may be. Q. How do you know that you're getting
the complete story about what happened with the
Sprayed Mineral Fibers Association, Mr. Levine,
in those years regarding your concerning
interest and testing for asbestos?
How do you know those documents are
complete?
A. I'm glad you asked me that question because this is something I volunteered with
Mr. Brownson not too long ago. That is, I was there at Mount Sinai
at that particular time, and I was involved in
most of those activities, and I had an impression as to what Mr. Levine's contribution
was, and thesedocuments reinforced my previously-held opinions and high regard for him.
q. was there any other asbestos
manufacturer at that time whoserepresentatives
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or general policies struck you as favorably as
Mr. Levine obviously has? MR. VALENZA: Objection.
MR. BROWNSON: I'll object to the
characterization of "asbestos
manufacturer." He didn't manufacture
asbestos, he manufactured products.
Subject to that objection, go ahead.
A. My interaction with manufacturers? Let's
say Johns-Manville was considerable at that time
and we were involved in certain joint studies
with the Johns-Manville Corporation, and I found
them to be generally helpful and open, generally so.
Q. Did you have any contact during that
time with representatives of U.S. Gypsum?
A. I don't think so, no.
Q. Did you have any contact with
representatives of W.R. Grace during that time?
A. I don't think so. You mean, in the late
'60s, early '70s?
Q. Yes.
A. I don't think so. I mean, I may have. I
may have misspoke. Is there someone who contacted
me, who wrote me a letter or called me on the
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telephone? It's possible, but I don't think
so. I cannot recall; that's a better response,
I cannot recall. Q. Remember the documents in here that
showed a report by Davershaw Cooper on air
sampling, taken in the building in California,
sir?
A. Yes. Q. That was in the Wells Fargo building
was it not 7
A. Yes.
Q. That product was Kafco, is that correct? A. I believe so.
Q. What's your opinion about the results of that air sampling?
I have the document tabbed, if you would like to look at it. A. May I?
Q. Yes. There's certainly more than one document, and I will try to give you the two
key ones.
For the record, the two documents I'm showing you, I believe, are the two main
reports. There are other documents that are in
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the transmittal letters, one dated September
23rd. A. This is 1970 and '71, isn't it?
MR. BROWNSON: Let him finish.
Q. There's a subsequent one dated
November 5th, 1970, and there are other letters,
but these are the two key ones. A. Here on my notes which I consider
important, and whether I get out of this, so to
speak.
Q. Please read your notes. I
appreciate it.
A. There's a letter of 23 September, 1970. This is Fowler of Davershaw Cooper of Mr.
Levine, a preliminary report. This is
Montgomery Ward.
Q. I think it was Wells Fargo.
A. Well, hold it a second. Let's go down to
Wells Fargo, Fowler to Cooper to the Sprayed Mineral Fiber Manufacturer's Association.
That's 5 November, '70.
Q. Those are the two I put in front of
you now?
A. Yeah. The values are 00085, 0011, 0019,
rounds it out to 001, 001, 002.
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How, they -- Q. Can we stop right there for a
second?
A. Sure.
Q. Are those the values of the report
or values that you have recalculated yourself
from other data?
A. The values in the report are as follows:
0.00085, 0.0011 and 0.0019, and I just rounded
them out.
They're all pretty much -- but say
that they are the same, for argument's sake.
Now, this was couched in 1970, according to the
TLV, to prevent asbestosis.
Again, the threshold limit values at
that time, 1970, this predates OSHA two years.
We are relying on the American Conference of
Governmental Industrial Hygienists, ACGIH, in
which the TLV, to prevent asbestosis, what they
call 5,000 fibers per liter, five fibers per CC,
no firm guidelines on levels, and they say that
these fiber levels, 001 or 01, probably such
amounts constitutes no risk.
So, given asbestosis, given the
TLV's at the time, given the fiber levels they
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found, it's not such an outrageous conclusion.
In fact, it's pretty good.
Q. Do you agree with the other
conclusion reached in both of those reports regarding the same study, that air passing along
the airplane line with Kafco has more asbestos
in it after passing through an air plenum, before
it passes through it, is in a statistical
increase in asbestos?
A. That is their conclusion.
Q. Do you have any reason to doubt
that?
A. No. Why should I?
Q. Do you have any further comments
about that?
You're looking at your notes, sir.
A.
No, I think that
--
Q. The next, category 9, "Air sampling
data obtained byAsbestospray
consultants in
other propertydamage litigation."
I have another quick question before
you
do that.
A. Shoot.
Q. Some of the other information
concerns a Marylandproject.
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What type of building or buildings was
that, do you know? A. My understanding is that this was from the
state office building,Preston Street.
Q. The next set of information I see
concerns samples taken in the Trans World
Airline terminal in Kennedy Airport in NewYork.
Is that the same TWA terminal which
had some samples taken in these documents that
we saw in Exhibit A, early samples, if you know?
A. I don't know.
Q. Okay. The next samples come from
Columbia, South Carolina, USC Business
Administration Building.
is that a university, if you know?
A. Yes.
Q. The next samples come from a
convention center in Wichita, Kansas, is that
correct, sir?
A. Yes.
Q. Next samples come from a West Virginia University coliseum in Bordentown, West
Virginia, is that correct?
A. Correct.
q. The last samples refer to a drop
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test in the coliseum in West Virginia
University. What was that test all about?
THE WITNESS: Do you have an
objection that you want to get on the
record?
MR. BROWNSON: No, I have no
objection to that question.
A. The contention at that time that concerned
plaintiff's data was that when and if this
material failed, it would drop to the ground and
generate dust, that there could be a catastrophic
failure if this material was on the coliseum
ceiling.
So, they constructed an isolated
chamber and they dropped some stuff at a certain
height, whether it was three meters or four
meters or 10 meters, whatever it was, and they
dropped the stuff into this glove box, which is,
for many human beings, it's 20 feet high and 10
feet by 10 feet, whatever the dimensions were,
and they dropped the stuff down and they
measured the amount of fiber liberated in this
drop test.
Q. Is the information that you have
here as part of this category 9, the
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plaintiffs's generated information on the drop
test, or is this information that you or
somebody in your office have prepared? A. I think I took the information from the
plaintiff's data and I may have recalculated a
couple of things, but that's it.
Q. Was the plaintiff's data published,
if you know?
A. I don't think so. Q. This was part of the West Virginia?
A. Wait a minute. I think it may be
published in part in theCollegium Ramazzini,
but I'm not sure.
Q. Was this drop test originally part
of the West Virginia litigation,asbestos
property damage litigation?
A. You mean, was it carried out for that
purpose?
Q. Yes, sir, if you know.
a. I think so.
Q. Now, I've gone through this category
9, and to the best of your recollection, have I listed all the different types of buildings and
locations of air sampling that you have under
this category 9?
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A. Well, actually, there's two other work
sites which use the Asbestospray T formulation.
One is the Wallbrook High School, and the other is the one that's in Maryland, and the other is the Erasmus Hall; I mean, it's arguably Asbestospray product, Erasmus Hall in Brooklyn, so there's two other data sets.
MR. MILLER: Mr. Brownson, I guess there's no objection to provide the other data sets?
MR. BROWNSON: I don't -- oh, we just didn't have the written reports?
THE WITNESS: I may have them in our
computer. MR. BROWNSON: To the extent that
they can be found and generated, we will provide them.
THE WITNESS: Of course.
Q. Can you tell me, in summary, what the air sampling data shows for each of those?
A. I think Erasmus Hall was 0008. That was
an average. The outside air was a little higher. The Wallbrook High School also was triple digits, some numeral.
MR. MILLER: Let's break.
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(Whereupon a brief recess vas taken
at 2:00 p.m.) (Whereupon the proceedings resumed
at 2:15 p.m.)
BY MR. MILLER: Q. Doctor, back to Exhibit 3, which is
the listing of materials in which you will rely
in this case, subsections 10 and 11both refer
to the symposium held at Harvard in 1988,
published in 1989.
I have questioned you about this
before. I would appreciate it if you can, very
briefly, summarize for me, if I were Mr. Brownson,
asking you to explain to the jury the lessons
from that symposium, including, of course, your
article which was part of it.
MR. BROWNSON: Well, the question is
broad and I'll object to it, but go ahead.
A. Very broad. However, I shall attempt to
answer it.
The papers presented at Harvard,
actually in the Kennedy School of Government,
reported on current epidemiological findings,
current experimental findings, current fiber
levels found in buildings. A synthesis of those
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data sets was attempted, and the conclusion was
that "the asbestos in the buildings problem,"
put that in quotes, was overblown in the United
States, to use a phrase, and that it was not as
serious a problem as envisionedby various
groups, including groups within the Federal
government, agencies within the Federal
government.
Q. With regard to the asbestos problem,
reference specifically was to the asbestos
problem for passive building occupants, is that
correct, sir?
A. No. As a matter of fact, we touched on
asbestos exposure in a range of settings, not
merely building occupants but, of course, the
nature of the symposium was the health effects
to building occupants, whichincludedpassive
occupants.
Q. If you can summarize briefly to the
jury your paper that was presented there and
published there.
A. That paper focused on fiber type and the
asbestos diseases.It reviewed much
epidemiological data, at least those studies
which delineated among thefiber types.
It
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touched upon environmental mesothelioma in
certain settings. It touched upon the papers
upon which risk is calculated, and clearly showed that there is a significant difference in
the mortality experience of workers exposed to
chrysotile asbestos as compared to those workers
exposed to one of the amphibole asbestos types.
The paper also included some data
pertaining to fibers in the lungs of workers in
the United States.
The paper also included some
interesting data on fiber types found in certain
kinds of products in the United States, and
basically concluded that there is a very
significant difference in fiber type potency to
reduce asbestos diseases, and that because
chrysotile was one of the prominent types that
we deal with in buildings, the risks associated
with such exposures weremuchless than
previously perceived.
Q. Category 12 of Exhibit 3 refers to
the "Health Effects Institute - Asbestos
Research," a 1991 document.Again,
I've asked
you about this before.
Would you be kind enough to
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suBuirize briefly, as if I were Mr. Brownson
asking you to summarize for the jury, the main
lessons for that document? MR. BROWNSON: Again, I'll object to
the form of the question, but go ahead.
A. Well, you have the document here.
Q. I do? A. There's an executive summary and --
Q. Would you simply refer to the lessons of the executive summary?
A. You're not going to give me that nice
*
volume that you have all marked up?
Oh, okay.
The executive summary includes a
general background to this problem with asbestos
in schools, some brief description of what
asbestos is and how it was used in the United
States, the methods by which the panel reviewed
and either accepted or rejected data sets, the
methodology of the review committee, the
presence of asbestos in public and commercial buildings, the general categories of exposed
groups within these buildings, how asbestos is
measured in various environments, what the
exposure to asbestos in buildings is based on
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the available data, some measurement of what the asbestos levels are in the ambient air, a focus
on the levels found inside of buildings with
asbestos-containing products, exposure
categories and distinctions made on the basis of
different categories of occupants, from passive
occupants to emergency workers who come in and
fight fires, the control of the asbestos
exposure in buildings through operations and
maintenance, the potential health effects
associated with exposure to fiber in buildings,
and the various physical chemical properties
which are thought to govern these risks, the
risks to building occupants based on the fiber
levels, and the various models used to calculate
risks, the presence of man-made mineral fibers,
the so-called vitreous fibers in buildings,
whether or not these are of concern, and
finally, research needs.
Q. Were the risks, as calculated in the
Summary to the Harvard Symposium, one in
100,000; that is, the risks of asbestos related
to the death for somebody in a school?
A. I don't recall. Why don't you give me the
paper and let me look at it.
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Okay. I think this is it, sir, and
this is the summary, also. Oh, that's what I
want, the summary of it. Q. I think that may be it.
A. Yes, Spengler and colleague's summary. MR. BROWNSON: I'm going to object
to the form of the question. I think the risk calculated was in buildings, not
schools. A. Buildings, yes.
Now, the question is: What is the
risk in?
Q. The Summary of the Harvard Symposium to persons in buildings? A. In conclusion, number seven in the executive summary, the following statements are
present:
Recent data indicate the average
concentration of asbestos in schools and other buildings is that asbestos-containing materials
is generally well below the 0.001 fibers per ML used in risk calculations performed for school
children. Of course that's mixed fibers of
different types T.
Using these conservative risk models
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of exposures higher than typically measured are
the projected lifetime risk from exposure to
mixed asbestos fibers is one death among the cohort of one in a thousand children.
Q. Now, to the HEI executive summary
table, l-l, Page l-ll, also has some lifetime
cancer risks, is that correct?
A. Yes, it does. Q. Do you consider those risks to be
substantially the same as the Harvard University
risks you just read for exposure in a building?
A. Are they the same, you said?
Q. Yes, sir.
A. Okay. Reading this table, we're talking
about school-aged children, 5 to 18 years, 180
days per year, five hours per day. Given two
different averages which are different by an
order of magnitude, one is five times higher
than the value that we just read .001, and the
other is one half that value.
So, therefore, we're dealing with an order of magnitude difference in dose, and then
given these various conditions, the premature
cancer deaths, the lifetime deaths per million.
So six per million would be .6 per 100,000.
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Let's say it's one per 100,000, at half the dose
given in the Harvard school, and the 60 per million or six per 100,000 is the risk which is
five times higher.
So, are these values the same? Yes,
they're pretty much the same. Q. Do you think that in making risk
predictions that a difference of five times
something you can say is basically the same?
A. No, no, no, I didn't say that.
Q. Okay. I misunderstood you, then.
A. The Harvard, Spengler, et al., executive
summary says, given the fiber level of .001, it's
approximately one in a 100,000. This table says
given a level .005, which is five times the
value of 001, is approximately 60 per million,
or six per 100,000. So, you have five times the
level, linear five times one is five, so they
have six instead of five. So, they're given the
numbers five and six. They are statistically
identical. So this is the same model and the
same people crunching the numbers.
Q. Thank you, Doctor.
On Page 1-6 on the AGI executive
summary, there is a figure 1-1.
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Can you turn to that, please?
A. Sure, yes. Q. Can you explain, if you have an
answer, to why the mean value for average air
board concentrations in non-litigation data by
building type is at least twice as high in
schools as in all buildings, residences and
public and commercial buildings?
MR. BROWNSON: Can you just read
that back for me, please? I'm not sure I
caught the question.
(Whereupon the pending question was
read back.) MR. VALENZA: Objection.
MR. BROWNSON: I'll object to the
form. Go ahead.
Q. Did you understand my question,
Doctor?
A. I understand the question that I'm looking
the data and that which you've stated is
correct, the schools are twice as high as these
other structures. So, if the schools are at
0005 and these are about 00025, they're about
half, that is correct.
Why is it higher in the school
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buildings? There have been a lot of interesting
hypotheses generated, it may be the activity in
the building, it may be any number of reasons,
but can I answer it definitively? No, no, I
can't do it.
Q. Do you have an answer that you
believe is more likely than others?
A. I'm sorry?
Q. Do you have an answer that you
believe is more likely than others?
MR. VALENZA: Objection. Speculation.
A. No.
Q. Okay. Doctor, the last category of
documents on which you will specifically rely in
this case as listed in Exhibit 3 is the article
you wrote with Drs. Wilson, Nolan, Gee and Ross,
titled: "Asbestos in New York City Public School
Buildings - Public Policy: Is There a Scientific
Basis?"
I have just a couple of questions to
ask you about that, and if you would like, I'd
put a copy of the article in front of you and if
you want to refer to it, you may now.
First of all, sir, did you receive
any type of grant or sponsorship, financial
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sponsorship from any asbestos litigation
defendant for writing this paper?
A. No. Q.
Do you know if anyone did?
A. No, I'm quite sure no one did.
Q. Okay. There is a specific reference
to an analysis of a sample in one of the New York City
schools. It's on Page 165.
Was that sample analyzed as part of
the litigation?
A. Yes. Pending litigation, correct.
Q. There are, of course -- I take it
that some asbestos defendant paid for the
analysis of that sample, is that correct?
A. Yes.
Q. Which defendant was it?
A. Asbestospray.
Q. Page 162 is the following statement,
the first full paragraph: "NYC's Board of
Education was not required by law to measure the
asbestos fiber concentrations in air, or the
type of asbestos present before any asbestos
abatements was to take place."
Do you believe that it should be so
required by law?
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A. Should it be required by law?
Well, I think before any abatement
activity, number one, you want to make sure you have asbestos in the product that you are abating, ripping out. You may think that this
is trivial and kind of stupid, but let's carry
on from there. Do I believe that it should be
required to measure fibers in the air? I think
that if someone gives advice, they should base this on some perceived, or some not perceived so
much, base this on some estimate of risk associated with each of these instances.
Q. Therefore, do you believe that, by law, they should be required to consider: Number one, concentrations in the air; and
number two, the type of asbestos present before
any asbestos abatement?
MR. BROWNSON: Well, I guess I'll object to the form of the question, and I'm
not sure what the exact legal objection is
other than the fact that you're asking for a
hypothetical legal conclusion.
A. Yeah, it is a legal conclusion.
MR. BROWNSON: Go ahead.
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A. I'm one of those people that believes you
ought to do what's right without having it
mandated. Q.
You believe certainly --
A. What do I believe? Let me just interrupt
you. Let me finish this.
If, let's say, you are a school district, like Chicago, New York or Los Angeles, and you're going to expend $125 million in an asbestos abatement activity, I think that you
should know something about the risks that you
are addressing. I think the way to do that is
to measure that which is the hazardous elements of environment, which means measure fiber in the air and come to some understanding as to, at
least prioritize various buildings and places
and so on and so forth. Some might have a measurable risk, others not, but you ought to do something in terms of establishing a data base
upon which you will base your decisions.
Q. Do you believe that another thing a school district should do in terms of
prioritizing is to establish the type of asbestos
present?
A. Yes, I do. I certainly believe that.
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Q. Would you, in general, all other
things being equal, recommend the removal of an
amosite-containing, asbestos-containing material
before the removal of a chrysotile
asbestos-containing material?
A. You mean, all of the factors equal?
Q. Sure.
A. Probably. Q. On Page 163 of your article is a
list of EPA guidance documents.
Did you have any connection with the
Yellow Book?
A. No.
Q. Did you have any connection with the
first Green Book of 1983?
A. I don't think so. I may have reviewed it,
but I'm not sure.
Q. Did you have any connection with the
Silver Book of 1984?
A. No, I don't think so.
Q. Do you still agree with this
sentence which is written on Page 166, "The
acceptance of risk is a social and moral issue,
and scientists are no better qualified than
anyone else to decide whether a risk is
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acceptable or not"?
A. Absolutely. Q. You also still believe that
scientists will provide thebest available data
for decision makers and thatscientists should
be consulted?
A. Sure.
Q. Okay. On Page 166, at the very
bottom, just before the heading "Other
approaches to address asbestos in schools," is
the statement: "If such removals have taken
place, the lifetime risk of school children and
teachers increase," citing the US EPA 1990,
which in the back, you indicate, I believe was
the Green Book, is there a specific statement in
the Green Book, this being the second Green Book
that says, "If there's a poorly executed removal
program, that will increase the lifetime risk of
school children and teachers"?
A. I think that's listed directly from the
Green Book, or is the citing of a study in Great
Britain, and I'm thinking of Bridet and Rude
(phonetic), their study which showed elevated
levels in buildings after abatement, school
buildings, that followed the six months after
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abatement. So, we're talking about the actual
fiber levels where they talk about that in that
particular reference. If you calculate the
fiber levels found before, during and after, the
lifetime risk of those individuals increased.
Q. This was done in the Green Book, you
believe?
A. I think so. As I sit here and recall it.
Q. I'm now going to ask you a couple
questions, Doctor, about your article titled
"chrysotile, its occurrence and properties as
variables controlling biologicaleffects,"
and I
have a copy of it in front of you, in case you
would like to look at it.
A. Sure.
Q. Do you know, first of all, which
industry sources gave funds to support this
conference?
A. No.
Q. Do you know any of them?
A. No. This conference was to be supported
by an international program for chemical safety,
but I think NIOSH withdrew some support, so they
generated funds from other places.
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Q. Do you believe that the mechanical
manipulation required for the industrial
application of chrysotile can increase the
carcinogenic effects of chrysotile?
A. Yes. Q. Specifically, what is there about
that mechanical manipulation which leads to an increased carcinogenic effect? A. We're talking in the generic sense now?
Q. Yes, sir. A. We're not talking about the degradation, the grinding of stuff -- "stuff", that's such a bad word -- the grinding of fiber which alters surface properties decreases crystallinity or
thermal shock brought about by friction product usage, things like that, that decreases it.
It is the opening of the fiber
bundle, for example, in the textile industry, in which you get a lot of long, thin fibers
generated in the willoving and spinning
processes in asbestos textiles, which increases the carcinogenic textile of this mineral fiber, yes.
Q. Do you believe that that manipulation
is responsiblefor the results
found in the
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Charleston textile mill study? A. It's one of the possible explanations,
yes. Q.
Do you know of any other possible
explanations? A. You mean, for lung cancer in Charleston,
South Carolina?
Q. Yes. A. As you know, Charleston, South Carolina is
the northernmost extent of the cancer belt in
the United States, which begins around the Gulf
Coast and goes up the coast of North America.
It's a paper written in 1976 by Bill, Blott and
Fraumin (phonetic). They talked about cancer
belts in the United States, and Charleston
happens to be one of the last of the high cancer
areas for lung cancer in which the SMR's,
standardized mortality ratios, for lung cancer
are elevated above national background. No one
knows quite why. That's one explanation, that
the referent population used by Dement was the
wrong one.
There's another one given by Corbit
McDonald. He seems to favor the use of oils in
the dust suppression as being another factor in
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this.
There is the third additional
factor, Charleston's a big port; city people are
exposed to amphiboles, that's another factor.
Then there's a factor that the
actual machinery, itself, broke apart the fiber
bundles and produced an aerosol with
characteristics unlike fibers found in other
environments.
Then there's another factor which is
not stated by Dr. Dement, that the tissue burden
study was done by Fred Pooley in Great Britain.
Fred Pooley found crocidolite, and those
tissues showed that those working in this plant
also spun crocidolite for a short time, so
there's many of these confounders and variables
which haven't been embellished or brought out in
the ideology of this peculiarly high incident of
lung cancer by physical chemical properties.
one of these -- and I'm one of the
people who believes that we were dealing with a
very interesting dust, meaning, it was -- I
don't understand the resolution of the light
microscope, it was very thin, but certainly
sufficiently high in long, thin fibers to be
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produced in the thesis.
Q. How do you know that Pooley found
crocidolite fibers?
A. He told me, and from -- the records also
report these, I think they're reported in the --
I think they are reported in the General Motors
biological effects of chrysotile, the meeting
which was held by Chris Vogna, in Cardiff, in
1986 or '85, something like that.
Q. Why hasn't that information, which is
now almost a decade old, in terms of going to
the public, become generally accepted as the
explanation for the excess lung cancer at
Charleston?
MR. VALENZA: Objection.
MR. BROWNSON: Object to the form of
the question. I think he said it was one
factor to be considered.
Q. Do you think that that could explain
all of the excess lung cancers?
A. Ho.
Q. Okay. That's fine.
Let me ask you --
A. The next time you're speaking with John
Dement, ask him a perfectly good question.
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Q. Did you read his article on the
subject in The Third Wave Book?
A. I've looked at it, I have not formed an
opinion as yet. Q. You referred to the harsh form of
chrysotile as opposed to the -- what's the word
you used?
A. Soft. Q. Do you believe that the harsh form
of chrysotile, which you say on Page 438, tends
to behave after amphibole asbestos fibers were
aerosolized?
A. Correct.
Q. Could be one of the reasons why
chrysotile can have a carcinogenic effect?
A. In part, yes. I think that was what I was
trying to do in that paper. I was trying to
explain the differences seen among different
cohorts around the world and explain the different experiences on the basis of the
physical chemical properties in the materials,
themselves. There are a number of factors which
have not been properly addressed, and this is
just another one of them, yes.
I'm just so tickled pink someone's
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read that paper. (Whereupon a discussion was held off
the record.)
BY MR. MILLER: Q. There's a statement on Page 448 that
"Tremolite and amphibole material is important
in this regard since tremolite asbestos in
appropriate concentration is considered to be
the agent responsible for pleural mesothelioma
found amongst chrysotile exposed workers."
Do you believe that?
A. Sure.
Q. By "chrysotile exposed workers," are
you talking about workers in the mines and mills
in Canada?
A. It's possible.
Q. Your last paragraph states,
"Mineral nature of chrysotile controls
biological behavior. These factors must be
considered in developing any health criteria
document in resolvinghealth effects following
exposure to dust."
It you were able to legislate or not
legislate, at least to give advice to someone,
what types of factors would you ask them to take
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into effect based upon your research in this
paper? MR. BROWNSON: Excuse me. I'll
object to the form of the question. Go
ahead.
Q. You really what, Doctor?
A. I'm not sure I would.
Why do I say this? I know that
there are all of these interesting observations,
experimental observations, epidemiological
observations, and they point to many, many
interesting aspects of this, but if I was a
regulator and I was given the fact that I'm
going to have an industrial hygienist, certified
industrial hygienist, a certified industrial
hygienist using a phase contrast microscope
going out to the field and doing dust counts, I
would not do that. I would have a bulk sample
read that generates fiber type and regulates
across the board.
If we're dealing with -- the best of
all possible worlds would be other techniques
and other instruments, but in dealing with the
pragmatics of everyday life, and the challenges
that face us all, I would probably not regulate
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thaw differently, I'd probably just use one
standard for chrysotile.
MR. MILLER: This is 6.
(P-6, report by Dr. Langer, received
and marked for Identification.)
Q. Dr. Langer, this picture I was
referring to when I said some of your pictures
come out pretty well, do you like the picture?
A. No, let's go on.
Q. Do you like the title?
A. Voice of reason, I think that that
underestimates my true value and worth.
Q. Tell me how you got involved with
U.S. News and World Report, in this article?
Somebody called you up, you called them or what?
A. Do you want to hear the whole story, for
the record?
Q. I do.
A. It's fascinating.
Q. I'd like to.
A. Well, normally when I tell the story, I
have a single malt Scotch in one hand.
Q. I wish I could.
A. It all started out with New York City's
asbestos episode in the fall of '93, and I wrote
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a latter to the editor of The New York Times. I
was joined with several colleagues, specifically
those who are on the paper in the Journal of
Regulatory, so on and so forth. The New York Times did not publish
this letter, and we basically said, ''Wait a
minute. Before you start to invest a lot of
money and time and it's evident that these
people really are at risk, why don't you do some
air measurements of data from other schools,
indicate that there's very small risks." We
just completed a small study in one New York
City school and we don't see very much, so on
and so forth.
The New York Times chose not to
publish that paper. To add insult to injury,
they published a letter to the editor by an
asbestos abatement contractor, the president of
the company, who said something grotesquely
stupid. It was ghastly.
The other was an Op-Ed piece by a
well-known ideologist, whose name I seem to have
forgotten, so I decided --
Q. Somebody who's at Mount Sinai?
A. Well, I've forgotten that, but I've
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decided that if this is the case, then we ought to get a paper out somewhere, and I did the
first draft of a major paper which was then
circulated among our colleagues. We sent it
down to the Journal of Regulatory Toxicology and
Pharmacology. We sent it out. When they
reviewed it, they said, "Hey, this is a hot
topic. Why don't you guys publish
this with a
few minor changes," and so on. Minor changes,
there was too much data description, but we
did. We cited a lot of chapters and verses and
all of the documents that are outlined, so we
had to reduce that significantly.
They said,
"Yes, we're going to publish this."
Now, this paper was then picked up
by someone in the Office of Technological
Assessments in Congress. They were doing a
study of problems in U.S. schools, and I was
contacted by this person. Of course, the
problems in U.S. schools focus on the physical
problems of radon, lead asbestos.They also had
several other interesting areas, like violence,
and so on.
This was picked up by the OTA.
Then, the preliminary report was circulated in
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Washington and someone picked it up from U.S.
News and World Report. They contacted me
saying, "We've seen this particular paper.
We're interested in the New York City experience
and that particular episode. What did the EPA
do, and what didn't they do?"
Of course, they interviewed me for
two hours and took what they wanted to take and misquoted me there, but nonetheless, they
contacted me and this was an evolutionary
process because The New York Times didn't
publish our letter to the editor.
Q. Is it fair to say that you gave the
reporter for U.S. News much or most of the information published here about the history of
the EPA?
A. I don't think so. I think the EPA's
history is eloquent and speaks for itself.
Q. I'm trying to figure out how the
reporter heard it.
A. The reporter spoke to many, many people,
and my selection is mysterious, to me, anyway.
q. Did you have any contact with John
Welch during this process of giving an interview
to U.S. World Report?
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A. No. As a matter of fact, John Welch, I
think, has left the Safe Building Alliance, as
far as I believe. Q. I believe he's quoted here as being
with the Safe Building Alliance, at least
president of a former manufacturer of asbestos
products.
So at the time of this interview,
he's thought to be there, but you had no contact during the course of this?
A. I don't think so.
Q. Is the U.S. News and World Report a journal on which you would customarily rely in
the medical or scientific field, that you would call authoritative?
A. Would I call it authoritative? Is this a real question?
Q. Yes.
A. I don't consider lay press as authoritative, any more than I would consider a
book by Paul Gradore (phonetic) to be
authoritative in the sense of an unimpeachable
source of scientific source.
Q. Let me ask you a few questions about the article. I believe you have a copy in front
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of you. Here it is. Sorry, it never quite got
over to you. The first paragraph of the article
talks about a number of precautions that are
taken by a man named Bill Lee before he can
replace an offending valve.
Do you believe those cautions are
wise and appropriate from a health point of
view?
A. I don't know.
MR. BROWNSON: Well, I'll object to
the form of the question.
A. I don't know. Is the pipe wrapped with
fiberglass or is it wrapped with crocidolite?
Q. Assume that it has some form of
asbestos around it.
A. Well, then, what is the percentage of that
asbestos; 90 percent or one percent?
Q. If it were 90 percent, you might
agree that the cautions were vise. If it was
one percent, you might say it's not wise, is
that how I should interpret that response?
A. No. What we're dealing with is some kind
of product on a pipe, and this product has been
in service for a number of years, but this
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product may have been in service for two months,
this product may have been in service for 50
years. One product may have an integral
physical condition, another may be more, to use your word, EPA's word, "friable," some of these
materials may contain a crocidolite, others may
contain goat hair, so there are different
materials, there are different conditions, there
are different scenarios, so to speak.
Now, let's assume that this is a
worst-case scenario. Let's say this is the
stuff that's a very high temperature pipe and
it's a combination of chrysotile and amosite.
Do I think these precautions are
warranted, sure.
Q. In the middle column on the first
page there's a sentence about six or seven lines
down, starts "Across the nation."
Do you see the sentence. Doctor?
A. Yes.
Q. "Across the nation, school districts
that can't buy new library books are spending
hundreds of thousands of dollars to deal with
asbestos because they mistakenly believe their
children are in great danger."
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Do you agree with that?
A. It's possible that there are some school districts that possibly cannot buy new library books that are possibly spending hundreds of thousands of dollars to deal with a problem in
which there's asbestos in the basement or asbestos above suspended tile ceilings in which
the levels in the buildings are 0001 and kids
can't buy new math books. I think that that would be a mistake
of the administration. These are your risks,
these are your quantitative risks. Here are your children, they're out there scoring 50 points
less than the school there on their math SAT's. What do I think?
I think you ought to reconsider
removal, yeah, I think that it depends.
Q. In your third column under the middle sentence, right next to the blurb, U.S.
News Investigative Report, it starts with,
"there is now broad consensus among scientists
and physicians that asbestos in public buildings is not much of a threat to health."
Do you agree with that?
A. Consensus, what does that mean to you.
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Counsel? Q.
I'm asking you. Doctor. I don't
know what it means. A. Well, I don't know what it means, either,
if there's a consensus.
MR. BROWNSON: I'm sorry, so we're clear, you're asking if he disagrees with
the consensus, or if he agrees with the
statement that there's that much of a
risk?
Q. The first statement: Do you believe
that asbestos in public buildings is not much of
a threat in health?
A. From the data that I've seen, I think it's
pretty close to the truth.
Q. Now, the next sentence: Do you
believe there's no broad consensus among
scientists and physicians that asbestos in
public buildings is not much of a threat in
health?
A. I haven't the foggiest.
Q. The last paragraph refers to a study
soon to be released by the federal Office of
Technology Assessment.
Is that the same study which you
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were referring to? A. What is its technology, yes.
Q. Have you seen any conclusions from
that study?
A. Not yet, no, I haven't, but they're quoting Richard Wilson, isn't that right?
Q. Not for that purpose, no.
A. I see. Okay.
Q. Do you have any idea what that study
will say regarding asbestos in schools?
A. No.
Q. On the next page, sir, there's a
sentence beginning at the top, "Less
understandable is the role of government
agencies, especially the federal Environmental
Protection Agency, which created a public panic
on the basis of paper-thin scientific
information."
Do you agree with that, sir?
A. What part of the statement?
Q. Any part. I'll divide it up as you
wish.
A. Less understandable is the role of the
government, yeah,that'sprobably
true,
especially the EPA, that's lessunderstandable,
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more or less understandable. You have to have
been there and to know the players in the game
as to who advised what, which created a public
panic. Well, it's not so much a public panic.
People didn't know what to do, so they did what they thought the EPA was telling them to do,
which was to take out asbestos from buildings on the basis of paper-thin scientific information.
Paper-thin scientific information,
well, what did we know? We knew asbestos in the workplace
was dangerous, but it was a function of dose based on the risk calculations and fiber
levels to which people were exposed. They had
less of a risk than we certainly knew, so it's kind of overwritten, yeah.
Q. They say -- they're quoting you in the sentence, "When everybody's shouting 'Fire!'
in a theater, the man who quietly stands in the
corner and says, 'There's no fire,' is rarely
heeded."
Is that a correct quotation of what you told the interviewer?
A. Not bad. Yeah.
Q. In fact, is that what you believe
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you told the EPA during the years when you were
there consulting as a peer-reviewer on those three
rainbow books we talked about?
A. Three rainbow books, 1979, I thought that
friable asbestos was a problem in school
buildings. I participated in that. As time
went on and more data became available, I was
less convinced that there was a problem, and
probably by the Health Effects Update.
As a matter of fact, I wanted to put
in a section on fiber type and risk associated
with the fiber types because I believe that an
inspection should focus on and prioritize
problems in buildings. So, I was much in favor
of a fiber-type kind of approach to this
problem.
Of course, as time went on and the
Health Effects Update was published in 1986 and
those documents were made available and people
were measuring fibers in buildings, that I no
longer believed that there was much of a problem
in buildings.
Q. You talked about your wanting a
section on fiber type.
Where, in regard to what paper did
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you want a section on fiber type? A. This was in the, I think it's the Purple
Book. When we talked about algorithms and such,
I was interested in a fiber type section.
Q. What happened to your request for
fiber type? A. It was never put in, they dismissed me,
like I was a shoe salesman. That's the phrase
that's used, I was dismissed; in more ways than
one. Q.
Were there other specific advices
that you gave during your work at any of the three rainbow books that were not followed that
you can recall at this time?
A. Well, yes, I think that at these very same
meetings, when a hearing was being formulated,
the Asbestos Hazard Emergency Response Act, I objected to the language and I objected to the
use of emotive words, and thought that the data
pointed the other way and that people should be
calm and not pushed into any action without some
data base; but that was never done, neither.
Q. Under the picture of you is the
statement, "Professor Arthur Langer kept trying
to warn policy makers they were overreacting."
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Is one of the places where you tried
to warn policy makers they were overreacting was
in AHERA conversations you just described?
A. I would say that every year I attended a
number of meetings, both nationally and
internationally, in which there were members of
various federal agencies, and many discussions,
public and private, followed. As time went on, I continued to
voice the opinion based on observations and
analysis and analysis of data, that the risks
posed by asbestos products in buildings were
overblown, and the very first time that I
participated in such an exercise with a
colleague, we evaluated documents brought
forward in the Service Employees International
Union lawsuit against the Environmental
Protection Agency, in which we evaluated the
risk analysis which was being proposed at that
time by a Bill Nicholson, and had lots of
concerns. I mean, there were official concerns,
there were unofficial concerns, but this was a
continuing kind of dialogue for various members
of government.
Q. When was the first time that you
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believe you tried to warn policy makers they
were overreacting, what year?
A. Probably 1983, maybe earlier, but
certainly by '83. Q. The Blue Book came out in 1983.
During the course of those discussions
and that review in the Blue Book, did you try to
warn anybody at that time that you thought they
were overreacting?
A. You keep using this word "warn.'* What's
"warn"? Don't walk on 42nd Street against the
light, that's a warning; open and broad and
frank discussions as to the contents of certain
documents, I thinkthat's moreappropriate.
Q. Okay.
A. Did I raise concern? Did I bring forward
any objections? Did Icritique the contents?
Did I recommend alterations of text? Did I
carry forward the normal kind of operations of
an outside reviewer and contribute to a federal
document? Yes; did I try to warn someone,
that's kind of, you know...
q. Take out the word "warn."
What I'm interested in is the idea
that you believe the federal policy makers were
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overreacting. Can you tell me when you first, in
some way, indicated to federal -- A. Well, the federal policy, did they
overreact? They didn't do anything other than
write these documents. Everyone else reacted,
some more than others, and therefore, you might
say they overreacted.
What did the feds do? The feds put
forward a position that generated position
documents, and they said, "This is our position:
We think you ought to have an inspector come
in. We think if you follow this algorithm of
conditions of materials and places in buildings
where they exist in proximity to air supply," so
on and so forth, "air supply units, and whether
or not these materials are easily accessible and
whether or not they are," and there's a whole
strategy for coming to some conclusion.
Each of these factors are then multiplied by some normalizing weighted factor,
and there's a number that's generated. On the
basis of that number, one proceeds in certain
ways to ameliorate. This is the algorithm
technique, without using microscopy to measure
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fibers in the air. This is a whole evolutionary
process that I did warn people, you discuss it;
"this algorithm may work, it may not." It turns turned out the algorithm
didn't work. It turns -- out the material was, as
to prognosis data, the fibers in the air neither
is fiber content nor fiber percentage or this or
that.
What is it that you have to do? You
have to measure fibers in the air, that's what's necessary to calculate risk. This is the bottom
line, and did I warn people? That's such an
unfortunate word in this instance; you discuss.
Q. On the second page in the third
column on the right-hand side there's a heading in bold, "On second thought," and then there's a
sentence, "But by 1985, some notable
environmental health scientists were backing
off."
Were you one of those?
A. I wouldn't characterize myself as a
notable, that's rather self-aggrandizing, don't
you think?
Q. Were environmental scientists backing off in 1985?
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A. You mean in asbestos school buildings?
Q. Just as discussed here, and you can
feel freeto look at the exact context. A. Let me read that. Where is that?
Q. Second page, right-hand side
underneath the picture. Under the picture it
says, "On second thought," in bold heading.
Would you say you were one of those
there they' re describing?
A.By 1985, yeah,
maybe I would consider by
1985, '86 for sure,
by the time the models were
more available and characterized by.
Q. Turning to the next page, the last page, it's Page 63, at the top in the first
column, bold headline, "Powerful convert:
Perhaps most important, Selikoff himself had
drifted from the position of being a neutral
research scientist to that of an antiasbestos
activist. He told a congressional panel in 1984
that risk posed by asbestos in schools was
intolerable."
Do you agree that by 1984, Dr. Selikoff
had drifted fromtheposition
of being a neutral
research scientist to that of an antiasbestos
activist?
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181
A. Do I think that's accurate?
Q. Yes, sir.
A. An antiasbestosactivist, no. Q. How would you characterize?
A. Who, Irv?
Q. Irv's position, changes,
modification, if there was any in the 1980's?
MR. BROWNSON: With respect to
asbestos in buildings?
Q. Yes.
MR. BROWNSON: Okay.
A. I don't know. Q. Do you believe he did change in any
way?
A. No. I think Irving wasalways himself. I
don't think he changed very much, no. He was
right on one side and I think he was wrong on
another issue.
Q. What side was he right on and what
side was he wrong?
A. I think that his work on exposure to
asbestos in, for example, the insulation work,
was the best work ever done in that field,
without question, that Selikoff, Hammond,
Cherring, Canastein (phonetic), and others.
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Q. You were part of that too, weren't you? A. I was involved in some of it.
Irving was brilliant and articulate
and charismatic and carried the day in that
area.
Q. Where was he wrong?
A. He was wrong in not considering dose
response.If hisinsulation worker
were exposed,
on average, to somewhere between 10 and 20 fibers
per cc of air, it begged that question: What
will exposure to .001 fiber -- and based on the
models generated by one of his own people,
Nicholson, the risk was very much less, and the
question begged was: Can we provide an
environmentwhich isabsolutely safe?
I think
that it is a question which is decided by
society. That's not an answer that anybody else
can reach a resolution on, those issues;
nevertheless, I think his contributions are many
in a brilliant and occupational sphere, but
diminished in the environmental setting. I
think that hisposition wasextreme, and it's
unfortunate.
Q. While you were still at Mount Sinai,
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did you ever bring up with either Dr. Selikoff or Dr. Nicholson or anyone else the points
you just made: What about .001 in the Nicholson
risk model? A. As you know, Dr. Nicholson served on the AGI
committee and we had many, many, many open discussions at a table just like this one, where
we discussed all of this. Everyone brought
forward their data, and Nicholson was on one
side, Peter was down there, and I was here, and
we argued this loudly. Nicholson also would not
be moved nor persuaded by our arguments, and I
was entitled to have a different discussion with
Irving Selikoff. No.
Q. My question wasn't clear. I
wondered if before you left Mount Sinai --
A. Did I have a discussion with Irving
Selikoff concerning this subject?
Q. Or anyone else there at that time
before 19 --
A. No. Nicholson, I'm sure we banged heads
on this once or twice.
q. Before you left?
A. Before 1988?
Q. Yes, sir, that was my question.
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A. Before 1988, yes, I think we discussed
this.
Q. Okay. MR. MILLER: Off the record. (Whereupon a brief recess was taken
at 3:15 p.m.) (Whereupon the proceedings resumed
at 3:20 p.m.) BY MR. MILLER:
Q. Doctor, do you agree that chrysotile can
cause pleural mesothelioma?
MR. SENFTLEBEN: Objection to the
form. A. If present in high enough concentration, possibly, yes.
Q. Do you believe that chrysotile can
be an initiator of lung cancer?
A. It's possible.
Q. Do you believe that amphiboles can
be initiators of lung cancer?
A. It's possible.
Q. Do you believe that asbestosis progresses after exposure to asbestos --
A. What grade asbestosis?
Q. That would make a difference to your
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results, sir? A. Yes.
Q. Explain the different grades of asbestosis and how your answer would differ.
A. I believe it is like a silicosis, the more
the pulmonary burden, the greater the chance of
it progressing after cessation of exposure, but
if the dose is low enough, I believe the process
stops. Q.
On what evidence do you base that
conclusion? A. On the basis of some animal studies which
showed very minimum scarring or no scarring
present after very low exposures to animals, but
with higher exposures, the lungs are progressively
scarred. As a matter of fact, I'm sitting here
thinking, maybe some of Chris Vogner's early
work showed that, but its outcome was malignancy
among scarring, but it's just reported.
Q. Can you state to a reasonable degree of scientific certainty that no one will ever
contract an asbestos-related disease in any of
the schools in the plaintiff's case in which
Asbestospray T is allegedly present?
MR. BROWNSONj I'll object to the
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form of the question. Go ahead, to a
reasonable degree of scientific
certainty. A. Well, to a reasonably degree of scientific
certainty, if you use the risk models, eventually,
maybe not this century or millennium, but in the
next, there may be someone who might develop
mesothelioma on the basis of exposure.
Q. Can you state to a reasonable degree
of scientific certainty that no one ever has, in
the past, contracted an asbestos-related disease
in those same schools?
MR. VALENZA: Objection.
MR. BROWNSON: I'll object to the
form of the question.
A. No.
MR. BROWNSON: Calling for
speculation.
A. Which of theindividuals areyou talking
about; occupants; maintenance workers?
Q. Any persons.
A. Any person?
Q. Any persons.
A.
No, I wouldthink thatsomeoneengaged
in
maintenance activities which are carried out
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without the proper precaution may have developed
some asbestos disease, it's possible.
Q. Would you ever refer to the presence of asbestos in a surface in a school where it's
not an ingredient in surface material, for
example, asbestos in surface dust, would you refer to that asbestos as a contamination?
MR. VALENZA: Objection. Calls for
legal conclusion.
.
Q. Using your scientific work?
MR. VALENZA: I object to the term
because it is susceptible of many
different reasons, one, which has a legal
scenario, and I object to the use of the
word, but the doctor may answer. A. You mean, if I find fiber in a settled
dust, and settled dust in an interior
environment normally does not contain that
particular kind of material, would I call it a
contaminant?
Q. Yes.
A. Yes, I probably would, or adventitious
particle.
Q. Is there any particular numerical
connotation of asbestos in the dust that would
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be required before you would call it a
contaminate? MR. VALENZA: Objection. Same
objection.
A. I wouldn't, but there may be some nomenclature which I'm not familiar with.
Q. Have any of your published articles
ever been supported by a grant from a defendant
in asbestos litigation?
A. Sure.
Q. Every time that's happened, has
there been an acknowledgment made in the
publication that it was supported, at least in
part, by a grant from a defendant?
A. Well,mostcertainly, of course.
Q. In those cases where an asbestos
defendant has the support by some form of grant,
an article you eventually published -- strike
that.
Would you believe that it would be
proper for you to give a draft or advance copy
of that article to the defendant or its counsel
for their review prior to submission of the
document for publication?
A. I think it's improper.
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189
Q. I take it you've never done that, is
that correct?
A. No. Q.
No, you have done it? It's a double
negative, that's why I want to be very clear.
A. No, I have not. Q. In the OSHA regulations that were
published Wednesday, August 10th, 1994, there
are a couple of statements I want to ask you
about . I have the regulations here, if you
want to see them.
A. Just read them to me.
Q. Do you believe that a significant
level of risk exists at 0.1 fibers per?
MR. VALENZA: Objection.
MR. SENFTLEBEN: Jon, this is a
hell of time to bring it up, but
I assume you're operating that an
objection by one is an objection
by all?
MR. MILLER: Yes.
A. 0.1, is that the level?
Q. Yes, sir.
A. The term used is significant, which is
OSHA'Ss term.
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190
Q. Yes, sir. A. 0.1, that would be 1/100 of insulation
work. All factors being equal, the risk of
mesothelioma would be one in a thousand, and, of
course, per thousand, let's just calculate this,
let's say there's a 17 percent -- 16 percent
excess lung cancer, itwould be 160 and a hundredth
of that would be 1.6, so it would be roughly
three excess cancer deaths per thousand workers.
Now, you say there's still a
significant risk. It's
okay. Three in a
thousand, yeah, I would say that's still a
significant risk.
Q. At approximately Page 40979, they
give their reasons for not separating the fiber
types.
A. Oh, this is good.
Q. Do you agree with their decision not to
separate fiber types?
A. What are their reasons?
Q. Well, there are lots of pages.
We'll start with at least three reasons.
A. Start, please.
Q. I have to show it to you, and you
can tell me if you agree with the reasons. I'm
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pointing to where they start with the three
reasons they made mention of.
A. "As OSHA explained in the Preamble to 1986
standards ... to summarize." MR. BROWNSON: Just for the record,
you're reading aloud here. (Whereupon a discussion was held off
the record.) MR. BROWNSON: Let's start over and
pose a question here.
A. You better ask me a question.
Q. Dr. Langer, as we have noted, OSHA
says that there are at least three reasons why they have decided not to separate fiber types.
Would you please read into the
record each one of the reasons, and whether or not you agree with them and the reasons for your
agreement or disagreement.
A. "To summarize the data on risk differential
by asbestos fiber type, human epidemiological
studies have suggested that occupational
exposure to amphiboles is associated with
greater risk of mesothelioma than is exposure to
asbestos."
I agree with that, so they're saying
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192
basically you're exposed to amphibole asbestos
fiber. It's worse for mesothelioma induction.
That certainly is true. However, they go on to say, "No
clear risk differential for lung cancer or other
asbestos-related disease has been demonstrated
by epidemiological studies." Not true.
The increased risk of gastrointestinal
cancer is found among amphibole exposed cohorts.
No clear risk differential for lung cancer is
true, and that is why I have written papers on
the properties of chrysotile, as related to the disease risk.
So, for lung cancer, that's probably true. So what OSHA had said is: Yes, there are
more mesotheliomas with amphiboles. They have a
mistaken notion that GI cancer is the same as
amphibole cohorts and chrysotile cohorts. Nay,
not so.
That there is no clear differential
for lung cancer, that is true.
Animal experiments. All of a sudden
they're drawing on animals. "However, have
indicated that chrysotile is a more potent
carcinogen than amphiboles when administered by
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DR. LANGER - Direct
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inhalation or intrapleural injection." This is generally true. This is
generally true, that when one uses route of administration, such as intracavitaries, as they
call it, you do induce more tumors with
chrysotile, this is so; by inhalation, is it
true?
That's the Vogner experiment, and
also John Davis. It's probably true. So the
animal studies support this, as well, but why they
have it with epidemiology, I don't understand.
It says OSHA agreed with the
testimony of Dr. Davis, who stated that, "the evidence cannot answer with certainty if one
fiber of amphibole is more dangerous than one
fiber of chrysotile."
That begs the question: Are the
amphiboles more dangerous? I think so, yeah.
Do I think that chrysotile exposure
in the textile industry is very dangerous? Yeah,
I certainly believe that.
Do I think the stuff in products in
buildings is more dangerous? Ho. I think the
properties differ significantly.
so, this first part of this OSHA,
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that first part of it is an embellishment of all
of these kinds of facts. "Second, as stated in the 1986
asbestos standard, even if OSHA were to accept the premise, which it does not, that chrysotile
may present a lower cancer risk than other type
of fiber types, occupational exposure to chrysotile asbestos still presents a significant risk of disease at the revised PEL." Yeah, it's
probably so. I think so, significant risk. The revised permissible exposure
level is -- let's say that they are interested
in 0.1, it's 100th of insulation worker. I
mean, all the factors equal, insulation workers
are exposed to amphiboles. Most of their --
much of their risk is enmeshed in mesothelioma,
which is an amphibole component exposure.
It's fascinating, this exercise is.
"In particular, asbestos, the disabling and often fatal fibrosis" -- I don't
believe that. It's not often fatal. Many
asbestosis cases live out their lives.
-- "of the deep portions of the lung is caused by exposure to all types of asbestos."
Yes, the evidence on this is strong.
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okay. All right. Let's say it's true.
Okay. "Lung cancer risks associated
with chrysotile exposures are also high - 6.7
lung cancer deaths per thousand workers exposed
to 0.2 fibers." Let's see whether that's true.
6.7 per thousand at 0.2, that would
be at, let's say 25 is for the insulation
workers would be a tenth. That would be eight percent -- no, there's somethingwrong with this
figure, by the way. I think this lung cancer
risk is wrong.
Now, it may be that this is a
Xeroxed copy, and that fiber, 6.7 lung cancer
deaths, is not correct.
Well, hold it a second.
Let's say you have 1,000 people and
you have a 16 percent among insulation
workers -- they're looking at all these other
data. Let's use insulation workers. There's a
16 percent excess, of course, 10 times greater
in current cigarette smokers than people who
never smoke. Nevertheless, let's say at 16
percent, would be 16 per hundred or 160 per
thousand. That's 20 fibers. You go down to two
fibers, it would be 16. If you go down to .2,
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196
it would be 1.6.
I think that their figure for lung
cancer risk includes the expected as well as the observed. Meaning, the observed as expected, plus observed. Observe over expected is your
standardized mortality ratio. I think it's a
little high.
Q. You're going through all three
reasons now?
A. No. Q.
Then let's do it, please.
A. Lung cancer risks associated. Dr. Crump acknowledged -- that's Kenny Crump, isn't it?
Q. Yes. MR. BROWNSON: Let me take that.
(Whereupon a brief recess was
taken.)
MR. BROWNSON: I think we'll continue.
A. I think that the risk of lung cancer is a
little bit high. Okay. Here we go.
I stated about OSHA estimated asbestosis risks at 0.25 per cc's exposure, as
an unacceptably high five cases per thousand. This is asbestosis on five cases per thousand.
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DR. LANGER - Direct
197
That would be 50 at .2 or 500 per thousand. Now, isn't that goofy? The highest
levels reported in Selikoff's cohort is 10 '
percent mesothelioma mortality, and they have 50
percent mesothelioma. I don't know where they
got that from, I really don't.
Okay. Let's say that they're
concerned, okay, "lung cancer risk associated" --
we've gone through that.
"Third, the record shows that
employees are likely to be exposed to mixed
fiber types at most construction and shipyard
industry work sites most of the time.
Assuming a higher PEL to chrysotile
with presenting agencies and analytical
difficulties, they should all be the same.
Okay. All right. What do I think?
Well, that's a real interesting
agglomeration of comments, but let's say that
you people aren't exposed to all of this stuff.
Let's say people are exposed to a defined fiber
type in a defined setting. Then you can use the
data specifically for that particular fiber
type. Why not?
If I were doing OSHA, I would also
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probably do it this way, and say let's not
distinguish the fiber types. In fact, I
recommended that in the HEI report. I was the
one that recommended it. Q. You mean, OSHA, would you also agree
with 1. fibers per cc as a level that shows --
A. That's as good as any level. Okay. .1 is
all right.
Q. Look at the standard hypothesis.
There's a short chrysotile fiber
which had been mechanically manipulated because
it went through the industrial process, and it
was harsh. By short, I mean less than five microns.
A. How short? Three microns.
Q. Okay. Three microns.
Do you believe that that fiber has
some carcinogenic potential?
A. It might, but you need a lot of it.
Q. If the fiber were not harsh, would you require more of them?
A. Probably not. Doesn't matter if it's soft
to harsh in that case. I was thinking of long
fibers, actually, rather than the curlicue in
which the effective diameter is the diameter of
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the spiral. The harsh fiber tends to be more
splintery and is more effective in getting to
the distal tissues.
Q. Doctor, is there a length or a ratio
of length to diameter or to width of a fiber,
either chrysotile or amphibole, let's say with chrysotile, below which you believe that it has
no carcinogenic potential?
A. I generally do not count fibers less than
one micron in length when I do a tissue burden
assay.
Q. Does that mean that you effectively
did not believe that fibers less than one micron
in length can have any potentialfor causing
cancer?
A. No, it means that I have examined the
cases in the general population, and those
fibers,chrysotile,
tend to be extremely short.
In fact, most of them lie below one micron in
length.
Q. Do you then have a limit below which
you believe a chrysotile fiber has no cancerproducing potentiality?
A. I'm shaking my head. You mean, none,
zero, it has nopotential?
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Q. Right. A. No, I think that it may be significantly
reduced, but is there some residual risk?
Well, it's probable. Probably so.
Q. Is there a point at which you make a
distinction in the cancer-causing potentiality
in a fiber based upon its life? For example --
A. Sure.
Q. -- what is that point or what is the
distinction?
A. Well, I am concerned mostly with fibers
greater than five microns in length, in that
this is the standard index against which we
measure risk or hazard, and everything else is
just embellishment.
What does it mean?
Q. Doctor, do you have an opinion about
the scientific value of the work done by the
Canadian Tumor Registry Panel?
A. The value of the work?
Well, it's like every registry, it
has value, of course.
Q. How can you describe that value?
MR. BROWNSON: Well, I'll object to
the form of that question.
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A. It gives us a better idea of what people
are dying with or from.
MR. MILLER: Thank you.
I have no more questions today.
However, I do want to leave open the
possibility of the return visit in this
case after receiving further documentation
from Mr. Brownson, which I understand will
be the full air sampling results and
back-up documents.
Given that, though, aside from that,
we're done in this case.
Thank you. Doctor.
(Whereupon the witness was excused
at 3:50 p.m.)
**
*
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CERTIFICATE
I, BERNADETTE H. MASTRANGELO, a Certified Shorthand Reporter, License No. XI01860, and Notary Public of the State of New York do hereby certify that prior to the commencement of the examination
DR. ARTHUR M. LANGER
was sworn by me to testify to the truth, the whole truth, and nothing but the truth.
I DO FURTHER CERTIFY that the foregoing is a true and accurate computer-aided transcript of the testimony as taken stenographically by and before me at the time, place, and on the date hereinbefore set forth.
I DO FURTHER CERTIFY that I am neither a relative of nor employee nor counsel nor attorney for any party in this action, and that I am not interested in the event nor outcome of this litigation.
('hyVKadOi. fYladnataUt)
Notary Public of the State of New York
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CERTIFICATION OF WITNESS
I have read the foregoing transcript of my deposition and find it to be true and accurate to the best of my knowledge and belief.
DR. ARTHUR M. LANGER
Sworn and subscribed to before me on thisday of, 1995
Notary____________________________ My Commission Expires.
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CURRICULUM VITAE
Arthur M. Langer, Ph.D.
CURRENT POSITIONS: Director, Environmental Sciences Laboratory of the Institute of Applied Sciences, Brooklyn College of the City University of New York, Brooklyn, NY 11210.
Deputy Director of the Center for Applied Studies of the Environment, Applied Science Coordinating Institute, City University of New York.
Professor of Geology, Department of Geology, Brooklyn College.
Professor of Geology, Ph.D., Graduate Faculty, Program: Earth and Environmental Sciences, City University, New York, NY, 1982-present.
Research Associate, Department of Mineral Sciences, American Museum of Natural History, New York, NY, 1979-present.
ADDRESS:
Office:
Home:
Environmental Sciences Laboratory Ingersoll Hall - Room 5144-6 Brooklyn College of the City University of New York Brooklyn, NY 11210 Telephone: (718) 951-4793; 951-4242 Facsimile: (718) 951-4199
6 Rochambeau Drive Hartsdale, NY 10530 (914) 428-3667
DATE OF BIRTH:
February 18, 1936
PLACE OF BIRTH:
New York, NY
LANGER, ARTHUR M.
PAGE 2
ACADEMIC DEGREES:
B.A., Geology, Hunter College-CUNY, New York, NY, 1956. M.A., Petrology (Geology), Columbia University, New York, NY, 1962. Ph.D., Mineralogy (Geology), Columbia University, New York, NY, 1965.
PREVIOUS POSmONS:
Associate Professor, Center for Polypeptide and Membrane Research, Mount Sinai School of Medicine, New York, NY 1986-1988.
Associate Professor, Mineralogy, Department of Community Medicine, Mount Sinai School of Medicine, New York, NY, 1968-1986, 1987-1988.
Science Administrator, Environmental Sciences Laboratory, Mount Sinai School of Medicine, New York, NY, 1983-1984.
Associate Director, Environmental Sciences Laboratory, Mount Sinai School of Medicine, New York, NY, 1969-1986.
Director of Laboratories, Environmental Sciences Laboratory, Mount Sinai School of Medicine, 1976-1986.
Head, Physical Sciences Section, Environmental Sciences Laboratory, Mount Sinai School of Medicine, New York, NY, 1969-1986.
Adjunct Associate Professor, Mineralogy, Graduate Division, City University of New York, NY, 1968-1969.
Assistant Professor, Mineralogy, Department of Community Medicine, Mount Sinai School of Medicine, New York, NY, 1967-1968.
Research Associate, Environmental Medicine, Department of Medicine, Mount Sinai Hospital, New York, NY, 1965-1967.
Lecturer, Geology, City College of City University of New York, NY, 1964-1965. Research Assistant, Mineralogy, Department of Geology, Columbia University, New
York, NY, 1961-1964. Teaching Assistant, Economic Geology, Department of Geology, Columbia University,
New York, NY, 1959-1960. Teaching Assistant, Department of Geology, Columbia College, New York, NY, 1958
1959. Field Assistant, Geology, Beartooth Mountains, MT, Columbia University, 6/57-8/57;
6/58-8/58. Exploration Geologist, Rosario Exploration Chibougamau Mining and Smelting, 6/56
8/56. Consulting Mineralogist, Columbia University, New York, NY:
-Texas Gulf Sulphur Company, 1965 (with Professor Kerr). -Tidewater Oil Company, 1962 (with Professor Kerr). -Consulting Mineralogist, Creole Oil Company, 1962 (with Professor Kerr). -Consulting Mineralogist, American Metals Climax, 1961 (with Professor Kerr). -TAMS Dam site, East Pakistan, WHO, 1960 (with Professor Fairbridge).
LANGER, ARTHUR M.
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FELLOWSHIPS:
Research Mineralogist, Department of Geology, Columbia University, New York, NY. Sponsored by the U.S. Air Force Cambridge Research Laboratories under the direction of Prof. P.F. Kerr, 3/61-9/63.
MEMBERSHIPS AND OTHER PROFESSIONAL ACTIVITIES:
Fellow, Geological Society of America. Fellow, Mineralogical Society of America. Fellow, New York Academy of Sciences. Fellow, Collegium Ramazzini. Geochemical Society. American Association for the Advancement of Science. Electron Microprobe Society of America. Sigma Xi, Kappa Chapter (Honorary Scientific). International Association of Bioinorganic Scientists.
HONORS AND AWARDS:
Honors, Department of Geology, Hunter College, New York, NY, 6/56. Sigma Xi, Kappa Chapter, Columbia University, New York, NY, 9/64. Dust Research, Polachek Foundation Award, 9/65-6/67. Career Scientist Award, National Institute of Environmental Health Sciences, 6/69
5/74. Phi Beta Kappa, Nu Chapter, Hunter College, New York, NY, 6/77. Hunter College Hall of Fame HC-CUNY, 6/78. Biography in: American Men of Science; Who's Who in the East; Who's Who in
America (September, 1992). Elected Fellow, Collegium Ramazzini, 1983.
EXPERT CONSULTANT:
Atlantic Legal Foundation. Amici Curiae in support of respondents to the Supreme Court of the United States. October Term, 1992. Daubert et.al. Petitioners v. Merrell Dow Pharmarceuticals.lnc., Respondent.
Environmental Protection Agency, Superfund Cases, 3/85-Present. National Institute for Occupational Safety and Health, 6/75-Present. National Institute for Environmental Health Sciences, 4/75-Present. Environmental Protection Agency, 6/75-Present. National Heart, Lung and Blood Institute, 6/75-Present. National Institutes of Health, Section of Grants, 6/74-Present. World Health Organization, Geneva, Biomedical Expert, 6/75-Present.
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GRADUATE THESES:
Geology of the Manhattan Formation. Submitted in partial fulfillment for the degree of Master of Arts, in the faculty of Pure Science, Columbia University, NY, p. 132, 1962.
Mineralogy and Physical Properties of Moiave Desert Plava Crusts. Submitted in partial fulfillment for the degree of Doctor of Philosophy, in the faculty of Pure Science, Columbia University, NY, p. 155, 1965.
EDITORIAL BOARD SERVICE:
Assistant Editor, Environmental Research, 1978-1985. Advisory Editor, Environmental Research, 1985-1987. Assistant Editor, American Journal of Industrial Medicine, 1980-1985. Associate Editor, American Journal of Industrial Medicine, 1985-1986. Editorial Review Board, Journal of Environmental Pathology and Toxicology, 1978
1982. Editorial Advisory Board, Advances in Modern Environmental Toxicology, 1981-1982. Editorial Review Board, Journal of Environmental Pathology Toxicology and
Oncology, 1983-Present.
REVIEW MANUSCRIPTS (Journals other than those cited above):
Journal of Histochemistry and Cytochemistry. Pharmacology Reviews. American Chemical Society Reviews. Advances in Chemistry Annals New York Academy Sciences. Science (AAAS). SEM-IITRI Symposia. Lung. American Review of Respiratory Diseases. American Mineralogist. Clay Minerals Society (Clays and Clay Minerals). Canadian J. Fisher Aquatic Sciences. Journal National Cancer Institute. American Journal Pathology. Annual Meeting SEMA. Chest. Laboratory Investigations. Journal of the American Medical Association. New England Journal of Medicine. Toxicology In Vitro. Journal of the American Industrial Hygiene Association.
LANGER, ARTHUR M.
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REVIEW MANUSCRIPTS (Journals other than those cited above) (contd):
Annals of Occupational Hygiene. Pathology Annual. Environmental Health Perspectives. Health Effects Institute. American College of Chest Physicians. Heterogeneous Chemistry Reviews. Bulletin of the Geological Society of America. Applied Occupational and Environmental Hygiene.
REVIEW AND AUTHOR FEDERAL, INDUSTRY DOCUMENTS (1978-PRESENT):
NIOSH Fibrous Glass Criteria Document, 1978. Reviewer. CDC-NIOSH. NIOSH Talc Criteria Document, 1979. Reviewer, contributor. CDC-NIOSH. OSHA
Fiber Manuscript (Definitions, Nomenclature, Properties), 1980. Reviewer. US Department of Labor. ALOSH Mineralogy Manuscripts (Occupational Lung Disease), 1979-1980. Reviewer, contributor. CDC-NIOSH. Asbestos, Smoking and Disease. The Scientific Evidence. Burns, DM, Ed., Commercial Union Insurance, 1982. Contributor. Environmental Protection Agency, Office of Pesticides and Toxic SubstancesAsbestos in Buildings Guidance Documents; Guidance Documents; Operations and Maintenance Programs. "Orange", "Purple", "Blue" Books. Reviewer, contributor. US Environmental Protection Agency. Surgeon General's Report: Cancer and Chronic Lung Disease in the Workplace: The Health Consequences of Smoking, 1985. Reviewer, contributor. Federal.
INTERNATIONAL COMMITTEES AND CONSULTATIONS:
US-Japan Cooperative Science Program Working Group on Air Pollution and Health. NIEHS-lnstitute of Health, Japan, February, 1969.
International Agency for Research on Cancer, WHO Development of V.14 Chemical Carcinogenesis Monograph Series, Evaluation of Carcinogenic Risk of Chemicals to Man: Asbestos. Member of working group, Lyon, France, 1976.
International Association of Geochemistry and Cosmochemistry, Working Group on the Geochemistry of Health and Disease, 1976-1979.
Consultant, South African Ministry of Mines, Asbestos Symposium, Johannesburg, South Africa, 1977.
Consultant, Institute of Public Health, Norway, Microscopy Facility, Oslo, Norway, 1977. Seminars on vitreous fibers as asbestos substitutes.
Consultant, International Metalworkers Federation, Problems Focusing on Asbestos Contamination of Nickel Ores, Geneva, Switzerland, 1980.
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INTERNATIONAL COMMITTEES AND CONSULTATIONS (cont):
Research Consultant, Societe Nationale de I'Amiante, Quebec, Canada, Work on Modified Fiber, 1984.
WHO-International Program on Chemical Safety (IPCS), Environmental Health Criteria Document on Asbestos and Other Natural Mineral Fibers, Section Chairman, Hannover, Federal Republic of Germany, 1985.
Organizing Committee, Third International Conference, ]n Vitro Effects of Mineral Dusts, Schluchsee, Federal Republic of Germany, 1984.
Societe Nationale de I'Amiante. Consultant. Modified Fiber and the Environmental Protection Agency Asbestos Ban, Montreal, Canada, 1986.
International Agency for Research on Cancer, WHO Development of V.42, Chemical Carcinogenesis Monograph Series, Evaluation of Carcinogenic Riskto Chemicals in Man: Silica and Some Silicates, Member of Working Group, Lyon, France, 1986.
Consultant, Asbestos Institute, Canada, Organization of Symposium on Biological Effects of Asbestos Substitutes, 1987.
Vllth International Pneumoconiosis Conference. International Organizing Committee; Organizer of Session: Hazard Recognition of Mineral Dust. Pittsburgh, PA, August, 1988. Chaired Two Sessions at Meeting: Mineral Recognition by Membranes and Mineral Toxicity; Mineral Fiber and Diseases of the Pleura.
International Federation of Building and Wood Workers, Conference on Interior Works, Geneva, Switzerland. Presented paper: Hazards in the Painting Trades. Panel on Hazards in the Interior Workplace, May 9-12, 1989.
Hoffman-LaRoche Pharmaceuticals. Development of Microscopy Method for the Analysis of Mineral Fiber in Parenteral Drugs. Edinburgh, April, 1993; Basel, March, 1994; Frankfurt, May, 1994.
Eurometaux - European Association of Metals. Protocol Format and Development of Proposed Meeting on Carcinogenicity of Metals and the Compounds. Brussels, November, 1994.
NATIONAL COMMITTEES, CONSULTATIONS:
Food and Drug Administration, Asbestos, Talc, Asbestos Bodies,and Consumer Talcums, Seminar, Washington, DC, June, 1968.
National Air Pollution Control Administration, Asbestos in Ambient Air. Arlington, VA, June, 1969.
National Institute for Occupational Safety and Health, USPHS, Asbestos Research in the United States, Cincinnati, OH, January, 1970.
Food and Drug Administration, Asbestos in Consumer Talcums, Seminar, Washington, DC, August, 1971.
Environmental Protection Agency, National Air Pollution Control Techniques Advisory Committee, Asbestos Emissions Document, Atlanta, GA, 1971.
NIOSH Task Force on Occupational Respiratory Diseases, 1975.
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NATIONAL COMMriTEES, CONSULTATIONS (cont):
Environmental Protection Agency, HERL, Ad Hoc Committee for the Fibrous Amphibole Study Protocol, Triangle Park, NC, 1976.
NIEHS-NIOSH-EPA-ERDA. Interagency Retreat and Colloquium. Man-made Vitreous fibers, Asbestos Substitutes and the Energy Crisis. Potential Risks to Health. Pinehurst, NC. 7-9 January, 1976.
NIOSH Criteria Document Recommended Standard For Occupational Exposure to Fibrous Glass, Reviewer. November, 1976.
Environmental Protection Agency, HERL, Biological Effects on Fibrous Inorganic Particles, 1977.
DHHS, Interagency Committee to Coordinate Environmental and Related Problems, Biological Effects of Fibrous Particulates: Serpentine-Containing Host Rocks, Amphibole-Containing Host Rocks, 1980.
Mount St. Helen's Volcanic Ash: Mineral Nature and Biological Activity, Interagency Task Force, Bethesda, MD, 1980.
NHLBI, Evaluation of Existing Inorganic Microparticulate Laboratories: Vermont Lung Group, Tulane Occupational Lung Hazards Group, 1978.
National Institute of Environmental Health Sciences, Workshop, Pathobiology of Mesothelioma, RTP, NC, January, 1983.
Occupational Safety and Health Administration, Expert to write and review Asbestos Standard-Talc Standard, US Department of Labor, 1983.
National Academy of Sciences, Division of Life Sciences Committee to Evaluate Risk to Low-Level Exposure to Asbestiform Fibers in the Environment, 1982-1984.
Environmental Protection Agency. Prepared for meeting Substitute Fibers. Pre-market Testing of Fibers. USEPA, Washington, DC. 26 June 1984.
Environmental Protection Agency, Committee Member, Review and Rewrite EPA "Guidance Document for Controlling Friable Asbestos-Containing Minerals in Buildings," 1985.
American Society Testing Materials, Committee, D22-lndoor Air Pollution Asbestos: D22.05-Methodology and Measurement of Fibers in Air. Electron Microscopy, 1985.
American Society Testing Materials, Organizing Committee, Silica and Silica-Induced Diseases, International Conference, 1985.
NIOSH, Mine Health Research Advisory Committee Meeting, Tucson, AZ: The Impact of Mineral-Asbestos Definitions on the Mining Industry, 1986.
Environmental Protection Agency. Develop guidance document for identifying asbestos hazards and implementing abatement programs in public buildings, Washington, DC, April, 1986.
NIOSH Review Panel for Project "Evaluation of Mesothelioma Production by Asbestos Substitutes." Cincinnati, OH, June, 1986.
Environmental Protection Agency, Guidance Document for Assessing and Managing Exposure to Asbestos in Building, Arlington, VA, September, 1986.
Environmental Protection Agency, Operations and Maintenance Programs for Asbestos-Containing Materials in Buildings: A Guide for Building Owners and Managers, Washington, DC, January, 1987.
LANGER, ARTHUR M.
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NATIONAL COMMrTTEES, CONSULTATIONS (cont):
NIEHS-NIOSH - National Toxicology Program. To formulate study protocol comparing biological activities of asbestiform and non-asbestiform amphibole minerals. NIEHS - Res. Triangle Park, NC, October 11, 1989.
Health Effects Institute. Asbestos Literature Review Panel. April, 1990 - August, 1991.
Health Effects Institute. Reviewer of Research Proposals - RPI-1-91.
TESTIMONY BEFORE FEDERAL AGENCIES:
Expert Testimony, Toxic Substances Control Act, 1973. Provided evidence to Sen. Tunney's committee hearing, Washington, D.C.
Expert Witness, Contamination of Lake Superior. On behalf of the Department of Justice of the United States, 1974. Minneapolis, MN.
Expert Testimony, Occupational Safety and Health Administration, US Department of Labor, Asbestos Standard, 1984. Washington, D.C.
Expert Testimony, Consumer Products Safety Commission. Asbestos in Play Sand. Washington, DC, December 7, 1988.
Expert Testimony Occupational Safety and Health Administration, U.S. Department of Labor. Asbestos Standard Revisions. April, 1990. Washington, D.C.
REGIONAL CONSULTATIONS:
Health Research Council of New York City, Subcommittee on Asbestos Hazards - Air Pollution Working Corp. Seminar on Asbestos in Construction Products, Rockefeller University, June 9, 1969.
New York City Board of Education: Asbestos in Schools, 1981-1983. New York City Department of Sanitation; Fire Department: Insulation Products, 1979
1983. New York State Consumer Affairs and Protection: Construction and Insulation
Products, 1980-1983. City University of New York-Brooklyn College: Asbestos Problems, 1981-1982. New York City Asbestos Taskforce. Inspector General's Office. Asbestos in schools. Analysis and evaluation of samples removed from school buildings. Fall, 1993.
ACADEMIC COMMITTEES:
Academic Council, Department Representative, MSSM, 1977-1979. Medical Center Safety Committee, MSSM, 1979-1988. Chemical Hazards Committee, MSSM, 1979-1988. Alternate Medical Safety Officer (in absence of Dr. S. Kochwa), MSSM, 1980-1983. Space Committee (Department and Institution), MSSM, 1982-1985.
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ACADEMIC COMMITTEES (cont):
New York Academy of Sciences, Conference Organizing Committee, 1976-1980. Visiting Professor Program. Mt. Sinai School of Medicine. 1971 -1973. Educational Policy Committee. Mt. Sinai School of Medicine. 1972-1974. Ad Hoc Reviewer, National Institutes of Health, Minneapolis Medical Center, 1977. Ad Hoc Reviewer, National Institutes of Health, Harvard Medical Center, 1978. Workshop Organizer and Chairman, Significance of Aspect Ratio in Asbestos
Diseases, New York Academy of Sciences, 1977. Workshop Organizer, Third International Workshop on ]n Vitro Testing of Mineral
Dusts, 1984. Executive Committee, Ph.D. Program, Earth and Environmental Sciences, City
University of New York, 1985-1993. Curriculum and Examination Committee, Ph.D. Program, Earth and Environmental
Sciences, City University of New York, 1987-1990. Faculty Membership Committee - Chairman - Ph.D. Program, Earth and Environmental Sciences, City University of New York, 1989-1992.
CONSULTANT:
Cyprus Minerals-Nature of US Talc Deposits, 1979. General Accident Insurance Company: Asbestos Compensation, 1981. Gulf Minerals: Modified Chrysotile Fiber, 1982. Oil, Chemical and Atomic Workers International Union, Safe Handling of Asbestos,
Training Film, 1976. Lung Center and NHLB-SCOR, in the Departments of Physiology, Medicine,
Pathology, and Engineering in the University of Vermont, Burlington, VT, 1984. Societe Nationale de I'Amiante: Phosphorylated Fiber; Asbestos Substitutes, 1984. W.R. Grace & Company: Asbestos and Indoor Air Pollution, 1984. Asbestos Institute of Canada, Asbestos and Asbestos Substitutes, 1985-1986. Litigations. Represented numerous plaintiffs, defendants, insurance carriers, US
Department of Justice, Brooklyn and New York counties' District Attorneys. R.T. Vanderbilt Company, Nature of Tremolite in the Gouverneur Talc Deposit, 1987. Safe Building Alliance, Problems of Asbestos in Buildings, 1987. Review applications for funding requests: FCAC, Quebec, Canada, 1985. Battelle Columbus Laboratories: Analysis of Microparticles by Analytical Electron
Microscopy, 1987-1989.
INDUSTRY PROJECT REPORTS:
Chrysotile and chrvsophosphate. A comparative study of their physicochemical properties and membrane activities. A report to SNA-Chrysophosphate, Canada. Langer AM, Nolan RP, p. 87, June 1, 1987.
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INDUSTRY PROJECT REPORTS (cont):
Wollastonite in the pulmonary tissues on animals. A report to Northrop Services Industries, National Toxicology Program. Langer AM, Nolan RP, p. 101, June 15, 1989.
Preparation, examination and characterization of mineral standards bv analytical electron microscopy. A report to Battelle Columbus Laboratories, Langer AM, Nolan RP, Pooley FD, Gieseke JA, Fisher G, 61 p., December 12, 1989.
Comparison and evaluation of the analytical capabilities of three U.S. laboratories for submicroscopic particulate analysis.
A report to Battelle Columbus Laboratories, Langer AM, Nolan RP, Gieseke JA, Fisher G. February, 1991.
Report on the measurement of fiber release from simulated steam-stressed Durabla gaskets (120. 160. 190 for 5 hours) for health hazard evaluation. A report to Durabla, Inc., Langer AM, Nolan RP, 21 p., January 31, 1992.
Measurement of fiber release from simulated steam-stressed Durabla gaskets for health hazard evaluation. A report to Durabla, Inc., Langer AM, Nolan RP, (revision 1/31/92 report) 29 p., July 16, 1992.
Report on the measurement of fiber release from simulated steam-stressed Durabla gaskets (120C. 160C. 190C for 5 days) for health hazard evaluation. A report to Durabla, Inc., Langer AM, Nolan RP, 23 p., November 6, 1992.
Report on the measurement of fiber release from simulated steam-stressed Durabla gaskets (250C. 30 days) for health hazard evaluation. A report to Durabla, Inc., Langer AM, Nolan RP, 25 p., December 9, 1992.
The Identification and Quantitation of Small Asbestos Fibres in Injectable Medicines. Technical Expert Report Prepared for Hoffman-LaRoche, Basel. 16 p. Addison J, Burdett GJ, Langer AM, Muhle H. 1993.
Analysis of rockwool fibers bv scanning electron microscopy. A report to Lapinus Fibres, B.V., Netherlands. Nolan RP, Langer AM, 13 p., November 29, 1993.
Analysis of rockwool fibers bv scanning electron microscopy. A report to Lapinus Fibres, B.V., Netherlands. Nolan RP, Langer AM, 25 p., November 29, 1993.
Release of Rockwool Fibers From Compressed Gasket Material. A report to Lapinus Fibres, B.V., Netherlands. Nolan RP, Langer AM, 38 p., Appendix, January 31, 1994.
Analysis of Quarry Sample #01. #08 and #18 for asbestos content. The Readymix Group, Australia, Nolan RP, Langer AM, 39 p., March 9, 1994.
Health Hazard Evaluation of the CD #1 Pit Empire Mine. Palmer. Michigan. A report to Cleveiand-Cliffs Corp., Nolan RP, Langer AM, Wilson R, 90 p., January, 1995.
GOVERNMENT PROJECT REPORTS:
Asbestiform Fibers - Non-Occupational Health Risks. National Academy of Sciences. National Research Council. National Academy Press, 334 p., 1984.
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GOVERNMENT PROJECT REPORTS (cont):
Report to the U.S. Consumer Product Safety Commission: Mineraloaical Analysis of Two "Play Sands" For Their Asbestos Contents. Langer AM, Nolan RP. 26 November, 1986. 9p., 4 Tables, 18 plates.
Report to the U.S. Consumer Product Safety Commission: Mineral Analysis of a Carbonate Plav Sand. Langer AM, Nolan RP. July, 1987. 19 p., 3 Tables, 8 plates.
Asbestos in Public and Commercial Buildings: A Literature Review and Synthesis of Current Knowledge. Health Effects Institute, Cambridge, MA and U.S. Environmental Protection Agency. 300 p., 1991.
INTERNATIONAL DOCUMENTS:
Evaluation of Carcinogenic Risk of Asbestos. Volume 14. Asbestos. Series. IARC Monographs on the Evaluation of Carcinogenic Risk of Chemicals to Man. Inti Agency for Res. on Cancer - WHO, Lyon. 106 p. 1977. Co-author, Panel Member.
International Programme on Chemical Safety (IPCS). WHO. Environmental Health Criteria 53: Asbestos and Other Natural Mineral Fibres. Hannover, Germany. 194 p., 1986. Co-author, Panel Member.
Silica and Some Silicates. Volume 42. Series IARC Monographs on the Evaluation of Carcinogenic Risk of Chemicals to Man. Inti Agency for Res. on Cancer WHO, Lyon. 289 p., 1987. Co-author, Panel member.
International Programme on Chemical Safety (IPCS), WHO. Workshop on Health Risks Associated With Chrvsotile Asbestos. Isle of Jersey. Ann. Occup. Hyg., 3, 397-646. Presenter. Author.
PUBLICATIONS IN PEER-REVIEWED JOURNALS:
1. Kerr PF, Thomas AM, Langer AM: The nature and synthesis of ferrimolybdite. Am Mineral 48:14-32, 1963.
2. Kerr PF, Langer AM: Mineralogical features of Mojave playa crusts. In: Mineralogy and Hydrology of US Plavas. Neal J (ed), US Air Force Cambridge Research Laboratories Environmental Research Paper No. 96, pp. 31-72,1965.
3. Langer AM, Kerr PF: Experimental variables influencing DTA curves of kaoiinite. Dupont Thermogram 3:1-4, 1966.
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PUBLICATIONS IN PEER-REVIEWED JOURNALS (cont):
4. Langer AM: Older paleozoic metamorphism and pegmatization in Bronx, New York. Ann NY Acad Sci 136:1-32, 1966.
5. Langer AM, Kerr PF: Mojave Desert playa crusts: physical properties and mineral content. J Sed Patrol 36:377-396, 1966.
6. Langer AM, Kerr PF: Evaluation of kaolinite and quartz differential thermal curves with a new high-temperature cell. Am Mineral 52:509-523, 1967.
7. Berkley C, Langer AM, Baden V: Instrumental analysis of inspired pulmonary particulates. Trans NY Acad Sci 30:331-350, 1967.
8. Neal JT, Langer AM, Kerr PF: Giant desiccation polygons of Great Basin playas. Bull Geol Soc Am 79:59-90, 1968.
9. Langer AM, Bowes DR: Polyphase deformation in the Manhattan formation of Manhattan Island, New York City. Memoir 115 Geol Soc Am 358-373, 1969.
10. Langer AM, Selikoff IJ, Sastre A: Chrysotile asbestos in the lungs of persons in New York City. Arch Environ Health 22:348-361, 1971.
11. Langer AM, Mackler AD, Rubin IB, Hammond EC, Selikoff IJ: Inorganic particles in cigars and cigar smoke. Science 174(4009) :585-587, 1971.
12. Selikoff IJ, Nicholson WJ, Langer AM: Asbestos air pollution in urban areas. Arch Environ Health 25:1-13, 1972.
13. Uebling R, Langer AM: Optical properties of fibrous brucite from asbestos, Quebec. Am Mineral 57:857-864, 1972.
14. Langer AM, Rubin IB, Selikoff, IJ: Chemical characterization of asbestos body cores by electron microprobe analysis. J Histochem Cytochem 20 (9):723-734, 1972.
15. Langer AM, Rubin IB, Selikoff IJ, Pooley, FD: Chemical characterization of uncoated asbestos fibers from lungs of asbestos workers by electron microprobe analysis. J Histochem Cytochem 20(9):735-740, 1972.
16. Bowes DR, Langer AM: Petrochemistry of the Manhattan formation, New York City. Krystalinikum 10:39-51, 1974.
17. Langer AM, Ashley R, Baden V, Berkley C, Hammond EC, Mackler AD, Maggiore CJ, Nicholson WJ, Rohl AN, Rubin IB, Sastre A, Selikoff IJ: Identification of asbestos in human tissues. J Occup Med 15:287-295,1973.
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PUBLICATIONS IN PEER-REVIEWED JOURNALS (cont):
18. Ehrenreich T, Mackler AD, Langer AM, Seiikoff U: Les fibers d'amiante dans les poumons humains: leur signification medicolegale dans les maladies de I'environment. Arch des Maladies Professionelles 34(4-5):189-204, 1973.
19. Bowes DR, Langer AM: Hornblende schists in the Manhattan formation in the Bronx, New York: Discussion. Bull Geol Soc Am 84:1483-1490, 1973.
20. Ehrenreich T, Mackler AD, Langer AM, Seiikoff U: Identification and characterization of pulmonary dust burden in pneumoconiosis. Ann Clin Lab Sci 3(2) :118-131, 1973.
21. Kleinfeld M, Messite J, Langer AM: A study of workers exposed to asbestiform minerals in commercial talc manufacture. Environ Res 6(2):132-143, 1973.
22. Nicholson WJ, Langer AM, Seiikoff IJ: Discussion: asbestos fibers in the air of towns. Atmosph Environ 7:666-668, 1973.
23. Langer AM: Inorganic particles in human tissues and their association with neoplastic disease. Environ Health Perspect 9:229-233, 1974.
24. Langer AM: Approaches and constraints to identification and quantitation of asbestos fibers. Environ Health Perspect 9:133-136, 1974.
25. Langer AM: Research perspectives concerning asbestos minerals and their effects on biological systems. Environ Health Perspect 9:335-338, 1974.
26. Langer AM: The subject of continuous vigilance. Environ Health Perspect 9:53 56, 1974.
27. Rohl AN, Langer AM: Identification of asbestos in talc. Environ Health Perspect 9:95-109, 1974.
28. Langer AM, Mackler AD, Pooley FD: Electron microscopical investigation of asbestos fibers. Environ Health Perspect 9:63-80, 1974.
29. Miller A, Langer AM, Teirstein, AS, Seiikoff U: "Non-specific" interstitial fibrosis: association with fibers detected by electron microscopy. N Engl J Med 292:91 93, 1975.
30. Rohl AN, Langer AM, Seiikoff IJ, Nicholson WJ: Exposure to asbestos in use of consumer spackling, patching, and taping compounds. Science 189(4204) :551553, 1975.
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PUBLICATIONS IN PEER-REVIEWED JOURNALS (cont):
31. Rohl AN, Langer AM, Klimentidis R, Wolff MS: Asbestos exposure during brake lining maintenance and repair. Environ Res 12:110-128, 1976.
32. Rohl AN, Langer AM, Selikoff IJ, Tordini A, Klimentidis R, Bowes DR, Skinner D: Mineral and chemical characterization of selected consumer talcum products. J Toxicol Environ Sci 2:255-284, 1976.
33. Bowes DR, Langer AM, Rohl AN: Nature and range of mineral dusts in the environment. Phil Trans Roy Soc London 286:593-610, 1977.
34. Rohl AN, Langer AM, Klimentidis R, Wolff MS, Selikoff IJ: Asbestos content of dust encountered during brake maintenance and repair. Proc Roy Soc Med 70:32-39, 1977.
35. Rohl AN, Langer AM, Selikoff IJ: Environmental asbestos pollution related to use of quarried serpentine rock. Science 196:1319-1322, 1977.
36. Langer AM, Wolff MS, Rohl AN, Selikoff IJ: Variation of properties of chrysotile asbestos, subjected to prolonged milling. J Toxicol Environ Sci 4:173-188,1978.
37. Fischbein A, Langer AM, Suzuki Y, Selikoff IJ: Carcinoma of the lung in a drywall taping worker: report of a case. Toxicol Letters 2:231-236, 1978.
38. Langer AM: Crystal faces and cleavage planes in quartz as templates in biological processes. Quart Rev Biophys 11:543-575, 1978.
39. Rohl AN, Langer AM, Selikoff IJ: Airborne asbestos in the vicinity of a freeway: discussion. Atmosph Environ 12:2030-2031, 1978.
40. Langer AM, Rohl AN, Selikoff IJ: Asbestos on Maryland's roads. The Lancet i: 1263-1264, 1978.
41. Fischbein A, Rohl AN, Langer AM, Selikoff IJ: Drywall construction and asbestos exposure. Am Industr Hyg Assoc 40:402-407, 1979.
42. Langer AM, Rohl AN, Selikoff U, Harlow G, Prinz M: Asbestos as a co-factor among nickel processing workers. Science 209:420-422, 1980.
43. Nolan RP, Langer AM, Harington JS, Oster G, Selikoff IJ: Quartz hemolysis as related to its surface functionalities. Environ Res 26:503-520, 1981.
44. Rohl AN, Langer AM, Moncure G, Fischbein AS, Selikoff U: Endemic pleural disease associated with mixed fibrous dust exposure in Turkey. Science 216(4545) :518-520, 1982.
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PUBLICATIONS IN PEER-REVIEWED JOURNALS (corrt):
45. Langer AM, McCaughey WET: Mesothelioma in a brake repair worker. The Lancet ii:1101-1103, 1982.
46. Ehrenreich T, Espinoza T, Langer AM, Rohl AN, Daum SM: Algorithm for forensic pathological diagnosis of asbestos-related diseases. Am J Foren Med Pathol 3(4):315-321, 1982.
47. Langer AM, Rohl AN, Fischbein AS, Selikoff IJ: Cancer in nickel processing workers in New Caledonia: discussion. Science 215:425-426, 1982.
48. Yeager H, Russo D, Yanez M, Gerardi D, Nolan RP, Kagan E, Langer AM: Preliminary observations on acute cytotoxicity of short fiber chrysotile asbestos for human alveolar macrophages. Environ Res 30:224-232, 1983.
49. Brody J, Miller A, Langer AM: Pneumoconiosis associated with exposure to glass and abrasive fragments. Am J Industr Med 6:339-346, 1984.
50. Constantopoulos S, Goudevenos J, Saratzis N, Charalampopoulos C, Laparidou S, Langer AM, Selikoff IJ, Moutsopoulos H: Endemic asbestos-like pleural calcifications in the absence of occupational asbestos exposure. Arch Hellenic Med 1 (10):165-171, 1984.
51. Sawyer R, Rohl AN, Langer AM: Fiber contamination in buildings resulting from removal of asbestos-containing materials. Environ Res 36(1):46-55, 1985.
52. Constantopoulos SH, Goudevenos JA, Saratzis N, Langer AM, Selikoff IJ, Moutsopoulos HM: Metsovo lung: pleural calcification and restrictive lung function in northwestern Greece. Environmental exposure to mineral fiber as etiology. Environ Res 38:319-331, 1985.
53. Langer AM, Nolan RP: Asbestos in potable water supplies and attributable risk of gastrointestinal cancer. Northeast Environ Sci 5(1/2):41-53, 1986.
54. Langer AM, Nolan RP, Constantopoulos SH, Moutsopoulos HM: Association of Metsovo lung and pleural mesothelioma with exposure to tremolite-containing whitewash. The Lancet i:965-967, 1987.
55. Nolan RP, Langer AM, Weisman I, Herson R: Surface character and membrolytic properties of the titania polymorphs. Br J Ind Med 44(10):687-698, 1987.
56. Nolan RP, Langer AM, Eskenazi RA, Herson GB, Foster KW: Membranolytic activities of quartz standards. Toxicol In Vitro 1(4):239-245, 1987.
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PUBLICATIONS IN PEER-REVIEWED JOURNALS (cont):
57. Langer AM, Nolan RP: Fiber type and mesothelioma risk. Symposium of Health Aspects of Exposure to Asbestos in Buildings. Energy and Environmental Policy Center, Kennedy School of Government. Harvard University. December, 1989, 91-141.
58. Freed JA, Miller A, Gordon R, Fischbein A, Kleinerman J, Langer AM: Desquamative interstitial pneumonia associated with chrysotile asbestos fibers. Br J Ind Med, 48: 332-337, 1991.
59. Nolan RP, Langer AM, Herson GB: Characterization of palygorskite specimens from different geological locales for health hazard evaluation. Br J Ind Med, 4: 463-475, 1991.
60. Langer AM, Nolan RP: Chrysotile biopersistence in the lungs of persons in the general population and exposed workers. Env. Health Perspect. Supplts., 102. Suppl. 5, 235-239, 1994.
61. Nolan RP, Langer AM, Addison J: Lung content analysis of cases occupationally exposed to chrysotile asbestos. Env. Health Perspect. Supplts., 102. Suppl. 5, 249-250, 1994.
62. Langer AM, Nolan RP: Chrysotile: Its Occurence and Properties as variables controlling Biological Effects. Ann. Occup. Hyg., 38, No. 4, 427-451. 1994.
63. Wilson R, Langer AM, Nolan RP, Gee JBL, Ross M: Asbestos in New York City public school buildings. Public Policy: Is there a scientific basis? Jour. Regulatory Toxicology and Pharmacology, 20, 161-169, 1994.
PUBLICATIONS IN SYMPOSIA PROCEEDINGS:
1. Berkley C, Langer AM, Sastre A, Arneson A: Electron microprobe analysis of asbestos bodies. In: Int'l Konferenz uber die Bioloaischen Wirkunaen des Asbestos. Dresden, 22-24 April, 1968. Holstein, Anspach M (eds), Deutsches Zentralinstitut fur Arbeitsmedizin, Berlin, DDR, pp. 12-22.
2. Schwartz J, Langer AM: Technique of removal and analysis of single fibrous particles from human lung tissue. In: lnt'1 Konferenz uber die Bioloaischen Wirkunaen des Asbestos. Dresden, 22-24 April, 1968. Holstein, Anspach M (eds), Deutsches Zentralinstitut fur Arbeitsmedizin, Berlin, DDR, pp.8-12.
3. Bowes DR, Langer AM: Polyphase deformation in the schist of the Bronx, New York City. In: Geology. Queens College, CUNY, Alexandroff E (ed) 1(3):17-32, 1969.
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PUBLICATIONS IN SYMPOSIA PROCEEDINGS (cont):
4. Langer AM: Electron microprobe analysis (study of asbestos fibers and bodies from lung tissue). In: Laboratory Diagnosis of Diseases Caused bv Toxic Agents. Sunderman FW, Sunderman Jr FW (eds), W.H. Green; St. Louis, MO, Chapt. 14, pp. 126-136, 1970.
5. Langer AM, Rubin IB, Selikoff U: Electron microprobe analysis of asbestos bodies. In: Pneumonconiosis. Proc Int'l Conf, Johannesburg. Shaprio HA (ed), Oxford University Press; Capetown, pp. 57-69, 1970.
6. Langer AM, Baden V, Hammond EC, Selikoff IJ: Inorganic fibers, including chrysotile in the lungs at autopsy: preliminary report. In: Inhaled Particles III. Proc Conf Brit Occup Hyg Soc, London. Walton WH (ed), Unwin Bros; Surrey, England, 2:683-694, 1971.
7. Langer AM, Selikoff IJ: Chrysotile asbestos of lungs in residents of New York City. In: 2nd Int'l Clean Air Conaress. Washington, DC. Englund HM, Beery, WT (eds), Academic Press; New York, pp. 161-165, 1971.
8. Langer AM, Pooley FD: Identification of single asbestos fibers in human tissues. In: Proc Int'l Agency for Research on Cancer. Biol. Effects of Asbestos, Lyon, 1972, Bogovski P, Gilson JC, Timbrell V, Wagner JC (eds), pp. 119-125, 1973.
9. Langer AM: Aspects of mineralogy of talc. In: Proc Svmp on Talc. Washington, DC, May 9, 1973. Goodwin A (ed), USBM 1C 8639, US Department of Interior, pp. 82-88, 1974.
10. Pooley FD, Rohl AN, Langer AM, Bowes DE, Skinner DL: Mineralogy and chemistry of British talc and consumer talcum products. In: 4th Int'l Svmp Inhaled Particles and Vapors. Walton WH (ed), Pr. 8.6, pp. 1-18, 1975.
11. Langer AM, Rohl AN, Wolff MS, KJimentidis R, Shirey SB: Review of current techniques for the analysis of fibers in talc. In: Electron Microscopy of Microfibers. Asher IM, McGrath PP (eds), US Government Printing Office, pp. 28-33, 1977.
12. Langer AM, Wolff MS: Asbestos carcinogenesis. In: Inorganic and Nutritional Aspects of Cancer. Adv Exper Med Biol Series, Schrauzer GN (ed), Plenum Press; New York, pp. 29-55, 1977.
13. Nicholson WJ, Langer AM, Selikoff U: Epidemiological evidence on asbestos, Part l: Human health effects; Part II: Extrapolation to other inorganic fibers. In; Proc of Workshop on Asbestos. NBS SP 506. Gravatt CC, LaFleur PD, Heinrich KFJ (eds), with discussion. US Government Printing Office, Washington, DC, pp. 71-93, 1978.
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PUBLICATIONS IN SYMPOSIA PROCEEDINGS:
14. Rohl AN, Langer AM: Fibrous mineral content of consumer talc-containing products. In: Dusts and Diseases. Lemen R, Dement JM (eds), Pathotoxicol Publishers, Inc; Forest Park, ll_ pp. 393-403, 1979.
15. Langer AM, Rohl AN, Wolff MS, Selikoff IJ: Asbestos, fibrous minerals, and acicular cleavage fragments: nomenclature and biological properties. In: Dusts and Diseases. Lemen R, Dement JM (eds), Pathotoxicol Publishers, Inc; Forest Park, IL, pp. 1-22, 1979.
16. Langer AM, Maggiore CJ, Nicholson WJ, Rohl AN, Rubin IB, Selikoff IJ: The contamination of Lake Superior with fibrous amphiboles. In: Proc NY Acad Sci Asbestos Conf. New York, NY, June 1978, Ann NY Acad Sci 330:549-572,1979.
17. Langer AM: Significance of aspect ratio in regulation of asbestos fiber exposure. Ann NY Acad Sci 330:601-604, 1979.
18. Langer AM, Selikoff IJ, Rosenberg C: Defining new asbestos high risk groups. In: Int'l Conf Critical Current Issues in Environmental Health Hazards. Tel Aviv, Israel, March 4-7, p. 11, 1979.
19. Langer AM, Sebastien P: Physical-chemical properties of fibers: discussion. In: Biological Effects of Asbestos. IARC, Vol. 1, Wagner JC (ed), WHO, Lyon, France, pp. 143-145, 1980.
20. Suzuki Y, Rohl AN, Langer AM, Selikoff IJ: Mesothelioma following intraperitoneal administration of zeolite. Fed Proc 39(3) :640, 1980.
21. Langer AM: Natural non-fibrous asbestos substitutes: talc. In: Proc Nat'l Workshop on Substitutes for Asbestos. Guimond RJ, Rowe J (eds), US Environmental Protection Agency, Office of Pesticides and Toxic Substances, Washington, DC, pp. 563-569, 1980.
22. Rohl AN, Ehrenreich T, Langer AM: Characterization and identification of asbestos materials in human tissues. In: Adv Pathol (Anat and ClinL Vol. 2, Anatomic Pathology, Cytopathology, Forensic Pathology and Toxicology, Levy E (ed), Pergamon Press, New York, pp. 523-525, 1982.
23. Langer AM, Weisman I, Adams A: Characterization of crystalline particles in human tissues by analytical electron microscopy. J Rheumatol 8(6) :1018-1019, 1982.
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PUBLICATIONS IN SYMPOSIA PROCEEDINGS (cont):
24. Nolan RP, Langer AM: Quartz and hemolysis: physico-chemical factors controlling membrane activity. In: 4th Ann RMCOEH Conf Health Issues Related to Minina and Minerals. Wagner W, Merchant J, Rom W (eds), Chapt. 4, Ann Arbor Press, Michigan, pp. 63-81, 1983.
25. Langer AM: Surface characteristics and biological properties of minerals. In: World Svmp on Asbestos. May 25-27, 1982, Canadian Asbestos Information Center, Montreal, Quebec, pp. 393-400, 1983.
26. Langer AM, Nolan RP: Minerals, rocks and ore bodies: sources of agents of human disease. In: Proc 2nd Int'l Conor on Applied Mineralogy in the Minerals Industry. Park WC, Hausen DM, Hagni RP (eds), pp. 1159-1179, 1985.
27. Harlow GE, Kimball MR, Dowty E, Langer AM: Observations on amositegrunerite dusts. In: Proc 2nd Int'l Conor on Applied Mineralogy in the Minerals Industry. Park WC, Hausen DM, Hagni RP pp. 1147-1157, 1985.
28. Nolan RP, Langer AM, Foster KW: Particle size and chemically induced variability in the membrolytic activity of quartz: preliminary observations. In: |n Vitro Effects of Mineral Dusts. 3rd Int'l Workshop. Beck EG, Bignon J (eds), Springer-Verlag, Berlin, pp. 39-50, 1985.
29. Langer AM, Nolan RP: Physico-chemical properties of minerals relevant to biological activities. State of the Art. In: In Vitro Effects of Mineral Dusts. 3rd Int'l Workshop. Beck EG, Bignon J (eds), Springer-Verlag, Berlin, pp. 9-24,1985.
30. Langer AM, Nolan RP: Physico-chemical properties of quartz and biological activity. In: Silica. Silicosis, and Cancer. Controversy in Occupational Medicine. Ca Res Monogr 2, Goldsmith DF, Winn DM, Shy CM (eds), Praeger, NY, pp. 125-136, 1986.
31. Langer AM, Nolan RP: The properties of chrysotile asbestos as determinants of biological activity. Variations in cohort experience and disease spectra as related to mineral properties. In: General Motors Workshop. Biol Effects Chrysotile Asbestos. Cardiff, 7-9 May, 1986, Wagner JC (ed), Accomplishments in Oncology. Lippincott Press, 1(2):30-51, 1986.
32. Langer AM: Fibers in friction products. Discussion and Summary of Symposium. In: Proc Fibres in Friction Materials Symposium. The Asbestos Institute, Montreal, Canada, pp. 67-74, October, 1987.
33. Nolan RP, Langer AM: Quantitative aspects of fiber morphology. In: Proc Fibres in Friction Materials Symposium. The Asbestos Institute, Montreal, Canada, pp. 75-97, October, 1987.
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PUBLICATIONS IN SYMPOSIA PROCEEDINGS (cont):
34. Langer AM, Nolan RP: Fiber type and burden found in parenchymal tissues of workers occupationally exposed to asbestos in the United States. In: Mineral Fibers in the Non-Occupational Environment. Spec. Publ. 90. IARC-WHO, Lyon, France, pp. 310-315, 1989.
35. Langer AM, Nolan RP, Bowes DR, Shirey: Inorganic particles found in cigarette tobacco, cigarette ash, and cigarette smoke. In: Biological Interaction of Inhaled Mineral Fibers and Cigarette Smoke. Wehner AP, Felton DL, Eds., Battelle Press, Columbus, OH, pp. 421-439, 1989.
36. Nolan RP, Langer AM, Herson GB: Physico-chemical properties and membranolytic activities of the titanium dioxide polymorphs compared to quartz. In: Biological Interaction of Inhaled Mineral Fibers and Cigarette Smoke. Wehner AP, Felton DL, Eds., Battelle Press, Columbus, OH, pp. 391-419, 1989.
37. Langer AM, Nolan RP: Mineral fibers in lung tissues of persons exposed to asbestos in the United States. In: Vllth Int'l Pneumoconiosis Conf. August 23 26, 1988.
38. Nolan RP, Langer AM: Physicochemical characteristics of quartz dust which controls its biological activity. In: Vllth Int'l Pneumoconiosis Conf. August 23-26, 1988.
39. Langer AM, Nolan RP: Distinguishing asbestiform tremolite from non-asbestiform tremolite. In: Vllth Int'l Pneumoconiosis Conf, August 23-26, 1988.
40. Nolan RP, Langer AM, Herson GB: Membranolytic activity of palygorskite and sepiolite. In: IVth Int'l Workshop: Effects of Mineral Dusts on Cells. Mossman BT, Begin RO, Eds., Springer-Verlag, Oxford, Quebec, Canada, September, pp. 37-48, 1989.
41. Langer AM, Nolan RP, Pooley FD,: Phyllosilicates:associated fibrous minerals. In: Health Related Effects of Phyllosilicates. J. Bignon, Ed., 59-74, SpringerVerlag, N.Y., 1990.
42. Langer AM, Nolan RP, Addison J: Physico-chemical properties of asbestos as determinants of biological potential. In: Mineral Fibers and Health. Liddell FDS, Miller K, Eds., CRC Press, Ann Arbor, Michigan, pp. 207-224, 1991.
43. Langer AM, Nolan RP, Addison J: Distinguishing between amphibole asbestos fibers and elongate cleavage fragments of their non-asbestos analogues. In: NATO Advanced Research Workshop on Mechanisms in Fibre Carcinogenesis. RC. Brown, J. Hoskins, N. Johnson, Eds., Albuquerque, New Mexico, October 22-25, 1990. p. 253-267. 1991.
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PUBLICATIONS IN SYMPOSIA PROCEEDINGS (cont):
44. Nolan RP, Langer AM, Oechsle GW, Addison J, Colflesh DE: Association of tremolite habit with biological potential. In: NATO Advanced Research Workshop on Mechanisms in Fibre Carcinogenesis. RC. Brown, J. Hoskins, N. Johnson, Eds., Albuquerque, New Mexico, October 22-25, 1990. p.231-251. 1991.
45. Langer AM, Nolan RP: Zeolite catalysts. Is there a health risk? The erionite experience and its application to other zeolites, h: Chemicals and the Environment. Symposium on Chemical Specialties USA, Philadelphia. Spring Innovations, Ltd. 91-95, 1992.
46. Nolan RP, Langer AM: Limitations of the Stanton Hypothesis. Chapt. 9. Health Effects of Mineral Dusts, Reviews in Mineralogy, 28, Guthrie GD., Jr. and Mossman BT, Eds, Mineral. Soc. America, Wash., D.C., 309-326, 1993
47. Ross M, Nolan RP, Langer AM, Cooper WC: Health effects of mineral dusts other than asbestos. Chap. 12. Health Effects of Mineral Dusts, Reviews in Mineralogy, 28, Guthrie GD., Jr. and Mossman BT, Eds, Wash., D.C., 361-407, 1993.
48. Langer AM, Nolan RP: Factors controlling the biological potential of inorganic dusts. Surface chemistry and character. In: Toxic and Carcinogenic Effects of Solid Particles in the Respiratory Tract, Mohr U, Dungworth DL, Mauderly JL, Oberdosrster G, Eds., ILSI Monographs, ILSI Press, Wash. D.C., 147-157,1994.
OTHER PUBLICATIONS:
1. Langer AM, Mackler AD: Mineral particles and human disease. In: Encyclopedia of Geochemistry and Environmental Sciences. Fairbridge RW (ed), Van Nostrand; Reinhold, NY, Vol. IVA, pp. 730-739, 1972.
2. Miller A, Teirstein AS, Langer AM, Selikoff IJ: Submicroscopical asbestos fibers and disease. Letter to the Editor. Reply to discussion. N Engl J Med 292:1195 1196, 1975.
3. Wolff MS, Langer AM, Shirey SB: Gas chromatographs: health effects. Letter to the Editor. Science 191:13, 1976.
4. Langer AM, Pooley FD: Mineralogy of asbestos minerals and methods of their characterization in human tissues. In: Asbestos: Volume 14 in the Chemical Carcinogenesis Series. Int'l Agency for Research on Cancer, p. 107, 1977.
5. Rom WN, Langer AM: Carcinogenicity of fibrous glass. Letter to the Editor. West J Med 126:413, 1977.
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OTHER PUBLICATIONS (cont):
6. Rohl AN, Langer AM, Selikoff IJ: Asbestos pollution. Letter to the Editor. Reply. Science 197:716-718, 1977.
7. Rohl AN, Langer AM, Selikoff U: Chrysotile asbestos, effects of human exposure. Letter to the Editor. Reply. Science 198:1202, 1977.
8. Langer AM, Holaday D, Nicholson WJ, Rohl AN: Chapter 2. Asbestos Minerals: Nature, Occurrence and Properties, pp. 34-50. Chapter 4. Identification and Quantitation of Asbestos Fibers, pp. 71-100. Chapter 5. Environmental Distribution, pp. 102-134. In: Asbestos and Disease. Selikoff IJ, Lee WHK (eds), Academic Press; NY, 1978.
9. Langer AM: Relationship between chemical and physical properties of mineral fiber and health effects. NBS SP506, Workshop on Asbestos: Definitions and Measurement Methods. Gravatt CC, LaFleur PD, Heinrich KFJ (eds), pp. 104 195:198, 1978.
10. Langer AM: Mineralogical factors in asbestos health effects. NSB. SP506, Workshop on Asbestos: Definitions and Measurement Methods. Gravatt CC, LaFleur PD, Heinrich KFJ (eds), pp. 91-92; 198, 1978.
11. Rohl AN, Langer AM, Wylie AG: Mineral characterization of asbestos-containing spray finishes. In: Asbestos-Containing Materials in School Buildings. US Environmental Protection Agency, Document EPA 450-2-78-014, US Government Printing Office; Washington, DC, pp. 59-64, 1979.
12. Fischbein AS, Rohl AN, Langer AM, Selikoff U: Pleura-und-peritoneal mesothelioma. Letter to the Editor. Med Klin 74(19):742, 1979.
13. Langer AM: Electron microscopy and x-ray applications to environmental and occupational health analysis. Book Review. J Toxicol Environ Health, 1979.
14. Langer AM: Mineralogy and physical properties of fibers. Talcs contaminated with other minerals. Discussions, in: Dusts and Disease. Lemen R, Dement J (eds), Pathotoxicol Pub Inc, Park Forest, ll_ 120:122-123, 341-344,1979.
15. Langer AM: Round-table discussion on talc as an asbestos substitute. Chairman's remarks. In: Proc Nat'l Workshop on Substitutes for Asbestos, Arlington, VA, Guimond RJ, Rowe JN, Levin A, Pillsbury H (eds), USEPA Office Toxic Substances, Washington DC, pp. 612-619, July 14-16, 1980.
16. Fischbein AS, Langer AM, Rohl AN, Selikoff U: Asbestos disease in drywall construction workers. Letter to the Editor. Am Industr Hyg Assoc 40(9):829830, 1980.
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OTHER PUBLICATIONS (cont):
17. LangerAM: Mineralogy of dust diseases. In: Maxcv-Rosenau Public Health and Preventive Medicine. Last J (ed), Appleton-Century-Crofts Publishers; NY, 11th Edition, pp. 637-641, 1981.
18. Fischbein AS, Rohl AN, Langer AM, Seiikoff IJ: Past asbestos exposure among cable splicers. Letter to the Editor. Am J Pub Health 71:1277-1278, 1981.
19. Langer AM: Host rocks and gangue materials in relation to pneumoconiosis and cancer. Editorial. Am J Industr Med 2:89-90, 1981.
20. Langer AM, Sebastien P: Physical-chemical properties of fibers. Rappateur Report, in: Biological Effects of Mineral Fibers, Int'l Agency for Research on Cancer, Wagner JC (ed), IARC-WHO, Lyon, France, 1:143-145, 1981.
21. Langer AM, Seiikoff IJ: Scientific data on chrysotile. Letter to the Editor. Chem Engineer News 2:59, 1984.
22. Zoltai T, Langer AM: Chapter 2. Asbestiform fibers: historical background, terminology, and physicochemical properties. In: Asbestiform Fibers - NonOccupational Health Risks. NAS-NRC, Nat'l Acad Sci, pp. 25-47, 1984.
23. Lynch J, Langer AM: Chapter 4. Measurement of exposure to asbestiform fibers. In: Asbestiform Fibers - Non-Occupational Health Risks. NAS-NRC, Nat'l Acad Sci, pp. 82-96, 1984.
24. Langer AM: Mineral dust and pulmonary lesions: etiological link or epiphenomenon? Editorial. Am J Industr Med 6:169-171, 1984.
25. Fischbein AS, Langer AM, Rohl AN: Asbestos-associated diseases: lessons from the past for the future. Letter. JAMA 254:1309-1310, 1985.
26. Langer AM, et al.: Asbestos and other natural mineral fibers. In: Int'l Program on Chemical Safety. Environ Health Criteria 53, Chapters II, III, IV, V, WHO, Geneva, 1986.
27. Langer AM, Nolan RP: Asbestos in play sand. Letter, N Engl J Med 316:882, 1987.
28. Constantopoulos SH, Langer AM, Saratzis N, Nolan RP: Regional findings in Metsovo lung. Correspondence, The Lancet ii:452-453,1987.
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OTHER PUBLICATIONS (cont):
29. Langer AM: Mineralogy. Chapter II. Presidential Report on Occupational Chest Diseases, Vol. 1, Characterization and Measurement of the Environment Merchant J, Dement J (eds), ALOSH-NIOSH-CDC; Washington, DC, pp. 3-40, 1987.
30. Langer AM: Asbestos. In: Collier's Encyclopedia. 3:4-5, 1987.
31. Langer AM, Nolan RP, Constantopoulos S: Endemic pleural calcification and mesothelioma. JAMA 260(3):339-340, 1988.
32. Langer AM: Chairman's Summary and Discussion. In: Biological Interaction of Inhaled Mineral Fibers and Cigarette Smoke. Wehner AP, Felton DL, Eds., Battelle Press, Columbus, OH, pp. 590-593, 1989.
33. Langer AM, Nolan RP, Ross M: Asbestos policy. Science, 249:1485, 1990.
34. Langer AM: Asbestos. In: Merit Students Encyclopedia. Macmillan Ed Pub, NY, pp. 270-271, 1990.
35. Langer AM, Nolan RP: Pleural mesothelioma resulting from exposure to amosite asbestos in a building. Letter. Respiratory Medicine 84:509-510, 1990.
36. Langer AM, Nolan RP, Addison J: Letter to the Editor concerning Editorial" On talc, tremolite and tergiversation," Reger R, Morgan WKC. British Journal Industrial Medicine 48:359-360, 1991.
37. Langer AM: Chrysotile: The mineral and its properties. Summary of Presentation In: Health Risks Associated with Chrysotile Asbestos. Ann. Occup. Hyg., 38, 407. 1994.
38. Langer AM: Asbestos chapter: American Medical Association Primer on Chemical and Radiation Exposures. Davis, A., Ed. in press.
39. Nolan RP, Langer AM: Mineralogy. Chapter 12. In: Occupational and Environmental Respiratory Disease, Harber P., Schenker M., Balmes J., Eds. jn press.
PUBLISHED ABSTRACTS:
1. Berkley C, Langer AM, Berkley LE, Anderson CA: Chemical changes in inspired asbestos fibers. Microscopy Symp, McCrone Laboratory, Chicago, IL, August, 1966.
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PUBLISHED ABSTRACTS (cont):
2. Berkley C, Langer AM, Selikoff U: Microbeam analysis and identification of the degradation products of inspired pulmonary particulates. Trans NY Acad Sci, May, 1967.
3. Langer AM, Kerr PF: The nature of soft and harsh chrysotile. jn: Conference on the Physics and Chemistry of Asbestos Minerals, Oxford, Pr. 2-2, July, 1967.
4. Berkley C, Langer AM, Arneson A: Electron microprobe and electron diffraction analysis of asbestos bodies. In: 2nd Inti. Conf. Biol. Effects Asbestos, p. 7, Dresden, 22-25 April, 1968.
5. Berkley C, Langer AM, Rubin IB: Electron Microprobe analysis of particles in tissues, jn: Proc 5th Nat'l Conf Elect Probe Soc Am, New York, Pr. 29A, July, 1970.
6. Selikoff IJ, Nicholson WJ, Langer AM: Asbestos air pollution in urban areas. In: Proc AMA Air Pollution Med Res Conf, New Orleans, October, 1970.
7. Ehrenreich T, Langer AM, Selikoff IJ: Asbestos fibers in human lungs: forensic significance in environmental disease. !q: 6th Int'l Mtg Forensic Sci, Edinburgh, Int'l Assn Forensic Sci, p. 86, September, 1972.
8. Teirstein AS, Miller A, Langer AM, Selikoff IJ: Sublight microscopic mineral particles in the etiology of pulmonary fibrosis. jn: Proc Am Thorac Soc, New York, pp. 33-34, May 21-21, 1973.
9. Everett GA, Langer AM: Environmental contamination of Lake Superior asbestos from amphiboles in taconite wastes, in: Proc Ann Mtg Geol Soc Am, Miami, p. 728, November 18-20, 1974.
10. Rohl AN, Anderson H, Langer AM: Asbestos exposure during brake lining maintenance and repair. jn: Proc Am Ind Hyg Assn, Miami, May 12-16, 1974.
11. Langer AM, Pooley FD, Bowes DR: Environment, disease and minerals. Read before the Geological Society, London, November 20, J Geol Soc, with discussion, 1974.
12. Langer AM, Wolff MS, Rubin IB: Environmental assay: particles in human tissues. jn: Proc Int'l Conf Environ Sensing and assessment, Environmental Protection Agency, Pr. 29-34, September 14-19, 1975.
13. Bowes DR, Langer AM, Rohl AN: Nature and range of mineral dusts in the environment. Roy Soc London (Geological), p. 18, April 7, 1976.
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PUBLISHED ABSTRACTS (cont):
14. Langer AM, Suzuki Y, Rohl AN: Identification of Microparticles. In: 3rd Inti. Symp. on Detection and Prevention of Cancer. ACS. New York, p. 91, April 26-May 1, 1976.
15. Fischbein A, Langer AM, Suzuki Y, Selikoff U: Carcinoma of the lung in a drywall taping worker: report of a case, in: Prog Abstr 3rd Int'l Symp Detect Prevent Cancer, No. 280, September 7, 1976.
16. Rom WN, Frank A, Suzuki Y, Langer AM, Selikoff IJ: Rapidly progressive interstitial pulmonary fibrosis from short term mixed dust exposure. 42nd An Sci Assem Am Coll Chest Phys, Atlanta, GA, October 21 -28, 1976.
17. Fischbein A, Langer AM: Shale oil: A future energy alternative. A review of the mineralogic and carcinogenic properties of shale oil. Tenth Ann Conf Trace Substances in Environ Health, Columbia, MO, June 8-10, 1976.
18. Langer AM: Identification of microparticles in tissues. Workshop 13, 3rd Int'l Sump on Detection and Prevention of Cancer, September 1976.
19. Langer AM, Wolff MS: Asbestos carcinogenesis. ]n: Inorganic and Nutritional Aspects of Cancer, La Jolla, CA, January 2-5, 1977.
20. Selikoff IJ, Nicholson WJ, Langer AM: Epidemiological evidence on asbestos. Workshop on Asbestos, Definition and Measurement Methods, NBS Workshop, Gaithersburg, MD, July, 1977.
21. Langer AM: Contamination by submicroscopic particles in the biosphere. Int'l Symp Anal Electron Microscopy in Biol and Environ Routine and Research Work, Nat'l Inst Pub Health, Oslo, May 10, 1977.
22. Langer AM: Asbestos minerals and their carcinogenic effect. Int'l Symp Anal Electron in Biol and Environ Routine and Research Work, Nat'l Inst Pub Health, Oslo, May 10, 1977.
23. Langer AM, Oster G: Crystal faces and cleavage planes in quartz as templates in biological processes. Biophys Soc Am Phys Soc Mtg, Washington, DC, March, 1978.
24. Liiis R, Langer AM: Chronic interstitial pulmonary disease with severe respiratory disfunction due to unsuspected occupational beryllium exposure. In: XIXth Int'l Congr Occup Health, Dubrovnik, Yugoslavia, pp. 89-90, September 25-30,1978.
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PUBLISHED ABSTRACTS (cont):
25. Rohl AN, Langer AM: Asbestos and asbestiform minerals in non-asbestos mines in the United States, in: XIXth Int'l Congr Occup Health, Dubrovnik, Yugoslavia, pp. 121-122, September 25-30, 1978.
26. Russo DA, Kagan E, Langer AM, Yeager H: Human alveolar macrophages: cytotoxicity of naturally-occurring short fiber. Federation Proceedings, Section Physiology, Federation American Societies of Experimental Biology, 1980.
27. Langer AM: Physical-chemical characteristics of chrysotile and their relationship to biological activity. In: 29th Ann Clay Minerals Conf, Baylor, Waco, TX, p. 58, October 5-9, 1980.
28. Bowes DR, Langer AM: Mineralogy and geochemistry of particulates in cigarette smoke. ]n: Ann Mtg Geol Soc, London, Environmental Mineralogy, January 11, 1980.
29. Langer AM: Particulate inhalants. In: Occupational Toxicology, College of Medicine and Dentistry, NJ, September 26, 1980.
30. Langer AM: Minerals as toxic agents: parameters and mechanisms. In: Proc 50th Geol Alumni Assn, Brooklyn College, CUNY, May 7-9, 1981.
31. Langer AM, Weisman, I, Adams A, Pooley FD: Characterization of crystalline particles in tissue by analytical electron microscopy. !o: Symp on Crystalline Deposits in Tissues, Toronto, August 13-14, 1981.
32. Russo DA, Kagan E, Langer AM, Nolan R, Yeager H: Cytotoxicity of naturallyoccurring short fiber asbestos. In: 20th Int'l Congr Occup Health, Cairo, September 25-October 1, 1981.
33. Ehrenreich T, Langer AM, Selikoff IJ: Algorithm of pathologic diagnosis of asbestos-related diseases for forensic purposes. Significance of quantitative and morphological correlates. Jn: 20th Int'l Congr Occup Health, Cairo, September 25-October 1, 1981.
34. Rohl AN, Langer AM, Suzuki Y, Litis R, Moncure G, Selikoff IJ: Endemic pleural disease associated with mixed fibrous dust exposure in Turkey. Ini 20th Int'l Congr Occup Health, Cairo, September 25-October 1, 1981.
35. Nolan RP, Langer AM: Surface functionalities of quartz and hemolytic activity. !q: Proc 2nd Int'l Workshop of the In Vitro Effects of Mineral Dusts, Arkadelphia, AR, April 5-6, 1982.
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PUBLISHED ABSTRACTS (cont):
36. Langer AM, Nolan RP: Membrolytic properties of quartz, in: Proc 2nd Ann Geol Alumni Assn, Brooklyn College, CUNY, p. 4, April 28-30, 1983 (abstracts).
37. Nolan RP, Langer AM, Foster KW: Recognition of quartz by erythrocyte membranes, in: 6th Int'l Penumoconiosis Conf, Bochum, FDR, September 20-23, 1983 (Abstract 198).
38. Nolan RP, Langer AM, Foster KW: Physico-chemical factors effecting membrolytic properties of quartz, in: 6th Int'l Pneumoconiosis Conf, Bochum, FDR, September 20-23, 1983 (Abstract 199).
39. Langer AM, Nolan RP: Surface properties of quartz and membrane activity, in: Int'l Symp Silica, Silicosis and Cancer, Chapel Hill, NC, April 3-5, 1984.
40. Langer AM: Asbestos, fibrous minerals, acicular cleavage fragments and the mineral industries, in: Int'l Congr Applied Mineralogy, Los Angeles, February, 1984.
41. Langer AM: Minerals and disease: the association between exposure to minerals and rock dust and the occurrence of human disease. In: Int'l Congr Applied Mineralogy, Los Angeles, February, 1984.
42. Langer AM: Mineral fiber in water? in Medical Geology Conference on Health Threatening Toxins in Water, Brooklyn College, NY, May, 1984.
43. Constantopoulos SH, Langer AM, Saratzis N, Goudevenos JA, Selikoff IJ: Metsovo lung: pleural calcifications and malignant pleural mesothelioma in northwestern Greece. Tremolite asbestos used for whitewashing as etiology, in: 3rd Int'l Conf Environmental Lung Disease, Am Coll Chest Phys, Montreal, Canada, October 15-18, 1986. Chest 91(2):300, 1987.
44. Langer AM, Nolan RP: Fiber type and parenchymal burden found in workers occupationally exposed to asbestos fiber in the United States. Implications for risk assessment in the general population. Mineral Fiber in the NonOccupational Environment, Lyon, France, October 8-10, 1987.
45. Langer AM, Nolan RP: Mineralogical and biological comparison of asbestiform and non-asbestiform tremolite. AIME Annual Meeting, Phoenix, AZ, January 25 28, 1988.
46. Langer AM, Nolan RP, Bowes DR, Shirey S: Inorganic particles in cigarettes and cigarette smoke, in: Biological Interaction of Inhaled Mineral Fibers and Cigarette Smoke. International Symposium/Workshop, Seattle, WA, April 10-14, 1988.
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PUBLISHED ABSTRACTS (cont):
47. Nolan RP, Langer AM, Herson GB: Physico-chemical properties of the titanium dioxide polymorphs and biological activity. in: Biological Interaction of Inhaled Mineral Fibers and Cigarette Smoke. International Symposium/Workshop, Seattle, WA, April 10-14, 1988.
48. Nolan RP, Langer AM, Herson GB: Membranolytic activity of palygorskite and sepiolite. !q: IVth International Conference on the Effects of Mineral Dusts on Cells, p. 15, Oxford, Quebec, Canada, 1988.
49. Langer AM, Nolan RP, Herson GB: Membranolytic activity and physicochemical characterization of phosphorylated Canadian chrysotile. ]q: IVth International Conference on the Effects of Mineral Dusts on Cells, p. 14, Oxford, Quebec, Canada, 1988.
50. Nolan RP, Langer AM, Herson GB: Physicochemical characteristics of quartz dust which controls its biological activity, in: Vllth International Pneumoconiosis Conference, DHHS (NIOSH) Publication No. 90-108, Part I, 754, 1990.
51. Langer AM, Nolan RP: Fiber type and risk of mesothelioma to building occupants. Jn: Health Aspects of Exposure to Asbestos in Buildings. Energy and Environmental Policy Center, John F. Kennedy School of Government, Harvard University, Cambridge, MA, December 14-16, 1988.
52. Langer AM, Pooley FD, Nolan RP: Phyllosilicates: Associated fibrous minerals, in: First International Conference on Health Related Effects of Phyllosilicates. NATO Advanced Research Workshop, Paris, France, March 16-18, 1989.
53. Nolan RP, Langer AM, Herson GB: Characterization of palygorskite specimens from different geological locales for health hazard evaluation, in: First International Conference on Health Related Effects of Phyllosilicates. NATO Advanced Research Workshop, Paris, France, March 16-18, 1989.
54. Langer AM, Nolan RP: The importance of mineral subpopulations in biological assays, in: NATO Advanced Research Workshops on Mechanisms in Fibre Carcinogenesis. Albuquerque, New Mexico, October 22-25, 1990. 49-50.
55. Nolan RP, Langer AM, Oechsle GW, Addison J, Colflesh DE: Association of tremolite habit with biological potential, in: NATO Advanced Research Workshops on Mechanisms in Fibre Carcinogenesis. Albuquerque, New Mexico, October 22-25, 1990. 57.
56. Langer AM: Fiber types, fiber sizes, and asbestos diseases. Andrews Communications Seminars. 2nd Asbestos Litigation. The Eye of the Storm. 25-26 February, 1991. Bal Harbour, Florida.
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PUBLISHED ABSTRACTS (cont):
57. Nolan RP, Langer AM, Oechsle GW, Johnson NF: Physicochemical characterization of selected zeolite minerals for health hazard evaluation. Fourth Inti. Conf. Env. Lung Disease. Montreal, Canada, September 25-28, 1991.
58. Nolan RP and Langer AM: Characterization of fibers for Health Hazard evaluation. Critical Research Needs. Workshop on Chemical and Biological Interactions of Glass. Bethesda, MD, 5-6 Mar, 1992.
59. Langer AM and Nolan RP (1992). Comparison of lung tissue mineral fibre retention of exposed workers and the general population. ]ri: Biopersistence of respirable synthetic fibres and minerals. 7-9 Sept, 1992. Lyon. p. 26.
60. Nolan RP, Langer AM, and Addison J (1992). Health hazard evaluation of the lung tremolite fiber content among Canadian chrysotile workers. In: Biopersistence of respirable synthetic fibres and minerals. 7-9 Sept, 1992. Lyon, p. 52.
61. Langer AM, Nolan RP (1993). Factors controlling the biological potential of inorganic dusts: surface character and chemistry. 4th International Inhalation Symposium, Hannover. Toxic and Carcinogenic Effects of Solid Particles in the Respiratory Tract, 1-5 March, 1993, Hannover. Abst. A-10, p. 48.
62. Langer AM, Nolan RP (1993) Physico-chemical characteristics of quartz dust which controls its biological activity. Clay Minerals Society Meeting. San Diego, 25-30 Sept, 1993.
MANUSCRIPTS IN PREPARATION:
Langer AM, Kimball MR, Harlow GE: A comparison of fibrillar structure in amosite and crocidolite.
Langer AM, Kimball MR, Harlow GE, Dowty E: Electron microscopy studies of amosite and grunerite dust particles.
Langer AM: Distribution of amosite and chrysotile fibers in the lung of an exposed worker: role of fiber size and type.
Nolan RP, Langer AM: Mineral surfaces and the anion transport system of the human red blood cell. Mechanism of hemolytic action.
Langer AM, et al: Amosite fiber size-distribution at a fabrication plant: mesothelioma and fiber dimension.
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MANUSCRIPTS IN PREPARATION (cont):
Langer AM, et aI: Asbestos bodies in two periods of time: review, secular trends, New York City, 1915-1970.
Langer AM, et gl: Asbestos bodies and age, sex, occupation of urban dwellers: asbestos bodies as indices of disease risk
PARTICIPATION IN POSTGRADUATE EDUCATION COURSES (1976-PRESENT):
6/70 - Page and Black Postgraduate, Mount Sinai School of Medicine: Asbestosis.
4/71 - NY Academy of Sciences, Seminars for Trade Union Representatives: Occupational Health Hazards.
4/71 - American College of Chest Physicians, Mount Sinai School of Medicine, NY: Interstitial Pneumonias-Acute and Chronic.
5/75 - United States-USSR Environmental Protection Agreement: Workshop on Basic Practical Approaches to Environmental Carcinogenesis, Mount Sinai.
6/76 - Page and Black Postgraduate, Mount Sinai School of Medicine: Environmental Lung Disease.
2/77 - Office of Continuing Education, Baylor Medical College, Houston, TX: Pulmonary Diseases and Carcinoma of the Lungs, Baylor Medical School, Houston, TX.
11/77- New York Lung Club, Cornell University Medical College: Silicosis revisited. New York City Tunnel and Caisson Workers: 1928-1977.
1/78 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Asbestos Carcinogenesis.
3/78 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Occupational and Environmental Pulmonary Diseases.
6/78 - Page and Black Postgraduate, Mount Sinai School of Medicine, NY: Health Effects of Asbestos Exposure. Course Director.
11/78 - Page and Black Postgraduate, Mount Sinai School of Medicine, NY: Asbestos Associate Diseases: With Particular Reference to United States Shipyards.
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PARTICIPATION IN POSTGRADUATE EDUCATION COURSES (1976-PRESENT) (cont):
1/79 - Page and Black Postgraduate, Mount Sinai School of Medicine, NY: Scientific Basis for Evaluation of Occupational and Environmental Asbestos Disease.
10/79 - Office of Continuing Education, Baylor Medical College, Houston, TX: Asbestos-Associated Diseases, Houston, TX.
1/80 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Asbestos in Shipyards.
3/80 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Management and Control of Asbestos in Public Buildings.
6/80 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Health Effects of Asbestos in Public Buildings.
9/80 - College of Medicine and Dentistry of New Jersey: Rutgers Medical School, Recent Advances in Occupational Toxicology, Graduate Center, NY.
11/81 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Recent Advances in Occupational Medicine.
12/82 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Scientific Basis for Evaluation of Asbestos-Associated Diseases.
3/85 - Page and Black Postgraduate, Mount Sinai School of Medicine, New York, NY: Asbestos in Schools and Public Buildings, Hasbrook Heights, NJ.
9/91 - Environmental Lung Disease. American College of Chest Physicians. Mineralogy Techniques. Montreal, Canada.
INVITED SEMINARS AND LECTURES:
Union College, Schenectady, NY, Department of Geology and Civil Engineering. "Asbestos and Lung Disease." April, 1967.
Barnard College, NY, Department of Geology. "Asbestos Fiber in the Ambient Air and Disease Potential." December, 1968.
The Rockefeller University, Air Pollution Working Group, Health Research Council. "Analysis of Air Samples for Inorganic Constituents." June 9, 1969.
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INVITED SEMINARS AND LECTURES (cont):
New York University Medical Center, New York Lung Club. "Sublight Microscopic Mineral Particles in Talc Pneumoconiosis. Physiology and Mineralogical Studies." November, 1970.
Leheigh College, Department of Material Sciences and Metallurgy. "Electron Probe Characterization of Particles in Tissues." April, 1971.
Oberlin College, OH, Department of Geology. "Asbestos Dust in the Workplace and Human Disease." May, 1971.
State University of New York at Buffalo, Department of Biophysical Sciences. "The Asbestos Problem. An Interdisciplinary Approach." April, 1972.
Queens College of the City University of New York, Department of Earth and Environmental Sciences. "Asbestos and Disease. How Wide is the Spectrum?" November, 1972.
Columbia University, NY, Department of Geology and Biological Sciences. "Contamination of the Environment with Mineral Particles." December, 1972.
College of South Hampton, Long Island University, Division of Chemical Sciences and Oceanography. " The Contamination of Lake Superior with Fibrous Silicates." January, 1973.
National Institutes of Health. Review of Asbestos Problem. "Mechanism of Fiber Action." National Institutes of Health, Bethesda, MD, February, 1973.
City College of the City University of New York, Department of Earth and Planetary Sciences. "Mineral Particles and Human Disease." May, 1973.
Queens College of the City University of New York, Department of Health Sciences. "Occupational Hazards as Harbingers of Environmental Hazards." September, 1973.
Rutgers University, Colonial Conference Tri-State Environmentalists. "Recognition of Environmental Hazards: Use of Academic Facilities for Monitoring and Control of Hazards." November, 1973.
Johns Hopkins University, School of Public Health. "Asbestos Minerals and Disease." Seminar. April, 1974.
Rutgers University, Department of Environmental Sciences. "Mineral Particles in the Air." June, 1974.
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INVITED SEMINARS AND LECTURES (cont):
Temple University, Department of Geology. "Amphibole Contamination of Lake Superior Water from Taconite Processing." September, 1974.
American Museum of Natural History, Department of Mineral Sciences. "Minerals and Disease." September, 1978.
Rutgers University, Department of Chemistry. "Minerals and Their Biological Activity." October, 1978.
University of North Carolina, Department of Medicine. 1) "Analytical Methods of Tissue Analysis, Asbestos Bodies and Asbestos Fibers"; 2) "Inorganic Particles in Cigarettes and Cigarette Smoke." November, 1978.
University of Maine, Farmington, Department of Geology. 1)'The Asbestos Problem"; 2) "Evolution of an Environmental Scientist, A Personal View." March, 1979.
School of Public Health, Columbia University. "Epidemiology of Asbestos Diseases." April, 1979.
New York Medical Examiner's Conference. "Analysis of Tissues for Asbestos Fiber and Asbestos Bodies." June, 1979.
Seton Hall University, Department of Chemistry. "Minerals and Disease: Interaction Mechanisms." October 2, 1979.
American Institute of Mining Engineers, Tucson. "The Biological Effects of Inorganic Fibers: The Case Against Asbestos" (invited speaker). October, 1979.
American Chemical Society, Staten Island Section. "Minerals and Diseases: Physical and Chemical Factors." February 19, 1980.
Society of Sigma Xi Lecture. "Man in Conflict with His Physical Environment." Invited Speaker for initiation ceremony, City University, New York, May 13,1980.
National Institutes of Health, Bethesda, Department of Health and Human Services CCERP-26. "Serpentine-Containing Host Rocks: Amphibole-Containing Host Rocks." November, 1980.
NIEHS-EPA-NIOSH Joint Committee at Bethesda. "Mount St. Helen's Volcanic Ash: Mineral Nature and Biological Activity." July 23, 1981.
The Rockefeller University. "Minerals and Toxic Agents, Parameters and Mechanisms." Invited speaker in series on Comparative Toxicology, February 12, 1981.
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INVITED SEMINARS AND LECTURES (cont):
Kettering Laboratory, University of Cincinnati Medical Center. "Silica and Silicosis: Mechanisms." April 8, 1981.
University of California, Irvine, Occupational and Environmental Health Center. "Physicochemical Properties of Inorganic Dusts and Biological Consequences of Exposure." Occupational Medicine Series, December 2, 1981.
Barlow Hospital, Los Angeles, University of Southern California Medical School. "Tissue Analysis for Asbestos, Comparative Techniques." Chest Service, Department of Medicine, December 3, 1981.
Appalachian Laboratory for Occupational Safety and Health NIOSH-CDC, Morgantown, West Virginia. "Surface Properties of Silica and Its Biological Activity." February 24, 1982.
National Institute for Occupational Safety and Health. "Silica and Silicosis." Cincinnati, March 24, 1982.
Society of Sigma Xi Lecture, Queens College of the City University of New York. "The Case Against Chrysotile Asbestos." December 2, 1982.
National Institute of Environmental Health Sciences, Pathobiology of Mesothelioma, Research Triangle Park, NC. "Physicochemical Character of Dust and its Role in Inducing Human Mesothelioma." January, 1983.
Geology Seminar, Long Island University, College at South Hampton. 'The Biological Effects of Asbestos." March 18, 1983.
Graduate Seminar, Rutgers University, Department of Geology. "Physical-Chemical Properties of Minerals and Their Biological Activities." April 13, 1983.
Symposium on Fibrous Minerals, American Lung Association Annual Meeting, Kansas City, MO. "Mineralogy of Mineral Fibers." May 8, 1983.
Faculty Research Collegium, Ph.D. Program Earth and Environmental Studies, CUNY, Graduate Center, NY. "Physicochemical Properties of Minerals Controlling Biological Activity." March 6, 1984.
Graduate Seminar, Department of Geology, Princeton University. "Minerals and Disease." Princeton, NJ, March 8, 1984.
Graduate Seminar, Department of Biology, New York University, New York, NY. "Asbestos: Biological Considerations." April 16, 1984.
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INVITED SEMINARS AND LECTURES (cont):
Medical Geology Conference on Health Threatening Toxins in Water. "Asbestos Fiber in Water." May 3, 1984.
Asbestos Information Association of North America, Annual Meeting, Alexandria, VA. "Asbestos Substitutes. How Safe are They?' September 18, 1985.
American Society for Testing Materials, Annual Meeting, Bal Harbour, FL "Method for Testing Indoor Air for Asbestos. Analytical Electron Microscopy." November 4, 1985.
University of Southern California Medical School. "Membranolytic Properties of Quartz and Phosphorylated Chrysotile Fiber." January 29, 1985. Main Campus. "Asbestos Fiber in Lung Tissue - Analysis by Analytical Electron Microscopy." January 30, 1986.
Milton Kannerstein Lecture. "Mineral Dust and Pulmonary Lesions." Annual Meeting of the Klemperer-Otani Society. Mount Sinai, June 5, 1986.
Northeast Regional Environmental Public Health Center, University of Massachusetts. Symposium: Asbestos in Play Sand. "Tremolite Analysis, Morphology, and Health Significance." February 11,1987.
The Defense Research and Trial Lawyers Association, Reno, NV. "Analytical Protocol for the Study of Sublight Microscopic Particles in Human Tissues"; 'Types and Amounts of Asbestos Fibers in the Pulmonary Tissues of Asbestos-Exposed Workers in the United States." October 30, 1987.
The Manville Settlement Trust Asbestos Disease Seminar, Washington, DC. "Asbestos Fibers: The Physico-Chemical Properties and Health Effects." March 16, 1988.
Asbestos Fiber Type and Risk of Disease, Conference: New York - New Jersey Environmental Expo: The Source for Environmental Solutions. Seminar 7E, The Management of Asbestos in the 1990's. Thursday, October 18, 1990.
American Industrial Hygiene Association, Georgia Section, Atlanta Georgia. Conference: The Asbestos Controversy. Has the Public Been Properly Informed? "Understanding the Asbestos Risk." November 30, 1990.
Armstrong Defense Group. Memphis, Tennessee. The Asbestos Saga and the Star Wars Trilogy: The Empire Strikes Back! Thursday, April 25, 1991.
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INVITED SEMINARS AND LECTURES (coot):
Department of Medicine - Pulmonary, New York University Medical Center. Pulmonary Grand Rounds. Particle Characteristics Responsible for Pneumoconiosis and Cancer. Tuesday, September 3, 1991.
Nelson Institute of Environmental Medicine, New York University School of Medicine. Surface Properties of Minerals as Determinants of Biological Activity. Tuxedo, New York. Wednesday, October 23, 1991.
Defense Research Institute. Current Issues in Asbestos in Buildings Seminar. Report From the Health Effects Institute. Orlando, Florida. Friday, January 24, 1992.
Characteristics of Mineral Fibers - Critical Research Needs. Invited to Workshop on Chemical and Biological Interactions of Glass. Bethesda, Maryland, March 5-6,1992.
Mealey's National Lead Litigation Conference. Presented: Bioavailabilitv of Lead. April 22-23, 1993. Philadelphia, Pennsylvania.
McGrath Fund Lectures. Current Topics in Environmental Science. Division of Natural Sciences, South Hampton Campus of Long Island University. Update on Asbestos. An Asbestos Risk Assessment. May 3, 1994.
United States Environmental Protection Agency. Environmental Asbestos Workshop: Chrvsotile Asbestos. Comparative carcinogenicity with the amohibole asbestos varieties. Monterey, California. October 7, 1994.
Defense Research Institute Symposium. Asbestos Medicine Seminar. Health risks associated with low-level asbestos exposures, (with Dr. B. Gee). New York. October 26-28, 1994.
INVITED INTERNATIONAL MEETINGS AND CONFERENCES:
Physics and Chemistry of Asbestos Minerals. Oxford, July, 1967. Presented: The Nature of Soft and Harsh Chrvsotile (with Kerr PF.).
Second International Conference on the Biological Effects of Asbestos. Dresden, DDR, April, 1968 (two papers with Berkley C, Sastre A, Ameson A and Schwartz J).
International Conference on Pneumoconiosis. Johannesburg, April 23-May 2,1969. Presented: Electron Microprobe Analysis of Asbestos Bodies (with Rubin IB and Selikoff IJ).
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INVITED INTERNATIONAL MEETINGS AND CONFERENCES (cont):
Working Group: Asbestos and the Asbestos Diseases. Cardiff, April 6-9, 1970. Presented: Identification of Asbestos Fiber in Tissue.
British Occupational Hygiene Society: Inhaled Particles and Vapors. Imperial College, London, June, 1970. Presented: Inorganic Fibers. Including Chrvsotile. in Lungs at Autopsy (with Baden V, Selikoff !J, and Hammond EC).
Fifth National Conference on Electron Microprobe Analysis. New York, July 21 -24, 1970. Invited paper: Electron Microorobe Analysis of Particles in Tissues (with Berkley C and Rubin IB).
Second International Clean Air Congress. Washington, DC, May 1971. Paper with Selikoff IJ.
International Agency for Research on Cancer. WHO, Lyon, September, 1972. Presented: Identification of Single Asbestos Fibers in Human Tissues with (Pooley FD).
6th International Meeting of Forensic Sciences. Edinburgh, September, 1972. Presented paper Asbestos Fiber in Human Lunos: Forensic Significance in Environmental Disease (with Ehrenreich T).
Dialogues in Microscopy, 1973, NY. Microscopical Society. Chaired Session: Identification of Asbestos, May, 1973.
International Conference on the Biological Effects of Ingested Asbestos. NIEHS sponsored. Pinehurst, NC, November, 1973.
International Conference on Environmental Sensing and Assessment. Las Vegas, NV, September, 1975 (paper with Rubin IB and Wolff M). Chaired 2 Sessions: Fine Particles-7; Fine Particles-2. United States Environmental Protection Agency.
Conference on Health Problems of Energy Technologies. Held by NIEHS, January, 1976.
Mineralogical Society of London. London, April, 1976. Presented paper Nature and Range of Mineral Dust in the Environment.
International Agency for Research on Cancer. The World Health Organization, Lyon. Invited member of working group, December, 1976. Chemical Carcinogenesis Monograph Series. Evaluation of Carcinogenic Risk of Asbestos. Vol. 14.
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INVITED INTERNATIONAL MEETINGS AND CONFERENCES (cont):
Royal Society of Medicine. Meeting Focusing on Occupational Diseases. London, April, 1977. Presented paper Asbestos Content of Dust Encountered During Brake Maintenance and Repair (with Rohl AN, Weisman I, and Klimentidis R).
Invited Consultant to Institute of Public Health. Oslo, Norway, May, 1977. Presented three papers: Contamination of the Biosphere bv Submicroscopic Particles: Asbestos Minerals and Their Carcinogenic Effects: Are There Safe Substitutes for Asbestos Materials?
Invited Consultant to the Ministry of Mines. Biological Effects of Asbestos. Johannesburg, October, 1977. Monograph, Ministry of Mines.
Health Hazards of Asbestos Exposure. New York, June 24-27, 1978. Presented paper on Contamination of Lake Superior with Amohibole Ganaue Minerals and organized and chaired workshop on Significance of Aspect Ratio in Regulation of Asbestos Fiber Exposure.
International Conference on Critical Current Issues in Environmental Health Hazards. Tel Aviv, March 4-7, 1979. Presented paper Defining New Asbestos High Risk Groups. Chairman Workshop: Laboratory Approaches in Environmental Medicine.
International Agency for Research on Cancer. WHO, Lyon, October, 1979. International Conference on the Biological Effects of Inorganic Fibers. Invited Rappateur. Physics and Chemistry of Asbestos Minerals.
Expert Consultant, International Metal Workers Federation. Geneva, September, 1980. Focus on problem: Asbestos Fiber Contamination of Metal Ore Deposits.
International Conference on Crystalline Deposits in Human Tissues. August 13-14, 1981. Mount Sinai Hospital, Toronto. Presented paper The Role of Analytical Electron Microscopy in Diagnosis of the Pneumoconiosis (with Weisman I, Adams A, and Pooley F).
Annual Symposium Scanning Electron Microscopy. Anaheim, CA, April, 1982. Presented paper Identification Characterization and Quantitation of Asbestos Fibers in Human Lung Tissue bv Analytical Electron Microscopy.
Fourth Annual Rocky Mountain Center for Occupational and Environmental Health Center. Salt Lake City, April, 1982. Invited to present two papers: Fibrous Minerals: Properties and Biological Activity: Relationship Between Surface Properties and Biological Activity in Health Issues Related to Metal and Non-Metallic Minina.
World Symposium on Asbestos. Montreal, May 24-27, 1982. Presented paper Mineral Fibers: Properties Imparting Biological Activity.
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INVITED INTERNATIONAL MEETINGS AND CONFERENCES (cont):
6th International Pneumoconiosis Conference. Bochum, Federal Republic of Germany, September, 1983. Presented two papers: Recognition of Quartz by the Ervthrocvte Membrane: Physicochemical Factors Effecting Membrolvtic Properties of Quartz (with Nolan RP, Foster KW, and Simenski R). Chaired Session: Etiopathogenesis of Silicosis.
Program of Research on Asbestos. University of Sherbrooke, December 5-6,1983. Presented lecture Physicochemical Properties of Minerals as Related to Biological Activities.
2nd International Congress on Applied Mineralogy. February 22-25,1984. Presented two papers: Asbestos. Fibrous Minerals. Acicular Cleavage Fragments and the Minerals Industries: Minerals and Disease: The Association Between Exposure to Minerals and Rock Dusts and the Occurrence of Human Disease.
Workshop on the Assessment of Mineral Fibers from Human Lungs. Oxford, England, September 17-19,1984. Keynote speaker Problems of Tissue Analysis with the Analytical Electron Microscope.
3rd International Workshop on the ]n Vitro Effects of Mineral Dusts. Schluchsee, Hochschwarzwald, FRG, October, 1984. Physicochemical Properties of Minerals Relevant to Biological Activities. State of the Art, (paper with Nolan RP); Alteration of surface of quartz and altered biological activity.
Workshop on Biological Effects of Chrysotile. General Motors Cancer Research Foundation. Cardiff, Wales, May 7-9,1986. The Mineralogy of Chrysotile Asbestos: Physicochemical Properties as Determinants of Disease Potential (with Nolan RP).
International Programme on Chemical Safety (IPCS). WHO Environmental Health Criteria 53. Asbestos and other Natural Mineral Fibres. Hannover, Germany. Session chairman, co-author. 194 p., 1986.
International Agency for Research on Cancer. The World Health Organization. Invited member of working group. Lyon, France, July, 1986. Chemical Carcinogenesis Monograph Series. Evaluation of Carcinogenic Risk of Fibrous Talc. Wollastonite. Palvoorskite. Sepiolite. Erion'ite and Crystalline Silica. Vols. 34-42.
Symposium: Mineral Fibers in the Non-Occupational Environment. IARC-WHO. Lyon, France, September 8-10, 1987. Summary of five presented papers; Round table discussant on Environmental Risk. Presented paper: Fiber Type and Parenchymal Burden Found in Workers Occupationally Exposed to Asbestos Fiber in the United States (with Nolan RP).
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INVITED INTERNATIONAL MEETINGS AND CONFERENCES (cont):
Symposium: Fibers in Friction Materials. Society Automotive Engineers and the Asbestos Institute. Symposium organizer and Co-Chairman. Fibers and Health Issues. Atlantic City, NJ, October, 1987. Presented summary paper; co-authored Physicochemical Properties of Fibers and Biological Potential (with Nolan RP).
Symposium: Safe Use of Asbestos Cement. Peruvian Ministry of Health and the Environment. The Asbestos Information Association of South America. Lima, Peru, March, 1988.
International Symposium/Workshop: Biological Interaction of Inhaled Mineral Fibers and Cigarette Smoke. Session Chairman. Presented paper: Inorganic Particles Found in Cigarette Tobacco. Cigarette Ash, and Cigarette Smoke: co-authored Physico-Chemical Properties and Membranolvtic Activities of the Titanium Dioxide Polymorphs Compared to Quartz. April 10-14, 1988.
Electron Microscopy Society of America; the Microscopical Society of Canada: Presented paper: Electron Diffraction of Mineral Fibers Vs. Acicular Cleavage Fragments. Milwaukee, Wl, August 7-12, 1988.
National Organizing Committee, Workshop Chairman: Hazard Recognition of Mineral Dusts. Presented: Mineral Fibers in the Lung Tissues of Persons Exposed to Asbestos in the United States: Distinguishing Between Tremolite Asbestos and Tremolite Cleavage Fragments. Vllth International Pneumoconiosis Conference, Scientific Organization Committee. Pittsburgh, PA, August, 1988.
Session Chairman: Asbestos. Pleural Pathology, and Luno Fiber Burden. Vllth International Pneumoconiosis Conference, Pittsburgh, PA, August, 1988.
Workshop on Asbestos Research. Health Effects Institute, American Academy of Arts and Sciences, Cambridge, MA, October 31-November 1, 1988.
Symposium on the Health Aspects of Exposures to Asbestos in Public Buildings. Presented: Fiber Type and Risk of Mesothelioma to Building Occupants. John F. Kennedy School of Government, Harvard University, Cambridge, MA, December 14 16, 1988.
First International Conference on Health Related Effects of Phyllosilicates. International Scientific Organizing Committee. Session Chairman: Health Related Effects After Non-Occuoational Exposure. Presented: Fibrous Minerals as Natural Contaminants of Phyllosilicates (with Pooley F). Paris, France, March 16-18,1989.
International Federation of Building and Woodworkers. Health Hazards in Painting and Allied Trades. Presented: Hazards in the Painting Trade. Panelist. Geneva, Switzerland, May 8-12, 1989.
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INVITED INTERNATIONAL MEETINGS AND CONFERENCES (cont):
NATO Advanced Research Workshop on Mechanisms in Fibre Carcinogenesis. Presented: The Importance of Mineral Subpopulations in Biological Assays. Alburquerque, NM, October 22-25, 1990. Co-authored paper: Association of tremollte habit with biological potential, co-authored with Nolan RP, Oechsle G, Addison J, Colfiesh D.
American Lung Association - American Thoracic Society. 1991 International Conference. Presented: Mechanisms of Asbestos-Induced Pulmonary Disease: Importance of Surface Properties of Asbestos and Non-asbestos minerals in cell interaction. Anaheim, California, May 12-15, 1991.
American College of Chest Physicians. Fourth International Conference Environmental Lung Disease. Presented: Diagnostic Methods in Occupational Luna Disease: Mineraloaic Techniques. Co-Chair session: Diagnostic Methods in Occupational Lung Disease. Montreal, Canada. September 25-28, 1991.
Chemical Industry Institute of Toxicology. Workshop on Approaches to Evaluating the Toxicity and Carcinogenicity of Man-made Fibers. Durham, North Carolina. November 11-13, 1991.
Institute for Glass Science and Engineering. Workshop on Chemical and Biological Interactions of Glass. Discussant on Biologically Important Properties. Bethesda, Maryland. March 5-6, 1992.
International Geological Congress. Environmental Mineralogy in relation to human health and activities. Session 1-3-49. Co-chair with N. Kohyama, J. Addison, Kyoto, Japan. 24 August-3 September, 1992. Introduction. Importance of environmental mineralogy. Summing up. 1 September, 1992.
Biopersistence of Respirable Synthetic Fibres and Minerals. Presented: Comparison of lung tissue mineral fibre retention of exposed workers and the general population. With RP Nolan, co-authored: Health hazard evaluation of the lung tremolite fibre content among Canadian chrvsotile workers. With RP Nolan and J Addison. Round Table discussant on: Role of Biopersistence in Pathogenicity. IARC, Lyon. September 7-9, 1992.
Symposium on Chemicals and the Environment. Chemical Specialities, '92. Invited presentation: Zeolite catalysts. Is there a health risk? Philadelphia, 3-4 November, 1992.
International Life Sciences Institute. 4th International Inhalation Symposium, Hannover. Invited Faculty Presentation: Factors Controlling the Biological Potential of Inorganic Dusts. Surface Chemistry and Character. With R.P. Nolan. Hannover, 1-5 March, 1993.
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INVITED INTERNATIONAL MEETINGS AND CONFERENCES (cont):
Hoffmann-LaRoche Pharmaceuticals. Analysis of Medicines for Asbestos. Microscopy Protocol. Edinburgh, U.K. Institute of Occupational Medicine. April 25-26, 1993.
Cellular and Molecular Effects of Mineral and Synthetic Dusts and Fibres. NATO Advanced Workshop. Invited paper: Phosphorvlated Canadian chrvsotile. With RP Nolan and G Herson. Chaired session: Physico-chemical properties of minerals in relation to their biologic effects. Paris, 11-13 October, 1993.
Workshop on the Health Risks Associated with Chrysotile Asbestos. Inti. Commission on Occupational Health - Inti. Program on Chemical Safety (WHO) Presented paper: Chrvsotile: The Mineral and Its Properties. Jersey, Channel Islands. 14-17, November, 1993.
Hoffmann-LaRoche Pharmaceuticals. Protocol for Analysis of Injectable Medicines for Asbestos. Workshop. Basel, Switzerland. March 2-4, 1994.
Verband der Chemischen Industries. Protocol for the Analysis of Chemicals for Asbestos. Workshop. Frankfurt, Germany. May 30-31, 1994.
Eurometaux (European Association of Metals) and the European Commission, Directorate General V. Scientific Organizing Committee InH. Seminar on Assessment of Respiratory Carcinogenic Risk from Occupational Exposure to Inorganic Substances. Meeting of the Scientific Committee. Brussels, 17-18 October, 1994.
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IN THE CIRCUIT COURT OF COOK COUNTY, ILLINOIS COUNTY DEPARTMENT, LAW DIVISION
#9021
BOARD OF EDUCATION OF THE CITY OF CHICAGO,
Plaintiff, v.
A, C AND S, INC., et al., Defendants.
)
) Nos. 92 L 9934 ) 92 L 9933
) 92 L 9932 ) 92 L 9927
)
) Honorable Michael Gallagher
DEFENDANT A2 EXPERT
Defendant Asbestospray Corporation ("Asbestospray") hereby
gives notice that, pursuant to Rule 220 of the Illinois Supreme Court Rules and this Court's Scheduling Order dated November 1,
1994, Asbestospray has served upon Daniel P. Albers, Esq., Liaison
Counsel for plaintiffs, and all defense counsel of record,
Asbestospray's Expert Witness Disclosures for the expert witnesses
it intends to call to testify at trial.
All of the witnesses
disclosed will serve as primary expert witnesses, although Dr.
Cooper will also serve as a backup medical, state-of-the-art
witness in the event Dr. Murray is unavailable to testify at trial.
The curriculum vitae for each witness is attached hereto.
In addition to the witnesses listed below, Asbestospray hereby incorporates by reference the Expert Witness Disclosures served by
or on behalf of each and every other defendant in these
consolidated cases and reserves the right to call any witness listed by the plaintiffs or any other defendant (including, but not
JIJ/KMOl.l
ArthurM, linger. Ph.D.
Environmental Sciences Laboratory Ingersoll Hall Brooklyn College of the City University ofNew York Brooklyn, NY 11210
A. QUALIFICATIONS
Dr. Arthur M. Langer is director of the Environmental Sciences Laboratory of the Institute of Applied Sciences, Brooklyn College of the City University ofNew York, Brooklyn College, City University ofNew York, Brooklyn, New York. He received a Bachelor of Arts degree in geology from Hunter College, City University ofNew York in 1956, M.A. in petrology from Columbia University in 1962, and a Ph.D. in mineralogy from Columbia University in 1965. From 1965 to 198S, Dr. Langer held various positions at the Mount Sinai School of Medicine in the Department ofMedicine, Department of Community Medicine, Environmental Sciences Laboratory, and Center for Polypeptide and Membrane Research.
Dr. Langer has published extensively in the area of asbestos, asbestos fiber type, asbestos mineralogy, asbestos-related disease, and asbestos disease mechanism. He has peer reviewed numerous manuscripts, served on numerous editorial boards, and authored and reviewed numerous documents for NIOSH, ALOSH, MSHA, and EPA. He has served on numerous international comminees and has spoken at numerous meetings.
B. TESTIMONY
Dr. Langer will testify that asbestos is a term for a number of minerals from around the world, some ofwhich had been used commercially and some of which have not. He wQI testify that there has been a great deal of confusion in the medical and scientific literature concerning the term asbestos and that it is often improperly defined. He will testify concerning historical and current scientific knowledge concerning how asbestos fiber enters the human body, the disease mechanism within the human body, and disease processes. He will testify that different asbestos fiber types are not equally biologically active. He will testify concerning medical, scientific, and
epidemiological literature with respect to persons occupationally exposed to asbestos. He will testify concerning low-level exposure to asbestos. He will testify concerning asbestos fiber, how h is used in asbestos products, the concept of "friability", the aerodynamics of ubcstos fibers, the dispersion of asbestos fibers in the air both inside and outside ofbiddings, and the nature of "fiber release" from asbestos fireproofing.
Dr. Langer w01 testify that air testing and monitoring is an appropriate method of determining whether an asbestos hazard exists within a building. He will testify concerning his own inspection of asbestos-containing materials in buildings, air monitoring in buildings, and asbestos levels both inside and outside of buildings.
Dr. Langer will testify concerning microscopic techniques, proper analysis of air and bulk asbestos samples, and the appropriate measurements of asbestos fiber levels.
Dr. Langer will testify concerning threshold limit values, both currently and historically, the EPA position on asbestos in buildings and threshold limit values, and the relation oflow level asbestos exposure to various diseases including asbestosis, lung cancer, peritoneal mesothelioma, and pleural mesothelioma.
Dr. Langer will testify concerning proper responses to asbestos in buildings including, but not limited to, the necessity of removal, the advisability of removal, and operations and maintenance of asbestos in buildings.
Dr. Langer will testify concerning the risks to building occupants from asbestos, the of epidemiological studies with respect to such risks, and the nature and extent of such risks based upon fiber levels.
Dr. Langer will testify concerning the state of the art with respect to low-level exposure to asbestos and risks of different asbestos fiber types.
Dr. Langer will rely on the following materials in this case:
1. The reports of William Longo, Jim Millette, and Richard Hatfield;
2. Deposition transcripts of William Longo, Jim Millette, and Richard Hatfield;
3. Plaintiffs' product identification reports for South Shore High School, Leif Ericson Elementary, District 2 Offices, Glenbrook South High School, and Hawthorne Elementary;
4. Plaintiffs' air sampling results for South Shore High School, Leif Ericson Elementary, District 2 Offices, Glenbrook South High School, and Hawthorne Elementary;
5. Plaintiffs' dust sampling results for South Shore High School, Leif Ericson Elementary, District 2 Offices, Glenbrook South High School, and Hawthorne Elementary;
6. Photographs in building survey and inspection reports taken by Asbestospray's consultants in South Shore High School, Leif Ericson Elementary, and District 2 Offices;
7. Air sampling data obtained by Asbestospray's consultants with respect to South Shore High School, Leif Ericson Elementary, and District 2 Offices;
8. Documents pertaining to Asbestospray and/or the Sprayed Mineral Fibers Manufacturing Association (SMFMA);
9. Air sampling data obtained by Asbestospray's consultants in other property damage litigation;
10. "Proceedings of the Symposium on Health Aspects ofExposure to Asbestos in Buildings" (Harvard University, December 14-16, 1988);
11. Spengler, Ozkaynak, McCarthy, and Lee, "Summary of Symposium on Health Aspects of Asbestos in Buildings" (Harvard University, August 1989);
12. Health Effects Institute - Asbestos Research, "Asbestos in Public and Cwumcncwl Buildings: A Literature Review and Synthesis of Current Knowledge" (1991); and
13. Wilson, Langer, Nolan, Gee, and Ross, "Asbestos in New York City Public School Buildings - Public Policy: Is There a Scientific Basis?" Reg. Toxicol. 1 harmocol. 20:161-169 (1994).
:\1jvAl 5 230MiscIoj..p
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ROBERT T MTJtH JOHN T ANGEU MICHAEL S. UEIDLt!*' STEVEN I. M'JTH ROBERT L. BROWNJON
D. SCOOT BALLOt
law orncES
STICH. ANGELL, KRE1DLER & MUTH. P.A.
THE CROSSINGS, SLTJZ 120 280 SEC0NI5 AVENUE SOUTH
MINNEAPOLIS. MINNESOTA 55401-2152 TELEPHONE <fl 12) SSS-6S8I FACSIMILE l'6l2' S3-:W
aim: admitted ti< PRACTICE IN WISCONSIN
May 26, 1995
Daniel P. Albers, Esq. COFIELD, UNGARETTI & HARRIS 3 500 Three First National Plaza Chicago, IL 60602
VIA FAX
Re: Chicago Board ofEducation, et ai v. Asbestospray, et al.
Our File No.: 12801/15230
")ear Mr. Albers
Enclosed are copies of the reports prepared by Dr. Arthur Langer.
If you have any questions, please do not hesitate to contact roe.
Very truly yours,
xtIBLKi H. r.\r.Gi THOM.UJ. LINNIHAV iCOIT 1> DRAWS' IAMES D. k.VL'DUN* Sl'SAN M. HANSEN JOSEPH M JTOCC0 GXIGORY L. WXIChT MICHAEL W. VAUGHAN. JR Ltd I. BRI5BOIS tXSVULA. B1HRXSDT JKFntEY A. MaGNL'S
Lynnae I. Waskoskv Legal Assistant
Enclosures
cc Jonathan Miller, Esq. (w/ enclosures) JeffJohnston, Esq. (w/ enclosures)
1 1/l:K * N-' '
B't O-Hi- 15 : :<:+4PM * V I ,VU 1 JLi LMfl
" Vie 951 4199 P.02
Table 1: Deaeration of tha aftee and typos of oeoond sir monitoring in South Shore High School.
Auditorium Front CLOSED BLANK
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TYPE OF SAMPLE
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Table 2: Rasufti of the anatyefe of the membrane fitters by transmission efaotion microscopy for asbestos in District 2 office.
TOTAL VOLUME OF MR
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Table 1: Detonation of the eitee and type of air monitoring In the Ldf
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Table 1: Description of the aim and typee of air monitoring In the Lett Erickson Elementary School
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Table 1: Peecrlpion of the sites end types of air monitoring in South Shore.
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SCIENCE & SOCIETY
The asbestos panic attack
closed its public schools for two week1 and spent S83 million on asbestos rein spection and removal because earlier as bestos inspections were shoddy. Tota spending nationwide has been S10 bil lion, the National School Boards Associ ation told Congress earlier this month Experts say it could reach S30 billion.
Much of the money, though, is proba bly being spent in vain. There is no telling
how much of the cleanup might have been necessary. No one has ever deter mined how much asbestos in the air is unsafe, and there is
How thefeds got schools to spend billions on a problem that really didn't amount to much
hen school custodians in Oak The reason for all the elaborate pre
Wland County, Mich., want to fix cautions: asbestos. A tough, heat-resis a leaky water valve, they don tant mineral fiber associated with lung head-to-toe spacesuits. "They madkieseyaosue, asbestos has long had the repu
sweat like a pig," says facilities consul tation of a workplace hit man. The repu
tant Bill Lee. Then, they test the air near tation has cost plenty. Across the nation,
the leaky valve. Next, they wet the insu school districts that can't buy new library
lation surrounding the pipe, chip it off, books are spending hundreds of thou
seal it in a bag, seal that in another bag, sands of dollars to deal with asbestos
then check the air again. Only after that because they mistakenly believe their
can they strip off their spacesuits and children are in great danger. In the fall
actually replace the offending valve.
of 1993, for instance, New York City
U.S.NEWS
tists and physicians that asbestos in public buildings is not much of a threat to health. "A phantom risk" is what Richard Wilson, a physics professor at Harvard University, calls it. A study soon to be released by the federal Office of Technology Assess ment will say much the same thing.
Paper-tWn rddaucu. How America's money-strapped schools could have so overreacted to the perceived threat from asbestos is an illuminating tale. One part, at least, is understandable: Parents and teachers pressed for drastic reme dies for something they were told was a
USJslEWS 4 WORLD REPORT. FEBRUUW2Q 1995
SCIENCE & SOCIETY
real danger. Less understand
able is the role of government
agencies, especially the federal
Environmental Protection
Agency, which created a public
panic on the basis of paper-
thin scientific information.
It's also a story about how
the instinct to act overwhelms
common sense. "When every
body's shouting `Fire!' in a the
ater. the man who quietly
stands in the comer and says.
`There's no fire,' is rarely heed
ed." observes Arthur Langer,
director of the Environmental
Sciences Laboratory at Brook
lyn College in New York, who
has served on numerous asbes
tos-study panels. Langer recalls
sitting with other experts in the
mid-1980s to draft EPA asbes
tos rules. The documents used
terms like "hazard" and "emer
gency. ""I said. `Let's change
the language of this: we are us Voice of reason. Prof. Arthur Longer kept trying to warn policy makers they were overreacting.
ing emotive words,' " Langer
said. "They just blew me out." John seen a high coincidence between smok issued its Blue Book, which said much
Welch, president of an association of for ing and asbestos-linked lung cancer. the same thing: Removal was "always
mer manufacturers of asbestos products, More important, the risk of all these dis appropriate, never inappropriate."
told an EPA panel in 1984 that since eases seemed to be tied to the exposure Joseph Breen, an EPA official who
asbestos was present in the air every level. How much was too much?
worked on the Orange Book, recalls,
where. the EPA should set standards for To help local officials, EPA published "There was a cry to get something out,
levels deemed permissible. "They didn't a "guidance" book in 1979. It had a and the best available information was
want to hear it." he says. " `No safe level' bright-orange cover and was known what had been done to date." Unfortu
was the foundation of their arguments." thereafter as the Orange Book. But in nately, at the time, little work had been
First warning. The saga began in the stead of declaring what levels of airborne done on the health effects of low levels
1950s when a brilliant, energetic physi asbestos were safe or unsafe for children, of asbestos in schoolroom air.
cian named Irving Selikoff documented it issued directions on how to search for While the Orange and Blue books
that workers in mines, shipbuilding and asbestos-containing materials. The only did not command that asbestos be tom
other asbestos-using industries had con "permanent" solution, it said, was to out. their dire admonitions--plus the
tracted lung cancer, asbestosis --a dis take it out. Four years later, the EPA availability of federal funds for asbestos
ease marked by stiffening of the lung
removal only --pushed schools into
tissues-and a rare lung cancer called
many needless removals. An asbestos-
mesothelioma. By the 1970s, Selikoffs research was the foundation for lawsuits against asbestos manufacturers. Many were driven out of business. (Others
RISKY BUSIMKSS
Many things expose people to a greater threat than exposure to asbestos
remediation industry sprang up over night; it would gross $4 billion to $5 bil lion annually. Schools would pay up to $1 billion a year; other building owners
continue to pay. Last October 3L for in stance. 10 large asbestos manufacturers
UMWw praMMCr et pnwalw* 4mUi IfatlMNI
paid the rest. On second thonght. But by 1985, some
settled with 15,000 school districts for $200 million in damages.)
Selikoffs findings scared school lead
Being a pedestrian hit by car Tobacco smoke
290 200
notable environmental health scientists were backing off. New data were show ing that levels of airborne asbestos in
ers because many walls, floor and ceil ing tiles, roofs and insulation materials contained asbestos fibers. If the sur
Diagnostic X-ray Bicycling
7S buildings with even flaking insulation
78
could be as low, or nearly as low, as the air outdoors. Asbestos was everywhere:
faces were abraded, asbestos could float into the air and be inhaled. Parents feared their kids could get asbestosis.
Miami/New Orleans drinking water 7
Lightning
3
It came from auto-brake pads and building and roofing materials. People breathe in a million fibers a year, and
That fear was misguided: Early stud
Humcanes
3 nearly everyone has asbestos in his
ies concluded that asbestosis came only after long-term inhalation of large vol
Asbestos in school buildings
1
lungs, scientists would say. The new thinking was that most as
umes of asbestos dust, as in dirty fac tories. A more likely risk to children was
USNM - tac a: bww and Emmrananw PoMy
Cams' of
Unafsmtj
bestos in schools should be left alone; in most cases, painting, spray-coating or
lung cancer. But again, early studies had
covering it up was the best and cheapest
62 u.s.News & world Report. February 2a 1995
solution. In a nod to the new thinking, the EPA's 1985 Purple Book placed "managing asbes tos'' ahead of "removal'' on some lists of options. But the shift was subtle, and it was hardly noticed. Opposing inter ests now had a stake in asbestos removal: They included labor unions who wanted it out of workplaces, parent-teacher groups fearful for children and a mushrooming asbestos-re
moval industry. Powerful convert. Perhaps
most important, Selikoff him self had drifted from the posi tion of being a neutral research scientist to that of an antiasbes tos activist. He told a congres
DON'T TAM OUR WORD ON IT The EPA gave confusing advice over the years about the best way to handle asbestos:
1979 Orange Book
"No safe level of expost Bated.... Removal completely eliminates the source of exposure to asbestos and is, there
fore, a permanent solution.**
1990 Green Book
"Based on available data, the average
school and office building! meant low overall risk. It point ed out that the dust created b> improper asbestos remova: could actually increase the dan ger. Still, some schools were not getting the message. In Ma> 1990, for instance, the Downers Grove school district near Chi cago prepared to spend the last $850,000 of a $5 million asbes tos-management project. But this time, it said, the work would be much harder because the as bestos was on pipes sealed inside the walls. That got the attention of then EPA Administrator Wil liam Reilly, who ordered the EPA to study how well it had advised the public on asbestos.
sional panel in 1984 that the risk
airborne levels in buildings seem to be very
The result, published in 1992.
posed by asbestos in schools was intolerable. The subcom mittee chairman, then Rep. Jim
lour.... Removal is often NOT a building owner's best course of action to reduce
was a remarkable -- and embar rassing-document. It found that the EPA had made matters
Florio of New Jersey, shepherd
asbestos exposure."
worse by publishing confusing
ed passage of a law called the
and shifting guidances.
Asbestos Hazard Emergency
In the absence of certainty at
Response Act in 1986. Florio
the federal level, six other
says he wanted to end the confusing maintenance officers, and they had all states have debated proposals like
stream of "guidances," put an end to submitted their [EPA required] plans Michigan's. None has been enacted be
the sloppy and corrupt asbestos-remov and were hellbent to rip it out."
cause of trial lawyers' protests and be
al firms and make the EPA write clear Some now regret it. Rick Simonson, cause of state attorneys general who
rules for schools to follow. The EPA the assistant superintendent for Oak argue that such a law could affect their
did some of that: It put asbestos in land County schools north of Detroit, own litigation against manufacturers.
spectors, planners and even haulers un estimates his district spent nearly $100 Eleven states have set permissible as
der strict regulations. It ordered million tearing out 70 percent to 80 per bestos-in-air standards for public build
schools to do inspections and create as cent of the schools' asbestos. He now ings; up to nine states include schools.
bestos-management plans. All that, of says they should have done just the re The level is about 10 times more strin
course, added millions of dollars to verse -- taken out 20 to 30 percent and gent than the one just set by the feder
schools' costs. The EPA. however, later sealed up the rest. "We spent a ton of al Occupational Safety and Health Ad
argued that Florio had fueled the as money removing that stuff when we ministration for industry.
bestos alarm. Florio counters that the didn't need to," he says.
But the EPA still refuses to say what
EPA dragged its feet and never set In its 1990 Green Book, the EPA ac levels are safe for schoolchildren to
safety standards.
knowledged that low levels of asbestos in breathe. The reason? Robert Jordan,
By 1990, however, a mass of new data
an environmental protection specialist
showed the true levels of asbestos in
at the EPA. says his agency fears that
schoolroom air around the nation were
"people could be lulled into compla
extremely low. Articles in the New Eng
cency" by taking infrequent air sam
land Journal of Medicine and Science
ples. If asbestos levels suddenly turned
magazine argued that the asbestos
worse, school officials wouldn't know
threat had been exaggerated. These
it, he argues.
caught the attention of John "Joe"
So asbestos madness continues. For
Schwarz, a physician and Michigan
fear of asbestos, in 1993 New York City
state senator, who feared the reasons to
ripped out tons of plaster from its
fear asbestos had been overstated from
schools, only to find that just 25 percent
the beginning. It took three years for
of it contained asbestos. How much of
him to win passage of a law requiring
that found its way into the air no one
school districts to keep asbestos-laden
knows. With Congress and the EPA
materials in place unless unique condi
now considering regulations for lead
tions made removal wiser. Opposition
and radon exposure, the 1992 EPA re
came from the state's trial lawyers, who
port on how it messed up its message
represented plaintiffs in asbestos cases,
on asbestos should be required reading.
parental groups, labor unions and even,
But that report is virtually impossible to
surprisingly, schools themselves. "What
find inside the EPA. Says one in-house
was particularly pernicious, a bu
expert, "I think it's been buried."
reaucracy had grown up," Schwarz now
says. "The schools all had asbestos- Florio. He wrote tough asbestos legislation.
By Peter Cary
U.S.NEWS & world Report. February 20.1996
63