Document RxG6keoVE5maLvaeNajQEkGE

CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: ASBESTOS CAS#: 1332-21-4 PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Hydrated mineral silicates (Si02) Common synonyms: Aetinolite, Amosite, Chrysotile, Crocidolite, Tremolite Anthophyllite Chemical/Physical Properties Molecular Wt. Varies Boiling Pt., Decomposes & Vapor Pressure Omm Specific gravity NA Solubility Insol. in H. Melting Pt. 1112^ / fcfcoS C, Physical state in air: In stack: gas In ambient air: gas X particulate X particula Ambient/background concentrations: Asbestos is a naturally occurring compound. Asbestos is found in beverages and talcs. Drinking waters may contain 1-65 mill Mean airborne asbestos cone, in U.S. cities ng/m? ;\around milling, 2.6-8. SxK^fym3. 2x-S.q*io~%jcc ' - Sampling/analytical methods: see attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY Exposure Limits: OSHA PEL: .2f/cc MSHA PEL: .Stfee ACGIH TLV: .2f/cc NIOSH REL: .lf/cc C..OGOlf|a 3^3 C35703 0508 Acute Toxicity Data; LDyy, for oral, dermal, inhalation. Inh-hmn TCL0=1.2f/cc Ipr-rat TDL0=.280mg/kg Include species. IDLH: ND (Ref: NIOSH ) ERPG: ND Chronic Toxicity Data:. Carcinogenic status: Z Yes No X Promoter (epigenetic) and/or Initiator (genotoxic): IARC Classification: A Other designations: NA NOAEL: No set NOABL (Ref: RTECS ) (no observable > adverse effect level) CAL. Prop 65 NSRL: lOOf/day (inh) (no significant risk level) CSF: ND (IRIS) Specific cancer end point: Lung Reproductive hazard: (inc. mutagen, terat) X Yes No Sister chromatid changes noted in lung tissue. Asbestos has cellular mutagenic activity. Also excess ovarian cancer rates. Sensitizer: No Non-cancer Rfd: ND End Point: ND Human toxicity data: Evidence of human carcinogenicity. Biological Monitoring: Available: X Yes Urine asbestos No ACGIH BEI: ND C35703 0509 III. ENVIRONMENTAL INFORMATION; Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA Effluent/Ambient Standards/established limits in various media: ^RQ=1 lb. 1 .. *' \ No dear excess cancer risk associated with asbestos in drinking water. RCRA - Cleanup levels \ SARA - Hazardous chemical T8CA - Chemical inventory DOT - Guide 58 Remediation Issues: RCRA cleanup levels; Air: 2E-02ug/m3; Water: ND; soil: ND. Reporting to Regulatory Agencies Worldwide: Regulated under Canadian and European TSCA equivalent; also worldwide air stds. Fate in the Environment: Asbestos is considered to be nonbiodegradable. Asbestos does not have an adsorptive affinity in soils. Relevant bioaccumulation data: Most asbestos forms do not bioaccumulate and are eliminated from the system in 10 days (max.); however, a small fraction of asbestos is not readily cleared and may be retained for long periods. Known toxicity in plants: No reported data relevant to environmenta1 concentrations. Known toxicity in fish: No reported data relevant to environmental concentrations. Known toxicity in other aquatic species: bioaccumulation in aquatic species. No evidence of C35703 0510 / t I t IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 X CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR High Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring _ available: Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) \ J. MACKAY (Environmental Issues) ^r r;-L.- JTITLEY (Dispersion Modelling) _____________________ (Environmental data/sampling/analysis) ______________________________ (Ind. Hygiene sampling information) H:\PLANTVMEMPRO.WAR 035703 057 7 VII. GLOSSARY OF ACRONYMS; ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC -- LDLO - LDjo LOAEL MEI NIOSH REL - - - - NOAEL NSRL PEL - - RfD - STEL - TDLO/TCLO -- flee - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: SME-Olmg/m3 = . 54mg/m3. For risk level,.E-6 .= 10"4 or 1 excess cancer in i million.) Emergency Response Planning Guide International Agency for Research on Cancer Ijnmediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose n. Ide<xo -paru CuJoLc VIII. RISK SUMMARY PomnmTH fcv; Employee: (Q8HA) No established data available for qualitative risk assessment .2f/cc Est. action level (for risk of E-6) 2E-03f/cc for community or ISOf/day (based on safety factor applied to ADI)* * Air levels should not exceed 2E-02ug/m3 (RCRA cleanup level) H:\PLANT\CHEMPRO.WAR C35703 0512 CADMIUM RISK SUMMARY; Health Effects Profile: Basis for Occupational Limits: The limits are based on the prevention of preclinical kidney dysfunction and increased risk of lung cancer. Carcinogenicity: Based on the EPA weight-of-evidence classification, cadmium is placed in the group Bl, probable human carcinogen. Although the evidence is limited cadmium is considered to present a significant risk of .lung cancer..- There is no evidence the cadmium, is carcinogenic via ingestion, which is the major route of human exposure, and that the upper limit of potency via ingestion would be at least 100 times lower than via inhalation. Mutagenicity: Results are mixed. Developmental Toxicitv: Cadmium has been demonstrated to be teratogenic and to decrease fertility in animal models following intravenous, intraperitoneal and subcutaneous injection. No studies on effects of inhaled cadmium. Reproductive Toxicitv: See above Systemic Toxicitv: Chronic exposure to cadmium via ingestion and inhalation is associated with renal toxicity in both humans and animal models once a critical body burden is reached. Acute occupational exposure to freshly generated fumes from heated cadmium results in a syndrome of tracheobronchitis, pneumonitis, and pulmonary edema which may not appear until several hours after exposure. Irritation: Exposure to high concentrations of freshly generated fumes from heated cadmium is reported to be severely irritating. Risk Profile: The ambient air level goal (AALG) for cadmium compounds is based on carcinogenicity. AALG: * 10E-6 point estimate-1. lE-4/xg/mJ 10E-5 point estimate-1. lE-3jig/mJ and C35703 O573 f CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: CADMIUM CAS#: 7440-43-9 it PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Cd Common synonyms: /lladmium/ CI77180, collodial cadmium Chemical/Physical Properties Molecular Wt. Boiling Pt. Solubility Cd dust f<1 fima Cd dust f!d fume 112.4 128.4 Specific Gravity 8.65 8.15 1409F Decomposes Insol. HjO Insol. H20 Vapor Pressure Omm Omm Melting Pt. 610F / 33|.^0G 25"#p/t42l3eG Physical state in air: In stack: gas In ambient air: gas X particulate X particulate Ambient/background concentrations: In body = 1.6 ug/1 geometric mean. Cd concentrations in rock/soil = .001 mg/kg (igneous rock) to 100 mg/kg (phosphatic rock). GW cone, of 312 mg/1 reported from electroplating operations. Atmospheric levels of .1-3 mg/m3 reported around working environments. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: fume = .1 mg/m3; dust = .2mg/m3 Ceiling limit OSHA = .3 mg/m3 (fume); 0.6 mg/m3 (dust) MSHA PEL: .2 mg/m3 ACGIH TLV: .05 mg/m3, dusts NIOSH REL: treat Cd as human carcinogen C35703 0514 f Acute Toxicity Data: LDj<rt for oral, dermal, inhalation. Include species. TCLO (inh) (man) = 88 ug/m3/8.6 yr LCLO (inh) (man) = 39 mg/m3 LDLO (unreported) (man) = 15 mg/kg LDLO (mouse) (inh) =170 mg/m3 LDLO (rabbit) (oral) = 70 mg/kg IDLH: dust = 50 mg/m3/ fume = 9mg/m3 (Ref: NIOSH) ERPG: ND Chronic Toxicity Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B1 Other designations: NA Risk assessment doses (e.g., EPA for 1X10"6) Limit Aaencv EPA Cal. Risk factor Unit Risk 6.1 E+O (mg/kg/day1) 6E-2 ug/m3 multi-stage extrapolation 0.05 ug/day NOAEL: .005mg/L (water) estimated (Ref: RTC8) (no observable adverse effect level) CAL. Prop 65 NSRL: 0.05 ug/day (inh) (no significant risk level) CSF: 6.10 E+O (mg/kg-day1) (inh-hmn) Specific cancer end point: respiratory system Reproductive hazard: (inc. mutagen, terat) X Yes No Limited human data and positive animal data. Sensitizer: Limited evidence is supportive for sensitization. Non-cancer Rfd: Old EPA 0.005 mg/kg/day (water) and 0.001 mg/kg/day (food) End Point: Kidney Human toxicity data: Known human carcinogen. Biological Monitoring: Available: X Yes Urine and blood No C35703 0515 ACGIH BEI: Yes 10 ug/g creatinine Cd in urine 10 ug/1 cadmium in blood III. ENVIRONMENTAL INFORMATION; Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA; MCL: .005mg/l. Effluent/Ambient Standards/established limits in various media: RCRA - cleanup levels established CWA - Listed DOT - Guide 58 SARA - Hazardous chemical TSGA - Chemical inventory WORLD WIDE LIMITS - ECOIN List Remediation Issues: RCRA cleanup levels: air: 6E-04 ug/m3; soil: 4E+01 mg/kg. Reporting to Regulatory Agencies Worldwide: Europe, Canada Fate in the Environment: Usually found in soil, however, it will leach slowly to groundwater. Primary fixation is in soil. Relevant bioaccumulation data: Cd has a long half-life in the human body. Cd will bioaccumulate slightly in animal and aquatic species. Known toxicity in plants: ND on-line. Known toxicity in fish: ND on-line. Known toxicity in other aquatic species: ND on-line. C35703 0516 UL. COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA X CALIFORNIA PROP 65 X Cd and Cd compounds potentially affect downstream sources. CONEG X HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING X LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate to high Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) _____________________ (Environmental data/sampling/analysis) ______________________________. (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0517 VII. GLOSSARY OF ACRONYMS: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E+01 - - -- ERPG IARC IDLH LC - - - LDLO LDjq LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - - - - - - - - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Careinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = .54mg/m3. For risk level, E-6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community; Cancer effects (Adult) 5.82E-08mg/m3 (Child) 8.l3E-08mg/m3 Non-cancer effects (Adult) 7.29E-05mg/m3 (Child) 2.08E-05mg/m3 (CSF=6.1E+0 mg/kg/day1) (Rfd=.001 mg/kg/day) Employee; .2mg/m3 (dust) .lmg/m3 (fume) .2mg/m3ceiling (fume) H:\PLANT\CHEMPRO.WAR C35703 0518 CADMIUM RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The limits are based on the prevention of preclinical kidney dysfunction and increased risk of lung cancer. Carcinogenicity; Based on the EPA weight-of-evidence classification, cadmium is placed in the group Bl, probable human carcinogen. Although the evidence is limited cadmium is considered to present a significant risk of lung cancer.. There.is no evidence the cadmium is carcinogenic via ingestion, which is the major route of human exposure, and that the upper limit of potency via ingestion would be at least 100 times lower than via inhalation. Mutagenicity: Results are mixed. Developmental Toxicity: Cadmium has been demonstrated to be teratogenic and to decrease fertility in animal models following intravenous, intraperitoneal and subcutaneous injection. No studies on effects of inhaled cadmium. Reproductive Toxicity: See above Systemic Toxicity: Chronic exposure to cadmium via ingestion and inhalation is associated with renal toxicity in both humans and animal models once a critical body burden is reached. Acute occupational exposure to freshly generated fumes from heated cadmium results in a syndrome of tracheobronchitis, pneumonitis, and pulmonary edema which may not appear until several hours after exposure. Irritation: Exposure to high concentrations of freshly generated fumes from heated cadmium is reported to be severely irritating. Risk Profile: The ambient air level goal (AALG) for cadmium compounds is based on carcinogenicity. AALG: 10E-6 point estimate-1. lE-4/ig/m3 10E-5 point estimate-1. lE-3/ig/mJ and C35703 05 79 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: CADMIUM CAS#: 7440-43-9 PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Cd Common synonyms: /kadmium, CI77180, collodial cadmium Chemical/Physical Properties Molecular Wt. Boiling Pt. Solubility Cd dust Cd fume Cd dust Cd fume 112.4 128.4 Specific Gravity 8.65 8.15 1409*F Decomposes Insol. HjO Insol. H2o Vapor Pressure Omm 0mm Melting Pt. 610F I 33i.^C 25,9'F/I4213`<. Physical state in air: In stack: gas In ambient air: gas X particulate X particulate Ambient/background concentrations: In body = 1.6 ug/1 geometric mean, cd concentrations in rock/soil = .001 mg/kg (igneous rock) to 100 mg/kg (phosphatic rock). GW cone, of 312 mg/1 reported from electroplating operations. Atmospheric levels of .1-3 mg/m3 reported around working environments. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: fume = .1 mg/m3; dust = .2mg/m3 Ceiling limit OSHA = .3 mg/m3 (fume); 0.6 mg/m3 (dust) MSHA PEL: .2 mg/m3 ACGIH TLV: .05 mg/m3, dusts NIOSH REL: treat Cd as human carcinogen C35703 0520 Acute Toxicity Data; LDjp, for oral, dermal, inhalation. Include species. TCLO (inh) (man) = 88 ug/mVs.O yr LCLO (inh) (man) =39 mg/m3 LDLO (unreported) (man) = 15 mg/kg LDLO (mouse) (inh) =170 mg/m3 LDLO (rabbit) (oral) = 70 mg/kg IDLH: dust = 50 mg/m3, fume = 9mg/m3 (Ref: NIOSH) ERPG: ND Chronic Toxicity Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B1 Other designations: NA Risk assessment doses (e.g., EPA for 1X10"6) Limit Aaencv EPA Cal. Risk factor Unit Risk 6.1 E+O (mg/kg/day*1) 6E-2 ug/m3 multi-stage extrapolation 0.05 ug/day NOAEL: .005mg/L (water) estimated (Ref: RTC8) (no observable adverse effect level) CAL. Prop 65 NSRL: 0.05 ug/day (inh) (no significant risk level) CSF: 6.10 E+O (mg/kg-day*1) (inh-hmn) Specific cancer end point: respiratory system Reproductive hazard: (inc. mutagen, terat) X Yes No Limited human data and positive animal data. Sensitizer: Limited evidence is supportive for sensitization. Non-cancer Rfd: Old EPA # 0.005 mg/kg/day (water) and 0.001 mg/kg/day (food) End Point: Kidney Human toxicity data: Known human carcinogen. Biological Monitoring: Available: X Yes Urine and blood No ^057oj 0521 ACGIH BEI: Yes 10 ug/g creatinine Cd in urine 10 ug/1 cadmium in blood III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA; MCL: .005mg/l. Effluent/Ambient Standards/established limits in various media: RCRA - cleanup levels established CVA - Listed DOT - Guide 58 SARA - Hazardous chemical TSCA - chemical inventory WORLD WIDE LIMITS - ECOIN List Remediation Issues: RCRA cleanup levels: air: 6E-04 ug/m3; soil: 4E+01 mg/kg. Reporting to Regulatory Agencies Worldwide: Europe, Canada Fate in the Environment: Usually found in soil, however, it will leach slowly to groundwater. Primary fixation is in soil. Relevant bioaccumulation data: Cd has a long half-life in the human body. Cd will bioaccumulate slightly in animal and aquatic species. Known toxicity in plants: ND on-line. Known toxicity in fish: ND on-line. Known toxicity in other aquatic species: ND on-line. C35703 0522 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 X Cd and Cd compounds potentially affect downstream sources. CONEG X HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING X LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate to high Significant public activity and/or key topics V*. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) _____________________ (Environmental data/sampling/analysis) ______________________________ (Ind. Hygiene sampling information) H:VPLANT\CHEMPRO.WAR C35703 0523 VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E01 ERPG IARC IDLH LC LDLO LDjo " LOAEL MEI NIOSH REL - NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10-4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Cancer effects (Adult) 5.82E-08mg/m3 (Child) 8.13E-08mg/m3 Non-cancer effects (Adult) 7.29E-05mg/m3 (Child) 2.08E-05mg/m3 (CSF=6.1E+0 "9/kg/day1) (Rfd=.001 "9/kg/day) Employee: 2mg/m3 (dust) .lmg/m3 (fume) .2mg/m3ceiling (fume) H:\PlANTVCHEMPRO.WAR C35703 0524 COPPER RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The limits for copper are based on the prevention of irritant effects. Carcinogenicity: Insufficient data Mutagenicity; Insufficient data Developmental Toxicity: Insufficient data Reproductive Toxicity: The spermicidal properties of copper are well established and have been applied to the prevention of pregnancy through the use of copper loops or wires placed in the uterus. Systemic Toxicity: In humans,ingestion of high concentrations of copper salts produces gastric irritation resulting in salivation, nausea, vomiting, gastric pain and hemorrhaging, and diarrhea. Irritation: Copper fumes are reported to cause upper respiratory tract irritation and a metallic or sweet taste. Risk Profile: The ambient air level goal (AALG) for copper is based on irritation. AALG: irritation (TLV-based), 8 hour TWAs dust and mists 0.02 mg(Cu)/m3 fume 0.02 mg(Cu)/m3 C35703 0525 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: COPPER CAS#: 7440-50-8 it PHYSICAL/CHEMICAL INFORMATION; Chemical formula: Cu, CufU^. , Cup/ ) common synonyms: Synonyms of other compounds vary depending upon the specific compound Cofpto tuWata , Ccfp*ACiu<k. Chemical/Physical Properties Molecular Wt. Boiling Pt. Solubility Flash Pt. 63.5 4703*F Insol. in HjO NA 7cPC\..\C Ionization Pressure Vapor Pressure Melting Pt. NA omm 1981F PEL MA LEL NA Specific Gravity 8.94 ItfcWC- Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: No data on-line. Ambient/background concentrations: Naturally occuring in the earth's crust at 70 ppm. Drinking water US cities (mean value) =60 ug/1; Air, U.s. cities range from 10-570 ng/m3. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: .05 mg/m3 MSHA PEL: .05 mg/m3 ACGIH TLV: .05 mg/m3 NIOSH REL: .05 mg/m3 Acute Toxicity Data: LDjo-, for oral, dermal, inhalation. Include species. LDLO oral - domestic animal species = 18,000-18,200 mg/kg C35703 0526 IDLH: 20 mg/m3 (Ref:NIOSH) ERPG: N.D. Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic): NA IARC Classification: D Other designations: NA NOAEL: LOAEL: ND (no observable adverse effect level) 5.3 mg (Ref: HEAST ) human - single dose CAL. Prop 65 NSRL: ND (no significant risk level) CSF: ND Specific cancer end point: ND Reproductive hazard: (inc. mutagen, terat) Yes X No Sensitizer: Possibly - some reports of sensitization. Non-cancer Rfd: 1.3 mg/1 (oral - human) End Point: GI - irritation Human toxicity data: human toxicity. Needed for biologic functioning. Low Biological Monitoring: Available: X Yes Blood analysis No ACGIH BEI: ND III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: TSCA - Chemical inventory Remediation Issues: No RCRA cleanup levels established. No CERCLA reportable quantity. Reporting to Regulatory Agencies Worldwide: No data found on line. C35703 0527  Fate in the Environment: Acidic conditions promote solubility of copper in soil. Copper undergoes a biotransformation reaction in water. Copper leaches in acidic environments (<3). Relevant bioaccumulation data: Copper exists as a mineral component in biological systems. It does not readily bioaccumulate to toxic levels. Known toxicity in plants: Mean plant value (natural) = 6.0 mg/kg. . .Known toxicity in fish: No relevant data for environmental concentrations. Known toxicity in other aquatic species: The limit of copper in water to protect aquatic organisms is 5.6 g/1. IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR LOW Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available: Summary attached: C35703 0528 VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) (Environmentaldata/sampling/analysis) (Ind. Hygiene sampling information) VII. GLOSSARY OF ACRONYMS; ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose H:\PLANT\CHEMPRO.WAR C35703 0529 VIII. RISK SUMMARY Community: Mon-cancer effects Est. Action Level 5.0E+1 mg/m3 Employee; (OSHA1 (No inhalation/oral Rfd available; action level based on safety factor applied to LOAEL) .OS mg/m3 H:\PtANT\CHEMPRO.WAR C35703 0530 HYDROGEN CYANIDE RISK SUMMARY: Health Risk Profile: Basis for Occupational Limits: The limits are based on the prevention of acute cyanide poisoning. In addition to acute toxic effects, the OSHA limit is also based on consideration of the irritant properties of cyanide salts. Carcinogenicity: NA Mutagenicity; NA Developmental Toxicitv: NA Reproductive Toxicitv: NA Systemic Toxicitv: At the cellular level, cyanide inhibits cytochrome c oxidase, the terminal enzyme in the electron transport chain, decreasing cellular ability to utilize oxygen and producing histotoxic hypoxia. Irritation: Some reports indicate that low level exposure to HCN vapor caused reddening of the skin or blotchy eruptions and cyanide salt aerosols are known to cause skin and upper respiratory tract irritation. Risk Profile: Given the uncertainty associated with data concerning the correlation of exposure and effect in the case of long term, low level exposures the more conservative NIOSH limit was selected as the basis for the ambient air level goal (AALG). AALG: systemic toxicity-0.09 ppm (0.095mg/mJ) ceiling C35703 0531 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: CYANIDE CAS#: 000057-12-5 I. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: CM, (HCM) Common synonyms: Hydrogen cyanide, potassium cyanide, sodium cyanide, cyanogen, cyanogen bromide, cyanogen chloride, cyanogen iodide, metal cyanides (ferro) Chemical/Physical Properties Molecular Wt. 65.1 Melting Pt. 1047F 5H3ct Physical state ambient air Boiling Pt. 2725P iW c Specific gravity 4.55 Solubility 58% in H.O Vapor Pressure 0 mm in air: * as HCN -gas- both in stack and In stack: X gas X particulate In ambient air: X gas X particulate If particulate, part size: No data Ambient/background concentrations: ND on-line. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 5 mg/m3 - NO STEL MSHA PEL: 5 mg/m3 (skin) ACGIH TLV: 5 mg/m3 (skin) NIOSH REL: ND 035703 0532 Acute Toxicity Data: LDjq., for oral, dermal, inhalation. Include species, cyanogen; LDLO = 15 mg/kg (dog); 13 mg/kg (rabbit); hydrogen cyanide; LDLO = .54 mg/kg (human); LCLO = 200 ppm/15 min. (human) IDLH: 50 mg/m3 (HCN) (Ref: NIOSH, 1990) ERPG: ND Chronic Toxicity Data: . Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic): NA IARC Classification: NA Other designations: NA NOAEL: 10.8 mg/kg/day (Ref: HEAST) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) CSP: NA Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) Yes X No Sensitizer: No Non-cancer Rfd: 2E-2 mg/kg/day (subchronic) End Point: degeneration Whole body, thyroid, nerve, myelin Human toxicity data: Highly acute human toxin. Biological Monitoring: Available: X Yes No ACGIH BEI: ND ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA C35703 0533 Effluent/Ambient Standards/established limits in various media: RCRA - Cleanup levels CWA - Listed DOT - Guide 58 TSCA - Chemical inventory CERCLA - Reportable quantity SARA - Extremely hazardous chemical Remediation Issues: RCRA cleanup level: no air data RQ = 1 lb MCLG = .2 mg/1 MCL=.2 mg/1 water = 7E-01; soil = 2E+03 mg/kg; Reporting to Regulatory reporting/stds worldwide. Agencies Worldwide: OEL Fate in the Environment: HCN readily leaches from soil to groundwater. Chemical transformed chemically by several mechanisms in soil and groundwater. HCN is generally not considered persistent. Relevant bioaccumulation data: bioaccumulate. CN and HCN do not Known toxicity in plants: concentrations. ND relevant to environmental Known toxicity in fish: Ambient water quality criteria; freshwater: acute = 2.2 E+lug/L; chronic = 5.2 E+0 ug/1; marine: acute =1.0 E+0 ug/1; chronic = none Known toxicity in other aquatic species: ND relevant to environmental concentrations. C35703 0534 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 NOT LISTED CONEG HAZARD COMMUNICATION X WORLDWIDE X . EC PACKAGING/RECYCLING X LIFE CYCLE ANALYSIS OUTRAGE FACTOR X HIGH Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) _______________________ (Environmental data/sampling/analysis) ______________________________ (Ind. Hygiene sampling inf ormation) H:\PLANTVCHEMPRO.WAR C35703 0535 VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC - LDLO - LDjq - LOAEL - MEI - NIOSH REL - NOAEL - NSRL - PEL - RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 * . 54mg/m3. For risk level, E-6 * 10"* or 1 excess .cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community; Mon-cancer effects (Adult) 2.92E-03 mg/m3 (Child) 8.33E-04 mg/m3 Employee: (OSHA) 5 mg/m3 (Rfd = 2E-2mg/kg/day) H:\PLANT\CHEMPRO.WAR C35703 0536 ETHYL BENZENE RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The exposure limits are based on skin and eye irritation since no systemic effects can be expected at levels producing distinctly disagreeable skin and eye irritation. Carcinogenicity: NA Mutagenicity: NA Developmental Toxicitv: NA Reproductive Toxicitv: NA Systemic Toxicitv: Insufficient data, however, in animal studies the target organs for acute high-level exposure (on the order of 5000 to 10,000 ppm) are the lungs and CNS but longer term lower exposure tends to result in effects on the visceral organs. Greatest potential for exposure is the use of technical grade xylene. Irritation: Ethyl benzene is a skin and eye irritant. Risk Profile: The ambient air level goals (AALG) are based on the subchronic animal study results and on irritation. AALG: systemic toxicity-0.03 ppm (0.13 mg/m3 24- hour TWA irritation-10.0 ppm (43.5 mg/m3) 24-hour TWA C35703 0537 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: ETHYLBENZENE CAS#: 100-41-4 I*. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: CHjCHjC^Hj Common synonyms: Ethylbenzol, phonylethane Chemical/Physical Properties Molecular Wt. 106.2 Boiling Pt. 277*F Solubility .01% in H20 Flash Pt. 55F ionization Potential 8.7eV Vapor Pressure 10mm LEL 1.0% Specific gravity' .87 Freezing Pt. -139"F <#>. i C UEL 6.7% Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Associated withgasoline use. Product of biomass combustion and a component of crude oil. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 100 ppm; STEL=125ppm MSHA PEL: lOOppm ACGIH TLV: lOOppm NIOSH REL: lOOppm; STEL=100ppm Acute Toxicity Data: LDyj-, for oral, dermal, inhalation. Include species, ocu-hmn TDL0=200ppm orl-rat LD^SSOOmg/kg ihl-rat LCL0=4000ppm C35703 0538 Remediation Issues: RCRA Cleanup levels; Air: HD; Soil: 8E+03mg/kg; Water: 4.0E+0mg/L. Reporting to Regulatory Agencies Worldwide: European TSCA equivalent. Canadian and Fate in the Environment: Moderate adsorption in soil. Most vaporizes to atmosphere. It does not hydrolyze in soil or groundwater. In water, it evaporates to air. In atmosphere, it exists as a vapor phase. Relevant bioaccumulation data: HD on-line. Known toxicity in plants: HD on-line. Known toxicity in fish: HD on-line. Known toxicity in other aquatic species: HD on-line. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low Significant public activity and/or key topics ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: C35703 0540 VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) ____________________ (Environmental data/sampling/analysis) __________________|___________ (Ind. Hygiene sampling information) VII. GLOSSARY OF ACRONYMS; ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Careinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose H:\PLANT\CHEMPRO.WAR C35703 0541 VIII. RISK SUMMARY Coimimn< fcy; Non-cancer effects (Adult) lMfiB-Olmg/m3 (Child) 4.17E-02B9/B3 (Rfd=lE+0mg/m3) Employee: (OSHA1 100 ppm 125 ppm H:\PLANT\CHEMPRO.WAR C35703 0542 FORMALDEHYDE RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The designation as a suspect human carcinogen is based on the occurrence of nasal tumors in rats. Carcinogenicity; Results from nine epidemiological studies suggest that lung, nasopharyngeal, sinonasal and oro-hypo-pharyngeal cancers are associated with formaldehyde exposure. Sufficient evidence exists that formaldehyde is carcinogenic via the. inhalation.route in experimental animals based on the occurrence of nasal squamous cell carcinomas in both sexes of multiple rat strains and mice. Mutagenicity: Formaldehyde has been shown to be genotoxic in a variety of test systems encompassing a broad range of endpoints and levels of phylogenetic complexity. Developmental Toxicity: NA Reproductive Toxicity: Limited data suggests some reproductive effects on menstrual irregularities and ovarian cysts as well as low birth weight babies and miscarriages. Systemic Toxicitv: Formaldehyde isa primary skin sensitizing agent inducing allergic contact dermatitis by a delayed-type (Type IV) hypersensitivity mechanism. Irritation: Formaldehyde is irritating to the eyes, nose, and throat, and although no clear thresholds have been established, most people experience these effects in the range of 0.1 to 3 ppm. Risk Profile: The air ambient level goal (AALG) for formaldehyde is based on carcinogenicity. AALG: 10E-6 95%UCL-0.05 ppb (0.062/xg/mJ) annual TWA 10E-5 95%UCL-0.51 ppb (0.62/ig/mJ) annual TWA C35703 0543 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: FORMALDEHYDE CAS#: 50-00-0 Xj. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: CHjO Common synonyms: Formalin/ Formic Acid, Methylene Glycol, Paraforn Chemical/Physical Properties gaseous form/aqueous form Molecular Wt. 30.0 Bolling Pt. Solubility 207 to 214*F Miscible to \c\.%co in hj Ionisation Potential 10.88eV Vapor Pressure >latm/lmm Flash Pt. NA(gas)/140-l85*F Freezing Pt. -134*F PEL 73% LEL 7.0% Specific Gravity (77*F): X.08-1.10 (formalin) Flammable gas Class IIIB combustible liquid Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: clean marine air: <0.034ppb.; urban/suburban, >25% ave. cone. = 2.7 ppb. cone, high where more traffic occurs; more photochem. activity. Studies of indoor air levels: >0.12ppm; as great as >3.0ppm (mobile homes). Sampling/analytical methods: See attached. C35703 0544 r II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits; OSHA PEL: lppm(c); 5ppm (STEL) MSHA PEL: 2ppm (3mg/nr) (c) AC6IH TLV: 0.3ppm NIOSH REL: 0.016ppm(c); O.lppm (STEL) Acute Toxlcitv Data: LDjo-, for oral, dermal, inhalation. Include species. LDjt oral (rat) : lOOmg/kg LOm skin (rabbit: 270mg/kg LCj, inhalation (mouse) x 400mg/m3 IDLH: 30 ppm (Ref: NIOSH ) ERPG: 500 lbs. Chronic Toxicity Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): Mutagenic in viruses, bacteria, yeast, fungi; most mammalian cells produce positive Ames test. IARC Classification: Other designations: A2 EPA Genetox Program: positive (mouse/rat carcinogencity) A2: ACG1H susp. human CA Bl: probable human CA Inhalation unit risk: 1.3E-5 per ug/m3 Extrapolation: linearized multistage procedure. Risk Level E-4 E--5 E-6 Cone. 8E+0ug/m3 8E-lug/m3 8E-2ug/m3 NOAEL: 15mg/kg/day oral rat: GI lesions, decrease in weight (Ref: HEAST) (no observable adverse effect level) CAL. Prop 65 NSRL: 15ug/day (no significant risk level) C8F: 4.5E-2 (24 mon. rat inhal.) Specific cancer end point: Nasal cavity tumors Reproductive hazard: (inc. mutagen, terat) X Yes No C35703 0545  Sensitizer: Yes. Compound also produces contact dermatitis and respiratory irritation. Non-cancer Rfd: 2E-1 mg/kg/day End Point: Rat 2 yr. (oral) bioassay; reduced weight gain; histopathology in rats. Human toxicity data: (Inhalat) irritant to eyes, resp. system, probable human carcinogen - nasal upper respiratory tract cancer Biological Monitoring: Available: X Yes No Blood ACGIH BEI: N.D. III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA Effluent/Ambient Standards/established limits in various media: DOT - Guide 29 SARA - Extremely hazardous subs. TSCA - Chemical inventory World wide limits - Occupational Canada std. Europe, Australia, Remediation Issues: CERCLA reportable quantity: 1000 lbs. EPA haz. waste #U1222. RCRA cleanup level; air = 8E-02ug/m3. Reporting to Regulatory Agencies Worldwide: Canada/Europe Fate in the Environment: Soil: will leach through soil; fate unknown. Aquatic: biodegrades to low levels in a few days. Fate in groundwater unknown. Atm fate: photolyzes and reacts rapidly with free radical (OH*); Tm a few hours; 1* oxidation product resulting in formic acid (acid rain component). Relevant bioaccumulation data: Food chain concentration: none. Rapid biodegradation in water/soil. Known toxicity in plants: NA on-line. Known toxicity in fish: 25mg/L/96 hour: channel catfish; LCj,,: 100--330ppm/48 hr; flounder; LC^lOmg/L/48 to 96 hr; striped bass. C35703 0546 EU COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA Chemical is an indirect food additve for use only as a component of adhesives; food additive for animal feeds. CALIFORNIA PROP 65 X CONEG HAZARD COMMUNICATION X . WORLDWIDE X .. EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR High Significant public activityand/or key topics Vs. ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: ______ IH exposure monitoringavailable:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) _______________________ (Environmental data/sampling/analysis) ______________________________ (Ind. Hygiene sampling information) H:VPLANT\CHEMPRO.WAR C35703 0547 VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL - MEI NIOSH REL - NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = ,54mg/m3. For risk level, E-6 = 10"4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Comumw-i try; Cancer effects (Adult) 7.89E-06mg/m3 (Child) 1.13E-05mg/m3 Non-cancer effects (Adult) 2.92E-02mg/m3 (Child) S.aaE-oamg/m3 (CSF=4.5E-2 mg/kg/day1) (Rfd=2E-l "9/kg/day) Employee: (OSHA) lppm 5ppm (STEL) H:\PLANT\CHEMPRO.WA R C35703 0548 LEAD RISK SUMMARY; Health Effects Profile: Basis for Occupational Limits: The occupational limits are set in consideration of systemic toxicity, principally effects on the blood, nervous system, and kidney. Carcinogenicity; Lead has been placed on the B2 group, probable human carcinogen by the EPA based on statistically significant increases in renal tumors in rats and mice. MUtaaenicitv: Reported to induce cell transformation and to affect DNA to DNA and DNA to RNA transcription. It has been suggested that lead also may have promotional activity. Developmental Toxicity: Fetotoxic effects have occurred in animals at chronic exposure of 600-800 ppm inorganic lead in the diet and more subtle effects (behavioral and neurological) appear to have been observed at 5-10 ppm lead in the drinking water and at levels of 10 /xg/mJ lead in air. Reproductive Toxicity: Adverse effects on sperm and decreased function of the prostate and/or seminal vesicles have been reported to result from chronic lead exposure with blood lead levels of 40-50/jg/dL. Systemic Toxicitv: Lead induces toxicity in a number of organ systems, hematological, neurological, and renal effects are generally considered the most significant because they are induced at lower levels of exposure. Lead inhibits the heme synthesis pathway at several specific steps and also causes disturbances in globin biosynthesis. Irritation: Lead is not generally regarded as an irritant. Risk Profile: The national ambient air quality standard (NAAQS) of 1.5 /ig/m3 is the most appropriate ambient air level goal (AALG), based on factors such as lead's ubiquitousness in the environment and recent controversies over which health effects are significant, as well as its legal status, i.e., a standard rather than a guideline. AALG: 1.5 jtg(Pb)/mJ as a monthly average. C35703 0549 r CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: LEAD CAS#: 007439-92-1 PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Pb .PbCrCq Pt>S `Common synonyms: Lead acetate, lead oxide, lead nitrate, lead . -Chiagide,) lead sulfide, inorganic lead, organic lead, lead flaked lead paint, tetraethyl lead xi Chemical/Physical Properties Molecular wt. Boilino Pt. 207.2 3164*F Solubility Vapor Pressure Insol. in H20 0 mm Melting Pt. Specific Gravity 621*7 11.34 iWrt.50G Physical state in air: In stack: gas X particulate In ambient air: gas X particulate * (Vapors occur after > 900*C) If particulate, part size: AED 0.1-1.0 um (predicted deposition in lung is 35%). Ambient/background concentrations: Ambient air (EPA) = 1.5 ug/m3. Urban background Pb: 6 ug/m3 (EPA - high value) to 0.0012 ug/m3 (EPA - low value). U.8. tap water average = 30 ppb. Sampling/analytical methods: See attached HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 50 ug/m3, TWA Blood level not to exceed 0.60 mg/lOOg blood MSHA PEL: 0.15 mg/m3 ACGIH TLV: 0.15 mg/m3 TWA; no STEL NIOSH REL: <100 ug/m3 C35703 0550 Acute Toxicity Data: LDgy, for oral, dermal. Inhalation. Include species. Or1-rat; TDLO=165gm/kg/48 weeks Ivn-rat; LD5=120mg/kg IDLH: 700 mg/rn3 (Ref: NIOSH, 1990) ERPG: 10 lbs. Chronic Toxicity Data: Carcinogenic status: X Yes No Risk based on XBUK model (ERA, 1989) Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B2 Other designations: NA NOAEL: ND (Ref: IRIS ) (no observable adverse effect level) LOAEL -- 10 ug/dl for children and 40 ug/dl for adults CAL. Prop 65 NSRL: 0.5 ug/day CSF: ND Specific cancer adenocarcinoma end point: (no significant risk level) Reports of renal Reproductive hazard: (inc. mutagen, terat) X Yes No Specify end point: In males, sterility Sensitizer: No Non-cancer Rfd: Recommended ADI of 3 mg/wk. End Point: deposition. Neurological (also bone effects from Human toxicity data: Use IUBK model for humans for intake/risk levels. Lead is known to have muscular and central nervous system effects for humans and animal species. Biological Monitoring: Available: X Yes Blood, urine analysis No ACGIH BEI: Pb in blood, urine; zinc protoporphyrin in blood. C35703 0551 III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: AAQ = l.S ug/m3; MCL = 50 ug/1. HAP status CAA. Effluent/Ambient Standards/established limits in various media: CERCLA R.Q. = 1 lb. RCRA - Listed CWA - Listed CAA - Listed DOT - Guide 17 SARA - Hazardous chemical list TSCA - Chemical inventory Remediation Issues: State specific cleanup levels - usually to BKg for soil or risk-based (using IUBK model). For groundwater, clean up to MCL. Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent. Also world-wide air and water stds. Fate in the Environment: Soil lead remains static for polonged periods; water not a major vehicle for the movement of lead. Pb retained in upper 2-5 cm. of soil. Relevant bioaccumulation data: Pb bioaccumulates in the bone matrix in humans. Pb bioconcentrates also in shellfish. Known toxicity in plants: Generally, the uptake of Pb from soil into plants is not sufficient for lead to be considered a plant toxin. Known toxicity in fish: At 50 mg/1, morphological changes have been reported in many species. Fish are more susceptable to poisoning in soft water. Single lethal doses reported. Known toxicity in other aquatic species: No data reported for environmental concentrations. C35703 0552 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA X FDA action limit = 70 ug/ml (pottery) CALIFORNIA PROP 65 X CONEG X HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING X LIFE CYCLE ANALYSIS OUTRAGE FACTOR X Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) ____________________ (Environmental data/sampling/analysis) ______________:________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0553 VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC - - LDLO LDm LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - - - - - - - - - - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/rf = .SAmg/m3. For risk level, E-6 = 10"4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Use site-specific IUBK model; no CSF or RFD established. (for risk level of E-6) Est. Action Level Cancer effects 5.0E--02/ug/m3 (air) (based on safety factor applied to PEL) Employee: (Q8HA) 50 ug/m3 H:\PLANT\CHEMPRO.WAR C35703 0554 MERCURY RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The occupational limits are based on neurotoxicity, the most sensitive endpoint for mercury toxicity. Carcinogenicity; NA Mutagenicity; Data is generally lacking some reports that methyl mercury blocks mitosis in plant cells and human cells treated in vitro and induces chromosome breaks in plant cells and mutations in Drosophila. Developmental Toxicitv: Data on developmental effects of inhaled mercury are lacking, however well documented effects of ingestion of methyl mercury producing psychomotor retardation in infants exist. Reproductive Toxicitv: Increased menstrual and spontaneous abortion have been reported based on occurrence of overt signs of mercury poisoning in instances, exposure levels were probably fairly high. Systemic Toxicitv: The types of systemic toxicity observed with mercury exposure are dependent on the chemical form. Chronic exposure to mercury vapor effects mainly the nervous system and kidney. Occupational exposure to inorganic mercury has been reported to cause peripheral nervous system damage. The primary effect of methyl mercury exposure is neurotoxicity. Irritation: NA Risk Profile: There are three separate ambient air level goals (AALG) that have been calculated based on the TLVs for the neurotoxicity effects of mercury vapor, inorganic mercury and for alkyl mercury. AALG: -systemic toxicity (TLV-based) mercury vapor-0.95 |xg(Hg)/m3 8-hour TWA inorganic mercury-0.48/xg(Hg) /m3 8-hr TWA alkyl mercury-0.05/ig(Hg)/m3 8 hour TWA C35703 0555 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: MERCURY COMPOUNDS CAS#: 7439-97-6 I. PHYSICAL/CHEMICAL INFORMATION; ^ Chemical formula: Hg TM Common synonyms: Collodial mercury; metallic mercury; quicksilver; organo mercury compounds; alkyl . mercury compounds. WWJLtyxsu* <J\Wde,, ) fVlmCuncMa^atc Chemical Physical Properties (listed below for mercury vapor) Molecular Wt. 200.6 Melting Pt. MA Boiling Pt. 674#p solubility Insol. in H2o Specific Gravity 13.6 Vapor Pressure .0012mm Physical state in air: In stack: X gas particulate In ambient air: X gas particulate As collodial or metallic mercury, physical state will be a vapor; as organo or alkyl compounds, physical state will be particulate. Ambient/background concentrations: Naturally occurring. Coal contains 10-8500 ppb; crude oil = 20-20,000 ppb. USA average stack Hg emmissions for roasting operations = 2-3%. Average atmospheric cone. = 2-10 ng/nr. Normal groundwater concentration below 1 ppb. Sampling/analytical methods: See attached. 035 703 0556 HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: Hg vapor=.05 mg/m3; Hg=.Ol mg/m3 MSHA PEL: .05 mg/m3 (vapor) ACGIH TLV: .05 mg/m3 (vapor) HIOSH REL: Hg vapor=.05 mg/m3; Hg=.01 mg/m3 Acute Toxicity Data: LDjo-t for oral, dermal, inhalation. Include species, inh - hmn TCLO=l69 ug/m3 ipr - rat TDLO=400 mg/leg orl - hmn TDLO=40 mg/kg (elemental Hg) IDLH: vapor=28 mg/m3; Hg=10 mg/m3 (Ref: HIOSH ) ERPG: ND chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic): NA IARC Classification: D Other designations: Under EPA review NOAEL: .009 mg/m3 CAL. Prop 65 NSRL: (Ref: IRIS ) (no observable adverse effect level) ND (no significant risk level) CSF: ND Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) X Yes No Chromosomal aberrations have been observed in whole blood cultures from exposed workers. Limited reports of teratogenicity in animals. Sensitizer: Organo/alkyl compounds are suspect sensitizers. Non-cancer Rfd: 3E-4 mg/m3 (inh - man; chronic and subchronic) End Point: Neurotoxicity Human toxicity data: Known human nephrotoxin and neurotoxin. Biological Monitoring: Available: X Yes No Blood, urine, hair, nails. Total inorganic Hg in blood=15mg/l C35703 0557 ACGIH BEI: ND Notice of intent to establish BEI of 35 ug/g creatinine. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: RCRA - Listed CWA - Listed DOT - Guide 39 SARA - Extremely hazardous chemical TSCA - Chemical inventory H2O=.001 mg/1, air= 5 ug/m3 recommended limits MC1=.002 mg/1 RQ=1 lb. Mercury is concentrated in the sludges from sewage treatment by a factor of several hundred to several thousand. Remediation Issues: RCRA cleanup levels: 1E+01 mg/kg - soil. Much of Hg deposited on land appears to vaporize within 2-3 days (organic Hg). Reporting to Regulatory Agencies Worldwide: European TSCA equivalent. Canada and Fate in the Environment: In aquatic systems, Hg binds to dissolved matter of fine particulates. Hg can be dissolved into the water column. Recycling of Hg from sediment to water to air and back is a rapid process. Volatilization from land and lakes was estimated to enhance atmospheric concentrations. Relevant bioaccumulation data: Hg bioaccumulates in humans and animal species and in food chains; concentration may be as much as 10,000 times that of water. Known toxicity in plants: Plants may bioaccumulate Hg, but to a lesser extent than animal species; however, the availability of soil Hg to plants is low. Max. levels of Hg are recommended at .5 ppm for plant tissue and 15 ppm for growth in soil. Known toxicity in fish: Permissable level in Canada for fish is .5mg/kg or less. Average fish cone.=100-200 ng/gr. C35703 0558  Known toxicity in other aquatic species: Bioconcentration factors of 63/000 for fresh water and 10,000 for salt water fish have been observed. IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X Bottled H20 must be tested and not contain in excess of the MCL value. Limits established in FD&C blue dye. 1 ppm Hg allowed in fish. CALIFORNIA PROP 65 ND CONEG X HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: H:\PLANT\CHEMPRO.WAR C35703 0559 VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) (Environmental data/sampling/analysis) ______________________________ (Ind. Hygiene sampling information) GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 -- - ERPG IARC IDLH LC - - " LDLO - LDjo - LOAEL - MEI - NIOSH REL - NOAEL - NSRL - PEL - RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10-4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose H:\PLANT\CHEMPRO.WAR C35703 0560 VIII. RISK SUMMARY ComnnirH fry; Mon-cancer effects Adult = 4.38E-05 mg/m3 Child = 1.25E-05 mg/m3 Employee: {QBHAl .05 mg/m3 (Rfd=3E-4mg/m3) H:\PLANT\CHEMPRO.WAR C35703 0561 METHYL ALCOHOL RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The occupational limits for methanol are based on prevention of recurring headaches in workers as well as consideration of narcotic and ocular effects and sensory irritation. Carcinogenicity; NA Mutagenicity: Methanol has been reported to interfere with DMA.repair, induce mutations, and cause chromosomal aberrations. Developmental Toxicitv: Fetotoxicity in rats has been reported in the form of decreased body weight, increased incidence of gross skeletal and visceral defects. Reproductive Toxicitv: Interference with spermatogenesis in mice. Systemic Toxicitv: Symptoms have been reported in cases of exposure via ingestion, dermal absorption, and inhalation which include, dizziness, nausea, and vomiting, various types of visual disturbances, vertigo, unsteady gait, multiple neuritis with paresthesia, pains in the extremities, and headaches. Irritation: Irritating to the eyes and mucous membranes at levels greater than are known to produce systemic effects. Risk Profile: There are two ambient air level goals for methanol; one based on developmental toxicity and another based on systemic toxicity using the TLV. AALG: systemic toxicity-2.3 ppm (3.1mg/m3) 8-hour TWA developmental toxicity-3.9 ppm (5.1 mg/m3) 24-hr TWA C05703 0562 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: METHYL ALCOHOL CAS#: 67-56-1 L. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: CH3OH Common synonyms: Methanol, wood spirits, wood alcohol, methyl hydroxide, carbinol, Columbian spirits. Chemical/Physical Properties Molecular Wt. 32.1 Specific Grayity .79 Boiling Pt. 147*P (??.<}*' Flash Pt. 52F Solubility Vapor Pressure Miscible in H2Q 92mm Ionization Potential 10.846V Freezing Pt. --114F - PEL 36% Physical state in air: LEL 6% In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Methanol is a natural emission product from various plants as well as decomposition of biological wastes. Largest source of release is from solvent use. Mean atmospheric cone. =7.9 ppb. Sampling/analytical methods: See attached C35703 0563 HEALTH INFORMATION - EMPLOYEE AND COMMUNITY; Exposure Limits: OSHA PEL: 200 ppm; MSHA PEL: 200 ppm ACGIH TLV: 200 ppm NIOSH REL: 200 ppm STEL=250 ppm Acute Toxicity Data: LDjo., for oral, dermal, inhalation. Include species. Orl - hmn LDL0 = 340 mg/kg; TDL0 hmn = 100 mg/kg; Ihl - hmn LCL0 = 300 ppm IDLH: 25,000 ppm (Ref: NIOSH ) ERPG: ND Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic): NA IARC Classification D Other designations NA NOAEL: 500 mg/kg/day (Ref: HEAST) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) CSF: ND Specific cancer end point: ND Reproductive hazard: (inc. mutagen, terat) X Yes No Reports of mutagenicity and teratogenicity in humans by oral route of exposure in excess of established exposure limits. Sensitizer: No Non-cancer Rfd: 5E+0mg/kg/day (oral - rat) End point: Blood alkaline phosphatase increase; increase SGPT; decreased brain weight. Human toxicity data: Causes central nervous system effects in humans. Reports of dermatitis are also known. C35703 0564  Biological Monitoring: Available: X Yes No Blood/ urine ACGIH BEI: Methanol - urine - 15 mg/L; 80 mg/gr creatinine (formac acid) III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: Effluents from wastewater ranged from 18-70 ppm RQ= 5000 lbs. DOT - Guide 59 TSCA - Chemical inventory Remediation Issues: No RCRA cleanup levels established. Reporting to Regulatory Agencies Worldwide: ND on-line. Fate in the Environment: If released into the atmosphere. Methanol degrades within Tia of 17.8 days. Methanol degrades both in soil and water. Methanol will leach to groundwater, but majority is evaporated from soil. Relevant bioaccumulation data: Methanol has a low bioconcentration factor in humans and animal species. Known toxicity in plants: No data relevant to environmental concentrations. Known toxicity in fish: No data relevant to environmental concentrations. Known toxicity in other aquatic species: No data relevant to environmental concentration. Methanol does not bioaccumulate in aquatic species. C35703 0565 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X Methanol is an indirect food additive for use only as a component of adhesives. CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION; Dispersion model summaryattached: ______ IH exposure monitoring available:_______ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) _____________________ (Environmental data/sampling/analysis) ______________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0566 VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL ME I NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 .54mg/m3. For risk level, E-6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY rnwmnn^ ty Mon-cancer effects (Adult) 7.29E-01mg/m (Child) 2.08E-01mg/m3 (Rfd=5E+0 mg/kg/day) Employee: (OSHA) 200 ppm 250 ppm STEL T>RO.WAR C35703 0567 METHYLENE CHLORIDE RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The occupational standards are based on the prevention of carboxyhemoglobinema in the absence of exposure to carbon monoxide, with consideration of suspect carcinogenic effects. Carcinogenicity; Methylene chloride has been placed in group B2 probable human carcinogen under the EPA weightof-evidence classification. Studies in mice of both sexes showed clear evidence of carcinogenicity based on incidence of alveolar/bronchiolar neoplasms and, hepatocellular neoplasms. Mutagenicity; Positive responses for methylene chloride have been reported in a number of assay systems. Developmental Toxicitv: A case-control study of women working in the Finnish pharmaceutical industry revealed an association between exposure to methylene chloride (and a number of other solvents) and the occurrence of spontaneous abortion. Exposure to high levels (1225 ppm via inhalation) of methylene chloride caused an increased incidence of minor skeletal anomalies in rats and mice fetuses. Reproductive Toxicitv: Refer to above Systemic Toxicitv: Methylene chloride is metabolized to carbon monoxide and produces elevated levels of COHb in both animals and humans. The COHb levels are related to both duration of exposure and concentration of methylene chloride. Irritation: Liquid methylene chloride is irritating if splashed in the eyes and may cause burns if it remains in contact with the skin. Risk Profile: The ambient air level goal for methylene chloride is based on carcinogenicity using the quantitative risk assessment performed on the NTP inhalation bioassay data. AALG: 10E-6 95%UCL-0.12Mg/m3 (0.036 ppb) annual TWA 10E-5 95%UCL-1.22/ig/m3 (0.357 ppb) annual TWA C35703 0568 CLEANWr ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: METHYLENE CHLORIDE CAS#: 75-09-2 I_s. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: CH2C12 Common synonyms: Dichloromethane, Methylenedichloride Chemical/Physical Properties Molecular Wt. 84.9 Vapor Pressure 350mm Boiling Pt. 104*F , AQ.0'0 Melting Pt. NA Solubility 2% in H20 Specific gravity 1.33 Physical state in air: In stack: Z gas In ambient air: Z gas particulate particulate Ambient/background concentrations: No natural sources. Methylene chloride is formed from the chlorination of water. Usual release to environment is through solvent use. Background global air conc.=32ppt. Water concentrations range from 10-5oppb. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 500ppm Ce=1000ppm STEL=2000ppm MSHA PEL: 500 ppm ACGIH TLV:50ppm;175mg/m3 NIOSH REL:CA - reduce to lowest feasible concentration C35703 0569 Acute Toxicity Data; LDjq., for oral, dermal, inhalation. Include species, inh-hnm TCL0=500ppm orl-dog LDL0=3000mg/kg ivn-dog LDL0=200mg/kg scu-rbt LDL0=2700mg/kg IDLH: Ca [SOOOppa] (Ref: MI08H ) ERPG: ND Chronic Toxicity Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B Other designations: ACGIH-A2 NOAEL: 694.8mg/m3; 5.85mg/kg/day (Ref: IRIS ) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) CSF: ND Specific cancer end point: ND Reproductive hazard: (inc. mutagen, terat) X Yes No Several reports of mutagenicity and teratogenicity in literature. Sensitizer: No Non-cancer Rfd: 3E+0mg/m3 (inh-rat); 6E-2mg/kg/day (oral-rat) End Point: liver, critical effect=toxicity Human toxicity data: group review. Suspect human carcinogen. Under EPA Biological Monitoring: Available: X Yes No Expired air, urine, blood. Liver function tests. ACGIH BEI: ND C35703 0570  * III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP chemical list CAA. Effluent/Ambient Standards/established limits in various media: RCRA - Listed DOT - Guide SARA - Hazardous chemical TSGA - Chemical inventory Remediation Issues: RQ=1000 lbs. RCRA cleanup levels; air: 3E-0lug/m3; H20: 5E-03mg/l; soil: 9E-f0lmg/kg. Reporting to Regulatory Agencies Worldwide: Canada and European TSCA equivalent. Fate in the Environment: Methylene chloride is released into atmosphere where it degrades by reaction with photochemical OH radicals. Releases to water and soil are primarily removed by evaporation. Methylene chloride will partially leach from soil to groundwater. It does not significantly absorb to sediment. Relevant bioaccumulation data: Methylene chloride is converted to CO and does not bioaccumulate. Known toxicity in plants: No reported data in literature for environmental levels. Known toxicity in fish: No reported data in literature for environmental levels. Known toxicity in other aquatic species: No reported data in literature for environmental levels. Compound does not bioaccumulate in aquatic species. C35703 0571 * IV COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA Z CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low to moderate Significant public activity and/or key topics Vi. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) _________________ (Environmental data/sampling/analysis) _______________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0572 VII. GLOSSARY OF ACRONYMS: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E01 - ERPG IARC IDLH LC -- - -- LDLO LDjo LOAEL MEI NIOSH REL - - NOAEL NSRL - - PEL - RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Careinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10"* or 1 excess cancer ,in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY nnmmin< fry; Non-cancer effects (Adult) 4.38E-01ag/n3 (Child) l.25E-0lmg/m3 Employee* (OSHA) (Rfc=3.0E+0mg/m3; 500 ppm 1000 ppm-ceiling 2000 ppm 8TEL Rf <1=6.0E-2mg/kg/day) H:\FLANT\CHEMPRO.WAR C35703 0573 METHYL ETHYL KETONE: RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The occupational limits are based on the minimization of eye an upper respiratory tract irritation. Carcinogenicity; NA Mutagenicity; NA Developmental Toxicitv: Exposure ofMEK vapors to rats showed a statistically significant decrease in fetal body weight and crown-rump length at the low (1126 ppm), but not at the high (2618 ppm), exposure level, and a dose- related increase in skeletal anomalies was observed. Reproductive Toxicitv: NA Systemic Toxicitv: Data onchronic inhalation and ingestion exposure to MEK are reported to be lacking in both animals and humans. Irritation: MEK is considered an eye and nose irritant. Risk Profile: Due to the lack of human data and the broad spectrum of effects associated with MEK exposure for animal studies the ambient air level goals were calculated for multiple endpoints-developmental, systemic and irritation. AALG: developmental toxicity-0.13 ppm (0.39mg/m3) 24-hour TWA systemic toxicity-0.11 ppm (0.33 mg/m3) 24- hour TWA(motor nerve function); 0.5 ppm (1.5mg/m3) 24-hour TWA (decreased body weight) irritation-4.0 ppm (12mg/m3) 24-hour TWA C35703 0574 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME KETONE CAS# 78-93-3 JU PHYSICAL/CHEMICAL INFORMATION: Chemical formula: C^O Common synonyms: MEX, methyl-acetone, 2-butanone ' Chem/Phvs Properties Molecular Wt. Boiling Pt. Solubility 72.1 175F 28% in H.O 'Mtfc Ionization Potential Specific Gravity 9.54eV 0.81 PEL (200*F) 11.4% LEL (200F) 1.4% Class IB flammable liquid Flash Pt. 16^ Vapor Pressure 71 mm Physical state in air: In stack: V X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Generally absent except during episodes of photochemical smog; atm cone: rural: ND; urban/sub: ND (181 samples); air pollution episodes: NJ: 64 ppt average from 33 samples. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 200ppm/300ppm STEL MSHA PEL: 200ppm ACGIH TLV: 200ppm NIOSH REL: 200ppm/300ppm STEL C35703 0575  Acute Toxicity Data: LDjo-. for oral, dermal, inhalation. Include species. LDm (oral) rats: 2,737 mg/kg/6.86 ml/)cg LCn rat: 23,5000 mg/m3/8 hr TCLO (inhal) human: 100 ppm/5 min IDLH: 3000 ppm (Ref: NIOSH ) ERPG: Mo data Chronic Toxicity Data: Carcinogenic status: Yes X No Mot classifiable as to human carcinogencity. Mo human data. Inadequate animal data. Only dermal study. Promoter (epigenetic) and/or Initiator (genotoxic):NA IARC Classification: NA Other designations: NA NOAEL: 693 mg/m3 (Ref: HEAST) (no observable adverse inhalation rat - none observed effect level) CAL. Prop 65 NSRL CSF: NA N.D. (no significant risk level) Specific cancer end point: MA Reproductive hazard: (inc. mutagen, terat) X Yes No Effects only at levels which are toxic to mother Arepro (unconfirmed human data). NOAEl 2978 mg/m3/LOAEL 8906 mg/m3. Experimental for repro toxicity. Sensitizer: No Non-cancer Rfd: 5E-lmg/m3 subchronic rat inhalation; 5E-2mg/m3 chronic rat inhalation RfC: lE+O mg/m3 Specify end point: Decreased fetal weight (mouse). ADI: 3.2 mg/day (oral) EPA SNARL: 7.5 mg/L (Suggested Mo Adverse Response Level) C35703 0576  Human toxicity ;data: The minimal toxic or lethal dose is not well established. May cause eye irritation, tachycardia, anesthetic type of respiratory depression, dyspnea as gasping, aspiration may result in chemical pneumonitis. CMS depression (headache, dizziness, fainting, tremor, coma, death). In combination w/n- Hexane, MEK can potentiate neurotoxin effects: peripheral neuropathy. Biological Monitoring: Available: X Yes No Urine and blood ACGIH BEI: MEK in urine end of shift: 2 mg/L III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: RCRA CWA CAA DOT SARA TSCA - listed - none - none - Guide t 26 - HOT extremely hazardous - listed Remediation Issues: CERCLA rep. qty: 5000 lbs. RCRA cleanup levels: air=3E+02mg/m3; water=2E+00mg/l; soil=4E+03mg/kg. Reporting to Regulatory Agencies Worldwide: Canada/Europe Fate in the Environment: Terrestrial Fate: partially evaporates into atm. and leaches into ground. Aquatic Fate: evaporates into atm.; t1/2 3-12 days in rivers/lakes. Atm. Fate: when released into atm., it will degrade by reaction with photochemically produced hydroxyl radicals (T,/2 2.3 days). Relevant bioaccumulation data: Mot expected to bioaccumulate in human or animal species. Known toxicity in plants: 32 samples southern pea MEK found in all; mean cone: 151 + 80 ppm. Known toxicity in fish: No data available on-line. C35703 0577  Known toxicity in other aquatic species: No data available relevant to environmental concentrations. MEK is not known to bioaccumulate in aquatic organisms. IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring _ available: Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) ` L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0578 VII. GLOSSARY OF ACRONYM^: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = .54mg/mJ. For risk level, E-6 * 10-* or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Commin i ty Non-cancer effects (Adult) 0.007 mg/m3 (Child) 0.002 mg/m3 (Rfc=5E-2mg/m3) Worker (OSHA) 200 ppm 300 ppm STEL H:\PLANT\CHEMPRO.WAR C35703 0579 SMETHYL ISOBUTYL KETONE RISK SUMMARY; Health Effects Profile: Basis for Occupational Limits: The occupational limits MIBK are based on prevention of irritation and possible nephrotoxicity. Carcinogenicity; NA Mutaaenicitv: NA Developmental Toxicitv: NA Reproductive Toxicitv: NA Systemic Toxicitv: The acute toxicity of MIBK is relatively low, based on both oral and inhalation data. Animal studies (rat) revealed that exposure to 100 to 200 ppm MIBK the only differences between the exposed and control animals was an increase in the absolute and relative kidney weights in the exposed groups and higher relative and absolute liver weights in the highest exposed group. The effects of MIBK exposure in humans have not been evaluated in epidemiologic studies. Case reports on humans have reported workers exposed to approximately 100 ppm MIBK having headaches and nausea. Irritation: At high concentrations (200 ppm) MIBK is irritating to the mucous membranes of the eyes, nose, and throat. Risk Profile: The basis of the ambient air level goals (AALG) is the longstanding Threshold Limit Value (TLV) established by the American Conference of Governmental Industrial Hygienists (ACGIH). AALG: systemic toxicity (TLV-based)--0.12 ppm (0.49 mg/m3) 8-hour TWA C35703 0580 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: METHYL ISOBUTYL KETONE CAS#: 108-10-1 I. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: (CE,)2CHCH2COCH3 Common synonyms: Hexane/ KIBK, Isobutyl methyl ketone hOh 3 Chemlcal/Phvsical Properties Molecular Wt. 100.2 Boiling Pt. 242F Mfc.S'c Solubility 2% in H20 Ionization Potential 9.30eV Specific Gravity 0.80 Flash Pt. 64F n.e>c'c Vapor Pressure 16mm FreeziroFpt, (-12Cft?y PEL (200F) 8.0% LEL (200F) 1.2% Class IB Flammable Liquid Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Qualitatively ID in Cuyahoga River. ID but not quantified in 1/704 samples of surface water from industrial areas in US. ID Delaware River; drinking water; leachate from landfill CT=l72-263/ug/L. Air levels - ND Allegheny Tunnel; NJ-O ppt (urban, sub) NJ at source dominated area=270 ppt; Kinbuc waste disposal site Edison, NJ (1976)=2.1-6.0/ug/m3. Sampling/analytical methods: See attached. C35703 0581 HEALTH INFORMATION \ EMPLOYEE AND COMMUNITY: Exposure Limits; OSHA PEL: 50ppm; 75ppm STEL MSHA PEL: 50ppm ACGIH TLV: 50ppm; 75ppm STEL NIOSH REL: 50ppm Acute Toxicity Data: LDjq., for oral, dermal, inhalation. Include species. Ihl - hmn TCLO=2 o Oppm orl - rat LDSfl=2080mg/kg IDLH: 3000ppm (Ref: NIOSH ) ERPG: 10,000 lbs. Chronic Toxicitv Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic): NA - No genetic studies found for MIBK. IARC Classification: D Other designations: NA NOAEL: 50mg/kg/day (Ref: HEAST ) (no observable adverse effect level) CAL. Prop 65 NSRL: NONE (no significant risk level) CSF: N.A. Specific cancer end point: N.A. Reproductive hazard: (inc. mutagen, terat) Yes X No Class E - no effects in animals, but no human data. Actually human repro unknown. Not embryotoxic or teratogenic in rats or mice. Sensitizer: No Non-cancer Rfd: 5E-1: oral rat subchronic 5E-2: oral rat chronic End Point: Liver/kidney effects Human toxicity data: Absorbed by ingestion, inhalation and dermal exposure. CV:tachycardia. Resp: anesthetic resp. depression, aspiration may result in chem. pneumonitis. Inhalation of high cone, causes CNS depression. Biological Monitoring: Available: X Yes No Blood, urine ACGIH BEI: ND C35703 0582 III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA Effluent/Ambient Standards/established limits in various media: RCRA - Listed DOT - Guide #26 TSCA - Chemicalinventory SARA - Hazardous chemical Remediation Issues: CERCLA reportable: 5000 lbs. RCRA cleanup levels: air=7.E+ lug/m3; water=2E+00mg/L; soil=4E+03mg/kg. Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent. Fate in the Environment: Soil: removed by photolysis on soil surface. Volatilization or aerobic biodegradation susceptable to extensive leaching. Aquatic: method of removal - volatilization (Tia:15-33 hr) and direct photolysis. Atm: direct photolysis (Tin 15 hrs); acetone is a major photo oxidation product. Relevant bioaccumulation data: Not expected to bioaccumulate in aquatic organisms or absorb to suspended solids or sediments in water. Does not bioaccumulate in humans. Known toxicity in plants: ND on-line. Known toxicity in fish: LCM goldfish 460mg/l/24h; LC^ fathead minnow 505mg/l/96h. Known toxicity in other aquatic species: ND on-line. C35703 0583 IV. COMMERCIAL ISSUES ftfop RELATED REGULATIONS: FDA Permitted for direct addition to food for human consumption as long as quantity added to food does not exceed amount reasonably required to accomplish its intended physical, nutritive or tech, effect in food. Indirect food additive with adhesives/polymers in contact with food. CALIFORNIA PROP 65 C0NE6 . HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summaryattached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) - L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PlANT\CHEMPRO.WAR C35703 0584 VII. GLOSSARY OF ACRONYM^: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10-4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community; Mon-cancer effects (Adult) 0.007mg/m3 (Child) 0.002mg/m3 Workers:(OSHA) 50 ppm; 75 ppm STEL (Rfd=5E-2mg/kg/day) H:\PLANT\CHEMPRO.WAR C35703 0585 \ NICKEL RISK SUMMARY; Health Effects Profile: Basis for Occupational Limits; There are several occupational limits for nickel depending on the form of the nickel. The American Conference of Governmental Industrial Hygienist (ACGIH) assessed the literature on the health effects of nickel and concluded that based on the available information that not all forms of nickel are carcinogenic. However, NIOSH has set recommended limits based on its conclusions that nickel metal and all inorganic nickel compounds when airborne should be considered carcinogenic. Carcinogenicity: Human epidemiologic data indicate that as least some forms of nickel are carcinogenic to humans via inhalation, but the evidence for the ingestion route is inadequate. Nickel sulfide ore smelting and refining processes have been associated with an excess risk of lung, nasal, and laryngeal cancers in a number of studies, as well as buccal, pharyngeal, prostate, and kidney cancers in a few instances. Nickel sulfide and nickel subsulfide from refinery dust are considered Group A, human carcinogens by the EPA. Nickel carbonyl is classified as a group B2, probable human carcinogen, by the EPA. Mutagenicity: It appears that nickel may induce gene mutations in bacteria and cultured mammalian cell, however, the evidence is fairly weak. In addition nickel appears to induce chromosomal aberrations in cultured . mammalian cells and sister chromatid exchanges in both cultured mammalian cells and human lymphocytes. Developmental Toxicitv: Nickel carbonyl has induced various fetal malformations and caused fetotoxicity in several species of mammals. Other nickel compounds, nickel chloride, nickel acetate, and nickel subsulfide, have been reported to be fetotoxic and/or teratogenic following ingestion or parenteral administration. Reproductive Toxicitv: Inhalation studies assessing the effects of nickel on the reproductive processes of laboratory animals are lacking. Ingestion and parenteral administration studies in male rats indicate that certain nickel salts may cause degenerative changes in the testes and epididymis as well as effects on spermatogenesis. Nickel as an implant may decrease embryo viability and inhibit the implantation process. Systemic Toxicitv: The most prominent effects associated with nickel are allergenicity and toxicity to the respiratory tract. Nickel can induce contact dermatitis in sensitized individuals. Irritation: Although dusts and fumes of various nickel compounds may be irritating to the respiratory tract and have been reported to cause perforation of the nasal C35703 0586 \ septum, other forms of toxicity generally occur at lower levels of exposure than those producing these effects. Risk Profile: The ambient air level goal (AALG) for those nickel compounds classified in groups A or B2 are treated as carcinogens. These include nickel subsulfide and nickel carbonyl. An additional AALG for nickel carbonyl was calculated based on developmental effects. The AALGs for soluble nickel compounds and insoluble nickel compounds are on systemic effects. AALG: nickel subsulfide 10E-6 95% UCL--0.4 ng (Ni)/m3 annual TWA 10E-5 95% UCL--4.2 ng (Ni) /m3 annual TWA nickel carbonyl carcinogenicity--0.024 /ig/m3 annual TWA developmental--210 MU/5 24-hour TWA soluble nickel compounds systemic toxicity--0.36 ng (Ni)/m3 24-hour TWA insoluble nickel compounds systemic toxicity--7.1 ng (Ni)/m3 24-hour TWA C35703 0587 * CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: NICKEL CAS#: 7440-02-0 Xjl PHYSICAL/CHEMICAL INFORMATION; ^ Chemical formula: -Common synonyms: Hi Nickel metal, nickel rUcK*u ' refinery dust*- Chemical/Physical Properties Molecular Wt. Boiling Pt. Solubility 58.7 5139*F Mb &C' Insol. in HzO Melting Pt. Specific Gravity 2831P \<c)S(p 8.90 Physical state in air: Vapor Pressure 0 mm In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: ND on-line. Ambient/background concentrations: Naturally occurring. Nickel constitutes .03% of the particulate matter suspended in the atmosphere. Average concentration of nickel in earth's crust is 60-90 mg/kg. Typical average levels of airborne nickel are 003-.03 ug/m3. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: .1 mg/m3 (soluble forms); 1 mg/m3 (metal and insoluble forms) MSHA PEL: 1 mg/m3 (metal soluble forms) ACGIH TLV: 1 mg/m3 (metal soluble forms) NIOSH REL: 0.015 mg/m3 (soluble forms) C35703 0588 Acute Toxicitv Data: LDjq., for oral, dermal, inhalation. Include species. IMS - rat TDLO = no mg/kg IVN - dog LDLO = 10 mg/kg IV - rat TDLO =133 mg/kg IMS - rat LDLO =25 mg/kg IDLH: No limit set (Ref: NIOSH) ERPG: 500 lbs. Chronic Toxicitv Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: D as metal Other designations: Nickel refinery dust = IARC class A carcinogen NOAEL: 100 ppm (Ref: IRIS) (no observable CAL. Prop 65 NSRL: adverse effect level) 0.80 ug/day (no significant risk CSF: 8.4E-l{mg/kg/day`l) level) Specific cancer end point: Respiratory system - tumors by inhalation (man) Reproductive hazard: (inc. mutagen, terat) X Yes No (HSDB) - reports of male and female reproductive capacity effects. Sensitizer: Yes; skin and respiratory Non-cancer Rfd: 2E-2 mg/kg/day subchronic oral Rfd (rat) End Point: Whole body; decreased weight Human toxicity data: Known human carcinogen to respiratory system (nasal cavity and lung) by inhalation. Oral toxicity is low. Nickel is well known as a cause of contact dermatitis. Biological Monitoring: Available: X Yes No Can be detected in serum/blood/urine. Normal blood 2.6-4.6 ug/L. Normal urine levels 5 ug/dl. levels ACGIH BEI: ND (no established criteria) C35703 0589 III. ENVIRONMENTAL INFOI&IATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: RCRA - Listed DOT - Guide 39 TSCA - Chemical inventory CERCLA - R.Q.=1 lb. Remediation Issues: RCRA Levels Nickel Nickel Dust Air (ug/m5) ND 4E-03 Water (mg/1) 7E-01 ND Soil (mg/kg) 2E+03 ND Reporting to Regulatory Agencies Worldwide: Europe, Canada Fate in the Environment: The atmosphere is a major conduit for nickel as particulate matter. Soilborne nickel may enter waters by surface runoff. Relevant bioaccumulation data: Does not readily bioaccumulate in man or animal species. Known toxicity in plants: Nickel is taken up by plants from soil. Soils in excess of 2000mg/kg may cause root/stem plant damage. Known toxicity in fish: No relevant data for environmental concentrations. Known toxicity in other aquatic species: No relevant data for environmental concentrations. Nickel is not known to bioaccumulate in aquatic species. C35703 0590 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA X CALIFORNIA PROP 65 X CONEG X HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate to high significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available: Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:VPLANTVCHEMPRO.WA R C35703 0591 VII. GLOSSARY OF ACRONYM^: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDso LOAEL MEI NIOSH NOAEL NSRL REL - PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = .54mg/m3. For risk level, E--6 * 10"4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Cancer effects (Adult) 4.22E-07mg/m3 (Child) 6.04E-07mg/m3 Non-cancer effects (Adult) 2.92E-03mg/m3 (Child) 8.33E-04mg/m3 (CSF: 8.4E-1 (mg/kg/day'1) (Rfd: 2.OE-2(mg/kg/day) Employee; (OSHA) 0.1mg/m3 soluble forms 1.0mg/m3 metal and insoluble forms H:\PLAKT\CHEMPRO.WAR C35703 0592 NITRIC ACID S RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The occupational limits for nitric acid are based on prevention of irritation. Carcinogenicity; NA Mutagenicity; NA Developmental Toxicitv: NA Reproductive Toxicitv: NA Systemic Toxicitv: Limited data available Irritation: The principal hazards associated with nitric ac;id exposure in humans are related to it irritant and caustic properties and include (1) chemical burns due to dermal contact, (2) corneal and other eye injuries due to "splash" accidents, (3) chemical burns around the mouth and mucous membranes following ingestion and (4) pulmonary irritation following inhalation exposure. Risk Profile: The basis for the ambient air level goal (AALG) for nitric acid is its irritant properties. AALG: irritation (TLV-based)--8ppb (21 /xg/m3) 8- hour TWA 035703 0593 cleanUir assurance team CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: NITRIC ACID CAS#: 7697-37-2 I. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: HNQ, Common synonyms: Engraver's acid, acide nitrique, aqua fortis chemical Physical Properties Colorless to yellow or reddish brown fuming corrosive liquid Molecular Wt. 63.01 Bolling Pt. 181*F $UC/ Specific Gravity (77) 1.50 Solubility Miscible in H* Flash Pt. NA Ionization Potential 11.95eV Vapor Pressure 48mm Freezing Pt. --44F ca PEL NA LEL NA Noncombustible liquid, combustible materials. but increases the flammability of Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 2ppm; MSHA PEL: 2ppm ACGIH TLV:2ppm; NIOSH REL:2ppm 4ppm STEL 4ppm STEL 035703 0594 Acute Toxicity Data\ LD50-, for oral, dermal, inhalation. Include species. Min. lethal dose (human): lOOmg/kg LCjj (inhal.) rat: 65ppm/4 hr LCj, (inhal.) mouse: 67ppm/4 hr IDLH: looppm (Ref: EPA(1985) ) ERPG: Threshold Planning Quantity 1000 lbs. Chronic Toxicity Data: Carcinogenic status: Yes Z No Promoter (epigenetic) and/or Initiator (genotoxic) :NA IARC Classification: NA Other designations: NA Genetic effects: increases the ability of other mutagens to induce structural rearrangements of chromosomes. Mixture of HNOj+NOj+PAH is a known mutagen. NOAEL: NA CAL. Prop 65 NSRL: (Ref: NA ) (no observable adverse effect level) ND (no significant risk level) Reproductive hazard: (inc. mutagen, terat) Yes No X Under review Repro hazard rating A-. Unconfirmed human repro. Studies inconclusive. due to methemoglobinema; hypothesis and possible relation of miscarriage in clean- room employees. Sensitizer: No, (irritant) Non-cancer Rfd: ND End Point: NA Human toxicity data: Nitric acid can be corrosive to the skin, eyes, nose, mucous membranes, resp. tract, gastrointestinal tract, or any other tissue; causes severe GI burns, ulceration, or perforation. Low ' concentrations are mildly irritating; pulmonary edema has been reported. Respiratory symptoms can be delayed several hours after exposure and gastrointestinal effects may be delayed several years. Chronic changes in respiratory function and chronic bronchitis have been reported. Biological Monitoring: Available: X Yes No Blood analysis ACGIH BEI: Yes, 1.5% methemoglobin in blood. C35703 0595 III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: Federal Water Pollution Control Act/CWA hazardous substance. HAP status CAA Effluent/Ambient Standards/established limits in various media: DOT - Guide #44 SARA - Extremely hazardous chemical list TSCA - Chemical inventory Remediation Issues: Reportable quantity 1000 lbs. No RCRA cleanup levels reported. Reporting to Regulatory Agencies Worldwide: Canada/Europe Fate in the Environment: Formed in troposhere by gas-phase chemistry/component of acid rain. Aquatic: elevated levels stimulate plankton and aquatic weed growth. Terrestrial: transport through soil to ground water. A contaminated plume will be produced with dilution and dispersion serving to reduce the acid concentr. Atm: wet and dry deposition mixed with other acids; primarily sulfuric acid. Deposition in W. PA in total rain: 47% H2S04, 23% BOj, 16% HN03+H2so4. Relevant bioaccumulation data: Compund is not known to bioaccumulate. Known toxicity in plants: Acid rain constituent; causes leaf/stem yellowing. Known toxicity in fish: Harmful to aquatic life at low cone.; LCSO (salt H2o cockle) 330-l000ppm/48hr. Known toxicity in other aquatic species: ND on-line. Compound is not known to bioconcentrate in aquatic species. C35703 0596 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA Compound is an indirect food additive for use only as a component of adhesives. CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low-Moderate Significant public activity and/or key topics Vjl ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoringavailable:__________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0597 VII. GLOSSARY OF ACRONYM^: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC -- -- LDLO LDjq LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO -- - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Careinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 => .54mg/m3. level, E-6 = 10-4 or 1 excess cancer For in risk 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY CftmimiTH fry; No established levels Employee: (OSHA) 2ppm; 4 ppm STEL Est. Action: 2.00E-02 ppm (based on safety factor applied to PEL) H:\PLANT\CHEMPRO.WAR C35703 0598 PHENOL \ RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The occupational limits for phenol are based on the prevention of systemic toxicity; the health effects considered include skin and eye irritation and central nervous system, liver, and renal toxicity. Carcinogenicity: Phenol is placed in Group D, unclassified substance, by the EPA weight-of-evidence classification. Skin painting studies suggest that phenol may be a promotor and/or a weak skin carcinogen in mice. Mutaaenicitv: Phenol has been reported to have some mutagenic properties but data is limited. Developmental Toxicitv: Limited data indicates that phenol exposure increased pre-implantation loss and early postnatal death in mice. Another study rats indicated no maternal toxicity at fetotoxic doses. Reproductive Toxicitv: No effects in rats exposed to 100- 1000 ppm phenol in drinking water for five generations and no toxic effects in animals exposed to 3000-5000 ppm in drinking water for three generations. Systemic Toxicitv: Chronic inhalation studies in animal models and human epidemiologic data are lacking for phenol. Subchronic study results were inconclusive. The acute toxicity of phenol is similar in humans and animals regardless of the route of exposure, and phenol is rapidly absorbed via all major exposure routes. In acute poisoning in humans, the heart rate is initially increased and then slows and becomes irregular; blood pressure increases initially and rapidly decreases, and this is often followed by salivation and labored breathing. This progression of physiological events is due to the effect of phenol on the CNS, and death usually results from respiratory failure. Irritation: Phenol vapors and liquid phenol are highly irritating to the skin, eyes, and mucous membranes. Since phenol is readily absorbed via the skin, prevention of contact with liquid phenol is emphasized in the documentation for the occupational limits. Risk Profile: The basis for the ambient air level goal (AALG) is the long-established Threshold Limit Value (TLV) of 5 ppm set by American Conference of Governmental Industrial Hygienist (ACGIH) divided by 42. AALG: systemic toxicity (TLV-based)--0.024 ppm (0.09 mg/m3) 8-hour TWA C35703 0599 cleanUir assurance team CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: PHENOLS CAS#: 108-95-2 Lu PHYSICAL/CHEMICAL INFORMATION: Chemical formula: CfHjOH Common synonyms: Carbolic acid, hydroxybenzene, monohydroxy benzene, phenyl alcohol, phenyl hydroxide, phenolic acid Chemical/Physical Properties Molecular Wt. Boiling Pt. Solubility Flash Pt. 94.1 359*F 9% (77F) in H20 175*F WU Ionization Potential Vapor Pressure ?9.5cC Melting Pt. 85 ev .4mm 109F PEL LEL Specific Gravity 42 ND 1.8% 1.06 Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Obtained from coal tar and the decomposition of organic waste. PSA average HzO cone. = 1 ppb. Mot usually found in ground water; 0-5 ppb found in surface water. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 5 ppm MSHA PEL: 5 ppm ACGIH TLV: 5 ppm NIOSH REL: 5 ppm C=15.6 ppm C35703 0600 Acute Toxicity Data; LDyy, for oral, dermal, inhalation. orl-hmn TDL0=14 mg/kg orl-hmn LDL0=140 mg/kg or1-rat LDj,=4l4 mg/kg Include species, IDLH: 250 ppm (Ref: MIOSH) ERPG: 1000 lbs. Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic) :NA IARC Classification: D Other designations: NA NOAEL: 60 mg/kg/day (orl-rat) (Ref: BEAST ) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) CSF: ND Specific cancer end point: ND Reproductive hazard: (inc. mutagen, terat) X Yes No Sensitizer: Suspect sensitizer Non-cancer Rfd: 6E-1 mg/kg/day (orl-rat-subchronic) End Point: Fetus - decreased weight Human toxicity data: membrane irritant. Known human respiratory and mucous Biological Monitoring: Available: X Yes No ACGIH BEI: Total phenol 250 mg/gr creatinine 035703 0607 III. ENVIRONMENTAL INFORMATION; Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: Effluent concentrations .01-.03 ppm RCRA - Cleanup levels CWA - Listed DOT - Guide 17 SARA - Hazardous Chemical. TSCA - Chemical inventory . Remediation Issues: RCRA Cleanup Levels; Air: ND; water: 2E+0mg/l; soil: 5E+4mgkg. Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent. Fate in the Environment: Primary removal through biodegradation. Biodegradation is rapid in air (2-5 days). Phenol is not expected to hydrolyze rapidly or readily. Phenol biodegrades and hydrolyzes in water. It exists primarily in the vapor phase of the atmosphere. Relevant bioaccumulation data: Phenol bioaccumulates slightly in many aquatic species. Known toxicity in plants: Effects not usually seen in low cone., however, in high concentrations, it may cause stem/root yellowing. Known toxicity in fish: Phenol exists in fish and aquatic species. May cause toxicity in very high cone. Known toxicity in other aquatic species: May cause harm in very high concentrations and bioconcentrates slightly (especially in shellfish). C35703 0602 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR LOW Significant public activity and/or key topics L. ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: _______ IH exposure monitoring _ available: Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0603 VII. GLOSSARY OF ACRONYM^: ACGIH TLV ADI BEI C/Ce (ca) CSF E01 -- - - , ERPG IARC IDLH LC -- -- LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO -- - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Pnmniiin fry; Mon-cancer effects (Adult) 8.75E-02mg/m3 (Child) 2.50E-02mg/m3 BnDlOVee: (OSHA) (RfD=6E-lmg/kg/day) 5 ppm H:\PLANT\CHEMPRO.WAR C35703 0604 SULFURIC ACID RISK SUMMARY; \ Health Effects Profile: Basis for Occupational Limits: The occupational limits for sulfuric acid are based on prevention of pulmonary irritation and damage to the teeth. Carcinogenicity; NA Mutagenicity; NA Developmental Toxicitv: Slight maternal toxicity noted in one study of mice and rabbits exposed to sulfuric acid aerosol. There was not evidence for teratogenic effects or embryofetal toxicity. Reproductive Toxicitv: NA Systemic Toxicitv: The major toxic effect of sulfuric acid is local irritation and since inhaled sulfuric acid is largely neutralized by NH, in the expired air, it is unlikely that significant systemic exposure to sulfuric acid occurs. Irritation: The predominant effects of both acute and chronic exposure to sulfuric acid are its irritant properties. It has been associated with dental erosion, eye irritation, and effects on the respiratory system. Risk Profile: The ambient air level goal (AALG) is based on irritation and the occupational exposure level of 1 mg/m3 divided by a total uncertainty factor of 500. AALG: irritation (TLV-based) --2/xg/m3 8-hour TWA C35703 0605 CLEArj AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: SULFURIC ACID CAS#: 7664-93-9 L. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: H2S04 Common synonyms: vitriol Hydrogen sulfate, battery acid, oil of Chemical/Physical Properties Molecular Wt. 98.1 Boiling Pt. 554*F Solubility Flash Pt. Miscible in HzO MA Specific Gravity Vapor Pressure Freezing Pt. PEL LEL 1.84 (96-98% acid) (295F): 1mm 51F NA MA I0.uc Moncombustible liquid, but capable of iqnitinq finely divided combustible materials. Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: l-10um size function of amount of H20. Toxicity of mist inversely proportional to particle size. is is Ambient/background concentrations: 3.0-16.6 mg/m3 dry day, low humidity. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: lmg/m3 MSHA PEL: lmg/m3 ACGIH TLV: lmg/m3; STEL: 3mg/m3 NIOSH REL: lmg/m3 MAK: lmg/m3 C35703 0606 Acute Toxicity Datai LDjo", for oral, dermal, inhalation. Include species. LDm rat: 2l40mg/kg LCW rat: 510 mg/nr; LCj, guinea pig: 18mg/m3 (most sensitive) IDLH: SOmg/m3 (Ref: MIOSH ) ERPG: 1000 lbs. Chronic Toxicity Data: Carcinogenic status: X Yes No X Promoter (epigenetic) and/or Initiator (genotoxic): With arsenic for respiratory carcinogenesis. IARC Classification: NA Other designations: Suggested C; cause irreversible effects which can be life threatening. Unconfirmed human repro. hazard with mixed exposures. NOAEL: 0.066-0.098mg/m3 (human respiratory effects.) (no observable adverse effect level) LOAEL: inhalation, human 3mg/m3/24 weeks. system (Ref: HEAST ) CAL. Prop 65 NSRL: N.D. (no significant risk level) Reproductive hazard: (inc. mutagen, terat) Yes X No Neither embryotoxic, fetotoxic, or teratogenic in mice and rabbits at inhaled doses producing some maternal toxicity. Rabbits muscloskeletal abnormality in fetal rabbits at a dose of 20mg/m3. Some human data w/Pb as gynecological diseases, miscarriage, abnormal pregnancy. Sensitizer: No. H2S04 is an intense irritant. Non-cancer Rfd: ND End Point: ND Human toxicity data: Acute: irritant/bronchio construction. Chronic: bronchitis reduced pulmonary function. Biological Monitoring: Available: X Yes No ACGIH BEI: No C35703 0607 ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: Public water systems 250mg/l. HAP status CAA Effluent/Ambient Standards/established limits in various media: CWA - Hazardous substance DOT - Guide 39 TSCA - Chemical inventory SARA - Hazardous chemical Remediation Issues: established. CERCLA 100 lbs. No RCRA cleanup levels Reporting to Regulatory Agencies Worldwide: Canada and Europe Fate in the Environment: Major component of acid rain. Aquatic fate: miscible w/H20; reacts with Ca and Mg and increases weathering of Ca from soil/rocks. Terrestrial fate: acid rain corrosion of steel, deterioration of limestone, marble, roofing slate. Atm fate: sulfuic acid aerosol nuclei uptake in droplets. Relevant bioaccumulation data: Potential for accumulation is negative. Known toxicity in plants: possible plant death. Causes yellowing of leaves and Known toxicity in fish: LDj, salt water prawns 42.5 ppm/48 hr; LDj, fresh water bluegill=24.5 ppm/24 hr. Known toxicity in other aquatic species: Lower forms of aquatic species are very sensitive to sulfuric acid in low concentrations. Sulfuric acid does not bioaccumulate, however, and elimination is rapid. C35703 0608 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low to moderate Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: _______ IH exposure monitoring available: Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0609 VII. GLOSSARY OF ACRONYMS; ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjq - LOAEL MEI NIOSH REL - NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level; E-6 =* 10"* or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Non-cancer effects (Adult) 0.10mg/m3 (Child) 0.03/mg/m3 Employees: (OSHA) 1 mg/m3 (NOAEL=.066mg/m3) H:\PLANTvCHEMPROWAR C35703 0610 1,1,2,2 TETRACHLOROETHYLENE RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The occupational limits are set for several factors to prevent discomfort from prolonged exposures resulting in dizziness and headaches. A short term limit is set to prevent anesthetic effects. More recently limits have been established in consideration of possible carcinogenic effects. Carcinogenicity; Evidence from epidemiologic studies is inadequate to evaluate the carcinogenicity of tetrachloroethylene because, although an association between cancer'risk and employment in the dry-cleaning industry has been reported, either the solvents used have not been reported or there was exposure to multiple solvents. Animal studies in mice have shown increased hepatocellular carcinomas from administration of tetrachloroethylene either by gavage or via inhalation. EPA classifies tetrachloroethylene as a group B2, probable carcinogen. Mutagenicity; Tetrachloroethylene has generally produced negative or weak responses in assays designed to evaluate gene mutations,chromosomal aberrations and effects on DNA. However, the epoxide of tetrachloroethylene has been found to be mutagenic and is generally recognized to be responsible for the carcinogenic potential of the compound. Developmental Toxicity: Studies have concluded that the teratogenic potential of tetrachloroethylene is not significant. Reproductive Toxicity: NA Systemic Toxicitv: In humans and animal models, neurotoxicity appears to be the most sensitive endpoint, with renal and hepatic effects generally reported only after prolonged exposure periods or high exposure levels. Irritation: In humans, short term exposures in the range of 100 to 200 ppm have been reported to cause irritation of the eyes and mucous membranes. Risk Profile: The ambient air level goal (AALG) for tetrachloroethylene 1 is based on carcinogenicity. AALG: 10E-6 95% UCL-0.12 ppb (0.86/zg/m3) annual TWA 10E-5 95% UCL-1.2 ppb (8.6fig/m3) annual TWA H:\CDE\CHEMPSUM CDE C35703 0611 cleatIair assurance team CHEMICAL PROFILE FORM DRAFT MODEL CHEMICAL NAME: 1,1>2,2 Tetrachloroethylene CAS#: 127-18-4 I. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: C2C14 Common synonyms: Perchloroethylene, Perchlorethylene. Perk, Ethylene Tetrachloride, carbon Dichloride Chem/Phvsic. Properties Molecular Wt. 165.8 Boiling Pt. 250*F lairc- Solubilitv (77*F) 0.02% in HjO Ionization Potential 9.32 eV Specific Gravity 162 Flash Pt. NA Vapor Pressure 14mm Freezing Pt. PEL --2*F NA - WO Noncombustible liquid LEL NA Physical state in air: In stack: X gas In ambient air: X gas particulate particulate Ambient/background concentrations: Does not occur naturally; groundwater: 27 PS cities=0.6 ppb (median), (0-1.2 ppb range); surface HzO: 154 PS cities=20 ppb median; rain (LA) =21 ppt; air: 557 PS sites=l mg/m3 median; rural: 128 pptr; urban: 0.290-0.590 ppb Sampling/analytical methods: See attached C35703 0612 II. HEALTH INFORMATION 4 EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 25 ppm MSHA PEL: 100 ppm ACGIH TLV: 25 ppm; 100 ppm STEL (A3) animal carcinogen NIOSH REL: CA Acute Toxicity Data: LDjp, for oral, dermal, inhalation. ldm (oral) rat: 2629 mg/kg; LCj, (inhl) rat: 34200 mg/m3; TCL0 (inhl) human: 96 ppm/7 hra. Include species. IDLH: 500 ppm (CA) (Ref: NIOSH) 50 ppm 200 ppm 400 ppm 600 ppm 1000 ppm 1500 ppm odor threshold minimal eye irritation, minimal lightheadedness strong unpleasant odor, eye irr. tolerable unpleas. odor, loss of inhalations after 10 min. intense, irritating odor, eye/resp. tract iritation "gagging" odor, intolerable eye and nose irritation, unconsciousness after 30 min. ERPG: No data Chronic Toxicity Data: Carcinogenic status: Yes X No * Under review by EPA (IRIS). Promoter (epigenetic) and/or Initiator (genotoxic): NA. Under review. Rat embryo cells in culture were transformed to cancer cells when grown with perk. IARC Classification: NA Other designations: NA ' NOAEL: Long-term adverse health effect; 14mg/kg/day (2L/day H20 consump.) (Ref: RTECS ) (no observable adverse effect level) LOAEL: 71mg/kg/day CAL. Prop 65 NSRL: 14ug/d (no significant risk level) C35703 0613 * Reproductive hazard: (inc. mutagen, terat) X Yes No A' repro effects. Chromosome abberations in lymphocytes from exposed workers. Sensitizer: No Non-cancer Rfd: IE-1 mg/kg/day End Point: Hepatotoxicity Human toxicity data: Acute exposure can cause CNS depression/ .liver/kidney damage and death from anesthetic effects. fatigue/ Over exp: headaches/ can cause malaise/ lightheadedness. dizziness/ Chronic: dermatitis/ irritation of eyes, upper respiratory tract irritation, hepatitis, confusion, irritation. Biological Monitoring: Available: X Yes No Monitor liver/renal function and urine analysis. ACGIH BEI: Perk in end exhaled air: prior to last shift of workweek: 10 ppm Perk in blood: prior to last shift of workweek: 1 mg/L; Trichloroacetic acid in urine end of workweek: 7 mg/L III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: Freshwater: acute=5280 mg/L, chronic=840 mg/L, saltwater: acute/chronic 10,200 mg/L, 450 mg/L. Hax. contam. level (^0) =0.005 mg/L. HAP status CAA. Effluent/Ambient media: Standards/established limits in various CWA - Listed DOT - Guide 74 TSCA - Chemical inventory SARA - Hazardous substance list RCRA - Cleanup levels established Remediation Issues: CERCLA RQ: 1 lb. considered hazardous waste. RCRA cleanup levels; soil: 6E+01mg/kg. Reporting to Regulatory Agencies Worldwide: Canadian/European TSCA equivalent. Also water stds. established. C35703 0614 Fate in the Environment: Terrestrial: Perk evaporates fairly rapidly and has low absoption into soil. Aquatic: 1* loss due to evaporation (T,y2=3 hrs-14 days) ; degradation expected to be slow. Atm: exists in vapor phase; some may wash out in rain. Degrades by reactions with hydroxyes. Relevant bioaccumulation data: Not expected to concentrate in organisms or sediment. Known toxicity in plants: No data available relevant to environmental concentration. Known toxicity in fish: toxicity. No data on fish or waterfowl Known toxicity in other aquatic species: No data available relevant to environmental concentrations. Not epected to bioconcentrate. Does not have BOD; unknown effect on aquatic life. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 X CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate Significant public activity and/or key topics ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: C35703 0615 VI. KEY ALCOA CONTACTS:^ R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) ____________________________ (Environmental data/sampling/ana lysis) _' (Ind. Hygiene sampling information) VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = . 54mg/m3. For risk level, E-6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect No significant risk level Permissible exposure limits level Reference dose Short term exposure limit Total lowest dose C35703 061 VIII. RISK SUMMARY Community * Non-cancer effects (Adult) 0.015 mg/m3 (Child) 0.004 mg/m Employee (OSHA) 25 ppm (RfD=lE-lmg/)cg/day) C35703 067 7 \ C35703 0678 mg** <JL^JLkj> * (LtuU* 0CtcfuJi/*+JL 'Ain,h i YA- uj--ju**uH (^ow Jt-e^Si f*~ /<wv^/(U<X r I*!>(?{,{ 0Hq. trnhJ'vi*. Sh/'TWfl " IOjo^/k^ SfEL >^pprr\ * C<VL6.t hoCj^uU^ * \Jft * 7'. /o fr fcOj3Zliiij) k -f+rthMi* (VwjLvudjuHylMu. t\Aii>ptrfJr^ ajW bcA. VKMCesd- IrrtbJyjy* l^sfd QrryJir * * vf$- G-*^ ^utcA-^jc*-- C/4rtt-^ U hiA+9- <A--k/k -Ca/n/ctO JL^ctJ'^ Cirri bJi**'} <L*tL-- (a cA*-\a *-cJU ^u- iy^~> &cctftl* EPfl * Aft Ur . iO E~^fi ^fUi/iJb ^ |*J3*_,3 fk^lh\? IO&"S p&-Ordr p>.hn*J& ~ Us '2* Vr^fnr?* C35703 0619 cleanWr assurance team CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: Hydrogen Sulfide CAS#: 7783-06-4 JL*. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: H2S Common synonyms: Sewer gas, hydrosulfuric acid Oiftm[Vhys"Properties Molecular Wt. 34.1 Boiling Pt. -77*F Solubility 0.4% in HjO Flash Pt. NA Ionization Potential 10.46eV Vapor Pressure >atm Freezing Pt. --122*F PEL 44.0% LEL 4.0% Flammable Gas Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Ambient levels tend to be low; 0.001 mg/m3; pollution episodes; 0.5 mg/m3; as high as 14.3mg/m3 have been reported. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 10 ppm/15 ppm 8TEL MSHA PEL: 10 ppm ACGIH TLV: 10 ppm/15 ppm STEL NIOSH REL: 10 ppm(C) Acute Toxicity Data: LDjo-, for oral, dermal, inhalation. Include species. LCjg rat: 444 ppm LCm mouse: 673 ppm C35703 0620 IDLH 300 (Ref: NI08H) ERPG Threshold planning quality: 500 lbs. Chronic Toxicity Data: Carcinogenic status: Yes X No Compound has not been evaluated for human carcinogenicity potential by EPA (IRIS). Promoter (epigenetic) and/or Initiator (genotoxic) :NA IARC Classification: HA Other designations: NA NOAEL: 3.1 mg/kg/day UFtlooo MF: 1 (no observable adverse effect level) LOAEL: 15 mg/kg/day (Ref: RTECS ) (pig, oral) CAL. Prop 65 NSRL: ND (no significant risk level) CSF: NA Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) Yes X No However, unconfirmed human repro. haz. because of mixed experiments with CSj (known human repro. haz.) ; inadequate dose/response data. Sensitizer: Not listed as a sensitizer; however, reports in the literature of respiratory sensitizations. Non-cancer Rfd: 9E-3 mg/kg/day subchronic RFC 9E-4 mg/m3 (med. confidence) Inflammation of nasal mucosa (mouse sub-chronic inhl study) End point: GI disturbance (low confidence with study); (pig-oral) Human toxicity data: Concentration of 50-150 ppm effect olfactory; 250 ppm causes irritation of mucosa memb. bronchitis, pulmonary edema; 500 ppm causes headache, nausea, weakness, disorientation and coma Humans Range of Toxicity Symptomatic Severely toxic Fatal H2S (cone) 50 ppm for 200 ppm for 800 ppm 600 ppm onset 1/2 hr. 1 min. immediate 30 min. C35703 0621  Biological Monitoring: Available: X Yes No Blood gas ACGIH BEI: No II3U ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) . Emission Standards: HAP status CAA. Ambient HjO quality chronics2.0E+0mg/L; fresh HjO= 2.0E+0mg/L; marine waters are the same. Effluent/Ambient Standards/established limits in various media: DOT SARA - TSCA - Guide #13 Extremely hazardous Chemical inventory substances list) Remediation Issues: RQ=100 lbs. Reporting to Regulatory Agencies Worldwide: Canada/EC Fate in the Environment: atm fate: affected by ambient temp.; at lower temp., increased residence time (1-40 days); soil: chemical evaporates. Relevant bioaccumulation data: Does not bioaccumulate in food chain. Chemical is not known to bioaccumulate in humans or animal species. Known toxicity in plants: May cause plant damage after repeated exposure or at high acute doses. Known toxicity in fish: No data on-line. Known toxicity in other aquatic species: No data on-line. Chemical does not bioaccumulate in aquatic species. C35703 0622 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summaryattached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:VPLANT\CHEMPRO.WAR C35703 0623 VII. GLOSSARY OF ACRONYM^; ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E01 - ERPG IARC IDLH LC - - -- LDLO - LDjo - LOAEL -- MEI - NIOSH REL - NOAEL -- NSRL - PEL - RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: S.AE-Olmg/m3 = . SAmg/m3. level, E-6 = 10-4 or 1 excess cancer For in risk 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Communityt Non-cancer effects (Adult) 1.13E-3 mg/m3 (Child) 3.75E-4 mg/m3 (RfD:9E-3 mg/kg/day) Employee: (OSHA); 10 ppm is ppm STEL H:\PLANT\CHEMPRO.WAR C35703 0624 /i|.I tir-o^A- I icon u \ iqjnnxtuuy UjiMK {U*h> ^rtjrilx> \ * '^(bttyfiJxrrfSL <4*fhds \ S5^i- fjUl&Uu^jhfrikbAJ--JLi^uJl ->/U*6uu> L*,lJlJtto>l &~-$-- fUU^inJT^ . 8h'TWf\: 6-ff(t\ j ST&-'-Upffn . * OlLLCuri^rtici^J. flJfr ' L : PoirhA' j\JwJLhr>r U* tJ^mr9^nr-Jl Ai-Mjt-eJisiua uk a Nft * ^l^t6<LucfldCT^X>cJ^ ! &-ft'-C. (j-tua 3 - tkak- 0 * SlfshJk'ilnrwfo: fljfr * Jlrrihuhii* 1 flu> cLiX-ft*. htfUuMJL. / rrihJitr- ^ f ly* <yd- /^CCpir^Jf^. 4r*d jo rrruU : (/00fiL) jhJL * j-- &^jLu/*vb <Xtf / w h~OA*X & ^5^ trr.f-tijr uufz . ftfyLAr u UiUKuS- tfv Qo '-T^O okuudl UjUmJ~JIjUmSL JrOr' JlUSrAA' pJy-^LuLm - Uio m-wJL u. JLcl^cS. i/y 'K4y>~- tfrruvJ JjUu^M- ML(r ? H)E--fLe> JUjsrJr lihfrtJbu ~ 3'(iB-4 *va, 10--S y-fk*<nJt taJ'invjlL, - 3 i &S~3 C35703 0625 CLEAjfr AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: HYDROGEN FLUORIDE CAS#: 7664-39-3 X. PHYSICAL/CHEMICAL INFORMATION! Chemical formula: HP Common synonyms: Anhydrous hydrogen fluoride Chemical/Physical properties Molecular Nt. 20.0 Boiling Pt. 67*F Solubility Miscible in H20 Plash Pt. NA Ionization Potential 15.98 eV Specific Gravity 1.00 (liq. at 67P) Vapor Pressure 71 atm Freezing Pt. --118F - t'i.4cC Nonflammable gas PEL NA LEL NA Physical state in air: In stack: X gas ' In ambient air: X gas particulate particulate Ambient/background concentrations: Groundwater 1-25 mg/L Sampling/analytical methods: See attached C35703 0626 HEALTH INFORMATION EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 3 ppm; (2.5 mg/m3) 8TEL: 6 ppm; (5.0 mg/m3) MSHA PEL: 3 ppm/6 ppm STEL ACGIH TLV: 3.0 ppm (Ce) NIOSH REL: 3 ppm/6 ppm STEL MAK: 3 ppm Acute Toxicity Data; LDjq., for oral, dermal, inhalation. Include species. LCj, (inhl) rat: 1276 ppm/1 hr. LCj, (inhl) monkey: 1774 ppm/l hr. TCLO (inhl) human: 100 mg/nr/min. LCLO (inhl) human: 50 ppm/30 min. IDLH: 30 ppm (Ref: NIOSH/Davis et al) (Meditext TOMES) ERPG: 100 lbs. Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic) :NA IARC Classification: NA Other designations: NA NOAEL: (rats) .0025 mg/m3 (Ref: TOMES ) (no observable adverse end pt: embryo toxic/teratogenic effect level) CAL. Prop 65 NSRL: N.D. (no significant risk level) CSF: NA Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) X Yes No Repro: Class 3 (irrev. effects which are a threat to life.) Class A (known reproductive hazard; mottling of baby teeth.) Chromosomal abberations in root tips. Some chromosomal abberations in bone marrow cells when inhaled by rats 1 mg/m3/6 hr/l month. Russian study: mottling of baby teeth C3s703 0627  Sensitizer: N^; however, chemical is a severe irritant. Non-cancer Rfd: N.D. End Point: N.D. Human toxicity data: Irritating to eyes, skin, severe burns; respiratory tract irritant. Biological Monitoring: Available: X Yes No ACGIH BEI: Fluoride in urine ACGIH (1992) 3mg/g creatinine/ST: prior to shift lOmg/g creatinine/ST: end of shift T| bone = 8-10 yrs. III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: Safe Drinking Water Act = 4 mg/L; 2nd max cont. = level 2 mg/L. HAP status CAA Effluent/Ambient Standards/established limits in various media: CAA - Listed RCRA - D/34 DOT - DOT Guide #15 SARA - Extremely hazardous substance TSCA - Chemical inventory Remediation Issues: CERCLA RQ=100 lbs. Reporting to Regulatory Agencies Worldwide: Canada/EC Fate in the Environment: N.D. on-line. Relevant bioaccumulation data: Food chain accumulation: none; chemical is not known to bioaccumulate in humans or animal species. Known toxicity in plants: 0.1 ppm for 3-4 hr.; minimal effects if pH > 6.5. High Ca content will immobilize fluoride which can be damaging to plants when present in acid soils. Known toxicity in fish: Fresh water fish - lethal cone.: 60 ppm; harmful: 40 ppm. Known toxicity in other aquatic species: Shrimp LDg, > 300 ppm. C357Q3 0628 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA HP is an indirect food additive for use only as a component of adhesives. Bottled H20 not to exceed 1.8 mg/L fluoride (to which fluoride not added). Bottled H20 > 1.2 mg/L fluoride in water to which fluoride is added CALIFORNIA PROP 65 CONEG .HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate Slcmifleant public activityand/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPROWAR C35703 0629 VII. GLOSSARY OF ACRONYM^: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E+01 - ERPG IARC IDLH LC ~ LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - - - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: SME-Olmg/m3 = .54mg/m3. For risk level, E-6 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Cone, bulk/dust (Adult) 3.5E-3 mg/kg (Child) 4.0E-4 mg/kg Cone, air/vapor (Adult) 3.65E-4mg/m3 (Child) 1.04E-4mg/m3 Employee: OSHA 3 ppm (2.5 mg/m?) 6 ppm (5.0 mg/m3) (NOAEL = .0025 mg/m3 Risk based on non-cancer effects) H:\Plj\KnCHEMPRO WAR C35703 0630 Cs (Lu^ 9 /Jt*UL&Jo frirjjJbu ' ................................-............... * &CuQ-6a ^tfL 0<tAbLO*Jinrd dwtih \ '^-JkL*_jLmib 4M-_ JuunJ^. <ftv %<lwiuh'/h O/^A^<r^7w>ice^^40 > ShfTVjfi - Qju/Ltvn-C\L*H*h ; Nft Ik aJ-n ^vk >tA . Nfi <UCLZ+J. yo QjUliry JLtmiH'Zeoflfitn 4 TlTXiti^ ^ /6/ <c ki<Jn^ to*\ Cojuajl. '$JlCu*-> chv>r*y- \*~JjUjuo &6 '-yyu.yntsruA, JjuLxxbgmo * h` (/^ u*u~ CUoUrif t-o-hi$UA^. hy^UQCtAj &r$-- &JJuJUd yzt> f&c-nMT'l 'JtrhjJr J&r ^l^OuMMuj fcnL&.....- "^KtAstf iUx> * -^'-fttLcJ3^ pr-1/Kt.ry JAjHbrJ^_ <pds , frrjfL-: -jp-Lt* V^o " kii'jufc' CtiV' Go^ *j4oJL C 0 dj^fitdL '-h^nxjk' ^bnLtJ-4- tNto'Q'QS} o-j lQ nujlfy^^ 0^ yiouA, \fMirlL jbiu* ZibAjuJ^ io <b-(oxJL " '(brL 3^'yip/n (*far*gt^ ) -ftf/u~<ri. . ^ <r?to hu*) C35703 0631 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: Carbon Monoxide CAS#: 630-08-0 Xj. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: CO Common synonyms: Carbon oxide, flue gas, monoxide Chem/Physic Properties Molecular Wt. Boiling Pt. Solubility 28.0 313*P lf5t.yt' 2% in H,0 Ionization Potential Vapor Pressure 14.01 eV >latm PEL 74% LEL 12.5% Flash Pt. NA (gas) Melting Pt. --337*F ~2.or>c Flammable Gas Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: ND on-line. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 35 ppm, 2 00 ppm(C) MSHA PEL: 50 ppm ACGIH TLV: 25 ppm NIOSH REL: 25 ppm Acute Toxicity Data: LDj^, for oral, dermal, inhalation. Include species. LCLO mouse: 2000 ppm (inhalation) LCI0S human: 4 000 ppm/hr LCjo rat: 1807 ppm/4h LCjo mouse: 2 444 ppm/4 hr C35703 0632  IDLH: 1500 ppi4 ERPG: N.D. (Ref: NIOSB) Chronic Toxicity Data: Carcinogenic status: Yes X No No studies on carcinogenicity. Promoter (epigenetic) and/or Initiator (genotoxic) :NA IARC Classification: NA Other designations: NA NOAEL: ND CAL. Prop 65 NSRL: CSF: ND (Ref: ND ) (no observable adverse effect level) ND (no significant risk level) Specific cancer end point: ND Reproductive hazard: (inc. mutagen, terat) X Yes No A* for repro; known human reproductive hazard w/no known safe dose. CO levels of 10% or higher can cause brain damage in fetus. Sensitizer: No Non-cancer Rfd: ND End Point ND Human toxicity data: Poisoning causes multitude of effects due to tissue hypoxia and cellular poisoning, atrial fibrillation, IV block, ventricillar arrythmia, headache, dizziness, convulsions, coma, death. Increased carboxyhemoglobin from CO exposure during pregnancy is teratogenic. Mild toxicity (COhb-10-30%). Mod toxicity (COhb-30-50%). Sev toxicity (COhb 50-80%). COhb = carbon monoxide + hemoglobin. Biological Monitoring: Blood; exhaled air. Available: X Yes No ACGIH BEI: COhb end of shift: <8% of hemoglobin; CO in exhaled air end of shift: less than 40 ppm. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: Air quality (NAAQS): 9ppm (10 mg/m3)/ 8hrs.; 35 ppm (40 mg/m3)/l hour. C35703 0633 Effluent/Ambient media: J^andards/established limits in various DOT - Guide #18 TSCA - Chemical inventory Remediation Issues: Ho CERCLA reportable quantities; no RCRA cleanup levels established. Reporting to Regulatory Agencies Worldwide: Canada/Europe Fate in the Environment: Will Disperse rapidly. Relevant bioaccumuiation data: No reported bioaccumulation in humans or animal species. studies of Known toxicity in plants: N.D. on-line. Known toxicity in fish: killed. 1.5 ppm/1-6 hr; minnows and sunfish Known toxicity in other aquatic species: Wild bird LC^ 1334 ppm. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate Significant public activity and/or key topics ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: C35703 0634 VI. KEY ALCOA CONTACTS:' R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) (Environmental data/sampling/analysis) (Ind. Hygiene sampling information) GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 - -- ERPG IARC IDLH LC - LDLO - LDjo LOAEL - - MEI . - NIOSH REL - NOAEL - NSRL - PEL - RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose C35703 0635 VIII. RISK SUMMARY Community: (NAAOS) 9 ppm (10 mg/m3)/8 hour 35 ppm (40 mg/m3)/l hour Employee: (OSHA) 35 ppm 200 ppm (C) C35703 0636 <ba \ * lia-Od. Prr^JU>: ' &<LaZ bctu-p/J-ts)*' U JtiL> ^0/ 4W- pJ*^n4^\r*^r. * O.CiALifi*\g/t'tt,lj^ Nfr * W\u.k-tLr\ tCtf^ *. ST^ tViXw.tH'j * *|^l^<J^rpfA^i-*i. *t~IXtfc<^ t kJft Up^jLA*- ft^lk SM- jb*-ci-er' jUvu-b+Jr <2> fv 4.n^J/n3 i A/y w I(U^'t~tAjUL^ *JU T~faILi(j *. AJ ^m^UatLu rwt&b> * SifS-flmi'q 'fgycr^! P/uaroiii * ^rn hyb/L : 4-yt- iW~Juu>fnrtJ-nj. f-*Jn/Y' /ut PtTjJiy t * H^-*--' h* (xir^j^ftS-e^bcO- (j^PiCC-'^ <v yJt/>*-C^tficfrO jU/v> tfu OfhnrLdLd^ gU<L^4tgTfc * A|*VUtr T /0 Judrtrr+Ai. --O, o?-5 fO >^Wr MhtrtJz, - j 0^3 <t'-^- C35703 0637 f' CLEAlj AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: FLUORIDES CAS#: 7681-49-4 I. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: NaF; Na,AlF( Common synonyms: Sodium fluoride/ cryolite, sodium aluminum fluoride Chemical/Pvsical Properties NaF/Na-AlF Molecular Wt. (42.0/209.9) Boiling Pt. 3099F/decomposes iqc6&c Solubility Flash Pt. 4%/.04% NA/NA in h2 Ionization Potential NA/NA Vapor Pressure 0/0mm Melting Pt. 1819/1832F PEL NA/NA LEL NA/NA Specific Gravity 2.78/2.90 Noncombustible solids Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: on-line. No data available , ^--------- Ambient/background concentrations:'. No data / Sampling/analytical methods: See attached C35703 0638 II. HEALTH INFORMATION V EMPLOYEE AND COMMUNITY; Exposure Limits: OSHA PEL: 2.5mg/m3 MSHA PEL: 2.5mg/m3 ACGIH TLV: 2.5mg/m3 NIOSH REL: 2.5mg/m3 MAK: 2.5 various countries European/Canadian Acute Toxicity Data: LDaj., for oral, dermal, inhalation. Include species. Human lethal dose:- (oral) x 50 mg/kg LDjg (IV) mouse: 22800 ug/kg IDLH: SOOmg/m3 (Ref: NIOSH ) ERPG: No data Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic) :NA EPA genetox prog. 1988: negative IARC Classification: Other designations: D NA; some evidence of animal carcinogenicity; DNA inhibition of mouse fibroblast at 130 ug/L (cell culture). NOAEL: 2.5mg/m3; optimal daily dose to prevent caries: o.05-0.07mg/kg/day (Ref: Repro Tox ) (no observable adverse effect level) CAL. Prop 65 NSRL ND (no significant risk level) CSF: NA Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) Yes 1 Current status is under review. X No Many arguements to classify as B (known repro. haz. where no effect level is known). Specify end point: Mottled teeth Sensitizer: No, irritant effects only at high concentrations. MDR (max. daily requirement) : 1.5-2mgin pregnancy to prevent caries. C35703 0639 Non-cancer Rfd: No^data End Point: No data Human toxicity data: Dental fluorosis, fluorosis from well water. Acute: ENT. irritation at 60ppm . Dndissociated fluoride is absorbed into the body more readily than fluoride ion; I20ppm highest level for 1 min. tolerated. Biological Monitoring: Available: X Yes No ACGIH BEI: Fluoride in urine (ACGIH/1992) 3mg/g creatinine/ST: prior to shift lOmg/g creatinine/ST: end of shift Tw bone=8-10 yrs. ' III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in.I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: DOT - DOT Guide #54 SARA - not listed TSCA - Chemical inventory Remediation Issues: CERCLA - not listed; no RCRA cleanup levels listed. Reporting to Regulatory Agencies Worldwide: Canada/Europe Fate in the Environment: No data found on-line. Relevant bioaccumulation data: Not known to bioaccumulate at low levels. Dairy cattle are sensitive to 40 ppm in the diet. Known toxicity in plants: Causes damage to vegetation at high concentrations. t Known toxicity in fish: Noudata-..ound on-line. Known toxicity in other aquatic species: No data found on -line. J i* C35703 0640 COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low to moderate Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0641 VII. GLOSSARY OF ACRONYM^: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = .54mg/mJ. For risk level, E-6 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest possible dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: cone, bulk/dust (adult) .07mg/kg (child) .0075mg/kg cone, air/part. (adult) .0073mg/m3 (child) . 0021mg/m3 Employee: OSHA 2.5mg/m3 (opt. daily dose = 0.05mg/kg/day for non-cancer effects) K:\PLANT\CHEMPRO.WAR C35703 0642 UjOLL&tTVjjC SiAJLj^JjU ` tittd&h tf^uh Prr^Jbu ` a DtejufoJinMj&tmth : a if.. J$y{....(hn ' i / *^ni t Jygrfi J -- timCMjZf-hirV^ yJ<n*/Ps l*~ JL''*H' i-l--a**--- Aju^ jCjuA/UsizbO} -Xr-tvf y>f / u. w ^ /tst~&fUrr> ft. Curapt+r^'ird**** dhrfHA- /t>flu. f*' * QAAb'ir'o^Au c,iip .' AJft * /tVUcJ-A-Vnic/?j S KJh ' 'p^rdL^b^KnJU tTrxit.11^ : AJ/V ' ft pr^oduA.cd'tr-cs ~ftnecj1fr A/fl- ' *}-/* c> i~fX/ait^ ' /h(JJj -hxic viL /hhJtJ-n, ^-Vu.htcjA Ovc*tfr*J">* ` _ fl-rrtf>~4~lrr* } {txjptrAJ'rvtj 'J-rrrhJ'isr- f~Usr-*^t /u^JL Q& QA*jt*uh^*Jinio . * frtyhj'. * S^>- o**J~uiJZ curJLuf-J^ CAAt't') OttUf aJictaL, JLj^uA, ^/jkicA JbuxJL n0 XqCfitAjtfKMfdhJL. ^ <kjLnA+ltk~ ff~v>y' h-* IHyaJlAM-c^-- C^mCjuO J-^pt/tlo * ftft ^iv- ** ^^-*3.0 t*-^}fK? C35703 0643 CLEAN\\IR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: CARBONYL SULFIDE CAS#: 463-58-1 I. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: COS Common synonyms: Carbon monoxide monosulfide Chem/Phvsical Properties Molecular Wt. 60.07 Melting Pt. -138*C Freezing Pt. --138#C Boiling Pt. Specific Gravity LEL -50.2'C 1.028 12% -58,3`F Solubility Soluble in H20 Reacts with oxidyzing mat./HS released When heated, releases CO PEL 2.1% Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: 3 6-51 ppt by vol at alt. 15.2-20 km over N. Calif, (natural sources production far exceed anthropogenic sources); urban/suburban: 0.27-0.80 mg/m3 rural: 60-180 ppt; Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: ND MSHA PEL: ND ACGIH TLV: ND NIOSH REL: ND Hungary/Russia: 10 mg/m3; 2 0 mg/m3 STEL C35703 0644 Acute Toxicity Data;' LDjo-, for oral, dermal, inhalation. Include species. LDm (Ip) rat: 23 mg/kg LCLO mouse: 1200 ppm/35 min LCLO mouse: 2900 ppm IDLH: ND (Ref: NIOSH ) ERPG: ND Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or Initiator (genotoxic) :NA IARC Classification: NA Other designations: NA NOAEL: ND (Ref: IRIS ) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) Reproductive hazard: (inc. mutagen, terat) Yes X No Sensitizer: No Non-cancer Rfd: ND End Point: ND Human toxicity data: Xildly toxic via inhalation. It is a narcotic in high cone., irritant to lungs and'^rachea. It is a CNS depressant/can be fatal by paralysis of the resp. system. In humans, COS is hydrolyzed by HzO to C02 and HS. Toxic symptoms and death ascribed to HS. COS has less prominent local irritating and olfactory warning properties than HCN. Biological Monitoring: Blood Available: X Yes No ACGlH BEI: ND C35703 0645 III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: DOT SARA TSCA -- Guide #18 -- Not on extremely hazardous -- Listed substances list. Remediation Issues: No RCRA cleanup levels established. Reporting to Regulatory Agencies Worldwide: ND on-line. Fate in the Environment: Oceans release into atm a significant fraction of yearly global emissions (volcanic sulforous H20) (8xiOn g/yr); long residence time 200-7300 days. Terrestrial: high mobility/rapid volatilization to atm.; Aquatic: rapid volatilization to atm. 1-1/2 - 2 hours. Relevant bioaccumulation data: Plants may be a major global sink for the gas. Will not bioaccumulate in fish/aquatic system. Known toxicity in plants: No data found on-line. Known toxicity in fish: No data found on-line. Known toxicity in other aquatic species: No data found on line. C35703 0646 / IV*. * COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low to moderate Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) C35703 0647 VII. GLOSSARY OF ACRONYM^: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = . 54mg/m3. For risk level, E-6 = 104 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure Total lowest dose limit VIII a. RISK SUMMARY (fBased on ^j^values - No published or established Rfd or ADI.) Worker Community /# 0 -- e?. C35703 0648 ^tht^OJdnUTuMJb . Bcun.ma^ \ ......................... ' HuJUL tfjcJb fr^du : * @HLaiji \jJis OfUjUujO^l^iJi, ^-1 rftApp ' **M Gaj-* <-$. 0*J ^lAjLiAJbd'li<v* fj~ ( JVydl^K&rJ& 7tX+jLL&44K*-) jZ-> ^Xc->^d~tVr~ir>. JLuMj ^ yL/HS<*4C4iA*cd- frnrjL & 'KqJ ^5^.*}? CJdUiu>v**-s f dQ^ca*u>\jiP/ uAj B&aaJL **' A -Iv-bL^Jt: - A*juLtit~o~> ^JjD^My^Ccdt'-r- j dtS ^ (U-~ fd&cdl t Juvryt&yj CXvuwij2^*d * / tttj , dt'fYr**<*eYir+$-.--' 6d~6-C<^{d'{tfaa ^Usj&^dod U*dP~ S-Xdus 'foAJP JLlg>4i/*io a^JLjtv tihtAA*/)^f4'50 Mh^itJz., * 1 AJfl * {U&MrAjJAjh^ 7W t ul^yj. PtfLSVb ^ '^JyJL . P- S'/O ^UJ fUjua^LLid fc&U-to^/H>o . ' r-S/^/1 f7s*v< lA. I nC-j.^/j ; <fin<^ohnr\ 4*-,/ MW v jLcoy~^ * '^rnbJ'trf' : -U 7u^ try^u^r^Ji^ jUjUtxJL Pt hs CUyryJLd^*-'. ~j^n*V. $ 6*C/hJ-- PjuX, Pr -f-<yX>-' CvfaM-iAT^Jz &MJ^p-rdl-- CPP^hLjy^ PQ-B^O J*a P%yCjL-^-' P^KA^tAyro^/>a 1 ^1 ^) fl/r^-~ *9 cOsttp-P ^X.aw> PZPft-^ (2. SP~ ^K^/-- dnM^rr/Jd JlPwp^a ^ C<A^f f~fJfPP- jOBcU^ /L^J-Kpc* (fr&J- pftl-Lr'u * /Ojtl~ C> ^Xt~orCC jLt/v rv\fjj^ ~ / D B- ~J/yd , ' l()P'5 -fX^-Crd pl^ttr-tdd " /* & - ~<J- )hi^ ( ftS>'C&LCt?c^U-u>d Jhd-iffvdz /. 4^ P-~~2. (*- )b ^rliK^cat- ) C35703 0649 cleanWr assurance team CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: POLYCHLORINATED BIPHENYLS CAS#: 1336-36-3 X*. PHYSICAL/CHEMICAL INFORMATION; Chemical formula: (C^)] Cl, based on mixtures) (no definitive formula; range Common synonyms: PCB's, Arochlor 1260, 1016, 1221, 1232, 1242, 1248, 1254: Chlorodiphenyl, Phenoclor, Chlophen, Kanachlor Chemical/Physical Properties Boiling pt. 385-42 0C Henry's Law Constant . 007/atm-m3/mol '136- W Solubility Specific gravity Sol. in solvents; 1.566 .080mg/l in H20 Log Koc Melting Pt. 6.42 31SC SI .31- F Vapor Pressure 4.50xl0'smm Moleuclar Wt. Ave=3.70 Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: No data on-line. Ambient/background concentrations: Blood Bkg=200ppbest. mean. PCB's are not naturally occurring substances. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: ND ACGIH TLV: lmg/m3 TWA for 42% chlorine; .5mg/m3 TWA for 54% chlorine NIOSH REL: .001ug/m3 (10 hr. TWA) MSHA PEL: ND C35703 0650 Acute Toxicity Datal LDjo-, for oral, dermal, inhalation. Include species. orl-ms-LDjaSlSOO ug/kg IDLH: ND (RefrNIOSH ) ERPG: ND Chronic Toxicity Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B2 Other designations: ND Maximum tolerated exposures OOug/kg ADI = .0035mg/l Est. carcinogenic potency=7.7mg/kg/day`l NOAEL: .Olmg/kg/day (Ref: HSDB ) (no observable adverse effect level) CAL. Prop 65 NSRL: o.lug/day (no significant risk level) CSF: ND (IRIS) Specific cancer end point: ND (IRIS) Reproductive hazard: (inc. mutagen, terat) X Yes No Reports of fetal injury. PCB's are suspect teratogens. Mutagenic activity induced in cytochrome P450 extract. Sensitizer: No Non-cancer Rfd: ND (IRIS) End Point: ND (IRIS) Human toxicity data: Major routes of exposure by food, drinking water inhalation - human studies of malignant neoplasms stomach, liver, lung, and lymphoma. and in Biological Monitoring: (Blood serum) Available: X Yes No ACGIH BEI: ND C35703 0651 ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient media: Standards/established limits in various RCRA - Cleanup levels CWA - Listed TSCA - Chemical inventory DOT - Guide 58 SARA - Hazardous chemical Remediation Issues: RCRA cleanup levels: water: 5E-06mg/l; soil: 9E-02mg/kg. Ambient water quality criteria: .OOlug/1. Recommended soil levels; less than lOmg/kg; ambient water level recommended at .79mg/l. Reporting to Regulatory Agencies Worldwide: regulations. Europe, Canada Fate in the Environment: PCB's are environmentally "cycled". Persistence increase with increase in chlorination. PCB's will generally not leach significantly in aqueous soil systems. If released to H20, adsorption to sediment is fate process. Relevant bioaccumulation data: PCB's readily bioaccumulate in fat tissue of humans and animals. PCB's are resistant to biologic degradation. Known toxicity in plants: Plant cultures exposed to PCB's had significantly decreased mitotic activity. Known toxicity in fish: PCB's bioconcentrate in aquatic organisms. have been shown to Known toxicity in other aquatic species: Criteria to protect fresh water aquatic species = . 014ng/l; for salt water species = .030ng/l. Eggshell thinning reported in birds exposed to nanogram levels of PCB's in soil and water. C35703 0652 Hi. COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA Z Tolerances have been established for FCB's in milk, poultry, meat and fish. CALIFORNIA PROP 65 X CONEG HAZARD COMMUNICATION X WORLDWIDE . X Canada and European TSCA equivalents set. EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR High Significant public activityand/or key topics V*. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) > (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANTiCHEMPRO.WAR C35703 0653 L VII. GLOSSARY OF ACRONYM; ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC - LDLO LDjq LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO -- - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 * .54mg/m3. For risk level, E-6- = 10-4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect No significant risk level Permissible exposure limits level Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: No established data available for quantative risk assessment Employee; (ACGIH) lmg/m3 for 42% Cl2 .5mg/m3 for 54% Cl2 Estimated Action Level: 1.00E-03mg/m3 for carcinogenic effects 1.4E-02mg/m3 for non-carcinogenic effects (Based on est. CSF=7.7mg/kg/day'1 and ADI=. 0035ppm) H:\PtANT\CHEMPRO.WAR ^35703 QtcJL Tur 'Pitch YtAuhiPb ficrPt) 3j*y> A $ialn*6/j YHyfrJrA, * * {h-fidjCk Pr^fdUs \ 4 &tLaJ*> AtAs Po/MjXjJ i/*jjL rd/fKob t . &AA,. ^ jtUu ^PLM*4snh*>K ^ J/tOLLaaP^ p^CoXj t/y '^r\uAhtr>r^h (/bot fikrThjft* O'Zn^lnt3 OtTP)/') ................................................" * C/flAjPj/* *<^1 {11^. * &Ha<Jt *** A '-Ifr-Lt^Jb-~^~- ~&JI <h^\CU Ch^A4<(^C^hjK)^ Cnt -kP '-ydt(J\ VthtJ-i JLl LaJ '^oioyv ft ^yit-ru-' pPiM, *+- /Ah*, ^WUyC) & 2 ^>y2W^M- Vu^HceW CA/vtUs/O^y** pHAImo ....... * fhuJ'iuynt&ttj '. Poit^lA, thtvrjf* Tl^ZZJL, /?iA>?n/?v/o^o tULCDufi) CLaj) <y /LijShs^ ............................. ................... * . TfX/Z/Aj l AJft ............ _. . ' PtjnArft(<rht ' ' T^'fthj . PoSiTtv^O yu^tXO^pLu^J^o /thvOsr-*^ /LtfLrvfccJ^ 4 ~hyEtL4vuJ'> ~Tsx i f (i Ct p i{ ^/y~h e*p (jUu-, jhP-h>. ^Oo - C*yjLy'-^t yd^co <WtK~ cUaSYt^JL (utj^cb 'QatJL hTptULuj * pJ'd -h^h*y\ * f irP./v^- ft^rAoA/r- jL/iAobdhr^ _________________ _________________________ PjaAj 'fi^f^iJjLf t . * fks-~ tA^ihjb UMj JjudL- ATP T i &+P X> (Vl aA Ca/TO 1C l/j Q/y$-- to d-tAtoirth- ^`'L'Vv. -hhx^ APP" CA^Cl^O tl+pju- fafrvhi- L'CSp). > hftHr i '^P^J't(yjttiu j/ikjio) '-CftPrJ' lohn-^to ' 6-~8 * / 0 ti y-M/rJC CaT>T'^Ac. ' (fi` Ap~ ? /*^/^tvP C35703 0655 CLEAN^AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODE!, CHEMICAL NAME: COAL TAR PITCH VOLITILES as BENZO(A) PYRENE CAS#: 50-32-8 I- PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Common synonyms: CTPV Chemical/Physical Properties (Yellow needles - monoclinic/orthorhombic) Molecular Wt. 252.3 Boiling Pt. >360C(760mmHg) Melting Pt. 179-179.3"C Specific Gravity 1.351 Solubility Miscible Flammable/combustible solid Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: No data on-line. Ambient/background concentrations: Drinking water 87% + in 15 DS cities; atm=0.l-2.1ppt. Sediment marine/estuarine; 8 sites (0-3030ppb) ; Atm cone. Il-I7km from atm=2-4ug/m3; rural: 0.062.35/m3 (Norway); urban-rural: 0.19-.09ug/m3 (Norway); 0.lug/m I13ug/m3 (Detroit). Sampling/analytical methods: See attached. IL. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 0.2mg/m3 as CTPV MSHA PEL: 0.2mg/m3 as CTPV ACGIH TLV: A2 carcinogen - as accurately detected. NIOSH REL: 0.1mg/m3 as CTPV low as C35703 0656 Acute Toxicity Datar LDjo-, for oral, dermal, inhalation. Include species. XiDj, (mouse IF) 250mg/kg IDLH: 700mg/m3 (Ca) (Ref: NIOSH ) ERPG: Mo data Chronic Toxicity Data; Carcinogenic status: X Yes No Positive in prokaryotes and mammalian cell assay systems. Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: A2 Other designations: NOAEL: 4.5ug/day probable human carcinogen (ACGIH) -------(7 I Dl (Ref: HEAST ) (no observable adverse effect level) CAL. CSF: Prop 65 NSRL: 0.06ug/d (no significant risk level) S^E+lmg/kg/day'1 (orl-rat); 6.1E+lmg/kg/day'` (inh) Specific cancer end point: Forestomach; resp. tract tumor Reproductive hazard: (inc. mutagen, terat) X Yes No Sensitizer: Yes Non-cancer Rfd: ND End Point: ND Human toxicity data: Fire may produce irritating gases/Probable human CA. Resp. tract/skin/bladder CA Biological Monitoring: Available: X Yes Coke oven std: medical surveillance Drinary cytology 8putum cytology No ACGIH BEI: ND C35703 0657 ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: Safe dringing water level = Omg/L (proposed); Max contaminant level = 0.0002ugL (proposed). HAP status CAA. Effluent/Ambient Standards/established limits in various media: RCRA CWA CAA DOT SARA TSCA - Listed Water and fish consumption 2.8E-ug/L Fish consumption 3.llE-2ug/L - Proposed stds. - Guide 39 - Extremely hazardous chemical - Chemical inventory Remediation Issues: CERCLA reportable quantity 1 lb. Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent; also water stds. Fate in the Environment: Adsorbs strongly into sediment. Not expected to appreciably leach into ground water. In aquatic environments, it will absorb into sediment and particulate matter. Atm: associates with particulate matter; long transport. Reacts with 03 and N02 to form hydroxyl radicals. Relevant bioaccumulation data: Expected to bioconcentrate in aquatic organisms that can not metabolize it. Known toxicity in plants: No data on-line. Known toxicity in fish: Acute LEC: 3.0E+2ug/L represent PNA as a class (lowest effective level) for fish species. Known toxicity in other aquatic species: May bioconcentrate in certain species. No data on-line. C35703 0658 EU COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 X CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR High Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0659 VII. GLOSSARY OF ACRONYM^: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO -- - - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Careinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = . 54mg/m3. For risk level, E-6 = 10"4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: (for risk level of E-6) Employee: OSHA 0.2mg/m3 cone, bulk/dust: (adult) 5.87E-7mg/kg (child) 6.29E-8mg/kg cone, air/part: (adult) 6.2x10"* /iujl(CSF=5.8mg/kg/day*) (child) 8.7x10^^/^ cone, air/yapor: (adult) 6.2x10'* (child) 8.7x10V/V H:\PLANT\CHEMPRO.WAR C35703 0660 HMitt Zjyj^jJh Pt-t^JLx,; ...... * QcjtUo Otiitf/J'ijyjJL l^iyJo .* OaJU H* fijJt tMutji^inrfiJLs JLtniik, -4/u Jo JlyMot sr 'Huu Jiu^vwr^dtX- ^ /H^6^vU*vX JLa^Jl chq&jfbsrTJ'isr* W\d- J/rtAjU^dL-^ Jlu^Uj tAwOtd. '~tbc* jLwAAfc, Jm Jh*4*d- cvl /X^U^r~tJt Yblifilo HsyJL Jjru^ A/^/7^2. (!j.L*yrbwd-d~ J-ioftA. " {Ul'Z- 'Of nr^lrnd, ' CoAtcnoyjntC'/h^ _! . QJUuzO >/jbcd'ltT' J &cu^JL ^ P^ 3Ptf -MlicUn^u^ Jo -flJklstuJL Jar-s Jjbu C^TUjj /3 3.^ ^oc-tbjlu . (IctA-CutU)^^ jzjJnXuo . ' ^ 'Puys*. a, /j ; ^ ' \ A)R * P-l^iM<Lu^J\fXs ~tofi/Ct^ ', A)ft _ .. 4 .- rp( iC't^ * . -JJtdvs^j. J/Py^ ' pm Aj~inri 700- *h/A CUijL* I/>~ /vuX^ _.^>d^y24-ca^M-C' . . ........................................ ^LoJb .fr-t^Jbu l * fth- CunrdjLjjPz Cut' jic***JL~ ^V?3L.( isJsn^t^jJ 10 Jj^Loud-- ty^> ftosit-tyr^o ^-/y*'Qsi-tJ- 1/0 6L(A^t <AtS^ 'j/Ltry^- yCl^*-' JjJIrlrha b*}-' (L&'tv'OaJ '^JLo-^IX' ^ 11/ Ct- S f~ y . ' iPfi-1-^ ^* Jp_-(j? pwt: JkJi rr-bJl--' -~ J J- <J & - eft /K^ jf. 3 //X/ ' jia-birrvi^, ' 6b> C35703 0661 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: HEXACHLOROBENZENE CAS/?: 118-74-1 I-r. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: C/Jl* Common synonyms: Perchlorobenzene, anticurie, buntcure Chemical/Physical Properties Molecular Wt. 284.8 Specific Gravity 2.04 Melting Pt. 231#C Boiling Pt. 326C ^.rF Solubility Sol. in benzene; insol. in H2o Physical state in air: Log Koc 3.59 In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Not naturally occurring and formed as waste product in the production of chlorinated hydrocarbons. Drinking water cone. ave.= .lppt. Soil (37 states) = .01 ppm average. Atmospheric cone. (USA) = .001.024 ppt. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: No stds set MSHA PEL: ND ACGIH TLV: ND NIOSH REL: ND C35703 0662 Acute Toxicity Data: LDjq., for oral, dermal, inhalation. Include species, orl - rat LDg, = 3500 mg/kg orl - cat LDjg = 1700 mg/kg orl - rabbit LD^ = 2600 mg/kg IDLH: ND (Ref: NIOSH) ERPG: ND Chronic Toxicity Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B2 Other designations: ND NOAEL: 0.08 mg/kg/day (Ref: HEAST) (no observable adverse effect level) CAL. Prop 65 NSRL: 0.4 mg/day (no significant risk CSF: 1.60E+0 mg/kg/day'1 level) (inh; rat) Specific cancer end point: liver - tumors Reproductive hazard: (inc. mutagen, terat) Yes X No No observed effects in limited animal studies and bioassays. Sensitizer: literature. No reports of sensitization reactions in Non-cancer Rfd: 8E-4 mg/kg/day End Point: Liver effects (inh - rat) Human toxicity data: The compound is possibly carcinogenic to effects dominant end point. Chemical under review. humans NIOSH and liver USEPA Biological Monitoring: Available: Urine and blood methods available. X Yes No ACGIH BEI: No C35703 0663 ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient media: Standards/established limits in various TSCA listed. No specific requirements, only performance literature search for RCRA & CAA. objective in Wastewater effluent concentrations ranged from X to 220 ppp. It has also been detected in fly ash from municipal refuse incinerators. Remediation Issues: no CERCLA R.Q.'S. No RCRA established levels for cleanup; Reporting to Regulatory Agencies Worldwide: No data.found. Fate in the Environment: HCB is a very persistent chemical. If released to atmosphere, it remains in vapor phase for long periods of time. Long range global transport is possible. If released to H20, it will partition from the water column to sediment and suspended particulates. Volatilization from H20 is rapid though. If released to soil, it is fairly resistant to leaching. Relevant bioaccumulation data: HCB bioconcentrates extensively in a number of fish and intervertebrates and water plant species. HCB bioconcentration has been reported in humans. Known toxicity in plants: Highly toxic to plant life - related to lindane, concentrations of 1-5 ng/gr was detected in fallen leaves. Known toxicity in fish: At high levels > 30 ppt, it is highly toxic. Detected in freshwater fish at .001-3.4 ppt. Known toxicity in other aquatic species: HCB is suspected of being highly toxic to aquatic species due to persistence and bioaccumulation. C35703 0664 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS; FDA X Regulated in food products. CALIFORNIA PROP 65 X CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: _______ IH exposure monitoring available:_________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANTYCHEMPRO.WAR C35703 0665 VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 - - ERPG IARC IDLH LC - - - LDLO - - LOAEL MEI NIOSH REL - - - NOAEL NSRL PEL RfD STEL TDLO/TCLO - - - - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10"* or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect No significant risk level Permissible exposure limits level Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community Cancer effects (for risk level E-6) Adult 2.22E-07 mg/m3 Child 3.17E-07 mg/m3 Non-cancer effects Adult 1.67E-04 mg/m3 Child 1.67E-5 mg/m3 (CSF=1.6E4-0 g/*g/day`) (Rfd=8E-4 mg/kg/day) Employee No set limits Est. action level = 1.67E-01 mg/m3 (based on LDj, and NOAEL) H:\PLAKHCHEMPRO.WA R C35703 0666 Suytyjyri^CLfu^ \ * idt&lck; tj^jujjfco ftr^t-^\Jju i &t<u<, ^VL- OcujLfuJ'tfKfS-' *//&_ oCUu^Ajhin^^ JilWUst JrU^iJL O* eXL^rux^ M^Jx^T/qJ CisrdL !*-*--> _ _ cLt^Luj-d-- j^xw~' (ytC^L-. fixfodSjio C 'Tcvk ' (so-v^la-1/^ i . bKAM^p^tsr-tJL^ Jx/yr^fc. - ,OT)XS /n^( ' &. t r ^J-< r*u; KsJb <LttJL- -- i/}ua/iir<^ cLuxJ[ J^umJL ^ 10 *' 0liAtOm-QC^n KL,I^ .' K)A IkUAJ-tX^xn >t / fj ; AJ^_ ' ~^Of-tsf ^ O^Vtlc- '^il-Qbuj-J-<(j- . . \^j+s<st^(strrJsy\. h.tr-+J~inr' h^Lut^l^ chcuiii y '^^svs~ixy(x^ jKAXXxJ^f) Xxy> Oxf>\ du i Ir~l-> ... ~T<f^/tt ^ ! CAi^o ft~. '^'X'C/^ ) fafi-l mxi - rrtc, j T# i <^ .' r-6U h,ir~ cLuwtu<^> P^flsxhlL jftdLfayisoy-j .QMAjyu*> /fr/ hJ'tfc ' i rri hJ~i^ jwUA^h^ ^ '-^ryurf& cUoz&. . -l _________________________________- ... _cJftsv^ux f^Last-x jf^r-t^xjtix '. y> * v^M-c- (ft ,f? .... r dAr>gO 6*n.L <0 ^4Ux*^,, ^V)n. A*JL..S**xCl...................... jlh^LCto sj^dLulsie. JU^fto; k.i/' 6-kodl ^ J^Uxx^^ -OjyfruM 1 fiftjft? - iuJLM. - . 6o<?Sh^lJl^. j \ftir - lO/vu^lr\ft. C35703 0667 CLEAN! AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: BISMUTH CAS#: 7440-69-9 it PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Bi Common synonyms: ND Chemical/Physical Properties Molecular Wt. 209.98 Boiling Pt. 1500C Specific Gravity 9.78 Melting Pt. 271*C Solubility Sol. in NO3/HCI/H2SO4 Insol. in H20 Siq.SF Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: Mo data on-line Ambient/background concentrations: Earth's crust=0.2 ppm; bismuth ore found extensively in South America, China, Korea, S. Africa, Australia. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: ND MSHA PEL: ND ACGIH TLV: ND NIOSH REL: ND Acute Toxicity Data: LDjo-( for oral, dermal, inhalation. Include species. LD;e (oral) rat: .025 mg/kg IDLH: ND (Ref: NIOSH ) ERPG: No data C35703 0668  Chronic Toxicilv Data: Carcinogenic status: Yes X NO Promoter (epigenetic) and/or NA Initiator (genotoxic): IARC Classification: D Other designations: NA NOAEL: N.D. CAL. Prop 65 NSRL: - CSF: ND (Ref: N.D. ) (no observable adverse effect level) N.D. (no significant risk level) Specific cancer end point: N.D. Reproductive hazard: (inc. mutagen, terat) Yes X No Class B- repro. (Limited data for animals, no quantitive human data available.) Sensitizer: No Non-cancer Rfd: ND End Point ND Human toxicity data*: Some mental changesnoted; nervousness, blood changes, lymphocytosis and bone marrow depression following prolonged inhalation of bismuth dust. No acute studies. Blue black line on gums, bad breath, stomach irritation, nausea, loss of appetite and weight, liver and kidney damage. Biological Monitoring: Available: X Yes No Blood and urine ACGIH BEI: ND III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA Effluent/Ambient Standards/established limits in various media: TSCA - Chemical inventory Remediation Issues: No RCRA cleanup levels established. CERCLA reportable quantities. No Reporting to Regulatory Agencies Worldwide: European TSCA equivalent Canadian and C35703 0669 Fate in the Environment: Bi appears in quench water system after removal from core during gasification by Hygas process. Relevant bioaccumulation data: Bismuth is not reported to bioaccumulate in human or animal species. Known toxicity in plants: M.D. on-line. Known toxicity in fish: M.D. on-line. Known toxicity in other aquatic species: N.D. on-line. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA Z CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low Significant public activity and/or key topics ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: C35703 0670 VI. KEY ALCOA CONTACTS: : R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) ________________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 - - ERPG IARC IDLH LC - * LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD ST EL TDLO/TCLO - - - - - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E--6 = 10"4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose C35703 0671 VIII. RISK SUMMARY No established action levels because no set CSF, NOAEL or Rfd. Estimated action level for community and employee .0025 mg/kg (based on safety factor applied to rat LDM) for bulk. Air not to exceed nuisance level of 10 mg/m3. c35 703 0672 QuJU Ju*nwyuL^.-* Ihkl ^Cffui, Pr^pJL; .......................................................... * fctly&Cg <yv Qctlupdl(rvJL dtrtlfk l 'H**' Ot&UjtKhrt^AjL jLrrvcU OAjl* Jhu*t& jUtA-i-J-Xjb+*. k^2*-u /Kt>7HJb*Asr-**J Ct*nK,_ UfLp*~) fU^LMsJfUj . pUufJd. . &CljbL+j~> fftjt+L 7ksYX*b*i^L, JLCA^O . ( #M*6 y/X(/i(^L) JZIaJ flC.(rt H 7~l~ l/ ~ ................. * * yyict/7' tClb: M* ` PoStl l*-*, ~J\M.t*~*m AsiAtf^ i^ruyrt^-^JL^ * '&UuJLp/ru-rJrf- ~^V- / d.^ i Mfi * Pl^auJLiuJ'I^ y 'puswyO ^Ui^yy^rJ /& / . - KdyO*-J~r . ........ * SojCL&muj 77x'6/4) * ' p)rrif+J'irp, ' prTi jUAspinJ^ briAct- ^ pU^>. /JtUSU*> faiyr-JfVMy*_ . ............. ..... .......................... ................... ............... P-loMj ^r-t^Xju ", H4^ C**n-bcLrJt' Cuas JLamJL *prwJL.(A-A ^r )- A> 'tstLA+Jl. />J . <0 /^JAAAAjA- ^1-/Vi y^-t> ir^j-tAJ.-iT-*-' cU^AJ ^ (2F_'0<?:_r><^ * BjLhkl' . = ._cP. ^2 ~t>3 7*^Jn:f,. . ._. C35703 0673 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: ETHYLENE GLYCOL MONOBUTYL ETHER CAS#: 111-76-2 I. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: UOCE1CE1OCfif Common synonyms: 2-Butoxyethanol, butyl callosolve, butyl glycol, hydroxylethyl butyl ether. Chemical/Physical Properties Molecular Wt. Specific Gravity 118.18 .90(20*0 Ref. Index 1.419 solubility Sol. in alcohol and water Boiling Pt. 171.2*C In Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Not naturally occurring. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: No std. set MSHA PEL: No std. set ACGIH TLV: SOppm (skin) NIOSH REL: No std. set C35703 0674 Acute Toxicity Data: LDjo-, for oral, dermal, inhalation. Include species, inh-hmn TDL0=195ppm orl-rat LDso=1480mg/kg ivn-rat LDso=340mg/kg IDLH: No std. set (Ref:NIOSE ) ERPG: ND Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or NA Initiator (genotoxic): IARC Classification: D Other designations: NA NOAEL: 121mg/m3 (inh - rat) (Ref: HEAST ) (no observable adverse effect level) CAL. Prop 65 NSRL ND (no significant risk level) CSF: NA Specific cancer end point: NA Reproductive hazard: Positive human and teratogenicity. (inc. mutagen, terat) X Yes No animal studies for mutagenicity and Sensitizer: Studies show potential for sensitization. Non-cancer Rfd: 2E-lmg/kg/day (oral-rat-subchronic) 2E-2mg/kg/day (oral-rat-chronic) End Point: Blood-altered hematology Human toxicity data: Known human reproduction hazard. Chemical causes irritation of the mucous membrane and upper respiratory track. Biological Monitoring: Blood and urine Available: X Yes No ACGIH BEI: ND \ C35703 0675 III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient Standards/established limits in various media: TSCA listed Remediation Issues: Ho RCRA cleanup levels established Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent. Fate in the Environment: Will adhere to soil matrix with slight leaching. Relevant bioaccumulation data: May bioaccumulate slightly. Known toxicity in plants: May cause yellowing of leaves. Known toxicity in fish: May be toxic to several fresh water fish species. Known toxicity in other aquatic species: May be toxic to fat head minnow - limited evidence, though. IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low to Moderate Significant public activity and/or key topics C35703 0676 V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) ________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLAKTiCHEMPRO.WAR C35703 0677 VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo - LOAEL MEI NIOSH REL - NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = . 54mg/m3. For risk level, E-6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide' International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community Non-cancer effects (Adult) 2.92E-03mg/m3 (Child) 8.33E-04mg/m3 Employee (ACGIH) SO ppm (skin) (Rfd=2E-02mg/kg/day) H:\PLANTVCHEMPRO.WAR t C35703 0678 Prt^ljs \ * PaALo OCjLXJu^ctiAY^dt ct///r>,-A * Viu^ 06&jLflaJ-tSF-*J-'^jtorruJo &AJU y^cXX 0^ $\xj ~^)/(juvMrd't'>-s ^ '^AJLtJU/ru^*c^v-S-- J/y\tiXfk4xi ^L(/vk' ^ ItjAjpir-iJ-n^ ^AsnAJs). 'ibu.B Lr ThJ/i - & 023- STL~0. 0^-6 Kg/fn3 /*/ J * OniCoKc^n 'C/{) * @4a*JL <v- Jluj &PA- L -- -U//Jjl*vc^ <UU-4XA^cuji'*>-o. J>-Ui^JiUM ? JmAa* CjL^<3t^iuJL l^ |*^Y .6* <2^ ^ *fl*^L<uJ~Uj _JhjLk.yT*-&T^ ^J-OHytn^> ho Jam //<r^ LulJL J-* ^ia^c^L 6.J /U^p^L^iA^rJi fix^L, ^ jLoyOsr~/J'/ij CAa^CJa yVluJr * TxumjL^ML Trt/f.,1^ ; /u/? ` J^aca 4j, fish'** * Unfit jk \ AJfl *^ 4 j/Vr/ /T^/^ flluAju jbh) jU+JL. J^^Uaaax, 'THAty C&Cc~tJ /U*-p< rtshif .^AAthJ li'y. ................. Puaju ~fc-r^Jjj \............................................................._. V&JL- (I'^J^XtJb O-IA ^^TvyK'VtrT^o. ^ - -^'QAAxrQ^.'yif.C'.dj Oat-J-- /a (h/LA/vtJx 4j^-*s liAArtoJ ^UfU^ ^ '^i*<J-rO ....._ LQKzib^^jQAHrtJb^ rrt^JX-_, r 4. Z2 Z? -&8y*^ ), /Ozz '^-Q^roC JL^^Im^Mfy __* ^s> -V- ^ t ks>__I C35703 0679 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: BERYLLIUM CAS#: 7440-41-7 PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Be Common synonyms: Beryllium dust; Glucinium, synonyms of other compounds vary depending upon specific compound. Chemical/Phvslcal Properties Molecular Wt. 9.0 Boiling Pt. 4532F *3o2cC Solubility Insoluble in h2o Melting Pt. 2349*F Specific Gravity 1.8S Vapor Pressure 0 mm Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: No data on-line. Ambient/background concentrations: Earth crust 2-10 ppm; range from .01 to 1.22 mg/L; soil range from .1-40 ppm H20 Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 0.002 mg/m3 C=0.005 mg/m3 STEL=0.025mg/m3 MSHA PEL: ND ACGIH TLV: ALARA-Aj (Ca) NIOSH REL: ALARA-not to exceed 0.0005 mg/m3 C35703 0680  Acute Toxicity Data: LDjo-, for oral, dermal, inhalation. Include species. Inh - hmm TCLO: 0.1 mg/m3 Scu - pig TDLO: 7 mg/kg IDLH: No limit established (RefrNIOSH) ERPG: ND Chronic Toxicity Data: Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B2 Other designations: ND NOAEL: 0.54 mg/kg/day (Ref: IRIS) (no observable CAL. CSF: Prop 65 NSRL: 0.1 8.4E+0 mg/kg/day'1 adverse effect level) mg/day (no significant risk level) Specific cancer end point: lung tumors Reproductive hazard: (inc. mutagen, terat) Yes X No Sensitizer: Yes Non-cancer Rfd: 5.0E-03mg/kg/day (ing. subchronic; rat) End Point: None observed (Ref: Heast) Human toxicity data: Known human respiratory carcinogen. Acute symptoms may include chemical pneumonitis and pulmonary edema. Biological Monitoring: Available: X Yes No Blood levels (LTT test) ACGIH BEI: ND t C35703 0681 ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP Status CAA. Effluent/Ambient Standards/established limits in various media: RCRA - Listed DOT - Guide 19 SARA - Hazardous chemical TSCA - Chemical inventory Remediation Issues: RCRA cleanup levels; H20: 8E-08 mg/1; Soil: 2E-01 mg/kg. Air: 4E-04 ug/m3; Reporting to Regulatory Agencies Worldwide: European TSCA equivalent. Canadian and Fate in the Environment: Occurs naturally. Has chain route from atmosphere to soil to water. It does not readily leach tinless in acidic environments. Relevant bioaccumulation data: Beryllium is not known to bioaccumulate readily in humans or animals. Known toxicity in plants: levels of .17 to 3 mg/kg Occurs naturally in plants in low Known toxicity in fish: No data on-line. Known toxicity in other aquatic species: No data on-line. C35703 0682 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 X CONEG X HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate to high Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoringavailable:_ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0683 VII. GLOSSARY OF ACRONYMS: ACGIH TLV ADI BEI C/Ce (Ca) CSF E+01 - - - ERPG IARC IDLH LC - LDLO LD,, LOAEL MEI - NIOSH REL - NOAEL - NSRL - PEL RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10-6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide' International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Cancer effects (Adult) 4.22E-08mg/m3 (Child) .04E-08mg/m3 Mon-cancer effects (Adult) 7.29E-05mg/m3 (Child) 2.08E-04mg/m3 (CSF=8.4E+0 mg/Jcg/day1) (Rfd=S.0E-3 "g/kg/day) Employee; (OSHA) . 002mg/m3 .OOSmg/nr5 ceiling .025mg/m3 STEL H:\PLANT\CHEMPRO.WAR C35703 0684 * fi&AU p^CtuOM/Jl , dtmi-k. * fltf-Jipfl -f-i/Y<~*JL .. . 0*) fclw --^/ij^jA^i^ ^ '-^JLtJiuruA^Ji' fasOfirtJ-&u^ 0^i^iuynycJ'i'y^ i/y\CAiO^ci iLovks !pbtjf><_rijh^ jCJOflAJs). '~t^.5 Lr JJo/} - O. OC'^. /v_^ / J 6 TiZl, 'O. oj.6 n^jm3 ............... .......... .......... C%iC*#-e^in 'C/'j. f &La*Jl 'V'-yt^o -^- -Ou/Zx^c^ dJL-<^<^<xxtJi J>-iA^JiUJtt ^ <<~hx/i) -b-OL/i*- j^xJcflU Q *> & %2^ <, ^/lUfKfrrJ //0O<iLLLLsXOjs^*^*j^frr3^ $J~htUyA_> faju fajij-u Jo (LouuJjulJL' -b -yviuj- oj /U^p^^t^rU. Ax^L 0 d MB. *Aji(Ui '*j^ ~T<rxitik) \ AJfl * StyS'tx-nuAx y*^ t^A/.C/^ _. , ,, v,.^.. . jtt:__-_J_*&*^J{Ju^tJLLQiky1 Ja ** * *PFrt HJrtny .' LiJ Jx*xL I i i I * iii i i j ^w>y CClXc^ J-Onj y ! 1 J - -- KnAlbiit'ys_______ _________________________ ________________________________________ flxaju 'P.CrjpIju. .*______ S/&-S-- .0-1C y^nvyuT-MT^. . /a _ C.fl'flJ~(r) Y'f*-'. iu.*r\ (Xyv-A-- . /nJ... jCAAJ^ Cbrxft-. / . iAxsL^vd-' /i kxi__> jZM*s iCfrr&J '......................... ....................... :(r..l_____tVLd^^iHSid:2 JF-&8 yu^ )m? . ____ /PA S' jLt^im^jljLi *V- y*1^/^ C35703 0685 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: BERYLLIUM CAS#: 7440-41-7 I_s_ PHYSICAL/CHEMICAL INFORMATION; Chemical formula: Be / Common synonyms: Beryllium dust; Glucinium. Synonyms of other compounds vary depending upon specific compound. Chemical/Physical Properties Molecular Wt. 9.0 Boiling Pt. 4S32*F 95o2*C Solubility Insoluble in HjO Melting Pt. 2349F Specific Gravity 1.85 - Vapor Pressure 0 Ml Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: No data on-line. Ambient/background concentrations: Earth crust 2-10 ppm; range from .01 to 1.22 mg/L; soil range from .1-40 ppm H20 Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 0.002 mg/m3 c=0.005 mg/m3 STEL=0.02Smg/m3 MSHA PEL: ND ACGIH TLV: ALARA-Aj (Ca) NIOSH REL: ALARA-not to exceed 0.0005 mg/m3 t C35703 0686  Acute Toxicity Data: LDjq., for oral, dermal, inhalation. Include species. Inh - hmm TCLO: 0.1 mg/m3 Scu - pig TDLO: 7 mg/kg IDLH: No limit established (Ref:NIOSH) ERPG: ND Chronic Toxicity Data; Carcinogenic status: X Yes No Promoter (epigenetic) and/or X Initiator (genotoxic): IARC Classification: B2 Other designations: ND NOAEL: 0.54 mg/kg/day (Ref: IRIS) (no observable CAL. CSF: Prop 65 NSRL: 0.1 8.4E+0 mg/kg/day'1 adverse effect level) mg/day (no significant risk level) Specific cancer end point: lung tumors Reproductive hazard: (inc. mutagen, terat) Yes X No Sensitizer: Yes Non-cancer Rfd: 5.0E-03mg/kg/day (ing. subchronic; rat) End Point: None observed (Ref: Heast) Human toxicity data: Known human respiratory carcinogen. Acute symptoms may include chemical pneumonitis and pulmonary edema. Biological Monitoring: Available: X Yes No Blood levels (LTT test) ACGIH BEI: ND C35703 0687 III. ENVIRONMENTAL INFORMATION; Existence in environment: (See Background data in I above) Emission Standards: HAP Status CAA. Effluent/Ambient Standards/established limits in various media: RCRA - Listed DOT - Guide 19 SARA - Hazardous chemical T8CA - chemical inventory Remediation Issues: RCRA cleanup levels; Air: 4E-04 ug/m3; HjO: 8E-08 mg/1; Soil: 2E-01 mg/kg. Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent. Fate in the Environment: Occurs naturally. Has chain route from atmosphere to soil to water. It does not readily leach unless in acidic environments. Relevant bioaccumulation data: Beryllium is not known to bioaccumulate readily in humans or animals. Known toxicity in plants: Occurs naturally in plants in low levels of .17 to 3 mg/kg Known toxicity in fish: No data on-line. Known toxicity in other aquatic species: No data on-line. I C35703 0688 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 X CONEG X HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate to high Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoringavailable:__________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0689 VII. GLOSSARY OF ACRONYMS: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E+01 - ERPG IARC IDLH LC - LDLO - LDjo LOAEL - MEI - NIOSH REL - NOAEL - NSRL PEL - RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10"4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide' International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Cancer effects (Adult) 4.22E-08mg/m3 (Child) 6.04E-08mg/m3 Mon-cancer effects (Adult) 7.29E-0Smg/m3 (Child) 2.08E-04mg/m3 (CSF=8.4E+0 og/hg/day'1) (Rfd=5.0E-3 mg/kg/day) Rmpiovee: (OSHA) . 002mg/m3 .OOSmg/m3ceiling .025mg/m3 STEL H:\PLANT\CHEMPRO.WAR C35703 0690 QujaJks Su {J-t&LcL i^-^ux^o ~f/ Qtux^x&iitHdL^iYYujL) )r^J_ I jL/nujL Qxjl) >rJ cOAt^tJL CL4^&h^J~ }p~C4x.`i't*3 Jy<i&j4- fr- 'TUMa-^L^uJ JLax^Hly^ J^f-uJ'f . 'H^c. 4CC- /1+JLoa ~ /kxJdL. cLuak^ tlAXl * CAAy6ju^(t-^t^\jycc^ ", ftfl P UsfJULtsr^ ^XA/VhJlS . 1**- * . Tcix.L,^ : Mft * -So^Xcyyru^u T*vxfj J 't)`.a-Jy*x* cLu^^U^jLitO kxLaJ \../U^xttJL>.. * >/UauMJi4- ` jljLaf>ir^J-rLy /AAA^chJis^ j /X^rttTrJ AxXhrxO flxxkj ........ ......................... . ._ . ......... .................... ............................ * (Cw^f?Usr& &oO Jl/ju-L@-- C 4.^.4^^ h </}3 -fasyZCXt^<A*3 Ctr^t/unt^ ..- fcrtL 3 h^f /JU^.-f^O MaJJo *r$~^ 6x*J?xjt.T-& CA'i/' /-oxIL C^-4*Jc^xx*strJLJ * t ..^..-.{'If. 6-tC " 4 ^Ou^ln^ C/* AXAryY~XY^7y^-S^> '^'Soc^- fyv <3j^Mb j&Uj JjxJJu^! Jaa^t =. 4 3h*^jl-Agj; ; Q,VlAjvJT^JXn~> t/yj C35703 0691 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: ALUMINUM CAS#: 7429-90-5 is. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: A1 Common synonyms: ND Chemical/Phvsical Properties Molecular Wt. 26*98 Specific Gravity 2*70 Boiling Pt. 3211F 1800<,c Solubility sol in HC1 .and h2so4 Melting Pt. p /660C Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: N.D. on-line. Ambient/background concentrations: Widely distributedin earth's crust. Bauxite/cryolite: 8.8% by weight; in soil=150- 600g/kg; urban air=10ug/nr; nonurban=0.Sug/m. Sampling/analytical methods: Seeattached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: ND MSHA PEL: ND ACGIH TLV: 10mg/m3 metal dust; 5mg/m3 welding fume NIOSH REL: 5mg/m3 resp; 10mg/m3 total \ C35703 0692 Acute Toxicity Data: LDjq., for oral, dermal, inhalation. Include species. No data for Al. For aluminum oxide; ihl-mouse, TCLO=357mg/m3. IDLH: ND (Ref: NIOSH) ERPG: ND Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or NA Initiator (genotoxic): IARC Classification: NA Other designations NA NOAEL: ND ADl=30-50mg/kg/day Body burden: 0.lg/70kg man; l7ugAl/l00ml blood (Ref: NA ) (no observable adverse effect level) CAL. Prop 65 NSRL: NA (no significant risk level) CSF: NA Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) Yes X No * Correlation between levels of Al in drinking water and CNS birth defects in humans - suggest association. Various animal studies show fetal effects. Repro A-- unconfirmed human repro hazard. Sensitizer: No - possible potroom asthma association. Non-cancer Rfd: ND End Point: ND Human toxicity data: No true acute effects. Fumes can cause delayed asthma type reaction. Pulmonary effects with exposure to flakes, dialysis dementia. Potroom asthma (S02+F?) Biological Monitoring: Available: Yes X No ACGIH BEI: ND C35703 0693 III. ENVIRONMENTAL INFORMATION: (yvjt/ Existence in environment: (See^Background *data in I above) Emission Standards: fjfl Effluent/Ambient Standards/established limits in various media: DOT - Guide #40 T8CA - chemical inventory Remediation Issues: No RCRA cleanup levels established. Reporting to Regulatory Agencies Worldwide: No data on-line. Fate in the Environment: No data on-line. Relevant bioaccumulation data: No data on-line. Known toxicity in plants: ND on-line. Known toxicity in fish: ND on-line. Known toxicity in other aquatic species: ND on-line. IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low Significant public activity and/or key topics C36703 0694 Vi. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANTVCHEMPRO.WAR i C36703 0696 VII. GLOSSARY OF ACRONYMS: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E+01 - ERPG IARC IDLH LC - - -- LDLO LD,o LOAEL MEI - - NIOSH REL - NOAEL - NSRL - PEL - RfD - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = . 54mg/m3. level, E--6 = 10"4 or 1 excess cancer For in risk 1 million.) Emergency Response Planning Guide' International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: cone, in bulk/dust (Adult) 42 mg/kg (Child) 4.5mg/kg ADI 30mg/kg/day Employee: ACGIH-TLV lOmg/m3 total 30mg/m3 STEL Smg/m3 respirable H:\PLANTNCHEMPStO.WAR c35703 0696 c^i'L'h alto PlaM, Sum/YlOJUi * PxJytL P+fiJjU : * &6_A^i 'jr-fQ @(JLLfid't /KjJLs cdJ *r\ /7^r .* S^U-oo t "^ 7/ " "^ l^vtjdha J-fu ]U,rhiULl . V^U- Jlt-iL^Xa ^.-xJ A-J JU**<?(: A^yU __^_. - ^JuLAA/dJly. jAd^y 'JLLCLC,Cwsi>~> djX/X-~Q {GKxtjfnS^' jj^O d^j OjyV JLat^Jl , Hu, JJtnJL y /^uJL /V 7Uoc4^ Ay\A-cJ-Ayjt Jb^judo H~A<a. Jb-uAybi j/iAndb -A Jj~oa-uS~~ (VJ ~^(Ullj 6<yCt^cuCy&b dajj fA\Jlri-Ly> VaIaxL. pj^f^fyr^yyUyYuJLjS---dj-o^Ld-i l/h-> d-h-*-3 ^dClA^bdlUi^--' , '^~A-ty A* `Z.S'nx^/yA^. * u* *#($* * '^t-**<^irvLj/rS-dL. t d/AJ-fKastb cO-^-Ccto ~Oviy .(lixjJUL<Jyt^ '-f'oJt/jriyQ Ayty flbb/yLCO dy(-fls<JL*y&Cf*U i * Ptf-dz/Tuu ~tifc <di^ - Pl'f-tuUArU ~nc^ ^ZXo- ^JuJytU^o , ^4-Lu-tArJ Jl/rdldu^ to..,JbywruJjAy M-trAdb dj^-cdt, a/a-g ^Ljt-SxXd-' ................................. .......... * ", &dLj-' jL*44l t u/fr, C&r\ (AyUUCh d/l^-CyLtyijJ> .Ay. u^/wr* .toy* ,CAAlaMa> JiAA-tuyuA'' CCUU*JL~ ^~AyUIXy COt~- -^yty 'Atom#. fctOHuJ 'jjbW .^MaAAUs*^ Q/L*y jAAyck-hrx^ '-ivubuico y^yd^Uurad ... ^CaJ^ {^rt-^xJbs '. .. / . d^^.&**AdA*XytL./L^ JLa*lIL fcrd- 30 d*/Uv<JL- flQi4r : Sb'HytlJ* iWA .jtrtuiytSr. *^A dcuJj a^v/^Xj-/S-JT.QPi.<____ ~tytf~c<^ M) C35703 0697 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: LITHIUM CAS#: 7439-93-2 I. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: Li Common synonyms: ND Chemical/Physical Properties Molecular Wt. 6.94 Specific Gravity 0.53(20*0) Boiling Pt. Solubility 1336*C sol. in H20 3k 4*%.* F Dust is a fire hazard. Melting Pt. 179*C 354.2F Physical state in air: In stack: gas X particulate In ambient air: gas X particulate If particulate, part size: N.D. on-line. Ambient/background concentrations: Earth's crust = 0.005% by weight; occurs in hydrosphere in low cone, (ll ppm sea HjO) ; Plant cone: traces in many plants grown in soils which contain as much as 100 ppm Li. Sampling/analytical methods: See attached II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: ND MSHA PEL: ND ACGIH TLV: ND NIOSH REL: ND Acute Toxicity Data: LDjo-, for oral, dermal, inhalation. Include species. LDm (inh) dog: 325 mg/kg LDL, (scu) rabbit: 40,000 mg/kg \ C35703 0698 IDLH: ND ERPG: ND (Ref:NIOSH) Chronic Toxicity Data: Carcinogenic status Yes X No Promoter (epigenetic) and/or Initiator (genotoxic): NA IARC Classification: D Other designations: NA NOAEL: ND (Ref: HEAST ) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) CSF: ND Specific cancer end point: ND ADI: (some foods contain Li) calculated ADI=2mg/day. Reproductive hazard: (inc. mutagen, terat) Yes X No Lithium carbonate has been associated with human defects in children taking it for manic depression. Repro: A- unconfirmed human repro effects. NOAEL: 100 mg/kg in rats Specify end point: Heart defects, hydrocephalus, spina bifida, neural tube; sperm may also be affected; reports of cleft palate, brain defects. Sensitizer: No Non-cancer Rfd: ND End Point ND Human toxicity data: Solid material may cause skin and eye burns since it reacts with moisture to form a caustic. Fumes from burning Li are highly irritating to the skin, eyes and mucous membranes. Li may injure kidneys, especially if sodium intake is limited. Biological Monitoring: Available: Blood and urine monitoring. X Yes No ACGIH BEI: ND C35703 0699 III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA Effluent/Ambient Standards/established limits in various media: DOT - Guide #40 TSCA - Chemical inventory Remediation Issues: No RCRA cleanup levels established. No CERCLA reportable quantity. Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent. Fate in the Environment: No data on-line. Relevant bioaccumulation data: No data on-line. Known toxicity in plants: Plants incorporate lithium when grown in lithium containing soils. Lithium has low plant toxicity. Known toxicity in fish: No data on-line. Known toxicity in other aquatic species: No data on-line. IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR LOW Significant public activity and/or key topics C35703 0700 L. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR t C35703 0701 VII. GLOSSARY OF ACRONYMS: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E+01 - - ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = .54mg/mJ. For risk level, E-6 = 10"4 or 1 excess cancer in 1 million.) Emergency Response Planning Guide' International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect No significant risk level Permissible exposure limits level Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: Non-cancer effects (Adult) 50 mg/kg (Child) 1.2 mg/kg (ADI 2 mg/day) Employee: Estimated employee action limit air = 10 mg/m3 (nuisance dust std.) bulk =2.5 mg/kg K:\PLANTVCHEMPRO.WAR l C35703 0702 'SuJjMju'bi'ilttt-; fthrTkJrf' (fypfr'J srfA^ S~^Lf/yj JNl^XJL rf>0 yOo- 'jLijusifdrino *1 ~C^u Cjul<Ual e^'-1 &4\ 4< Nft \<Uji^hnUJLJionuJz i yyud-Ci^L^LJa^-'- ^bMfrcL&ma^^/'blOA^iUit&i-hry^tJ: S'ft&jlj^/l*cjQsCtzJL Zj2-lU-L : ' b-pSJi&uth'r^ T~*X4Lj?L " rht^xsftJ- <L cJLl. jUUluM, <ry^ LaJ-,JUA &rJL AjJLyO, U^dL. u^c/i rtf^ZCJL. ^ in^ V d * Si^c^jjyvu^ ~Tfttdir^: AM * ^rCfhrJ~l/y\ ; Oo^\aurxJ^ Lo tv A-Cc^L^/y yCVtsc./xJ-<^, ft0 ^y/~Usr~^^ * 'Pr-i^Lt-j * K~/iCts /Qyr*M<A^i^ CXliT JLus-tjL ^tfJL CO JsKA<-JL /jlA-4s jri^X^tOr^ JL^-C<J'-i. A//tA <3? S ,, c?/rr\t{% ft ftl~b-~ jAlAft Q & ~ $ d^njiuc^Si, -4 Ar. Tnt^t rYUt/n dsT\(U*^ft'*ft<r>^> j < A/-/2f*/rU - C35703 0703 CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: SULFUR DIOXIDE CAS#: 7446-09-5 i. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: SO* Common synonyms: Sulfurous acid anhydride, sulfurous oxide, sulfur oxide Chemical/Physical Properties Molecular Wt. 98.1 Bolling Pt. 554#F Solubility Miscible in HjO Flash Pt. MA Ionization Potential Specific Gravity 1.84 (96-98% acid) Vapor Pressure (29SF): 1 mm Freezing Pt. 51F Moncombustible liquid but capable of igniting finely divided combustible material. Physical state in air: In stack: X gas In ambient air: X gas particulate particulate Ambient/background concentrations: 0.6 - 94.2 nmol/m3 Sampling/analytical methods: See attached. I C35703 0704 II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY; Exposure Limits: OSHA PEL: MSHA PEL: ACGIH TLV: NIOSH REL: MAK: 2 ppm; (5 mg/m3); 5 ppm; STEL (10 mg/m3) 5 ppm 2 ppm; 5 ppm STEL 2 ppm; S ppm STEL 2 ppm Acute Toxicitv Data: LDjo-a for oral, dermal, inhalation. Include species. LDj,: inhalation rat: 2520 ppm/lH; LDM: inhalation mouse: 3000 ppm/30m IDLH: 100 ppm (Ref: NIOSH ) ERPG: 500 lbs. Chronic Toxicitv Data: Carcinogenic status: Yes X No X Promoter (epigenetic) and/or Initiator (genotoxic): With arsenic, equivocal tumorgenic agent; (RTECS) lung, thorax, effects at 500 ppm (chronic exposure of 5 min. duration). IARC Classification: NA Other designations: NA NOAEL ND (Ref: HEAST ) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) CSF: NA Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) X Yes No Mouse: 25 ppm/7h; Rat: 4 mg/m3/24H for 72D prior to mating; menstrual cycle changes; effects on fertility; fetal delay growth. DNA inhibition. DNA deunage: (lymphocyte) 5700 ppb; Sensitizer: No, irritant effects only Non-cancer Rfd: ND End Point ND * C35703 0705 Human toxicity data: Irritating to the eyes, nose, throat and lungs. Pulmonary edema reported. Biological Monitoring: Available: X Yes No Medical Surveillance OSHA/ILO: PFT 29CFR 1910.20 ACGIH BEI: No ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: 1* NAAQS: 0.03 ppm (80 mg/m3); annual arith. mean: 0.14 ppm (365 mg/m3); 24 hr. max. not to be exceeded more than once yearly. 2s: 0.02 ppm; ann. arith. mean: 0.1 ppm; 24 hr. max. = 0.5 ppm 3 hr. max. Effluent/Ambient media: Standards/established limits in various CAA - Listed DOT - Guide #16 SARA - SARA Title III 500 TSCA - on TSCA inventory lbs. Remediation Issues: R.Q. = 1 lb. Reporting to Regulatory Agencies Worldwide: Major pollutant worldwide; European and Canadian standards. air Fate in the Environment: ^Complex cycles^/Major component of acid rain. Accelerate^ leaching 4m heavy metals from plumbing and ore deposits. ^ Relevant bioaccumulation data: potential: none. Food chain concentration Known toxicity in plants: Acid induced leaching of nutrients from the soil is a cause of nutritional imbalance in trees. Effects on pines and broad leaf. Known toxicity in fish: LD^S ppm/1 hr. fresh HzO trout. Known toxicity in other aquatic species: No data on-line. Chemical is not known to bioaccumulate in aquatic species. c35703 0706 IV. COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Moderate - high Significant public activity and/or key topics JL ADDITIONAL ALCOA INFORMATION; Dispersion model summary attached: _______ IH exposure monitoring available: Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L.rTITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) H:\PLANTVCHEMPRO.WAR \ C35703 0707 VII. GLOSSARY OF ACRONYMS: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E01 - - ERPG IARC IDLH LC - -- LDLO LDjg LOAEL - - MEI NIOSH NOAEL REL - NSRL - PEL RfD - - STEL - TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level, E-6 = 10-6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Community: (NAAOS) 0.03 ppm (80 mg/m3) annual aritta mean 0.14 ppm (305 mg/m3) 24 hr. max Employee: fOSHA) 2 ppm 5 ppm STEL H:\PLANTVCHEMPRO.WAR C35703 0708 ^LaMj SfjLjrfljlYLGJU^. * lljuJLcL fU&X; . '. GyOjUo ~4rfV Ocvu^^iJKtJL ^oJ fST^ yUo ^ 'm-hJnr^ J^yuM Joiop irfiJ'ITUj f-y$ZjLrr). bh^ TU#~ 3-pfn'j 5m.- L Oylftr* ('/in cj A* vij/An / i-/^j *i Nft 'TtUuf',CL^on UL. * {Lf^HsnJfA/J'l'r^s ~t~&1-iLtk . * ^Uy&TjAfYUA j~t~VX` t, I fj \ /Oft. * j^-t-^,4-,.A^_ v P^hJ.rM'Y' MsfUt^n^ f^J- m iYuiA^ juOuA'td'i/y* i\>$K Prt^Ui A^tUytx). -- ~ * A"'&4*v6 rir..pAML.Y^--t^Lir) to JhuJL rA a^o4b W- * 44^ cUwJ- dUw . 4^w 'lfu^ /*- ^ ,rr\bJr^ ' JUiLjs e ^> 3?-5~F-5"r^l^3 L ^ dcn^ .......-.............. 35703 0709 cleanUir assurance team CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: PHOSPHINE CAS#: 7803-51-2 I*. PHYSICAL/CHEMICAL INFORMATION: Chemical formula: PH3 Common synonyms: Hydrogen Phosphide Chemical/Physical Properties Molecular Wt. 34.0 Boiling Pt. 126*F Solubility Slight in H2Q Plash Pt. NA (gas) Ionization Potential 9.96 eV Flammable gas Vapor Pressure >latm Freezing Pt. -209*F - j 3^ e C- Physical state in air: In stack: X gas particulate In ambient air: X gas particulate Ambient/background concentrations: Not naturally occurring. Sampling/analytical methods: See attached. II. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: 0.3 ppm; 1.0 ppm STEL MSHA PEL: 0.3 ppm ACGIH TLV: 0.3 ppm; 1.0 ppm STEL NIOSH REL: 0.3 ppm; 1.0 ppm STEL MAK: 0.1 ppm Acute Toxicity Data: LDy,., for oral, dermal, inhalation. Include species. LDjq (oral) rat: 12/mg/kg LDjo (inhalation) rat: 11 ppm/4hr LDL0 (oral) human: 80 mg/kg LCL0 (inhalation) human: 1000 ppm/5 min * Toxicity listed for zinc phosphine. IDLH: 200 ppm (Ref: NIOSH ) C35703 0710 ERPG: 500 lbsj Chronic Toxicity Data: Carcinogenic status: Yes X No Not classifiable as to human carcinogencity. in pesticide workers, there are some reports of chromosomal damage (chromatid deletions, gaps/breaks translocations). Promoter (epigenetic) and/or Initiator (genotoxic) :NA IARC Classification: NA Other designations: NA Animal carcinogenic studies are inadequate. Quantitative est. of CA risk (oral): none. Quantitative est. of CA risk (inhal): none. NOAEL: 0.026 mg/kg/day (oral rat: whole body decrease in weight) Ref: HEAST ) (no observable adverse effect level) CAL. Prop 65 NSRL: CSF: NA N.D. (no significant risk level) Specific cancer end point: NA Reproductive hazard: (inc. mutagen, terat) Yes X No Sensitizer: No Non-cancer Rfd: 3E-4 mg/kg/day End Point: Body weight clinical parameters Human toxicity data: Inhalation exposures characterized by severe pulmonary irritation, cough, headache, chest tightness, dizziness, lethargy, stupor, delayed onset of pulmonary edema Biological Monitoring: Available: X Yes No Blood ACGIH BEI: No III. ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA C35703 0711 Effluent/Ambient media: Standards/established limits in various DOT - Guide #18 SARA - Extremely hazardous substance TSCA - Chemical inventory list Remediation Issues: CERCLA reportable quantity: 100 pounds. Reporting to Regulatory Agencies Worldwide: Canada/Europe Fate in the Environment: Pure phosphine is inert. oxidize under influence of radiation and UV light. Will Relevant bioaccumulation data: bioaccumulate. Phosphine is not reported to Known toxicity in plants: Hay cause leaf/stem damage. Known toxicity in fish: No data on-line. known to bioaccumulate in aquatic species. Phosphine is not Known toxicity in other aquatic species: No data COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR High Significant public activity and/or key topics ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: ______ IH exposure monitoring available:_______ Summary attached: C35703 VI. KEY ALCOA CONTACTS R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _________________ ____________ (In.d. Hygiene sampling information) VII. GLOSSARY OF ACRONYMS: ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Careinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = ,54mg/m3. For risk level, E--6 = 10"6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose c35703 0713 VIII. RISK SUMMARY flommtinity; 1 Non-cancer effects (Adult) 4.375E-5mg/ja5 (Child) l.25E-5ng/m3 Employee; (OSHA) (RfD=3E-4mg/kg/day) 0.3 ppm; 1.0 pptn (STEL) C35703 0714 )PKa^Sac<Lp PropenXujO 0^^kujlmj &aiy\X. : 6ciJLu\cj PouaX. '. ^V4LCua^ Pouvf: YYix/^y^ Rn-*utr; PlasV* PcafrvT ' 'JC -Phi? ^oJ-iSV- (V~ 6-$-Q pA^-^Jh-, L. ^&aJ-icaJ-^ (LkzrntcdLcLc-c. UeJ' fuurc. JLtU-fJ tr> N- Z>. ^LfrjJL toru^Jwn PotSUUi-P.' R-COOU/Tj6 - fcf ra*a ' DpfUiiyfeyn/t LcnuxCUfeL).' Lcu,t^y^MCK.U t^1 (U3-) Ooao ,' ^ ^oc : 4tAcL6 /uuwt A m C35703 0715  Acute Toxicity Data: LDjo-, for oral, dermal, inhalation. Include species, inh - hum TCLO = 500 ppm IDLH: 20,000 ppm ERPG: ND (Ref: NIOSH) Chronic Toxicity Data: Carcinogenic status: Yes X No Promoter (epigenetic) and/or NA Initiator (genotoxic): IARC Classification: D Other designations: NA NOAEL: 100 mg/kg/day (Ref: BEAST) (no observable adverse effect level) CAL. Prop 65 NSRL: ND (no significant risk level) CSF: ND Specific cancer end point: ND Reproductive hazard: (inc. mutagen, terat) Yes X No Sensitizer: No Non-cancer Rfd: 1E+0mg/kg/day End Point: kidney - nephrotoxicity Human toxicity data: Moderate toxicity in humans. May cause corneal erosion, headache, gastritis and nausea. Odor perception in air = 1.6 ppm. Biological Monitoring: Available: X Yes No Urine and blood ACGIH BEI: ND C35703 0716 ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: HAP status CAA. Effluent/Ambient media: Standards/established limits in various RQ=5000 lbs. Sanitary landfill leachates mean value of .6g/l. RCRA - Listed DOT - Guide 39 TSCA - Chemical inventory Remediation Issues: RCRA Cleanup Levels; Air: ND; Water: 4E-100 mg/1; Soil: 8E+3 mg/kg. Reporting to Regulatory Agencies Worldwide: Canadian and European TSCA equivalent. Fate in the Environment: Biodegradable in H20; adsorption in sediment not significant. In soil, it both volatizes and leaches. In atmosphere, it is lost by photolyses. Relevant bioaccumulation data: Acetone does not bioaccumulate in humans or animal species. Known toxicity in plants: Acetone has been identified in vegetation as a naturally occurring metabolite. Known toxicity in fish: environmental concentration. No relevant data for Known toxicity in other aquatic species: No relevant data for environmental concentrations. Acetone does not bioaccumulate in aquatic species. C35703 0717 IV. * COMMERCIAL ISSUES AND RELATEDREGULATIONS: FDA X Indirect food additive used in adhesives CALIFORNIA PROP 65 CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING LIFE CYCLE ANALYSIS OUTRAGE FACTOR Low Significant public activity and/or key topics V. ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: VI. KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) .ck-v'1'- J.^HACKAY (Environmental Tssi L. TITLE'S (Dispersion/Modell^ng> (Environmental data/sampling/analysis) ______ (Ind. Hygiene sampling information) H:\PLANT\CHEMPRO.WAR C35703 0718 \ VII. GLOSSARY OF ACRONYMS; ACGIH TLV - ADI BEI C/Ce (Ca) CSF E+01 ERPG IARC IDLH LC LDLO LDjo LOAEL MEI NIOSH REL " - NOAEL NSKL PEL RfD STEL TDLO/TCLO - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Careinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/mJ = .54mg/m3. For risk level, E-6 = 10-6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure Total lowest dose limit VIII. RISK SUMMARY Community: Non-cancer effects (Adult) l.45E-0lmg/m3 (Child) 4.17E-02mg/m3 (RfD: 1.0E+0 mg/kg/day) Employee: (OBHA) 750 ppm; 1000 ppm STEL H:VPLANTVCHEMPRO.WAR C35703 0719 ACETONE RISK SUMMARY: Health Effects Profile: Basis for Occupational Limits: The exposure limits are based on respiratory and eye irritation. Carcinogenicity; NA Mutagenicity: Negative for most assays il Toxicitv: NA cicitv: Long-term study of workers exposed .ly to acetone vapors (600-2150 ppm) revealed rally significant effects. Slight to moderate eye, nose, throat,and itation at 500 and 1000 ppm for 6 hours. air level goal (AALG) is based n the irritant effects of acetone which are noncumulative in nature and tend to be concentration-dependent rather than time dependent. The AALG is based on the long-standing ACGIH TLV-TWA. However, since there is reasonable evidence that irritation may occur at levels below the TLV, it is treated as a LOAEL, and a total uncertainty factor of 50 (10 for interindividual variation X 5 for a LOAEL) is used. AALG: irritation (TLV/50) 15 ppm (36mg/m3) 8-hour TWA. (X r^L O'-' Crprt^i Ud ^ ion JL ivorloo^ C< // ; \ pj^ \ Chopra, C35703 0720 f/9 W /la till * dt`rCc4t Ori^jjricL frvpJj, ty. //' \ P^mJ 1 CM&^prt * \JfrC- O C/h\H^t.h>. bjcnj^ ,, , ^La^c, (zy fji^jL<_ C~VL&/r^*3 &a_. i~f iHiff js+~* o\\ prrf^de, /-'.,. - () ..AW*-: ,, -- , OfUlM%i<nuy .,, A Jo Art" ~~ /x ttu- C kh^j^) * &A~ l^dUU0 C*XX *"H Ak<_J p-Ur^U^ jyx. ~btr&c+''>s C^t/h^U^jbJ) 'flu. Ku>UrULL * CV. Ju^. p/U-^JL- clur*y_ C^oio 7/Z.^ -fz> /2^ ^<4/1^0//fc- C^tcC. QfyJ^ds i^zUb^. * (Ljj^.k 'TMu / <># /^oZXr^ A .................................... ,, 4c u 0s<^y^._p^g~4'r_ '-L4-U,, TUJl ALq 4< i>c by CUrL C<44 -Ac I^'Li'^aJL^ ' Asf jLr-J- g ^inv\ ^7/z. r~~ py\vtc-ft <Tk lUj K (foK /Tv- ^$A/r~L*cs ^C--* U>hs,\jL. 4-Su, C&iyu^ ^*T A'H-v /l^TU. ^ C35703 0721 \ \ Qk, JLklA _ (Ju+nu.JL' 4hsu _________________ -. V^Uo ^XM-ryifri /*_ . .. U<JlJU /m^ 4iL- -js-^- .^fhoz . .. . _. U^k ... r..................................... . ' 11 /, 'pk&4*rs tKjt*u ca{^ &i*-VfU7\ 6/lxj tiAj- CJLr--t'^7^ ffY\ . "` *^7 Sifhyc, Lx*-* jltyi/L Urtif-tsy' fi ' \ ^ ^ ~hjttJL A> Jjfy- 7 ^5^/), ,, Li U rti/' /t 7%- AKy/- -('LOO Ou<lb J-taJ' UaM' ^At<X vry)p- ti<uu~j{\rJL(i_ C35703 0722 t 3,3, -TCbL) * iUJLtE Zjj-ttk fruits'. * -^-yu Ou^J .CU\Jl />u ^-rj-cvrv>^i ' V"^- N105 ff -- )j{sjl444MXs {Icv-tA-- <J -- /STO dyOA^Ct^^^M^rj Ctsr-Q----- JLj^-uJZ 6sy*4-- ts cLjLAAJS<A-y JfTbOyfM-- S*J C^HJ/yrydE yOr* jCid-ELy J^CAUZ-o (jUjyiJLy 4-*^tjlAuduty'--' JL. (jEd" ^y^JAyryJla * NlO^H /3 / [(} L - ^Oyi^J^ ySb^Gl)f/>bhdik.JQfif*"& * CyClA(jjUr^t>^lyn t f.t (y t J^VTv^C- Cy^Lidtj^lyf^t^htrr* (jyty &y jkuA*u/J CWistun-*<-^u-> S^~ " --f2A-OCyJjL~> * ?>V1 /<*-( /^- 4 'j^LyQ4yt*^py^^ -- 7/XV ,, Ajfi ...................... ......... ^/'ffn({<JL ^ /Uyp<yr^UAytyf!r-^y 4*XJC-/^ llvtcL^OU jE /huAr^tyryo . * ^H^zXlfaiLiSTtrf t Ll ^j > ^ h/J.tJ'i/yy ^ .AjA./n*S- Cir^C ytr^tsobf}^ /^O^gMAA^ ^yUy^L^f'-iJ^ /ffirf/fy'ity J^fccck. -JVQD.to aJ .............................. 6i yE^JL ry* ^ykt/ujlEJL d^lhd'._____.... ...................' jj&*AjJid'uyy *- ^XPurn/JL-_________ ___________ tAyst-t-s p *, n , * FEaaj p-uJLv 4 (/faEdytiE QjAJ Ji/jnC^1 ...... _____ ___ _____ _ (- ft fil-ty^ ^/X <dJA*J C35703 0723 2> ^2-0Z)_ ... . .......>(?. >^o^--/^'. /CQamj*) . jp-^r.p^> *~& fyL............................................................................................................................................................ t-hfiEE. *_ _*_$$$ lb ~ it. y?^?cdr MiJipvJ^u ^ 05"-- loyx^h ~fcO ............... ...................................^ ................................ _rr'"\. ________________ .............. * \t) E-L> J^-c^bt' tah.rbdE - yf^j/rx3 A/r JllLaycJl. /V* / 0"# IrdJl_ SA4jl 4-ir-rJ^-- <vu AjtOtH EEL. ) CLEAN AIR ASSURANCE TEAM CHEMICAL PROFILE FORM - DRAFT MODEL CHEMICAL NAME: 2,3,7,8 TCDD CAS#: 001746-01-6 JU PHYSICAL/CHEMICAL INFORMATION: Common synonyms: Tetrachlorodibenzofuran, Tetrachlorodibenzodioxin Ck2(YUUf> C(LU4<X^0^ Physical state in air: In stack: gas X particulate In ambient air: gas X particulate (mostly) TCDD has been shown to volatilize and to occur in air in both the gas and particulate phase. Estimated 1/2 life of gas TCDD=8.3 days. If particulate, part size: No data on-line. Ambient/background concentrations: Air bKg range of .02 to . 08pg/m3. TCDD has been positively detected in 2% of 491 OSEPA sornet stations. Not detected in storm H20. Average emissions of .97 to 3.8 ng/m3. TCDD detected in 1.3% of sornet sediment stations. Sampling/analytical methods: see attached sheet. IL. HEALTH INFORMATION - EMPLOYEE AND COMMUNITY: Exposure Limits: OSHA PEL: ND I3 MSHA PEL: ND ACGIH TLV: ND NIOSH REL: M (8hr TWA) \ Estimated maximum tolerated dose (YUU. inhalation l0-30ppm in oil and l00-300ppm (soil/water mixture) t C057Q3 0724 Acute Toxicity Data: LDjo-, for oral, dermal, inhalation. Include species. TDLO human skin 107 ug/kg IiDjo oral rat = 22 ug/kg LDjg oral monkey = 50 ug/kg IDLH: ND (Ref: NA ) Lethal dose in man=100 ug/kg. ERPG: ND Chronic Toxicity Data: - Carcinogenic status: X Yes No X Promoter (epigenetic) and/or Initiator (genotoxic): IARC Classification: B2 Other designations: Mixtures classified as Bl Risk assessment doses (e.g., EPA for 1X10"6) Risk Estimates 30.j(Kj/g composite limit 60 jg/m3 air exposure NOAEL: Suggested 70 ng/day (Ref: FDA ) (no observable adverse effect level) CAL. Prop 65 NSRL: 0.000005 ug/day (no significant risk CSF: (oral) =1.5E+5 (mg/kg/dar1) level) (inh)=1.5E+5 (mg/kg/day') Specific cancer end point: Lung/liver tumors EPA unit risk . fvVN Oral = 4.5E+o (ug/1)*1 Jtf Inhalation = 3.35E-5 (pg/m3)^ Reproductive hazard: (inc. Limited evidence; only confirmative evidence. mutagen, terat) have animal X Yes No studies as Sensitizer: It is a suggested sensitizer based on theoretical data. Non-cancer Rfd: ND End Point: ND (acute, chloroacne) WHO-suggested LOAEL = 3mg/kg/day (World Organization) FDA-suggested No effect level = 70ng/man/day Health \ C35703 0725 * If level in dust/dirt is 1 ppb, the amount inhaled has been calculated as 1.4 pg/day. Human toxicity data: Dioxin has been suggested as the most toxic chemical to man and the environment. Biological Monitoring: Available: (Limited, difficult, expensive) X Yes No ACGIH BEI: ND ENVIRONMENTAL INFORMATION: Existence in environment: (See Background data in I above) Emission Standards: ND - No limit set. HAP status CAA. Effluent/Ambient Standards/established limits in various media: RCRA - No RCRA cleanup level reported CWA - Listed TSCA - Chemical inventory DOT - Guide 58 SARA - Extremely hazardous chemical Remediation Issues: 8oil levels of 1 ppb are estimated to increase the risk of developing cancer by l in 1 million. Reporting to Regulatory Agencies Worldwide: France, Germany, Switzerland OEL, Finland, Fate in the Environment: Particulate phase may be physically removed from air by vet and dry deposition. In h20, TCDD will be associated with soils and sediments. Persistence half life is estimated to be in excess of 1.5 years in lakes. If released to soil, TCDD is not expected to leach. Water transport is limited because solubility in H20 is only 2 ppb. Relevant bioaccumulation data: Bioconcentration in aquatic organisms has been demonstrated. Soil 1/2 life = 1-3 years. TCDD bioaccumulates less than DDT. Bioconcentration factors (BCF) for aquatic species are available from EPA. Long biological 1/2 life in humans. Known toxicity in plants: Findings show ability of plants to take in dioxin and translocate it through conductive vessels to aerial parts where it is eliminated. Known toxicity in fish: in fish species. Fairly high BCF's have been reported C35703 0726 Known toxicity in other aquatic species: have been reported in aquatic species. Fairly high BCF's COMMERCIAL ISSUES AND RELATED REGULATIONS: FDA X CALIFORNIA PROP 65 X CONEG HAZARD COMMUNICATION X WORLDWIDE X EC PACKAGING/RECYCLING X LIFE CYCLE ANALYSIS OUTRAGE FACTOR High Significant public activity and/or key topics ADDITIONAL ALCOA INFORMATION: Dispersion model summary attached: _______ IH exposure monitoring available:________ Summary attached: KEY ALCOA CONTACTS: R. M. JAMES (Health Issues) S. M. HARVEY (Legal Issues) J. MACKAY (Environmental Issues) L. TITLEY (Dispersion Modelling) __________________________ (Environmental data/sampling/analysis) _____________________________________ (Ind. Hygiene sampling information) C35703 0727 VII. GLOSSARY OF ACRONYMS: ACGIH TLV -- ADI BEI C/Ce (Ca) CSF E+01 - ERPG IARC IDLH LC - - LDLO LDjq LOAEL MEI NIOSH REL NOAEL NSRL PEL RfD STEL TDLO/TCLO - - - American Conference of Governmental Industrial Hygienists Threshold Limit Value Acceptable Daily Intake Biological exposure indices Ceiling level Carcinogen/cancer Cancer slope factor Expotential notation (Example: 5.4E-01mg/m3 = .54mg/m3. For risk level-, E-6 = 1C6 or 1 excess cancer in 1 million.) Emergency Response Planning Guide' International Agency for Research on Cancer Immediately dangerous to life or health Lethal concentration at which 50% of exposed animals die Lowest lethal dose Lethal dose at which 50% of exposed animals die Lowest observable adverse effect level Maximally exposed individual NIOSH Recommended Exposure Limits No observable adverse effect level No significant risk level Permissible exposure limits Reference dose Short term exposure limit Total lowest dose VIII. RISK SUMMARY Employee: (for risk level of E-6) Action limit based on MEI and ADI (no est. C8F or Rfd) Cancer effect is estimated at: pg/m3 (air) 4^ppb (bulk) C5p-&0m$ C35703 0728 I C35703 0729 I c35703 0730 Table 1 C A A M ethods and Costs ai <o < Xo O O to 0 O UJ CL < H (U-UJ < to Q < Z E o <0 TO 1 04 3 o < Z E o co TO CD 3 o 2 e> o CM tO <o t- < 2 S2to to < tr> CO CO o CO CO co oo oo M1 CM T-- o r-- CM 00 <2 q to o >; x < z HI < rCM CM 5 So *Oo &W 5 TO O CTTO- TO o -- Q <0 Q OQ 2 SE a: < o <o 6 2. < Q. UJ <o CL /n UJ cd- E o to E o e E <o to to *o T3 > XI E o to JO EE o to o to 5 TO o 3 04 3 O) Oo E E to CO CO CL CL O to CD 3 O CD CD 33 Oo o Ooo o o oo o o M* to to to to o o o 'M' oTf otT ^-- -- CO V-- co XJ xO: _ <D Q 2 <o ui 2 00 *3 O -- <B Q 2E <o m2 >s x: o. CD & CM O *o aj e wo 2g <c Q. 0 UJ O >s -C CL CTJ <d e> CM O *0 O fZ 0c. 25 Qn"- 0C UJ 53 CO to O 9 *c tJL ^ U. <0 Q. (i) uj y, CO T-- TO O x: <D Q 2 O<. O UJ co *0 JO3 Q 2 <O m2 CT"O *JO0Z Q 2E U<. 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