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STANDARO FORW FOR
EMPLOYER'S FIRST REPORT OF INJURY OR ILLNESS
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Name and Mdrtss of to--taf n Mo--i _
Dr. Gay~ Industrial Medicine
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,,7/18/75 r,m -- For records
The Dow Chemical Co. Felix Ainsworthoit.ii.
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INDUSTRIAL ACCIDENT BOARD REQUIRES COMPLETION OF ALL APPLICABLE ITEMS ON THIS FORM
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standaro form for
EMPLOYER'S FIRST REPORT OF INJURY OR ILLNESS
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7/18/75
For records
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Sijned by.
r,mThe How Chemical Co.
Felix Ainsworth
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403276
DPaTmNT OF inOuSTBIAI malIH and MEDICINE
MEDICAL REPORT
BlOCa no A OE^ABTMCNT
supervisor on duty
jOILER shop
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MASTCM a clock NO
1 52 J
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DATE OP ONSET
Jilagnosls 6/18/75
7/-ro/
CLASSIFICATION
AMBULANCE REASON FOR Cii.L OR HISTORY
.as .6j'i to4il1er maker and exposed to asces tos-s i nee worxing for jc.v.
RfCOMUENOATiONS TO SUPERVISOR
ATTENOJNG PHYSICIAN AND OR NURSE
to RETURN i1
NECESSARY
_____________
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INSURANCE DEPT. COPY
,__; INDUSTRIAL [T INSURANCE decision 0 PERSONAL
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403277
Paul M. Sttvini, M.D. Cliitf, Pulmonary Section
The Methodist Hospi tad
6516 Bartnac 8oulvard
Houston, Texas 7702
TEXAS MXDICAL CXKTXA
Ml Teo Bowlk President
Ms. Tom Foukqvuam
Executive Vice President
June 6, 1975
Am Coe 713-790-20'
____Cobi
METHHQi
W. H. Sears, M.D. 106 N. Parking Place Lake Jackson, Texas 77566
RE:
Dear Dr. Sears:
Thank you so much for referring this very pleasant gentleman, whose problem was that of progressive dyspnea, associated with diffuse radiographic pulmonary infiltration in volving primarily both lower lobes and pleura. You had been taking care of this patient primarily because of an intercurrent respiratory tract infection but the patient felt that progression of symptomatology perhaps signified an underlying, progressive dysfunction of unknown etiology. On admission, questioning revealed that he had worked with Dow Chemical for many years and been exposed to a variety of irritating gases but, in addi tion, had been involved in handling sheets of asbestos and been present in the environ ment when these had been handled and considerable amounts of asbestos dust was in the air. This exposure has been off and on for the past twenty to thirty years. In addi tion, he is a moderately heavy smoker and had complained of relatively recent increase in the amount of cough and sputum production. As you pointed out in your woric-up, some respiratory dysfunction was present as long as thirteen years ago, at which time he was evaluated both at St. Joseph's Hospital and M. D. Anderson, with removal of several hundred cc's of serosanguineous pleural effusion but no definite evidence of malignancy. No tissue-diagnosis was made at this time but the possibility of tumor was excluded for the patient.
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Physical examination at this time revealed a modestly obese, anxious, mildly dyspneic, white male in no acute distress. There were bibasilar rales and decreased breath sounds at both bases . No evidence of cor pulmonale or clubbing was present.
Review of chest x-rays revealed that both in 1972 and at the present time, there were rather dense, interstitial increase in markings with slight honeycombings in both lung fields, primarily at the bases, with dense pleural thickening and some pleural calcification
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403278
particularly in the left hemidiaphragm.
This picture most closely resembles the distribution of asbestosis and, in order to both
make the diagnosis and exclude the possibility of an underlying carcinoma, the patient underwent transnasal fiberoptic bronchoscopy. This was carried out under local anes thesia and was well tolerated by the patient and revealed essentially clear airways with no evidence of interbnonchial lesions. Slight bronchospasm and mucosal erythema was noted. A transbronchial lung biopsy was performed under fluoroscopy and tissue ob tained from several subsegments of the right lower lobe all showed interstitial fibrosis. In addition, an alveolar lavage was obtained and cytological examination of the ceils revealed multiple ferruginous bodies, diagnostic of asbestos. No positive cytologies were obtained. Pulmonary function studies, a copy of which is enclosed, revealed the presence of combined modestly severe restrictive ventilatory defect, with the superimposed presence of mild airway obstruction that does not seem to respond to bronchodilators . The remainder of the work-up was basically non-revealing.
Under these circumstances, 1 would feel fairly certain of the basic diagnosis of asbestosis with both pleural and interstitial fibrosis . In addition, there is some superimposed airway disease, probably related to cigarette smoking, that does not seem to be particularly re versible with bronchodilators . The extent of pulmonary dysfunction more than adequately accounts for the patient's symptomatology. He is therefore entitled to some amount of disability and/or compensation.
I have made
aware of his diagnosis and extent of disability. I suggested to
him that, although there is very little directed therapy to reverse the underlying disease
process, he should avoid environmental pollutants, stop smoking and continue to remain
as active as possible with exercise up to tolerance, and a certain amount of weight re
duction.
He will be returning to your care and has requested me to give him a copy of the letter I am sending to you.
Thank you very much again for allowing me to help you with this very interesting patient.
ST053 20 Of
PMS/ej cc:
Paul M. Stevens, M.D. Professor of Medicine Baylor College of Medicine
403279